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Artigo de periodico
β-adrenergic stimulation-induced PVAT dysfunction in male sex: a role for 11β-Hydroxysteroid Dehydrogenase-1 (2024)
Victorio, Jamaira Aparecida ; Santos, Leticia Barsotti dos ; Aprahamian, Tamar ; Costa, Raul Gobato da ; Mousovich Neto, Felippe ; Oliveira, Helena Coutinho Franco de ; Mori, Marcelo Alves da Silva ; Davel, Ana Paula ; Rossoni, Luciana Venturini
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, TECIDO ADIPOSO, AORTA TORÁCICA, ÓXIDO NÍTRICO, ESTRESSE OXIDATIVO, RECEPTORES ADRENÉRGICOS, MACRÓFAGOS
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ABNT
VICTORIO, Jamaira Aparecida et al. β-adrenergic stimulation-induced PVAT dysfunction in male sex: a role for 11β-Hydroxysteroid Dehydrogenase-1. Endocrinology, v. 165, n. 5, p. 7 , 2024Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqae053. Acesso em: 15 nov. 2024.
APA
Victorio, J. A., Santos, L. B. dos, Aprahamian, T., Costa, R. G. da, Mousovich Neto, F., Oliveira, H. C. F. de, et al. (2024). β-adrenergic stimulation-induced PVAT dysfunction in male sex: a role for 11β-Hydroxysteroid Dehydrogenase-1. Endocrinology, 165( 5), 7 . doi:10.1210/endocr/bqae053
NLM
Victorio JA, Santos LB dos, Aprahamian T, Costa RG da, Mousovich Neto F, Oliveira HCF de, Mori MA da S, Davel AP, Rossoni LV. β-adrenergic stimulation-induced PVAT dysfunction in male sex: a role for 11β-Hydroxysteroid Dehydrogenase-1 [Internet]. Endocrinology. 2024 ; 165( 5): 7 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqae053
Vancouver
Victorio JA, Santos LB dos, Aprahamian T, Costa RG da, Mousovich Neto F, Oliveira HCF de, Mori MA da S, Davel AP, Rossoni LV. β-adrenergic stimulation-induced PVAT dysfunction in male sex: a role for 11β-Hydroxysteroid Dehydrogenase-1 [Internet]. Endocrinology. 2024 ; 165( 5): 7 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqae053
Artigo de periodico
Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats (2024)
Florido Neto, Armando Ribeiro ; Agostini, Lucas ; Silva Junior, Luciano Pedro da ; Nunes, Maria Tereza
Source: Endocrinology. Unidades: ICB, FSP
Subjects: FISIOLOGIA, RATOS WISTAR, MODELOS ANIMAIS DE DOENÇAS, GLICEMIA, CITOCINAS, DIABETES MELLITUS, HIPOTIREOIDISMO ANIMAL, RESISTÊNCIA À INSULINA, FÁRMACOS (METABOLISMO E NUTRIÇÃO), TRIIODOTIRONINA, HORMÔNIOS TIREOIDIANOS
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FLORIDO NETO, Armando Ribeiro et al. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats. Endocrinology, v. 165, p. 14 , 2024Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqae066. Acesso em: 15 nov. 2024.
APA
Florido Neto, A. R., Agostini, L., Silva Junior, L. P. da, & Nunes, M. T. (2024). Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats. Endocrinology, 165, 14 . doi:10.1210/endocr/bqae066
NLM
Florido Neto AR, Agostini L, Silva Junior LP da, Nunes MT. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats [Internet]. Endocrinology. 2024 ; 165 14 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqae066
Vancouver
Florido Neto AR, Agostini L, Silva Junior LP da, Nunes MT. Therapeutical potential of T3 as adjuvant therapy in male alloxan-induced diabetic rats [Internet]. Endocrinology. 2024 ; 165 14 .[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqae066
Artigo de periodico
Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons (2022)
Chaves, Fernanda Machado; Wasinski, Frederick ; Tavares, Mariana Rosolen ; Mansano, Naira da Silva ; Frazão, Renata ; Gusmão, Daniela O. ; Bergonsi, Paula Gabriele Fernandes Quaresma ; Pedroso, João Alfredo Bolivar ; Elias, Carol F. ; List, Edward O. ; Kopchick, John J. ; Szawka, Raphael E. ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, HIPOTÁLAMO, HORMÔNIO DO CRESCIMENTO, FATOR DE CRESCIMENTO INSULIN-LIKE I, NEURÔNIOS, SOMATOSTATINA, EXPRESSÃO GÊNICA, CAMUNDONGOS
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CHAVES, Fernanda Machado et al. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons. Endocrinology, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac045. Acesso em: 15 nov. 2024.
APA
Chaves, F. M., Wasinski, F., Tavares, M. R., Mansano, N. da S., Frazão, R., Gusmão, D. O., et al. (2022). Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons. Endocrinology. doi:10.1210/endocr/bqac045
NLM
Chaves FM, Wasinski F, Tavares MR, Mansano N da S, Frazão R, Gusmão DO, Bergonsi PGFQ, Pedroso JAB, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons [Internet]. Endocrinology. 2022 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac045
Vancouver
Chaves FM, Wasinski F, Tavares MR, Mansano N da S, Frazão R, Gusmão DO, Bergonsi PGFQ, Pedroso JAB, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Effects of the isolated and combined ablation of growth hormone and IGF-1 receptors in somatostatin neurons [Internet]. Endocrinology. 2022 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac045
Artigo de periodico
Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion (2022)
Santos, Willian Oliveira dos ; Wasinski, Frederick ; Tavares, Mariana Rosolen ; Campos, Ana Maria Peracoli; Elias, Carol F. ; List, Edward O. ; Kopchick, John J. ; Szawka, Raphael E. ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIO DO CRESCIMENTO, HIPOTÁLAMO, LEPTINA, CAMUNDONGOS, RNA MENSAGEIRO, GLUTAMATOS, INSULINA
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ABNT
SANTOS, Willian Oliveira dos et al. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion. Endocrinology, v. 163, n. 8, p. 1-17, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac103. Acesso em: 15 nov. 2024.
APA
Santos, W. O. dos, Wasinski, F., Tavares, M. R., Campos, A. M. P., Elias, C. F., List, E. O., et al. (2022). Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion. Endocrinology, 163( 8), 1-17. doi:10.1210/endocr/bqac103
NLM
Santos WO dos, Wasinski F, Tavares MR, Campos AMP, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion [Internet]. Endocrinology. 2022 ; 163( 8): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac103
Vancouver
Santos WO dos, Wasinski F, Tavares MR, Campos AMP, Elias CF, List EO, Kopchick JJ, Szawka RE, Donato Junior J. Ablation of growth hormone receptor in GABAergic neurons leads to increased pulsatile growth hormone secretion [Internet]. Endocrinology. 2022 ; 163( 8): 1-17.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac103
Artigo de periodico
Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells (2022)
Gusmão, Daniela de Oliveira ; Sousa, Maria Eletícia de ; Tavares, Mariana Rosolen ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, HORMÔNIO DO CRESCIMENTO, HORMÔNIO LIBERADOR DE HORMÔNIO DE CRESCIMENTO, GLÂNDULA PITUITÁRIA, RNA MENSAGEIRO, FATOR DE CRESCIMENTO INSULIN-LIKE I, CAMUNDONGOS, HIPOTÁLAMO, INSULINA
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ABNT
GUSMÃO, Daniela de Oliveira et al. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells. Endocrinology, v. 163, n. 11, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqac151. Acesso em: 15 nov. 2024.
APA
Gusmão, D. de O., Sousa, M. E. de, Tavares, M. R., & Donato Junior, J. (2022). Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells. Endocrinology, 163( 11), 1-13. doi:10.1210/endocr/bqac151
NLM
Gusmão D de O, Sousa ME de, Tavares MR, Donato Junior J. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells [Internet]. Endocrinology. 2022 ; 163( 11): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac151
Vancouver
Gusmão D de O, Sousa ME de, Tavares MR, Donato Junior J. Increased GH secretion and body growth in mice carrying ablation of IGF-1 receptor in GH-releasing hormone cells [Internet]. Endocrinology. 2022 ; 163( 11): 1-13.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqac151
Artigo de periodico
Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice (2021)
Bergonsi, Paula Gabriele Fernandes Quaresma ; Wasinski, Frederick ; Mansano, Naira da Silva ; Furigo, Isadora Clivatti ; Teixeira, Pryscila Dryelle Sousa ; Gusmão, Daniela O. ; Frazão, Renata ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, HIPOTÁLAMO, OBESIDADE, DIABETES MELLITUS, INGESTÃO, LEPTINA, CAMUNDONGOS, GABA, CITOCINAS
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ABNT
BERGONSI, Paula Gabriele Fernandes Quaresma et al. Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice. Endocrinology, 2021Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqab168. Acesso em: 15 nov. 2024.
APA
Bergonsi, P. G. F. Q., Wasinski, F., Mansano, N. da S., Furigo, I. C., Teixeira, P. D. S., Gusmão, D. O., et al. (2021). Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice. Endocrinology. doi:10.1210/endocr/bqab168
NLM
Bergonsi PGFQ, Wasinski F, Mansano N da S, Furigo IC, Teixeira PDS, Gusmão DO, Frazão R, Donato Junior J. Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice [Internet]. Endocrinology. 2021 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqab168
Vancouver
Bergonsi PGFQ, Wasinski F, Mansano N da S, Furigo IC, Teixeira PDS, Gusmão DO, Frazão R, Donato Junior J. Leptin receptor expression in GABAergic cells is not sufficient to normalize metabolism and reproduction in mice [Internet]. Endocrinology. 2021 ;[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqab168
Artigo de periodico
Ghrelin-induced food intake, but not GH secretion, requires the expression of the GH receptor in the brain of male mice (2021)
Wasinski, Frederick ; Barrile, Franco ; Pedroso, João Alfredo Bolivar ; Bergonsi, Paula Gabriele Fernandes Quaresma ; Santos, Willian O. dos ; List, Edward O. ; Kopchick, John J. ; Perelló, Mario ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, CITOCINAS, HORMÔNIO DO CRESCIMENTO, HIPOTÁLAMO, GRELINA, CÉREBRO, CAMUNDONGOS, NEUROPEPTÍDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
WASINSKI, Frederick et al. Ghrelin-induced food intake, but not GH secretion, requires the expression of the GH receptor in the brain of male mice. Endocrinology, v. 162, n. 7, p. 1-15, 2021Tradução . . Disponível em: https://doi.org/10.1210/endocr/bqab097. Acesso em: 15 nov. 2024.
APA
Wasinski, F., Barrile, F., Pedroso, J. A. B., Bergonsi, P. G. F. Q., Santos, W. O. dos, List, E. O., et al. (2021). Ghrelin-induced food intake, but not GH secretion, requires the expression of the GH receptor in the brain of male mice. Endocrinology, 162( 7), 1-15. doi:10.1210/endocr/bqab097
NLM
Wasinski F, Barrile F, Pedroso JAB, Bergonsi PGFQ, Santos WO dos, List EO, Kopchick JJ, Perelló M, Donato Junior J. Ghrelin-induced food intake, but not GH secretion, requires the expression of the GH receptor in the brain of male mice [Internet]. Endocrinology. 2021 ; 162( 7): 1-15.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqab097
Vancouver
Wasinski F, Barrile F, Pedroso JAB, Bergonsi PGFQ, Santos WO dos, List EO, Kopchick JJ, Perelló M, Donato Junior J. Ghrelin-induced food intake, but not GH secretion, requires the expression of the GH receptor in the brain of male mice [Internet]. Endocrinology. 2021 ; 162( 7): 1-15.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/endocr/bqab097
Artigo de periodico
Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice (2019)
Teixeira, Pryscila Dryelle Sousa ; Lobo, Angela Maria Ramos ; Furigo, Isadora Clivatti ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, CAMUNDONGOS, LACTAÇÃO ANIMAL, EPIGÊNESE GENÉTICA, METABOLISMO ANIMAL, ADIPOSIDADE, INSULINA
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ABNT
TEIXEIRA, Pryscila Dryelle Sousa et al. Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice. Endocrinology, v. 160, n. 12, p. 2903-2917, 2019Tradução . . Disponível em: https://doi.org/10.1210/en.2019-00639. Acesso em: 15 nov. 2024.
APA
Teixeira, P. D. S., Lobo, A. M. R., Furigo, I. C., & Donato Junior, J. (2019). Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice. Endocrinology, 160( 12), 2903-2917. doi:10.1210/en.2019-00639
NLM
Teixeira PDS, Lobo AMR, Furigo IC, Donato Junior J. Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice [Internet]. Endocrinology. 2019 ; 160( 12): 2903-2917.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2019-00639
Vancouver
Teixeira PDS, Lobo AMR, Furigo IC, Donato Junior J. Brain STAT5 modulates long-term metabolic and epigenetic changes induced by pregnancy and lactation in female mice [Internet]. Endocrinology. 2019 ; 160( 12): 2903-2917.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2019-00639
Artigo de periodico
Suppression of prolactin secretion partially explains the antidiabetic effect of bromocriptine in ob/ob mice (2019)
Furigo, Isadora Clivatti ; Suzuki, Miriam Fussae ; Oliveria, João Ezequiel de ; Lobo, Angela Maria Ramos; Teixeira, Pryscila Dryelle Sousa; Pedroso, João Alfredo Bolivar; Alencar, Amanda de; Zampieri, Thais Tessari; Buonfiglio, Daniella do Carmo; Bergonsi, Paula Gabriele Fernandes Quaresma; Prada, Patrícia de Oliveira; Bartolini, Paolo; Soares, Carlos Roberto Jorge ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, PROLACTINA, OBESIDADE, DIABETES MELLITUS, RECEPTORES DE DOPAMINA, BROMO, INSULINA, GLICOSE, RESISTÊNCIA À INSULINA
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ABNT
FURIGO, Isadora Clivatti et al. Suppression of prolactin secretion partially explains the antidiabetic effect of bromocriptine in ob/ob mice. Endocrinology, v. 160, n. 1, p. 193-204, 2019Tradução . . Disponível em: https://doi.org/10.1210/en.2018-00629. Acesso em: 15 nov. 2024.
APA
Furigo, I. C., Suzuki, M. F., Oliveria, J. E. de, Lobo, A. M. R., Teixeira, P. D. S., Pedroso, J. A. B., et al. (2019). Suppression of prolactin secretion partially explains the antidiabetic effect of bromocriptine in ob/ob mice. Endocrinology, 160( 1), 193-204. doi:10.1210/en.2018-00629
NLM
Furigo IC, Suzuki MF, Oliveria JE de, Lobo AMR, Teixeira PDS, Pedroso JAB, Alencar A de, Zampieri TT, Buonfiglio D do C, Bergonsi PGFQ, Prada P de O, Bartolini P, Soares CRJ, Donato Junior J. Suppression of prolactin secretion partially explains the antidiabetic effect of bromocriptine in ob/ob mice [Internet]. Endocrinology. 2019 ; 160( 1): 193-204.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2018-00629
Vancouver
Furigo IC, Suzuki MF, Oliveria JE de, Lobo AMR, Teixeira PDS, Pedroso JAB, Alencar A de, Zampieri TT, Buonfiglio D do C, Bergonsi PGFQ, Prada P de O, Bartolini P, Soares CRJ, Donato Junior J. Suppression of prolactin secretion partially explains the antidiabetic effect of bromocriptine in ob/ob mice [Internet]. Endocrinology. 2019 ; 160( 1): 193-204.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2018-00629
Artigo de periodico
Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells (2015)
Bargi-Souza, Paula; Kucka, Marek; Bjelobaba, Ivana; Tomic, Melanija; Janjic, Marija M.; Nunes, Maria Tereza ; Stojikovic, Stanko S.
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, GLÂNDULA PITUITÁRIA, EXPRESSÃO GÊNICA
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ABNT
BARGI-SOUZA, Paula et al. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells. Endocrinology, v. 156, n. 1, p. 242-254, 2015Tradução . . Disponível em: https://doi.org/10.1210/en.2014-1281. Acesso em: 15 nov. 2024.
APA
Bargi-Souza, P., Kucka, M., Bjelobaba, I., Tomic, M., Janjic, M. M., Nunes, M. T., & Stojikovic, S. S. (2015). Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells. Endocrinology, 156( 1), 242-254. doi:10.1210/en.2014-1281
NLM
Bargi-Souza P, Kucka M, Bjelobaba I, Tomic M, Janjic MM, Nunes MT, Stojikovic SS. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells [Internet]. Endocrinology. 2015 ; 156( 1): 242-254.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2014-1281
Vancouver
Bargi-Souza P, Kucka M, Bjelobaba I, Tomic M, Janjic MM, Nunes MT, Stojikovic SS. Loss of basal and TRH-stimulated Tshb expression in dispersed pituitary cells [Internet]. Endocrinology. 2015 ; 156( 1): 242-254.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2014-1281
Artigo de periodico
The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway (2014)
Serrano-Nascimento, Caroline ; Teixeira, Silvania da Silva ; Nachbar, Renato Tadeu ; Nicola, Juan Pablo ; Masini-Repiso, Ana Maria; Nunes, Maria Tereza
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, SODIO, IONS, HORMÔNIOS TIREOIDIANOS, IODO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SERRANO-NASCIMENTO, Caroline et al. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, v. 155, n. 3, p. 1145-1156, 2014Tradução . . Disponível em: https://doi.org/10.1210/en.2013-1665. Acesso em: 15 nov. 2024.
APA
Serrano-Nascimento, C., Teixeira, S. da S., Nachbar, R. T., Nicola, J. P., Masini-Repiso, A. M., & Nunes, M. T. (2014). The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway. Endocrinology, 155( 3), 1145-1156. doi:10.1210/en.2013-1665
NLM
Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2013-1665
Vancouver
Serrano-Nascimento C, Teixeira S da S, Nachbar RT, Nicola JP, Masini-Repiso AM, Nunes MT. The acute inhibitory effect of iodide excess on sodium/iodide symporterexpression and activity involves the PI3K/Akt signaling pathway [Internet]. Endocrinology. 2014 ; 155( 3): 1145-1156.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2013-1665
Artigo de periodico
Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice (2014)
Silva, Regina Pereira da; Zampieri, Thais Tessari; Pedroso, João Alfredo Bolivar; Nagaishi, Vanessa S.; Ramos-Lobo, Angela Maria; Furigo, Isadora Clivatti; Câmara, Niels Olsen Saraiva ; Frazão, Renata ; Donato Junior, José
Source: Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA, IMUNOLOGIA
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ABNT
SILVA, Regina Pereira da et al. Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice. Endocrinology, v. 155, n. 11, p. 4226-4236, 2014Tradução . . Disponível em: https://doi.org/10.1210/en.2014-1276. Acesso em: 15 nov. 2024.
APA
Silva, R. P. da, Zampieri, T. T., Pedroso, J. A. B., Nagaishi, V. S., Ramos-Lobo, A. M., Furigo, I. C., et al. (2014). Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice. Endocrinology, 155( 11), 4226-4236. doi:10.1210/en.2014-1276
NLM
Silva RP da, Zampieri TT, Pedroso JAB, Nagaishi VS, Ramos-Lobo AM, Furigo IC, Câmara NOS, Frazão R, Donato Junior J. Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice [Internet]. Endocrinology. 2014 ; 155( 11): 4226-4236.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2014-1276
Vancouver
Silva RP da, Zampieri TT, Pedroso JAB, Nagaishi VS, Ramos-Lobo AM, Furigo IC, Câmara NOS, Frazão R, Donato Junior J. Leptin resistance is not the primary cause of weight gain associated with reduced sex hormone levels in female mice [Internet]. Endocrinology. 2014 ; 155( 11): 4226-4236.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2014-1276
Artigo de periodico
Cellular mechanism by which estradiol protects female ovariectomized mice from high-fat diet-induced hepatic and muscle insulin resistance (2013)
Camporez, João Paulo Gabriel; Jornayvaz, François R.; Lee, Hui-Young; Kanda, Shoichi; Guigni, Blas A.; Kahn, Mario; Samuel, Varman T.; Carvalho, Carla Roberta de Oliveira ; Petersen, Kitt Falk; Jurczak, Michael J.; Shulman, Gerald I.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CAMPOREZ, João Paulo Gabriel et al. Cellular mechanism by which estradiol protects female ovariectomized mice from high-fat diet-induced hepatic and muscle insulin resistance. Endocrinology, v. 154, n. 3, p. 1021-1028, 2013Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1989. Acesso em: 15 nov. 2024.
APA
Camporez, J. P. G., Jornayvaz, F. R., Lee, H. -Y., Kanda, S., Guigni, B. A., Kahn, M., et al. (2013). Cellular mechanism by which estradiol protects female ovariectomized mice from high-fat diet-induced hepatic and muscle insulin resistance. Endocrinology, 154( 3), 1021-1028. doi:10.1210/en.2012-1989
NLM
Camporez JPG, Jornayvaz FR, Lee H-Y, Kanda S, Guigni BA, Kahn M, Samuel VT, Carvalho CR de O, Petersen KF, Jurczak MJ, Shulman GI. Cellular mechanism by which estradiol protects female ovariectomized mice from high-fat diet-induced hepatic and muscle insulin resistance [Internet]. Endocrinology. 2013 ; 154( 3): 1021-1028.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1989
Vancouver
Camporez JPG, Jornayvaz FR, Lee H-Y, Kanda S, Guigni BA, Kahn M, Samuel VT, Carvalho CR de O, Petersen KF, Jurczak MJ, Shulman GI. Cellular mechanism by which estradiol protects female ovariectomized mice from high-fat diet-induced hepatic and muscle insulin resistance [Internet]. Endocrinology. 2013 ; 154( 3): 1021-1028.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1989
Artigo de periodico
Triiodothyronine rapidly alters the TSH content and the secretory granules distribution in male rat thyrotrophs by a cytoskeleton rearrangement-independent mechanism (2013)
Bargi-Souza, Paula; Romano, Renata Marino; Salgado, Renato de Mayrinck; Silva, Francemilson Goulart da ; Brunetto, Erika Lia; Zorn, Telma Maria Tenório; Nunes, Maria Tereza
Source: Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARGI-SOUZA, Paula et al. Triiodothyronine rapidly alters the TSH content and the secretory granules distribution in male rat thyrotrophs by a cytoskeleton rearrangement-independent mechanism. Endocrinology, v. 154, n. 12, p. 4908-4918, 2013Tradução . . Disponível em: https://doi.org/10.1210/en.2013-1508. Acesso em: 15 nov. 2024.
APA
Bargi-Souza, P., Romano, R. M., Salgado, R. de M., Silva, F. G. da, Brunetto, E. L., Zorn, T. M. T., & Nunes, M. T. (2013). Triiodothyronine rapidly alters the TSH content and the secretory granules distribution in male rat thyrotrophs by a cytoskeleton rearrangement-independent mechanism. Endocrinology, 154( 12), 4908-4918. doi:10.1210/en.2013-1508
NLM
Bargi-Souza P, Romano RM, Salgado R de M, Silva FG da, Brunetto EL, Zorn TMT, Nunes MT. Triiodothyronine rapidly alters the TSH content and the secretory granules distribution in male rat thyrotrophs by a cytoskeleton rearrangement-independent mechanism [Internet]. Endocrinology. 2013 ; 154( 12): 4908-4918.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2013-1508
Vancouver
Bargi-Souza P, Romano RM, Salgado R de M, Silva FG da, Brunetto EL, Zorn TMT, Nunes MT. Triiodothyronine rapidly alters the TSH content and the secretory granules distribution in male rat thyrotrophs by a cytoskeleton rearrangement-independent mechanism [Internet]. Endocrinology. 2013 ; 154( 12): 4908-4918.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2013-1508
Artigo de periodico
The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion (2012)
Lellis-Santos, Camilo; Sakamoto, Luciano H.; Bromati, Carla Rodrigues; Nogueira, Tatiane Cristina de Araújo; Leite, Adriana R.; Yamanaka, Tatiana S.; Kinote, Andrezza; Anhê, Gabriel F.; Bordin, Silvana
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LELLIS-SANTOS, Camilo et al. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, v. 153, n. 8, p. 3668-3678, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1135. Acesso em: 15 nov. 2024.
APA
Lellis-Santos, C., Sakamoto, L. H., Bromati, C. R., Nogueira, T. C. de A., Leite, A. R., Yamanaka, T. S., et al. (2012). The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, 153( 8), 3668-3678. doi:10.1210/en.2012-1135
NLM
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1135
Vancouver
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1135
Artigo de periodico
Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment (2012)
Oliveira, Ariclécio Cunha de; Andreotti, Sandra ; Farias, Talita da Silva Mendes; Torres-Leal, Francisco Leonardo; Proença, André Ricardo Gomes de; Campaña, Amanda Baron; Souza, Arnaldo Henrique de; Sertié, Rogério Antonio Laurato; Carpinelli, Angelo Rafael ; Cipolla-Neto, José; Lima, Fábio Bessa
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA, Ariclécio Cunha de et al. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment. Endocrinology, v. 153, p. 2178-2188, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2011-1675. Acesso em: 15 nov. 2024.
APA
Oliveira, A. C. de, Andreotti, S., Farias, T. da S. M., Torres-Leal, F. L., Proença, A. R. G. de, Campaña, A. B., et al. (2012). Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment. Endocrinology, 153, 2178-2188. doi:10.1210/en.2011-1675
NLM
Oliveira AC de, Andreotti S, Farias T da SM, Torres-Leal FL, Proença ARG de, Campaña AB, Souza AH de, Sertié RAL, Carpinelli AR, Cipolla-Neto J, Lima FB. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment [Internet]. Endocrinology. 2012 ; 153 2178-2188.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2011-1675
Vancouver
Oliveira AC de, Andreotti S, Farias T da SM, Torres-Leal FL, Proença ARG de, Campaña AB, Souza AH de, Sertié RAL, Carpinelli AR, Cipolla-Neto J, Lima FB. Metabolic disorders and adipose tissue insulin responsiveness in neonatally STZ-induced diabetic rats are improved by long-term melatonin treatment [Internet]. Endocrinology. 2012 ; 153 2178-2188.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2011-1675
Artigo de periodico
Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice (2012)
Carvalho Filho, M. A.; Carvalho, B. M.; Oliveira, A. G.; Guadagnini, D.; Ueno, M.; Dias M. M.; Tsukumo, D. M.; Hirabata, Sandro Massao; Reis, L. F.; Curi, Rui; Carvalheira, J. B. C.; Saad, Mario J. A.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO FILHO, M. A. et al. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, v. 153, n. 11, p. 5261-5274, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1400. Acesso em: 15 nov. 2024.
APA
Carvalho Filho, M. A., Carvalho, B. M., Oliveira, A. G., Guadagnini, D., Ueno, M., Dias M. M.,, et al. (2012). Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, 153( 11), 5261-5274. doi:10.1210/en.2012-1400
NLM
Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1400
Vancouver
Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1400
Artigo de periodico
Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels (2012)
Kinote, Andrezza; Faria, Juliana A.; Roman, Erika A.; Solon, Carina; Razolli, Daniela S.; Ignacio-Souza, Letícia M.; Sollon, Carolina S.; Nascimento, Lucas F.; Araújo, Thiago M. de; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Velloso, Licio A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KINOTE, Andrezza et al. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, v. 153, n. 8, p. 3633-3645, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1341. Acesso em: 15 nov. 2024.
APA
Kinote, A., Faria, J. A., Roman, E. A., Solon, C., Razolli, D. S., Ignacio-Souza, L. M., et al. (2012). Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, 153( 8), 3633-3645. doi:10.1210/en.2012-1341
NLM
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1341
Vancouver
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2012-1341
Artigo de periodico
KISS1R Intracellular Trafficking and Degradation: Effect of the Arg386Pro Disease-Associated Mutation (2011)
Bianco, Susy D. C.; Vandepas, Lauren; Correa-Medina, Mayrin; Gereben, Balázs; Mukherjee, Abir; Kuohung, Wendy; Carroll, Rona; Teles, Milena G.; Xavier, Ana Claudia Latronico; Kaiser, Ursula B.
Source: Endocrinology. Unidade: FM
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BIANCO, Susy D. C. et al. KISS1R Intracellular Trafficking and Degradation: Effect of the Arg386Pro Disease-Associated Mutation. Endocrinology, v. 152, n. 4, p. 1616-1626, 2011Tradução . . Disponível em: https://doi.org/10.1210/en.2010-0903. Acesso em: 15 nov. 2024.
APA
Bianco, S. D. C., Vandepas, L., Correa-Medina, M., Gereben, B., Mukherjee, A., Kuohung, W., et al. (2011). KISS1R Intracellular Trafficking and Degradation: Effect of the Arg386Pro Disease-Associated Mutation. Endocrinology, 152( 4), 1616-1626. doi:10.1210/en.2010-0903
NLM
Bianco SDC, Vandepas L, Correa-Medina M, Gereben B, Mukherjee A, Kuohung W, Carroll R, Teles MG, Xavier ACL, Kaiser UB. KISS1R Intracellular Trafficking and Degradation: Effect of the Arg386Pro Disease-Associated Mutation [Internet]. Endocrinology. 2011 ; 152( 4): 1616-1626.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2010-0903
Vancouver
Bianco SDC, Vandepas L, Correa-Medina M, Gereben B, Mukherjee A, Kuohung W, Carroll R, Teles MG, Xavier ACL, Kaiser UB. KISS1R Intracellular Trafficking and Degradation: Effect of the Arg386Pro Disease-Associated Mutation [Internet]. Endocrinology. 2011 ; 152( 4): 1616-1626.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2010-0903
Artigo de periodico
Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response (2011)
Nogueira, Tatiane Cristina de Araújo; Santos, Camilo de Lellis; Jesus, Daniel Simões de; Taneda, Marco; Rodrigues, Sandra Campos; Amaral, Fernanda Gaspar do; Lopes, Ana Maria Souza; Cipolla Neto, José; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NOGUEIRA, Tatiane Cristina de Araújo et al. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, v. 152, n. 4, p. 1253-1263, 2011Tradução . . Disponível em: https://doi.org/10.1210/en.2010-1088. Acesso em: 15 nov. 2024.
APA
Nogueira, T. C. de A., Santos, C. de L., Jesus, D. S. de, Taneda, M., Rodrigues, S. C., Amaral, F. G. do, et al. (2011). Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response. Endocrinology, 152( 4), 1253-1263. doi:10.1210/en.2010-1088
NLM
Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2010-1088
Vancouver
Nogueira TC de A, Santos C de L, Jesus DS de, Taneda M, Rodrigues SC, Amaral FG do, Lopes AMS, Cipolla Neto J, Bordin S, Anhê GF. Absence of melatonin Induces night-time hepatic insulin resistance and increased gluconeogenesis due to stimulation of nocturnal unfolded protein response [Internet]. Endocrinology. 2011 ; 152( 4): 1253-1263.[citado 2024 nov. 15 ] Available from: https://doi.org/10.1210/en.2010-1088

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