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Artigo de periodico
NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes (2021)
Costa, Frederico Ribeiro Campos ; Leite, Jefferson Antonio; Rassi, Diane Meyre ; Silva, Josiane Fernandes da ; Oliveira, Jefferson Elias ; Guimarães, Jhefferson Barbosa ; Freitas, Maria Cristina Foss de ; Câmara, Niels Olsen Saraiva ; Pontillo, Alessandra ; Passaglia, Rita de Cassia Aleixo Tostes ; Silva, João Santana da ; Carlos, Daniela
Source: Cell Reports. Unidades: FMRP, ICB
Subjects: IMUNOLOGIA, DIABETES MELLITUS, DOENÇAS AUTOIMUNES, PÂNCREAS, CAMUNDONGOS, POLIMORFISMO, SISTEMA IMUNE, INTERLEUCINAS
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ABNT
COSTA, Frederico Ribeiro Campos et al. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, v. 35, p. 1-16, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.celrep.2021.109176. Acesso em: 16 nov. 2024.
APA
Costa, F. R. C., Leite, J. A., Rassi, D. M., Silva, J. F. da, Oliveira, J. E., Guimarães, J. B., et al. (2021). NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes. Cell Reports, 35, 1-16. doi:10.1016/j.celrep.2021.109176
NLM
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Vancouver
Costa FRC, Leite JA, Rassi DM, Silva JF da, Oliveira JE, Guimarães JB, Freitas MCF de, Câmara NOS, Pontillo A, Passaglia R de CAT, Silva JS da, Carlos D. NLRP1 acts as a negative regulator of Th17 cell programming in mice and humans with autoimmune diabetes [Internet]. Cell Reports. 2021 ; 35 1-16.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.celrep.2021.109176
Artigo de periodico
Interleukin-10 negatively modulates extracellular signal-regulated kinases 1 and 2 in aorta from hypertensive mouse induced by angiotensin II infusion (2019)
Bressan, Alecsander F.; Fonseca, Gisele A.; Tostes, Rita de Cássia Aleixo ; Webb, R. Clinton; Lima, Victor Vitorino; Giachini, Fernanda Regina Casagrande
Source: Fundamental and Clinical Pharmacology. Unidade: FMRP
Subjects: CITOCINAS, PROTEÍNAS QUINASES, INTERLEUCINAS
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ABNT
BRESSAN, Alecsander F. et al. Interleukin-10 negatively modulates extracellular signal-regulated kinases 1 and 2 in aorta from hypertensive mouse induced by angiotensin II infusion. Fundamental and Clinical Pharmacology, v. 33, n. 1, p. 31-40, 2019Tradução . . Disponível em: https://doi.org/10.1111/fcp.12409. Acesso em: 16 nov. 2024.
APA
Bressan, A. F., Fonseca, G. A., Tostes, R. de C. A., Webb, R. C., Lima, V. V., & Giachini, F. R. C. (2019). Interleukin-10 negatively modulates extracellular signal-regulated kinases 1 and 2 in aorta from hypertensive mouse induced by angiotensin II infusion. Fundamental and Clinical Pharmacology, 33( 1), 31-40. doi:10.1111/fcp.12409
NLM
Bressan AF, Fonseca GA, Tostes R de CA, Webb RC, Lima VV, Giachini FRC. Interleukin-10 negatively modulates extracellular signal-regulated kinases 1 and 2 in aorta from hypertensive mouse induced by angiotensin II infusion [Internet]. Fundamental and Clinical Pharmacology. 2019 ; 33( 1): 31-40.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/fcp.12409
Vancouver
Bressan AF, Fonseca GA, Tostes R de CA, Webb RC, Lima VV, Giachini FRC. Interleukin-10 negatively modulates extracellular signal-regulated kinases 1 and 2 in aorta from hypertensive mouse induced by angiotensin II infusion [Internet]. Fundamental and Clinical Pharmacology. 2019 ; 33( 1): 31-40.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/fcp.12409
Artigo de periodico
Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model (2018)
Martins, Larissa M. S.; Perez, Malena M.; Pereira, Camila A.; Costa, Frederico R. C.; Dias, Murilo S.; Tostes, Rita de Cássia Aleixo; Ramos, Simone Gusmão; Zoete, Marcel R. de; Ryffel, Bernhard; Silva, João Santana da; Carlos, Daniela
Source: Immunology. Unidade: FMRP
Subjects: INFLAMAÇÃO, LINFÓCITOS, INTERLEUCINAS, DOENÇAS METABÓLICAS, OBESIDADE
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ABNT
MARTINS, Larissa M. S. et al. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, v. 154, n. 4, p. 624-636, 2018Tradução . . Disponível em: https://doi.org/10.1111/imm.12946. Acesso em: 16 nov. 2024.
APA
Martins, L. M. S., Perez, M. M., Pereira, C. A., Costa, F. R. C., Dias, M. S., Tostes, R. de C. A., et al. (2018). Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model. Immunology, 154( 4), 624-636. doi:10.1111/imm.12946
NLM
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/imm.12946
Vancouver
Martins LMS, Perez MM, Pereira CA, Costa FRC, Dias MS, Tostes R de CA, Ramos SG, Zoete MR de, Ryffel B, Silva JS da, Carlos D. Interleukin-23 promotes intestinal T helper type17 immunity and ameliorates obesity-associated metabolic syndrome in a murine high-fat diet model [Internet]. Immunology. 2018 ; 154( 4): 624-636.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1111/imm.12946
Artigo de periodico
NLRP3 inflammasome mediates aldosterone-induced vascular damage (2016)
Bruder-Nascimento, Thiago; Ferreira, Nathanne S.; Zanotto, Camila Z.; Ramalho, Fernanda; Pequeno, Isabela O.; Olivon, Vania C.; Neves, Karla B.; Alves-Lopes, Rheure; Campos, Eduardo; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens; Carlos, Daniela; Mestriner, Fabiola Leslie Antunes Cardoso; Prado, Douglas; Pereira, Felipe V.; Braga, Tarcio; Luiz, Joao Paulo M.; Cau, Stefany B.; Elias, Paula C.; Moreira, Ayrton Custódio; Câmara, Niels Olsen Saraiva ; Zamboni, Dario Simões; Alves Filho, José Carlos Farias; Tostes, Rita de Cássia Aleixo
Source: Circulation. Unidades: FMRP, ICB
Subjects: ALDOSTERONA, IMUNOLOGIA, INFLAMAÇÃO, DOENÇAS VASCULARES, INTERLEUCINAS
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ABNT
BRUDER-NASCIMENTO, Thiago et al. NLRP3 inflammasome mediates aldosterone-induced vascular damage. Circulation, v. 134, n. 23, p. 1866-1880, 2016Tradução . . Disponível em: https://doi.org/10.1161/circulationaha.116.024369. Acesso em: 16 nov. 2024.
APA
Bruder-Nascimento, T., Ferreira, N. S., Zanotto, C. Z., Ramalho, F., Pequeno, I. O., Olivon, V. C., et al. (2016). NLRP3 inflammasome mediates aldosterone-induced vascular damage. Circulation, 134( 23), 1866-1880. doi:10.1161/circulationaha.116.024369
NLM
Bruder-Nascimento T, Ferreira NS, Zanotto CZ, Ramalho F, Pequeno IO, Olivon VC, Neves KB, Alves-Lopes R, Campos E, Silva CAA da, Fazan Júnior R, Carlos D, Mestriner FLAC, Prado D, Pereira FV, Braga T, Luiz JPM, Cau SB, Elias PC, Moreira AC, Câmara NOS, Zamboni DS, Alves Filho JCF, Tostes R de CA. NLRP3 inflammasome mediates aldosterone-induced vascular damage [Internet]. Circulation. 2016 ; 134( 23): 1866-1880.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1161/circulationaha.116.024369
Vancouver
Bruder-Nascimento T, Ferreira NS, Zanotto CZ, Ramalho F, Pequeno IO, Olivon VC, Neves KB, Alves-Lopes R, Campos E, Silva CAA da, Fazan Júnior R, Carlos D, Mestriner FLAC, Prado D, Pereira FV, Braga T, Luiz JPM, Cau SB, Elias PC, Moreira AC, Câmara NOS, Zamboni DS, Alves Filho JCF, Tostes R de CA. NLRP3 inflammasome mediates aldosterone-induced vascular damage [Internet]. Circulation. 2016 ; 134( 23): 1866-1880.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1161/circulationaha.116.024369
Artigo de periodico
P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction (2013)
Chiao, Chin-Wei; Santos, José Eduardo da Silva; Giachini, Fernanda R.; Tostes, Rita de Cássia Aleixo; Su, Ming‑Jai; Webb, R. Clinton
Source: Clinical Science. Unidade: FMRP
Subjects: OXIGENASE, INTERLEUCINAS, LIPOPOLISSACARÍDEOS, ÓXIDO NÍTRICO, SEPSE, CITOCINAS, DOENÇAS VASCULARES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHIAO, Chin-Wei et al. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction. Clinical Science, v. 125, n. 3, p. 131-141, 2013Tradução . . Disponível em: https://doi.org/10.1042/CS20120479. Acesso em: 16 nov. 2024.
APA
Chiao, C. -W., Santos, J. E. da S., Giachini, F. R., Tostes, R. de C. A., Su, M. ‑J., & Webb, R. C. (2013). P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction. Clinical Science, 125( 3), 131-141. doi:10.1042/CS20120479
NLM
Chiao C-W, Santos JE da S, Giachini FR, Tostes R de CA, Su M‑J, Webb RC. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction [Internet]. Clinical Science. 2013 ; 125( 3): 131-141.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/CS20120479
Vancouver
Chiao C-W, Santos JE da S, Giachini FR, Tostes R de CA, Su M‑J, Webb RC. P2X7 receptor activation contributes to an initial upstream mechanism of lipopolysaccharide-induced vascular dysfunction [Internet]. Clinical Science. 2013 ; 125( 3): 131-141.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/CS20120479

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