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Artigo de periodico
Inhibition of inducible nitric oxide synthase protects against the deleterious effects of sub-lethal sepsis and ethanol in the cardiorenal system (2021)
Sousa, Arthur Henrique de; Vale, Gabriel Tavares do; Nascimento, José A.; Awata, Wanessa Mayumi Carvalho; Silva, Carla Beatriz Pereira da ; Assis, Victor Oliveira; Alves, Juliano Vilela; Tostes, Rita de Cássia Aleixo ; Tirapelli, Carlos Renato
Source: Canadian Journal of Physiology and Pharmacology. Unidades: FMRP, EERP
Subjects: CREATINA, CITOCINAS, INIBIDORES DE ENZIMAS, ETANOL, ÓXIDO NÍTRICO, ESPÉCIES REATIVAS DE OXIGÊNIO, SEPSE
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ABNT
SOUSA, Arthur Henrique de et al. Inhibition of inducible nitric oxide synthase protects against the deleterious effects of sub-lethal sepsis and ethanol in the cardiorenal system. Canadian Journal of Physiology and Pharmacology, v. 99, n. 12, p. 1324-1332, 2021Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2021-0239. Acesso em: 16 nov. 2024.
APA
Sousa, A. H. de, Vale, G. T. do, Nascimento, J. A., Awata, W. M. C., Silva, C. B. P. da, Assis, V. O., et al. (2021). Inhibition of inducible nitric oxide synthase protects against the deleterious effects of sub-lethal sepsis and ethanol in the cardiorenal system. Canadian Journal of Physiology and Pharmacology, 99( 12), 1324-1332. doi:10.1139/cjpp-2021-0239
NLM
Sousa AH de, Vale GT do, Nascimento JA, Awata WMC, Silva CBP da, Assis VO, Alves JV, Tostes R de CA, Tirapelli CR. Inhibition of inducible nitric oxide synthase protects against the deleterious effects of sub-lethal sepsis and ethanol in the cardiorenal system [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 12): 1324-1332.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1139/cjpp-2021-0239
Vancouver
Sousa AH de, Vale GT do, Nascimento JA, Awata WMC, Silva CBP da, Assis VO, Alves JV, Tostes R de CA, Tirapelli CR. Inhibition of inducible nitric oxide synthase protects against the deleterious effects of sub-lethal sepsis and ethanol in the cardiorenal system [Internet]. Canadian Journal of Physiology and Pharmacology. 2021 ; 99( 12): 1324-1332.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1139/cjpp-2021-0239
Artigo de periodico
Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human (2020)
Costa, Eduardo D.; Silva, Josiane Fernandes da; Garcia, Daniela C.; Wainstein, Alberto J.; Rezende, Bruno A.; Tostes, Rita de Cássia Aleixo ; Teixeira, Mauro Martins; Côrtes, Steyner F.; Lemos, Virgínia S.
Source: Nitric Oxide. Unidade: FMRP
Subjects: ENVELHECIMENTO, TABAGISMO, ENDOTÉLIO VASCULAR, METABOLISMO, INIBIDORES DE ENZIMAS, FARMACOLOGIA, MEIA IDADE, ÓXIDO NÍTRICO
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ABNT
COSTA, Eduardo D. et al. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human. Nitric Oxide, v. 98, p. 20-28, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2020.02.003. Acesso em: 16 nov. 2024.
APA
Costa, E. D., Silva, J. F. da, Garcia, D. C., Wainstein, A. J., Rezende, B. A., Tostes, R. de C. A., et al. (2020). Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human. Nitric Oxide, 98, 20-28. doi:10.1016/j.niox.2020.02.003
NLM
Costa ED, Silva JF da, Garcia DC, Wainstein AJ, Rezende BA, Tostes R de CA, Teixeira MM, Côrtes SF, Lemos VS. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human [Internet]. Nitric Oxide. 2020 ; 98 20-28.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.niox.2020.02.003
Vancouver
Costa ED, Silva JF da, Garcia DC, Wainstein AJ, Rezende BA, Tostes R de CA, Teixeira MM, Côrtes SF, Lemos VS. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human [Internet]. Nitric Oxide. 2020 ; 98 20-28.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.niox.2020.02.003
Artigo de periodico
Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA) (2019)
Igual, Michelle Ogava; Nunes, Paulo Sergio Gonçalves; Costa, Rafael Menezes da ; Mantoani, Susimaire Pedersoli; Tostes, Rita de Cássia Aleixo ; Carvalho, Ivone
Source: Carbohydrate Research. Unidades: FMRP, FCFRP
Subjects: SOBREVIVÊNCIA CELULAR, CÉLULAS CULTIVADAS, INIBIDORES DE ENZIMAS, ESTRUTURA MOLECULAR (QUÍMICA TEÓRICA), MÚSCULO LISO VASCULAR
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ABNT
IGUAL, Michelle Ogava et al. Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA). Carbohydrate Research, v. 471, p. 43-55, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.carres.2018.10.007. Acesso em: 16 nov. 2024.
APA
Igual, M. O., Nunes, P. S. G., Costa, R. M. da, Mantoani, S. P., Tostes, R. de C. A., & Carvalho, I. (2019). Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA). Carbohydrate Research, 471, 43-55. doi:10.1016/j.carres.2018.10.007
NLM
Igual MO, Nunes PSG, Costa RM da, Mantoani SP, Tostes R de CA, Carvalho I. Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA) [Internet]. Carbohydrate Research. 2019 ; 471 43-55.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.carres.2018.10.007
Vancouver
Igual MO, Nunes PSG, Costa RM da, Mantoani SP, Tostes R de CA, Carvalho I. Novel glucopyranoside C2-derived 1,2,3-triazoles displaying selective inhibition of O-GlcNAcase (OGA) [Internet]. Carbohydrate Research. 2019 ; 471 43-55.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1016/j.carres.2018.10.007
Artigo de periodico
Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms (2015)
Chignalia, Andreia Z; Oliveira, Maria Aparecida; Debbas, Victor; Dull, Randal O; Laurindo, Francisco R. M; Touyz, Rhian M; Carvalho, Maria Helena Catelli de; Fortes, Zuleica Bruno; Tostes, Rita de Cássia Aleixo
Source: Clinical Science. Unidades: FMRP, FM, ICB
Subjects: ANDRÓGENOS (FARMACOLOGIA), INIBIDORES DE ENZIMAS, LEUCÓCITOS (METABOLISMO), TRANSDUÇÃO DE SINAL CELULAR, TESTOSTERONA
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ABNT
CHIGNALIA, Andreia Z et al. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, v. 129, n. 1, p. 39-48, 2015Tradução . . Disponível em: https://doi.org/10.1042/CS20140548. Acesso em: 16 nov. 2024.
APA
Chignalia, A. Z., Oliveira, M. A., Debbas, V., Dull, R. O., Laurindo, F. R. M., Touyz, R. M., et al. (2015). Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms. Clinical Science, 129( 1), 39-48. doi:10.1042/CS20140548
NLM
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/CS20140548
Vancouver
Chignalia AZ, Oliveira MA, Debbas V, Dull RO, Laurindo FRM, Touyz RM, Carvalho MHC de, Fortes ZB, Tostes R de CA. Testosterone induces leucocyte migration by NADPH oxidase-driven ROS- and COX2-dependent mechanisms [Internet]. Clinical Science. 2015 ; 129( 1): 39-48.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1042/CS20140548
Artigo de periodico
Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and S-nitrosylation in mouse aorta (2011)
Choi, Hyehun; Tostes, Rita de Cassia Aleixo; Webb, Clinton
Source: Journal of Cardiovascular Pharmacology. Unidade: FMRP
Subjects: ACETOFENONAS, ANTIOXIDANTES (FARMACOLOGIA), AORTA TORÁCICA (EFEITOS DE DROGAS), INIBIDORES DE ENZIMAS, PERÓXIDO DE HIDROGÊNIO (METABOLISMO), ESTRESSE OXIDATIVO, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CHOI, Hyehun e TOSTES, Rita de Cassia Aleixo e WEBB, Clinton. Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and S-nitrosylation in mouse aorta. Journal of Cardiovascular Pharmacology, v. 58, n. 5, p. 522-527, 2011Tradução . . Disponível em: https://doi.org/10.1097/FJC.0b013e31822d80a5. Acesso em: 16 nov. 2024.
APA
Choi, H., Tostes, R. de C. A., & Webb, C. (2011). Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and S-nitrosylation in mouse aorta. Journal of Cardiovascular Pharmacology, 58( 5), 522-527. doi:10.1097/FJC.0b013e31822d80a5
NLM
Choi H, Tostes R de CA, Webb C. Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and S-nitrosylation in mouse aorta [Internet]. Journal of Cardiovascular Pharmacology. 2011 ; 58( 5): 522-527.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1097/FJC.0b013e31822d80a5
Vancouver
Choi H, Tostes R de CA, Webb C. Thioredoxin reductase inhibition reduces relaxation by increasing oxidative stress and S-nitrosylation in mouse aorta [Internet]. Journal of Cardiovascular Pharmacology. 2011 ; 58( 5): 522-527.[citado 2024 nov. 16 ] Available from: https://doi.org/10.1097/FJC.0b013e31822d80a5

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