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Artigo de periodico
Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction (2024)
Morelo, David Fernando Colon ; Wanderley, Carlos Wagner de Souza ; Turato, Walter Miguel; Borges, Vanessa F.; Franchin, Marcelo; Castanheira, Fernanda Vargas e Silva; Nascimento, Daniele Carvalho Bernardo; Prado, Douglas da Silva; Lima, Mikhael Haruo Fernandes de ; Volpon, Leila Costa; Kavaguti, Silvia K.; Carlotti, Ana Paula de Carvalho Panzeri ; Carmona, Fabio ; Franklin, Bernardo S.; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: British Journal of Pharmacology. Unidades: FCF, FMRP
Subjects: MACRÓFAGOS, SEPSE, CRIANÇAS, IMUNOSSUPRESSÃO, SISTEMA IMUNE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORELO, David Fernando Colon et al. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, v. 181, n. 8, p. 1308-1323, 2024Tradução . . Disponível em: https://doi.org/10.1111/bph.16286. Acesso em: 07 nov. 2024.
APA
Morelo, D. F. C., Wanderley, C. W. de S., Turato, W. M., Borges, V. F., Franchin, M., Castanheira, F. V. e S., et al. (2024). Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction. British Journal of Pharmacology, 181( 8), 1308-1323. doi:10.1111/bph.16286
NLM
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/bph.16286
Vancouver
Morelo DFC, Wanderley CW de S, Turato WM, Borges VF, Franchin M, Castanheira FV e S, Nascimento DCB, Prado D da S, Lima MHF de, Volpon LC, Kavaguti SK, Carlotti AP de CP, Carmona F, Franklin BS, Cunha TM, Alves Filho JCF, Cunha F de Q. Paediatric sepsis survivors are resistant to sepsis‐induced long‐term immune dysfunction [Internet]. British Journal of Pharmacology. 2024 ; 181( 8): 1308-1323.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1111/bph.16286
Artigo de periodico
Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT (2022)
Lima, Mikhael Haruo Fernandes de; Hiroki, Carlos Hiroji; Borges, Vanessa de Fátima; Cebinelli, Guilherme Cesar Martelossi ; Santos, Jessica; Rosa, Marcos Henrique; Silva, Camila Meirelles de Souza; Wanderley, Carlos Wagner ; Gonçalves, Augusto Velozo; Quirino, Gustavo Fernando da Silva; Zamboni, Dario Simões ; Cunha, Thiago Mattar ; Alves Filho, José Carlos Farias ; Cunha, Fernando de Queiroz
Source: The Journal of Infectious Diseases. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, SEPSE, MACRÓFAGOS, CITOMETRIA DE FLUXO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Mikhael Haruo Fernandes de et al. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, v. 225, n. 3, p. 531-541, 2022Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiab405. Acesso em: 07 nov. 2024.
APA
Lima, M. H. F. de, Hiroki, C. H., Borges, V. de F., Cebinelli, G. C. M., Santos, J., Rosa, M. H., et al. (2022). Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT. The Journal of Infectious Diseases, 225( 3), 531-541. doi:10.1093/infdis/jiab405
NLM
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1093/infdis/jiab405
Vancouver
Lima MHF de, Hiroki CH, Borges V de F, Cebinelli GCM, Santos J, Rosa MH, Silva CM de S, Wanderley CW, Gonçalves AV, Quirino GF da S, Zamboni DS, Cunha TM, Alves Filho JCF, Cunha F de Q. Sepsis-induced immunosuppression is marked by an expansion of a highly suppressive repertoire of FOXP3+ T-regulatory cells expressing TIGIT [Internet]. The Journal of Infectious Diseases. 2022 ; 225( 3): 531-541.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1093/infdis/jiab405
Artigo de periodico
IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population (2017)
Nascimento, Daniele C.; Melo, Paulo H.; Piñeros, Annie R.; Ferreira, Raphael G.; Colón, David F.; Donate, Paula B.; Castanheira, Fernanda V.; Gozzi, Aline; Czaikoski, Paula G.; Niedbala, Wanda; Borges, Marcos de Carvalho; Zamboni, Dario Simões; Liew, Foo Y.; Cunha, Fernando de Queiroz; Alves Filho, José Carlos Farias
Source: Nature Communications. Unidade: FMRP
Subjects: IMUNOSSUPRESSÃO, INTERLEUCINAS, PACIENTES, SEPSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NASCIMENTO, Daniele C. et al. IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population. Nature Communications, v. 8, 2017Tradução . . Disponível em: https://doi.org/10.1038/ncomms14919. Acesso em: 07 nov. 2024.
APA
Nascimento, D. C., Melo, P. H., Piñeros, A. R., Ferreira, R. G., Colón, D. F., Donate, P. B., et al. (2017). IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population. Nature Communications, 8. doi:10.1038/ncomms14919
NLM
Nascimento DC, Melo PH, Piñeros AR, Ferreira RG, Colón DF, Donate PB, Castanheira FV, Gozzi A, Czaikoski PG, Niedbala W, Borges M de C, Zamboni DS, Liew FY, Cunha F de Q, Alves Filho JCF. IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population [Internet]. Nature Communications. 2017 ; 8[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/ncomms14919
Vancouver
Nascimento DC, Melo PH, Piñeros AR, Ferreira RG, Colón DF, Donate PB, Castanheira FV, Gozzi A, Czaikoski PG, Niedbala W, Borges M de C, Zamboni DS, Liew FY, Cunha F de Q, Alves Filho JCF. IL-33 contributes to sepsis-induced long-term immunosuppression by expanding the regulatory T cell population [Internet]. Nature Communications. 2017 ; 8[citado 2024 nov. 07 ] Available from: https://doi.org/10.1038/ncomms14919
Artigo de periodico
Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice (2016)
Mota, José M.; Leite, Caio A.; Souza, Lucas E.; Melo, Paulo H.; Nascimento, Daniele C.; Wagatsuma, Virginia M. de Deus; Temporal, Jessica; Figueiredo, Florêncio; Noushmehr, Houtan; Alves Filho, José Carlos Farias; Cunha, Fernando de Queiroz; Rego, Eduardo Magalhães
Source: Cancer Immunology Research. Unidade: FMRP
Subjects: MACRÓFAGOS, SEPSE, INFECÇÕES OPORTUNISTAS, IMUNOSSUPRESSÃO, MORTALIDADE, NEOPLASIAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOTA, José M. et al. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice. Cancer Immunology Research, v. 4, n. 4, p. 312-322, 2016Tradução . . Disponível em: https://doi.org/10.1158/2326-6066.CIR-15-0170. Acesso em: 07 nov. 2024.
APA
Mota, J. M., Leite, C. A., Souza, L. E., Melo, P. H., Nascimento, D. C., Wagatsuma, V. M. de D., et al. (2016). Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice. Cancer Immunology Research, 4( 4), 312-322. doi:10.1158/2326-6066.CIR-15-0170
NLM
Mota JM, Leite CA, Souza LE, Melo PH, Nascimento DC, Wagatsuma VM de D, Temporal J, Figueiredo F, Noushmehr H, Alves Filho JCF, Cunha F de Q, Rego EM. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice [Internet]. Cancer Immunology Research. 2016 ; 4( 4): 312-322.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1158/2326-6066.CIR-15-0170
Vancouver
Mota JM, Leite CA, Souza LE, Melo PH, Nascimento DC, Wagatsuma VM de D, Temporal J, Figueiredo F, Noushmehr H, Alves Filho JCF, Cunha F de Q, Rego EM. Post-sepsis state induces tumor-associated macrophage accumulation through CXCR4/CXCL12 and favors tumor progression in mice [Internet]. Cancer Immunology Research. 2016 ; 4( 4): 312-322.[citado 2024 nov. 07 ] Available from: https://doi.org/10.1158/2326-6066.CIR-15-0170

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