Source: Clinics. Conference titles: Simpósio avanços em pesquisas médicas. Unidade: FM
Subjects: RESUMOS (SIMPÓSIOS), INSULINA, GLUTAMATOS, FOSFATOS, AUTOANTICORPOS, DIABETES MELLITUS, INSULINA (DEPENDENTE)
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ABNT
CRISOSTOMO, L. G. et al. Insulin (IAA), glutamic acid decarboxylase-65 (antigad) and tyrosine-phosphatase (anti-IA2) autoantibodies: prevalence and association with hla-dr haplotypes, ins vntr, and ctla4 in siblings of patients with type 1 diabetes in Brazil. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo. . Acesso em: 08 out. 2024. , 2007APA
Crisostomo, L. G., Davini, E., Gamberini, M., Alves, L. I., Fukui, R. T., Rocha, D. M., & Silva, M. E. R. (2007). Insulin (IAA), glutamic acid decarboxylase-65 (antigad) and tyrosine-phosphatase (anti-IA2) autoantibodies: prevalence and association with hla-dr haplotypes, ins vntr, and ctla4 in siblings of patients with type 1 diabetes in Brazil. Clinics. São Paulo: Faculdade de Medicina, Universidade de São Paulo.NLM
Crisostomo LG, Davini E, Gamberini M, Alves LI, Fukui RT, Rocha DM, Silva MER. Insulin (IAA), glutamic acid decarboxylase-65 (antigad) and tyrosine-phosphatase (anti-IA2) autoantibodies: prevalence and association with hla-dr haplotypes, ins vntr, and ctla4 in siblings of patients with type 1 diabetes in Brazil. Clinics. 2007 ; 62 S188.[citado 2024 out. 08 ]Vancouver
Crisostomo LG, Davini E, Gamberini M, Alves LI, Fukui RT, Rocha DM, Silva MER. Insulin (IAA), glutamic acid decarboxylase-65 (antigad) and tyrosine-phosphatase (anti-IA2) autoantibodies: prevalence and association with hla-dr haplotypes, ins vntr, and ctla4 in siblings of patients with type 1 diabetes in Brazil. Clinics. 2007 ; 62 S188.[citado 2024 out. 08 ]