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Artigo de periodico
Macrophages: the bridge between inflammation resolution and tissue repair? (2018)
Garlet, Gustavo Pompermaier; Giannobile, W. V.
Source: Journal of Dental Research. Unidade: FOB
Subjects: MACRÓFAGOS, INFLAMAÇÃO, REGENERAÇÃO (FENÔMENOS BIOLÓGICOS), HOMEOSTASIA PERIODONTAL
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ABNT
GARLET, Gustavo Pompermaier e GIANNOBILE, W. V. Macrophages: the bridge between inflammation resolution and tissue repair?. Journal of Dental Research, v. 97, n. 10, p. 1079-1081, 2018Tradução . . Disponível em: https://doi.org/10.1177/0022034518785857. Acesso em: 20 jul. 2024.
APA
Garlet, G. P., & Giannobile, W. V. (2018). Macrophages: the bridge between inflammation resolution and tissue repair? Journal of Dental Research, 97( 10), 1079-1081. doi:10.1177/0022034518785857
NLM
Garlet GP, Giannobile WV. Macrophages: the bridge between inflammation resolution and tissue repair? [Internet]. Journal of Dental Research. 2018 ; 97( 10): 1079-1081.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1177/0022034518785857
Vancouver
Garlet GP, Giannobile WV. Macrophages: the bridge between inflammation resolution and tissue repair? [Internet]. Journal of Dental Research. 2018 ; 97( 10): 1079-1081.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1177/0022034518785857
Artigo de periodico
PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia (2016)
Borges, Beatriz de Carvalho; Garcia-Galiano, David; Rorato, Rodrigo; Elias, Lucila Leico Kagohara; Elias, Carol Fuzeti
Source: Molecular Metabolism. Unidade: FMRP
Subjects: HIPOTÁLAMO, INFLAMAÇÃO, LEPTINA, METABOLISMO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Beatriz de Carvalho et al. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia. Molecular Metabolism, v. 5, n. 6, p. 379-391, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.molmet.2016.03.003. Acesso em: 20 jul. 2024.
APA
Borges, B. de C., Garcia-Galiano, D., Rorato, R., Elias, L. L. K., & Elias, C. F. (2016). PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia. Molecular Metabolism, 5( 6), 379-391. doi:10.1016/j.molmet.2016.03.003
NLM
Borges B de C, Garcia-Galiano D, Rorato R, Elias LLK, Elias CF. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia [Internet]. Molecular Metabolism. 2016 ; 5( 6): 379-391.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.molmet.2016.03.003
Vancouver
Borges B de C, Garcia-Galiano D, Rorato R, Elias LLK, Elias CF. PI3K p110β subunit in leptin receptor expressing cells is required for the acute hypophagia induced by endotoxemia [Internet]. Molecular Metabolism. 2016 ; 5( 6): 379-391.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.molmet.2016.03.003
Artigo de periodico
PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges (2016)
Borges, Beatriz de Carvalho; Elias, Carol Fuzeti; Elias, Lucila Leico Kagohara
Source: Molecular and Cellular Endocrinology. Unidade: FMRP
Subjects: HIPOTÁLAMO, INFLAMAÇÃO, METABOLISMO ENERGÉTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Beatriz de Carvalho e ELIAS, Carol Fuzeti e ELIAS, Lucila Leico Kagohara. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges. Molecular and Cellular Endocrinology, v. 438, p. 36-41, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.07.005. Acesso em: 20 jul. 2024.
APA
Borges, B. de C., Elias, C. F., & Elias, L. L. K. (2016). PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges. Molecular and Cellular Endocrinology, 438, 36-41. doi:10.1016/j.mce.2016.07.005
NLM
Borges B de C, Elias CF, Elias LLK. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 36-41.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.mce.2016.07.005
Vancouver
Borges B de C, Elias CF, Elias LLK. PI3K signaling: a molecular pathway associated with acute hypophagic response during inflammatory challenges [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 36-41.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.mce.2016.07.005
Trabalho de evento-resumo
Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats (2011)
Ribeiro Junior, Luciano Filgueiras; Martins, Joilson de Oliveira ; Serezani, Carlos Henrique; Montes, Marlise Bonetti Agostinho; Negro, Sonia Jancar
Source: Abstracts. Conference titles: World Congress on Inflammation. Unidades: FCF, ICB
Subjects: DIABETES MELLITUS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIBEIRO JUNIOR, Luciano Filgueiras et al. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. 2011, Anais.. Paris: International Association of Inflammation Societies, 2011. . Acesso em: 20 jul. 2024.
APA
Ribeiro Junior, L. F., Martins, J. de O., Serezani, C. H., Montes, M. B. A., & Negro, S. J. (2011). Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. In Abstracts. Paris: International Association of Inflammation Societies.
NLM
Ribeiro Junior LF, Martins J de O, Serezani CH, Montes MBA, Negro SJ. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. Abstracts. 2011 ;[citado 2024 jul. 20 ]
Vancouver
Ribeiro Junior LF, Martins J de O, Serezani CH, Montes MBA, Negro SJ. Increased SOCS-1 inhibition of MYD88 expression correlates with milder sepsis-induced ali in diabetic rats. Abstracts. 2011 ;[citado 2024 jul. 20 ]
Artigo de periodico
Quercetin inhibits UV irradiation-induced inflammatory cytokine production in primary human keratinocytes by suppressing NF-kB pathway (2011)
Vicentini, Fabiana T. M. C.; He, Tianyuan; Shao, Yuan; Fonseca, Maria José Vieira; Verri Júnior, Waldiceu A.; Fisher, Gary J.; Xu, Yiru
Source: Journal of Dermatological Science. Unidade: FCFRP
Subjects: FLAVONÓIDES, RADIAÇÃO ULTRAVIOLETA, PELE, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VICENTINI, Fabiana T. M. C. et al. Quercetin inhibits UV irradiation-induced inflammatory cytokine production in primary human keratinocytes by suppressing NF-kB pathway. Journal of Dermatological Science, v. 61, n. 3, p. 162-168, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.jdermsci.2011.01.002. Acesso em: 20 jul. 2024.
APA
Vicentini, F. T. M. C., He, T., Shao, Y., Fonseca, M. J. V., Verri Júnior, W. A., Fisher, G. J., & Xu, Y. (2011). Quercetin inhibits UV irradiation-induced inflammatory cytokine production in primary human keratinocytes by suppressing NF-kB pathway. Journal of Dermatological Science, 61( 3), 162-168. doi:10.1016/j.jdermsci.2011.01.002
NLM
Vicentini FTMC, He T, Shao Y, Fonseca MJV, Verri Júnior WA, Fisher GJ, Xu Y. Quercetin inhibits UV irradiation-induced inflammatory cytokine production in primary human keratinocytes by suppressing NF-kB pathway [Internet]. Journal of Dermatological Science. 2011 ; 61( 3): 162-168.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.jdermsci.2011.01.002
Vancouver
Vicentini FTMC, He T, Shao Y, Fonseca MJV, Verri Júnior WA, Fisher GJ, Xu Y. Quercetin inhibits UV irradiation-induced inflammatory cytokine production in primary human keratinocytes by suppressing NF-kB pathway [Internet]. Journal of Dermatological Science. 2011 ; 61( 3): 162-168.[citado 2024 jul. 20 ] Available from: https://doi.org/10.1016/j.jdermsci.2011.01.002

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