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Artigo de periodico
Silencing of nuclear factor kappa b 1 gene expression inhibits colony formation, cell migration and invasion via the downregulation of interleukin 1 beta and matrix metallopeptidase 9 in renal cell carcinoma (2019)
Teixeira, Luiz Felipe S. ; Peron, Jean Pierre Schatzmann ; Bellini, Maria Helena
Source: Molecular Biology Reports. Unidade: ICB
Subjects: IMUNOLOGIA, CÉLULAS CULTIVADAS DE TUMOR, NEOPLASIAS RENAIS, EXPRESSÃO GÊNICA, INTERLEUCINA 1, APOPTOSE, QUIMIOTERAPIA, REAÇÃO EM CADEIA POR POLIMERASE, WESTERN BLOTTING, ELISA
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TEIXEIRA, Luiz Felipe S. e PERON, Jean Pierre Schatzmann e BELLINI, Maria Helena. Silencing of nuclear factor kappa b 1 gene expression inhibits colony formation, cell migration and invasion via the downregulation of interleukin 1 beta and matrix metallopeptidase 9 in renal cell carcinoma. Molecular Biology Reports, v. 47, n. 2, p. 1143–1151, 2019Tradução . . Disponível em: https://doi.org/10.1007/s11033-019-05212-9. Acesso em: 08 nov. 2024.
APA
Teixeira, L. F. S., Peron, J. P. S., & Bellini, M. H. (2019). Silencing of nuclear factor kappa b 1 gene expression inhibits colony formation, cell migration and invasion via the downregulation of interleukin 1 beta and matrix metallopeptidase 9 in renal cell carcinoma. Molecular Biology Reports, 47( 2), 1143–1151. doi:10.1007/s11033-019-05212-9
NLM
Teixeira LFS, Peron JPS, Bellini MH. Silencing of nuclear factor kappa b 1 gene expression inhibits colony formation, cell migration and invasion via the downregulation of interleukin 1 beta and matrix metallopeptidase 9 in renal cell carcinoma [Internet]. Molecular Biology Reports. 2019 ; 47( 2): 1143–1151.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1007/s11033-019-05212-9
Vancouver
Teixeira LFS, Peron JPS, Bellini MH. Silencing of nuclear factor kappa b 1 gene expression inhibits colony formation, cell migration and invasion via the downregulation of interleukin 1 beta and matrix metallopeptidase 9 in renal cell carcinoma [Internet]. Molecular Biology Reports. 2019 ; 47( 2): 1143–1151.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1007/s11033-019-05212-9
Artigo de periodico
BAY 41-2272 inhibits human neutrophil functions (2019)
Rosa, Paola Vendramini Ferreira; Carvalho, Marina Uchoa Wall Barbosa de; Pereira, Paulo Vitor Soeiro ; Harumi, Renata Cruz; Albuquerque, Rafael Sales de ; Antunes, Edson ; Condino Neto, Antonio
Source: International Immunopharmacology. Unidade: ICB
Subjects: NEUTRÓFILOS, IMUNOLOGIA, ADJUVANTES IMUNOLÓGICOS, MONÓCITOS, CITOCINAS
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ROSA, Paola Vendramini Ferreira et al. BAY 41-2272 inhibits human neutrophil functions. International Immunopharmacology, v. 75, n. art. 105767, p. 7 , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105767. Acesso em: 08 nov. 2024.
APA
Rosa, P. V. F., Carvalho, M. U. W. B. de, Pereira, P. V. S., Harumi, R. C., Albuquerque, R. S. de, Antunes, E., & Condino Neto, A. (2019). BAY 41-2272 inhibits human neutrophil functions. International Immunopharmacology, 75( art. 105767), 7 . doi:10.1016/j.intimp.2019.105767
NLM
Rosa PVF, Carvalho MUWB de, Pereira PVS, Harumi RC, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human neutrophil functions [Internet]. International Immunopharmacology. 2019 ; 75( art. 105767): 7 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.intimp.2019.105767
Vancouver
Rosa PVF, Carvalho MUWB de, Pereira PVS, Harumi RC, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human neutrophil functions [Internet]. International Immunopharmacology. 2019 ; 75( art. 105767): 7 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.intimp.2019.105767
Trabalho de evento-resumo periodico
In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites (2019)
Alvarez, José Maria ; Santana D. Y.; Silva do Nascimento, R.; Fevereiro, M.; Borges da Silva, H.; Barreto-Chaves, Maria Luiza Morais ; Marinho, Claudio Romero Farias ; Epiphanio, Sabrina ; Câmara, Niels Olsen Saraiva ; Lima, Maria Regina D'Império
Source: European Journal of Immunology. Unidades: ICB, FCF
Subjects: DOENÇA DE CHAGAS EM ANIMAL, TRYPANOSOMA CRUZI, CARDIOPATIAS, CAMUNDONGOS, ANIMAIS PARASITOS, IMUNOLOGIA, IMUNOPARASITOLOGIA, ANTIPARASITÁRIOS
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ALVAREZ, José Maria et al. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. Disponível em: https://doi.org/10.1002/eji.201970400. Acesso em: 08 nov. 2024. , 2019
APA
Alvarez, J. M., Santana D. Y.,, Silva do Nascimento, R., Fevereiro, M., Borges da Silva, H., Barreto-Chaves, M. L. M., et al. (2019). In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. doi:10.1002/eji.201970400
NLM
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/eji.201970400
Vancouver
Alvarez JM, Santana D. Y., Silva do Nascimento R, Fevereiro M, Borges da Silva H, Barreto-Chaves MLM, Marinho CRF, Epiphanio S, Câmara NOS, Lima MRD'I. In vivo roles of the immune response and cardiomyocytes in the heart response to Trypanosoma cruzi parasites [Internet]. European Journal of Immunology. 2019 ; 49 1196-1197.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/eji.201970400
Trabalho de evento-resumo periodico
Thermal and metabolic biphasic response to LPS in chicks (2019)
Bícego, Kênia Cardoso ; Amaral-Silva, Lara do ; Steiner, Alexandre Alarcon ; Silva, Welex Cândido da ; Batalhão, Luciane Helena Gargaglioni
Source: Revista Cubana de Investigaciones Biomedicas. Conference titles: PanAmerican Congress of Physiological Sciences. Unidade: ICB
Subjects: IMUNOLOGIA, TEMPERATURA ANIMAL, TEMPERATURA CORPORAL, INFLAMAÇÃO, MODELOS ANIMAIS, ANÓXIA, METABOLISMO ANIMAL, PINTOS, RATOS, ENDOTOXINAS, AVES DOMÉSTICAS, MAMÍFEROS
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BÍCEGO, Kênia Cardoso et al. Thermal and metabolic biphasic response to LPS in chicks. Revista Cubana de Investigaciones Biomedicas. Havana: Editorial Ciencias Medicas. Disponível em: http://www.revibiomedica.sld.cu/index.php/ibi/issue/view/17. Acesso em: 08 nov. 2024. , 2019
APA
Bícego, K. C., Amaral-Silva, L. do, Steiner, A. A., Silva, W. C. da, & Batalhão, L. H. G. (2019). Thermal and metabolic biphasic response to LPS in chicks. Revista Cubana de Investigaciones Biomedicas. Havana: Editorial Ciencias Medicas. Recuperado de http://www.revibiomedica.sld.cu/index.php/ibi/issue/view/17
NLM
Bícego KC, Amaral-Silva L do, Steiner AA, Silva WC da, Batalhão LHG. Thermal and metabolic biphasic response to LPS in chicks [Internet]. Revista Cubana de Investigaciones Biomedicas. 2019 ; 38( Supl. Especial ( 5): 2 .[citado 2024 nov. 08 ] Available from: http://www.revibiomedica.sld.cu/index.php/ibi/issue/view/17
Vancouver
Bícego KC, Amaral-Silva L do, Steiner AA, Silva WC da, Batalhão LHG. Thermal and metabolic biphasic response to LPS in chicks [Internet]. Revista Cubana de Investigaciones Biomedicas. 2019 ; 38( Supl. Especial ( 5): 2 .[citado 2024 nov. 08 ] Available from: http://www.revibiomedica.sld.cu/index.php/ibi/issue/view/17
Artigo de periodico
Understanding the metabolic profile of macrophages during the regenerative process in zebrafish (2019)
Paredes, Lais Cavalieri ; Câmara, Niels Olsen Saraiva ; Braga, Tárcio Teodoro
Source: Frontiers in Physiology. Unidade: ICB
Subjects: IMUNOLOGIA, PEIXES DE ÁGUA DOCE, TELEOSTEI, PEIXES TROPICAIS, MACRÓFAGOS, REGENERAÇÃO (FENÔMENOS BIOLÓGICOS), NEOVASCULARIZAÇÃO PATOLÓGICA, CITOCINAS
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PAREDES, Lais Cavalieri e CÂMARA, Niels Olsen Saraiva e BRAGA, Tárcio Teodoro. Understanding the metabolic profile of macrophages during the regenerative process in zebrafish. Frontiers in Physiology, v. 10, p. 19 , 2019Tradução . . Disponível em: https://doi.org/10.3389/fphys.2019.00617. Acesso em: 08 nov. 2024.
APA
Paredes, L. C., Câmara, N. O. S., & Braga, T. T. (2019). Understanding the metabolic profile of macrophages during the regenerative process in zebrafish. Frontiers in Physiology, 10, 19 . doi:10.3389/fphys.2019.00617
NLM
Paredes LC, Câmara NOS, Braga TT. Understanding the metabolic profile of macrophages during the regenerative process in zebrafish [Internet]. Frontiers in Physiology. 2019 ; 10 19 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fphys.2019.00617
Vancouver
Paredes LC, Câmara NOS, Braga TT. Understanding the metabolic profile of macrophages during the regenerative process in zebrafish [Internet]. Frontiers in Physiology. 2019 ; 10 19 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fphys.2019.00617
Artigo de periodico
Aedes aegypti saliva impairs M1-associated proinflammatory phenotype without promoting or affecting M2 polarization of murine macrophages (2019)
Barros, Michele S.; Lara, Priscila G; Fonseca, Monique T; Moretti, Eduardo H ; Filgueiras, Luciano R; Martins, Joilson de Oliveira ; Capurro, Margareth de Lara ; Steiner, Alexandre Alarcon ; Nunes, Anderson de Sá
Source: Parasites Vectors. Unidades: FCF, ICB
Subjects: AEDES, FENÓTIPOS, MACRÓFAGOS, IMUNOLOGIA, PARASITOLOGIA
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BARROS, Michele S. et al. Aedes aegypti saliva impairs M1-associated proinflammatory phenotype without promoting or affecting M2 polarization of murine macrophages. Parasites Vectors, v. 12, p. 1-15 art. 239, 2019Tradução . . Disponível em: https://doi.org/10.1186/s13071-019-3487-7. Acesso em: 08 nov. 2024.
APA
Barros, M. S., Lara, P. G., Fonseca, M. T., Moretti, E. H., Filgueiras, L. R., Martins, J. de O., et al. (2019). Aedes aegypti saliva impairs M1-associated proinflammatory phenotype without promoting or affecting M2 polarization of murine macrophages. Parasites Vectors, 12, 1-15 art. 239. doi:10.1186/s13071-019-3487-7
NLM
Barros MS, Lara PG, Fonseca MT, Moretti EH, Filgueiras LR, Martins J de O, Capurro M de L, Steiner AA, Nunes A de S. Aedes aegypti saliva impairs M1-associated proinflammatory phenotype without promoting or affecting M2 polarization of murine macrophages [Internet]. Parasites Vectors. 2019 ; 12 1-15 art. 239.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1186/s13071-019-3487-7
Vancouver
Barros MS, Lara PG, Fonseca MT, Moretti EH, Filgueiras LR, Martins J de O, Capurro M de L, Steiner AA, Nunes A de S. Aedes aegypti saliva impairs M1-associated proinflammatory phenotype without promoting or affecting M2 polarization of murine macrophages [Internet]. Parasites Vectors. 2019 ; 12 1-15 art. 239.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1186/s13071-019-3487-7
Artigo de periodico
Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients (2019)
Pedroso, Juliana de Fatima ; Lotfollahi, Zahra ; Albattarni, Ghadeer; Schulz, Maiara Arruda; Monteiro, Andrea Moreira; Sehnem, André Luiz; Gidlund, Magnus Ake ; Figueiredo Neto, Antonio Martins ; Jardini, Maria Aparecida Neves
Source: Scientific Reports. Unidades: IF, ICB
Subjects: PERIODONTITE, DIABETES MELLITUS, DOENÇAS CARDIOVASCULARES, HIPERGLICEMIA, INFLAMAÇÃO, COLESTEROL, IMUNOLOGIA
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PEDROSO, Juliana de Fatima et al. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients. Scientific Reports, v. 9, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-52498-7. Acesso em: 08 nov. 2024.
APA
Pedroso, J. de F., Lotfollahi, Z., Albattarni, G., Schulz, M. A., Monteiro, A. M., Sehnem, A. L., et al. (2019). Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients. Scientific Reports, 9. doi:10.1038/s41598-019-52498-7
NLM
Pedroso J de F, Lotfollahi Z, Albattarni G, Schulz MA, Monteiro AM, Sehnem AL, Gidlund MA, Figueiredo Neto AM, Jardini MAN. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients [Internet]. Scientific Reports. 2019 ; 9[citado 2024 nov. 08 ] Available from: https://doi.org/10.1038/s41598-019-52498-7
Vancouver
Pedroso J de F, Lotfollahi Z, Albattarni G, Schulz MA, Monteiro AM, Sehnem AL, Gidlund MA, Figueiredo Neto AM, Jardini MAN. Influence of periodontal disease on cardiovascular markers in diabetes mellitus patients [Internet]. Scientific Reports. 2019 ; 9[citado 2024 nov. 08 ] Available from: https://doi.org/10.1038/s41598-019-52498-7
Artigo de periodico
NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis (2019)
Braga, Tárcio Teodoro ; Brandão, Wesley Nogueira ; Azevedo, Hatylas ; Terra, Fernanda Fernandes; Feitosa, Amanda Campelo Lima Melo; Pereira, Felipe Valença; Oliveira, Vinicius de Andrade ; Hiyane, Meire Ioshie; Peron, Jean Pierre Schatzmann ; Câmara, Niels Olsen Saraiva
Source: Clinical Science. Unidade: ICB
Subjects: IMUNOLOGIA, AUTOIMUNIDADE, NUCLEOTÍDEOS, CITOCINAS, DOENÇAS AUTOIMUNES, LINFÓCITOS T, ENCEFALOMIELITE ANIMAL, MODELOS ANIMAIS, FATORES DE TRANSCRIÇÃO
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BRAGA, Tárcio Teodoro et al. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, v. 133, n. 17, p. 1901-1916, 2019Tradução . . Disponível em: https://doi.org/10.1042/CS20190506. Acesso em: 08 nov. 2024.
APA
Braga, T. T., Brandão, W. N., Azevedo, H., Terra, F. F., Feitosa, A. C. L. M., Pereira, F. V., et al. (2019). NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis. Clinical Science, 133( 17), 1901-1916. doi:10.1042/CS20190506
NLM
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1042/CS20190506
Vancouver
Braga TT, Brandão WN, Azevedo H, Terra FF, Feitosa ACLM, Pereira FV, Oliveira V de A, Hiyane MI, Peron JPS, Câmara NOS. NLRP3 gain-of-function in CD4+ T lymphocytes ameliorates experimental autoimmune encephalomyelitis [Internet]. Clinical Science. 2019 ; 133( 17): 1901-1916.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1042/CS20190506
Artigo de periodico
Viral receptors for flaviviruses: Not only gatekeepers (2019)
Oliveira, Lilian Gomes de ; Peron, Jean Pierre Schatzmann
Source: Journal of Leukocyte Biology. Unidade: ICB
Assunto: IMUNOLOGIA
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OLIVEIRA, Lilian Gomes de e PERON, Jean Pierre Schatzmann. Viral receptors for flaviviruses: Not only gatekeepers. Journal of Leukocyte Biology, v. 106, n. 3, p. 695-701, 2019Tradução . . Disponível em: https://doi.org/10.1002/JLB.MR1118-460R. Acesso em: 08 nov. 2024.
APA
Oliveira, L. G. de, & Peron, J. P. S. (2019). Viral receptors for flaviviruses: Not only gatekeepers. Journal of Leukocyte Biology, 106( 3), 695-701. doi:10.1002/JLB.MR1118-460R
NLM
Oliveira LG de, Peron JPS. Viral receptors for flaviviruses: Not only gatekeepers [Internet]. Journal of Leukocyte Biology. 2019 ; 106( 3): 695-701.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/JLB.MR1118-460R
Vancouver
Oliveira LG de, Peron JPS. Viral receptors for flaviviruses: Not only gatekeepers [Internet]. Journal of Leukocyte Biology. 2019 ; 106( 3): 695-701.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/JLB.MR1118-460R
Trabalho de evento-resumo periodico
Assessing the role of the complement system in the elimination of Leptospira SPP (2019)
Silva, Priscilla Yuri Okochi Alves da; Midon, Leonardo Moura; Heneimann, Marcos Bryan ; Barbosa, Angela Silva ; Isaac, Lourdes
Source: Molecular Immunology. Conference titles: European Meeting on Complement in Human Disease - EMCHD. Unidades: FMVZ, ICB
Subjects: IMUNOLOGIA, IMUNOPROTEÍNAS, LEPTOSPIROSE, WESTERN BLOTTING, ELISA, LECTINAS
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SILVA, Priscilla Yuri Okochi Alves da et al. Assessing the role of the complement system in the elimination of Leptospira SPP. Molecular Immunology. Oxford: Elsevier Ltd. Disponível em: https://doi.org/10.1016/j.molimm.2019.08.016. Acesso em: 08 nov. 2024. , 2019
APA
Silva, P. Y. O. A. da, Midon, L. M., Heneimann, M. B., Barbosa, A. S., & Isaac, L. (2019). Assessing the role of the complement system in the elimination of Leptospira SPP. Molecular Immunology. Oxford: Elsevier Ltd. doi:10.1016/j.molimm.2019.08.016.
NLM
Silva PYOA da, Midon LM, Heneimann MB, Barbosa AS, Isaac L. Assessing the role of the complement system in the elimination of Leptospira SPP [Internet]. Molecular Immunology. 2019 ; 114 463-464.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.molimm.2019.08.016.
Vancouver
Silva PYOA da, Midon LM, Heneimann MB, Barbosa AS, Isaac L. Assessing the role of the complement system in the elimination of Leptospira SPP [Internet]. Molecular Immunology. 2019 ; 114 463-464.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.molimm.2019.08.016.
Artigo de periodico
Leukotriene‐B4 modulates macrophage metabolism and fat loss in type 1 diabetic mice (2019)
Ramalho, Theresa Raquel de Oliveira ; Ramalingam, Latha ; Ribeiro Junior, Luciano Filgueiras; Festuccia, William Tadeu Lara ; Jancar, Sonia ; Moustaid-Moussa, Naima
Source: Journal of Leukocyte Biology. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, METABOLISMO ANIMAL, LEUCOTRIENOS B, METABOLISMO ENERGÉTICO, CAMUNDONGOS, TECIDO ADIPOSO, HIPERGLICEMIA, HIPERLIPIDEMIA, GLUCAGON, MACRÓFAGOS
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RAMALHO, Theresa Raquel de Oliveira et al. Leukotriene‐B4 modulates macrophage metabolism and fat loss in type 1 diabetic mice. Journal of Leukocyte Biology, v. 106, n. 3, p. 665-675, 2019Tradução . . Disponível em: https://doi.org/10.1002/JLB.MA1218-477RR. Acesso em: 08 nov. 2024.
APA
Ramalho, T. R. de O., Ramalingam, L., Ribeiro Junior, L. F., Festuccia, W. T. L., Jancar, S., & Moustaid-Moussa, N. (2019). Leukotriene‐B4 modulates macrophage metabolism and fat loss in type 1 diabetic mice. Journal of Leukocyte Biology, 106( 3), 665-675. doi:10.1002/JLB.MA1218-477RR
NLM
Ramalho TR de O, Ramalingam L, Ribeiro Junior LF, Festuccia WTL, Jancar S, Moustaid-Moussa N. Leukotriene‐B4 modulates macrophage metabolism and fat loss in type 1 diabetic mice [Internet]. Journal of Leukocyte Biology. 2019 ; 106( 3): 665-675.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/JLB.MA1218-477RR
Vancouver
Ramalho TR de O, Ramalingam L, Ribeiro Junior LF, Festuccia WTL, Jancar S, Moustaid-Moussa N. Leukotriene‐B4 modulates macrophage metabolism and fat loss in type 1 diabetic mice [Internet]. Journal of Leukocyte Biology. 2019 ; 106( 3): 665-675.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/JLB.MA1218-477RR
Trabalho de evento-resumo periodico
Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway (2019)
Bomfim, Caio César Barbosa ; Amaral, Eduardo Pinheiro ; Mittereder, Lara ; McCann, K.; Costa, Diego Luís ; Namasivayam, Sivaranjani ; Swamydas, M.; Moayeri, Mahtab ; Lima, Maria Regina D'Império ; Sher, Alan
Source: European Journal of Immunology. Conference titles: International Congress of Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, IMUNOLOGIA, MYCOBACTERIUM TUBERCULOSIS, ENZIMAS, METABÓLITOS, ATIVAÇÃO ENZIMÁTICA, MACRÓFAGOS, FAGOCITOSE, INTERFERONS, MEMBRANA PLASMÁTICA
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BOMFIM, Caio César Barbosa et al. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. Disponível em: https://doi.org/10.1002/eji.201970400. Acesso em: 08 nov. 2024. , 2019
APA
Bomfim, C. C. B., Amaral, E. P., Mittereder, L., McCann, K., Costa, D. L., Namasivayam, S., et al. (2019). Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway. European Journal of Immunology. Weinheim: Wiley-VCH Verlag GmbH & Co. KGaA. doi:10.1002/eji.201970400
NLM
Bomfim CCB, Amaral EP, Mittereder L, McCann K, Costa DL, Namasivayam S, Swamydas M, Moayeri M, Lima MRD'I, Sher A. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway [Internet]. European Journal of Immunology. 2019 ; 49 1129.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/eji.201970400
Vancouver
Bomfim CCB, Amaral EP, Mittereder L, McCann K, Costa DL, Namasivayam S, Swamydas M, Moayeri M, Lima MRD'I, Sher A. Irg1 induction in macrophages infected with Mycobacterium tuberculosis depends on STING/IFNAR-dependent signaling pathway [Internet]. European Journal of Immunology. 2019 ; 49 1129.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1002/eji.201970400
Artigo de periodico
Increased grp78 transcription is correlated to reduced tlr4 transcription in patients surviving sepsis (2019)
Stan, R. C; Bonin, C. Pinto; Porto, R; Soriano, Francisco Garcia ; Camargo, Maristela Martins de
Source: Clinical and experimental immunology. Unidades: FM, ICB
Subjects: TRANSCRIÇÃO GÊNICA, SEPSE, RECEPTORES IMUNOLÓGICOS, EXPRESSÃO GÊNICA, REGULAÇÃO GÊNICA, IMUNOLOGIA
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STAN, R. C et al. Increased grp78 transcription is correlated to reduced tlr4 transcription in patients surviving sepsis. Clinical and experimental immunology, v. 198, n. 2, p. 273-280, 2019Tradução . . Disponível em: https://doi.org/10.1111/cei.13348. Acesso em: 08 nov. 2024.
APA
Stan, R. C., Bonin, C. P., Porto, R., Soriano, F. G., & Camargo, M. M. de. (2019). Increased grp78 transcription is correlated to reduced tlr4 transcription in patients surviving sepsis. Clinical and experimental immunology, 198( 2), 273-280. doi:10.1111/cei.13348
NLM
Stan RC, Bonin CP, Porto R, Soriano FG, Camargo MM de. Increased grp78 transcription is correlated to reduced tlr4 transcription in patients surviving sepsis [Internet]. Clinical and experimental immunology. 2019 ; 198( 2): 273-280.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1111/cei.13348
Vancouver
Stan RC, Bonin CP, Porto R, Soriano FG, Camargo MM de. Increased grp78 transcription is correlated to reduced tlr4 transcription in patients surviving sepsis [Internet]. Clinical and experimental immunology. 2019 ; 198( 2): 273-280.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1111/cei.13348
Artigo de periodico
Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein (2019)
Cavalcante, Paula Andréa Malveira ; Alenina, Natalia ; Budu, Alexandre ; Lima, Leandro Ceotto Freitas ; Silva, Thaís Alves da ; Agudelo, Juan Sebastian Henao ; Qadri, Fatimunnisa ; Câmara, Niels Olsen Saraiva ; Bader, Michael ; Araújo, Ronaldo de Carvalho
Source: Mediators of Inflammation. Unidade: ICB
Subjects: IMUNOLOGIA, NEFROPATIAS EM ANIMAL, MACRÓFAGOS, PROTEÍNAS, MEDULA ÓSSEA DE ANIMAL, TÍBIA, PRESSÃO SANGUÍNEA, RATOS, REAÇÃO EM CADEIA POR POLIMERASE, ELISA EM ANIMAL, WESTERN BLOTTING
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ABNT
CAVALCANTE, Paula Andréa Malveira et al. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein. Mediators of Inflammation, v. 2019, p. 14 , 2019Tradução . . Disponível em: https://doi.org/10.1155/2019/9086758. Acesso em: 08 nov. 2024.
APA
Cavalcante, P. A. M., Alenina, N., Budu, A., Lima, L. C. F., Silva, T. A. da, Agudelo, J. S. H., et al. (2019). Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein. Mediators of Inflammation, 2019, 14 . doi:10.1155/2019/9086758
NLM
Cavalcante PAM, Alenina N, Budu A, Lima LCF, Silva TA da, Agudelo JSH, Qadri F, Câmara NOS, Bader M, Araújo R de C. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein [Internet]. Mediators of Inflammation. 2019 ; 2019 14 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1155/2019/9086758
Vancouver
Cavalcante PAM, Alenina N, Budu A, Lima LCF, Silva TA da, Agudelo JSH, Qadri F, Câmara NOS, Bader M, Araújo R de C. Nephropathy in hypertensive animals is linked to M2 macrophages and increased expression of the YM1/Chi3l3 protein [Internet]. Mediators of Inflammation. 2019 ; 2019 14 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1155/2019/9086758
Artigo de periodico
Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model (2019)
Rempel, Lisienny Campoli Tono ; Faustino, Viviane Dias; Foresto Neto, Orestes ; Fanelli, Camilla ; Arias, Simone Costa Alarcon; Moreira, Gizely Cristina da Silva; Nascimento, Thalita Fabiana do; Ávila, Victor Ferreira de; Malheiros, Denise Maria Avancini Costa ; Câmara, Niels Olsen Saraiva ; Fujihara, Clarice Kazue; Zatz, Roberto
Source: American Journa Physiology: Renal Physiology. Unidades: FM, ICB
Subjects: IMUNOLOGIA, ANÓXIA, INSUFICIÊNCIA RENAL AGUDA, RATOS WISTAR, NEFROPATIAS EM ANIMAL, ESTRESSE OXIDATIVO, IMUNIDADE, IMUNOHISTOQUÍMICA
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ABNT
REMPEL, Lisienny Campoli Tono et al. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model. American Journa Physiology: Renal Physiology, v. 317, n. 5, p. F1285-F1292, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00367.2018. Acesso em: 08 nov. 2024.
APA
Rempel, L. C. T., Faustino, V. D., Foresto Neto, O., Fanelli, C., Arias, S. C. A., Moreira, G. C. da S., et al. (2019). Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model. American Journa Physiology: Renal Physiology, 317( 5), F1285-F1292. doi:10.1152/ajprenal.00367.2018
NLM
Rempel LCT, Faustino VD, Foresto Neto O, Fanelli C, Arias SCA, Moreira GC da S, Nascimento TF do, Ávila VF de, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model [Internet]. American Journa Physiology: Renal Physiology. 2019 ; 317( 5): F1285-F1292.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1152/ajprenal.00367.2018
Vancouver
Rempel LCT, Faustino VD, Foresto Neto O, Fanelli C, Arias SCA, Moreira GC da S, Nascimento TF do, Ávila VF de, Malheiros DMAC, Câmara NOS, Fujihara CK, Zatz R. Chronic exposure to hypoxia attenuates renal injury and innate immunity activation in the remnant kidney model [Internet]. American Journa Physiology: Renal Physiology. 2019 ; 317( 5): F1285-F1292.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1152/ajprenal.00367.2018
Artigo de periodico
Flagellin/NLRC4 pathway rescues NLRP3-inflammasome defect in dendritic cells from HIV-infected patients: perspective for new adjuvant in immunocompromised individuals (2019)
Reis, Edione Cristina dos ; Leal, Vinicius Nunes Cordeiro ; Silva, Jaine Soares Lima da; Fernandes, Fernanda Pereira; Lima, Dhêmerson Souza de ; Bargieri, Bruna Cunha de Alencar ; Pontillo, Alessandra
Source: Frontiers in Immunology. Unidades: ICB, FM
Subjects: IMUNOLOGIA, MOLÉCULAS DE ADESÃO CELULAR, CÉLULAS DENDRÍTICAS, INFECÇÕES POR HIV, CITOCINAS, ADJUVANTES IMUNOLÓGICOS, MONÓCITOS, INIBIDORES QUÍMICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
REIS, Edione Cristina dos et al. Flagellin/NLRC4 pathway rescues NLRP3-inflammasome defect in dendritic cells from HIV-infected patients: perspective for new adjuvant in immunocompromised individuals. Frontiers in Immunology, v. 10, p. 13 , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.01291. Acesso em: 08 nov. 2024.
APA
Reis, E. C. dos, Leal, V. N. C., Silva, J. S. L. da, Fernandes, F. P., Lima, D. S. de, Bargieri, B. C. de A., & Pontillo, A. (2019). Flagellin/NLRC4 pathway rescues NLRP3-inflammasome defect in dendritic cells from HIV-infected patients: perspective for new adjuvant in immunocompromised individuals. Frontiers in Immunology, 10, 13 . doi:10.3389/fimmu.2019.01291
NLM
Reis EC dos, Leal VNC, Silva JSL da, Fernandes FP, Lima DS de, Bargieri BC de A, Pontillo A. Flagellin/NLRC4 pathway rescues NLRP3-inflammasome defect in dendritic cells from HIV-infected patients: perspective for new adjuvant in immunocompromised individuals [Internet]. Frontiers in Immunology. 2019 ; 10 13 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.01291
Vancouver
Reis EC dos, Leal VNC, Silva JSL da, Fernandes FP, Lima DS de, Bargieri BC de A, Pontillo A. Flagellin/NLRC4 pathway rescues NLRP3-inflammasome defect in dendritic cells from HIV-infected patients: perspective for new adjuvant in immunocompromised individuals [Internet]. Frontiers in Immunology. 2019 ; 10 13 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.01291
Artigo de periodico
Endothelial cells tissue-specific origins affects their responsiveness to TGF-β2 during endothelial-to-mesenchymal transition (2019)
Ferreira, Fernanda Ursoli ; Souza, Lucas Eduardo Botelho ; Thomé, Carolina Hassibe ; Pinto, Mariana Tomazini; Origassa, Clarice Silva Taemi ; Salustiano, Suellen Gomes; Faça, Vitor Marcel ; Câmara, Niels Olsen Saraiva ; Haddad, Simone Kashima ; Covas, Dimas Tadeu
Source: International Journal of Molecular Sciences. Unidades: FMRP, ICB, FCFRP
Subjects: CÉLULAS ENDOTELIAIS, CÉLULAS CULTIVADAS, CITOMETRIA DE FLUXO, FENÓTIPOS, IMUNOLOGIA, FIBROBLASTOS
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ABNT
FERREIRA, Fernanda Ursoli et al. Endothelial cells tissue-specific origins affects their responsiveness to TGF-β2 during endothelial-to-mesenchymal transition. International Journal of Molecular Sciences, v. 20, n. 3, p. [14] , 2019Tradução . . Disponível em: https://doi.org/10.3390/ijms20030458. Acesso em: 08 nov. 2024.
APA
Ferreira, F. U., Souza, L. E. B., Thomé, C. H., Pinto, M. T., Origassa, C. S. T., Salustiano, S. G., et al. (2019). Endothelial cells tissue-specific origins affects their responsiveness to TGF-β2 during endothelial-to-mesenchymal transition. International Journal of Molecular Sciences, 20( 3), [14] . doi:10.3390/ijms20030458
NLM
Ferreira FU, Souza LEB, Thomé CH, Pinto MT, Origassa CST, Salustiano SG, Faça VM, Câmara NOS, Haddad SK, Covas DT. Endothelial cells tissue-specific origins affects their responsiveness to TGF-β2 during endothelial-to-mesenchymal transition [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 3): [14] .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3390/ijms20030458
Vancouver
Ferreira FU, Souza LEB, Thomé CH, Pinto MT, Origassa CST, Salustiano SG, Faça VM, Câmara NOS, Haddad SK, Covas DT. Endothelial cells tissue-specific origins affects their responsiveness to TGF-β2 during endothelial-to-mesenchymal transition [Internet]. International Journal of Molecular Sciences. 2019 ; 20( 3): [14] .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3390/ijms20030458
Artigo de periodico
Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation (2019)
Marques, Otavio Cabral ; Schimke, Lena Friederike; Oliveira Junior, Edgar Borges de ; Khawanky, Nadia El ; Ramos, Rodrigo Nalio; Al-Ramadi, Basel K. ; Segundo, Gesmar Rodrigues Silva ; Ochs, Hans D.; Condino Neto, Antonio
Source: Frontiers in Immunology. Unidade: ICB
Subjects: IMUNOLOGIA, CITOMETRIA DE FLUXO, DOENÇAS IMUNOLÓGICAS, IMUNODEFICIÊNCIA DE VARIÁVEL COMUM, MUTAÇÃO GENÉTICA, SEQUENCIAMENTO GENÉTICO, GENOMAS, FENÓTIPOS, AUTOIMUNIDADE
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ABNT
MARQUES, Otavio Cabral et al. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation. Frontiers in Immunology, v. 10, p. 23 , 2019Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2019.02742. Acesso em: 08 nov. 2024.
APA
Marques, O. C., Schimke, L. F., Oliveira Junior, E. B. de, Khawanky, N. E., Ramos, R. N., Al-Ramadi, B. K., et al. (2019). Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation. Frontiers in Immunology, 10, 23 . doi:10.3389/fimmu.2019.02742
NLM
Marques OC, Schimke LF, Oliveira Junior EB de, Khawanky NE, Ramos RN, Al-Ramadi BK, Segundo GRS, Ochs HD, Condino Neto A. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation [Internet]. Frontiers in Immunology. 2019 ;10 23 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.02742
Vancouver
Marques OC, Schimke LF, Oliveira Junior EB de, Khawanky NE, Ramos RN, Al-Ramadi BK, Segundo GRS, Ochs HD, Condino Neto A. Flow cytometry contributions for the diagnosis and immunopathological characterization of primary immunodeficiency diseases with immune dysregulation [Internet]. Frontiers in Immunology. 2019 ;10 23 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.3389/fimmu.2019.02742
Artigo de periodico
MiR-23~27~24–mediated control of humoral immunity reveals a TOX-driven regulatory circuit in follicular helper T cell differentiation (2019)
Wu, Cheng-Jang ; Cho, Sunglim ; Huang, Hsi-Yuan ; Lu, Chun-Hao ; Russ, Jasmin; Cruz, Leilani O.; Cunha, Flavia Franco da ; Chen, Mei-Chi ; Lin, Ling-li; Warner, Lindsey M. ; Liao, Hsin-Kai ; Utzschneider, Daniel T. ; Quon, Sara ; Berner, Jacqueline; Câmara, Niels Olsen Saraiva ; Zehn, Dietmar ; Belmonte, Juan Carlos Izpisua ; Chen, Li-Chen; Huang, Shiang-Fu ; Kuo, Ming-Ling ; Lu, Li-Fan
Source: Science Advances. Unidade: ICB
Subjects: IMUNOLOGIA, LINFÓCITOS T, DIFERENCIAÇÃO CELULAR, IMUNOLOGIA CELULAR, EXPRESSÃO GÊNICA, TRANSCRIÇÃO GÊNICA, MICRORNAS, ÁCIDOS NUCLEICOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WU, Cheng-Jang et al. MiR-23~27~24–mediated control of humoral immunity reveals a TOX-driven regulatory circuit in follicular helper T cell differentiation. Science Advances, v. 5, p. 13 , 2019Tradução . . Disponível em: https://doi.org/10.1126/sciadv.aaw1715. Acesso em: 08 nov. 2024.
APA
Wu, C. -J., Cho, S., Huang, H. -Y., Lu, C. -H., Russ, J., Cruz, L. O., et al. (2019). MiR-23~27~24–mediated control of humoral immunity reveals a TOX-driven regulatory circuit in follicular helper T cell differentiation. Science Advances, 5, 13 . doi:10.1126/sciadv.aaw1715
NLM
Wu C-J, Cho S, Huang H-Y, Lu C-H, Russ J, Cruz LO, Cunha FF da, Chen M-C, Lin L-li, Warner LM, Liao H-K, Utzschneider DT, Quon S, Berner J, Câmara NOS, Zehn D, Belmonte JCI, Chen L-C, Huang S-F, Kuo M-L, Lu L-F. MiR-23~27~24–mediated control of humoral immunity reveals a TOX-driven regulatory circuit in follicular helper T cell differentiation [Internet]. Science Advances. 2019 ; 5 13 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1126/sciadv.aaw1715
Vancouver
Wu C-J, Cho S, Huang H-Y, Lu C-H, Russ J, Cruz LO, Cunha FF da, Chen M-C, Lin L-li, Warner LM, Liao H-K, Utzschneider DT, Quon S, Berner J, Câmara NOS, Zehn D, Belmonte JCI, Chen L-C, Huang S-F, Kuo M-L, Lu L-F. MiR-23~27~24–mediated control of humoral immunity reveals a TOX-driven regulatory circuit in follicular helper T cell differentiation [Internet]. Science Advances. 2019 ; 5 13 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1126/sciadv.aaw1715
Artigo de periodico
BAY 41-2272 inhibits human T lymphocyte functions (2019)
Carvalho, Marina Uchôa Wall Barbosa de; Rosa, Paola Vendramini Ferreira; Kubo, Christina Arslanian; Pereira, Paulo Vitor Soeiro ; Albuquerque, Rafael Sales de ; Antunes, Edson ; Condino Neto, Antonio
Source: International Immunopharmacology. Unidade: ICB
Subjects: IMUNOLOGIA, TESTE DE TUKEY, LINFÓCITOS T, DOENÇAS AUTOIMUNES, FÁRMACOS IMUNOSSUPRESSORES, INTERFERONS, IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO, Marina Uchôa Wall Barbosa de et al. BAY 41-2272 inhibits human T lymphocyte functions. International Immunopharmacology, v. 77, p. 7 , 2019Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2019.105976. Acesso em: 08 nov. 2024.
APA
Carvalho, M. U. W. B. de, Rosa, P. V. F., Kubo, C. A., Pereira, P. V. S., Albuquerque, R. S. de, Antunes, E., & Condino Neto, A. (2019). BAY 41-2272 inhibits human T lymphocyte functions. International Immunopharmacology, 77, 7 . doi:10.1016/j.intimp.2019.105976
NLM
Carvalho MUWB de, Rosa PVF, Kubo CA, Pereira PVS, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human T lymphocyte functions [Internet]. International Immunopharmacology. 2019 ; 77 7 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.intimp.2019.105976
Vancouver
Carvalho MUWB de, Rosa PVF, Kubo CA, Pereira PVS, Albuquerque RS de, Antunes E, Condino Neto A. BAY 41-2272 inhibits human T lymphocyte functions [Internet]. International Immunopharmacology. 2019 ; 77 7 .[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.intimp.2019.105976

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