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Artigo de periodico
AHR is a Zika virus host factor and a candidate target for antiviral therapy (2020)
Giovannoni, Federico ; Bosch, Irene ; Polonio, Carolina Manganeli ; Torti, María F.; Wheeler, Michael A.; Li, Zhaorong ; Romorini, Leonardo ; Varela, María S. Rodriguez; Rothhammer, Veit ; Barroso, Andreia ; Tjon, Emily C.; Sanmarco, Liliana M.; Takenaka, Maisa C.; Modaresi, Seyed Mohamad Sadegh ; Vázquez, Cristina Gutiérrez; Zanluqui, Nágela Ghabdan ; Santos, Nilton Barreto dos ; Munhoz, Carolina Demarchi ; Wang, Zhongyan; Damonte, Elsa B.; Sherr, David; Gehrke, Lee ; Peron, Jean Pierre Schatzmann ; Garcia, Cybele C. ; Quintana, Francisco J.
Source: Nature Neuroscience. Unidades: ICB, FM
Subjects: IMUNOLOGIA, FARMACOLOGIA, ZIKA VÍRUS, VÍRUS, FLAVIVIRUS, MICROCEFALIA, TRANSCRIÇÃO GÊNICA, GENOMAS, HIDROCARBONETOS, INTERFERON TIPO I, ANTIVIRAIS, REPLICAÇÃO VIRAL, NANOPARTÍCULAS
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ABNT
GIOVANNONI, Federico et al. AHR is a Zika virus host factor and a candidate target for antiviral therapy. Nature Neuroscience, v. 23, p. 939–951, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41593-020-0664-0. Acesso em: 11 nov. 2024.
APA
Giovannoni, F., Bosch, I., Polonio, C. M., Torti, M. F., Wheeler, M. A., Li, Z., et al. (2020). AHR is a Zika virus host factor and a candidate target for antiviral therapy. Nature Neuroscience, 23, 939–951. doi:10.1038/s41593-020-0664-0
NLM
Giovannoni F, Bosch I, Polonio CM, Torti MF, Wheeler MA, Li Z, Romorini L, Varela MSR, Rothhammer V, Barroso A, Tjon EC, Sanmarco LM, Takenaka MC, Modaresi SMS, Vázquez CG, Zanluqui NG, Santos NB dos, Munhoz CD, Wang Z, Damonte EB, Sherr D, Gehrke L, Peron JPS, Garcia CC, Quintana FJ. AHR is a Zika virus host factor and a candidate target for antiviral therapy [Internet]. Nature Neuroscience. 2020 ; 23 939–951.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1038/s41593-020-0664-0
Vancouver
Giovannoni F, Bosch I, Polonio CM, Torti MF, Wheeler MA, Li Z, Romorini L, Varela MSR, Rothhammer V, Barroso A, Tjon EC, Sanmarco LM, Takenaka MC, Modaresi SMS, Vázquez CG, Zanluqui NG, Santos NB dos, Munhoz CD, Wang Z, Damonte EB, Sherr D, Gehrke L, Peron JPS, Garcia CC, Quintana FJ. AHR is a Zika virus host factor and a candidate target for antiviral therapy [Internet]. Nature Neuroscience. 2020 ; 23 939–951.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1038/s41593-020-0664-0
Artigo de periodico
Effect of protein denaturation and enzyme inhibitors on proteasomal-mediated production of peptides in human embryonic kidney cells (2019)
Dasgupta, Sayani ; Fishman, Michael A. ; Castro, Leandro M. ; Tashima, Alexandre Keiji ; Ferro, Emer Suavinho ; Fricker, Lloyd D.
Source: Biomolecules. Unidade: ICB
Subjects: FARMACOLOGIA, INIBIDORES DE ENZIMAS, PEPTÍDEOS, AMINOÁCIDOS, ESPECTROMETRIA DE MASSAS, RIM, EMBRIÃO
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ABNT
DASGUPTA, Sayani et al. Effect of protein denaturation and enzyme inhibitors on proteasomal-mediated production of peptides in human embryonic kidney cells. Biomolecules, v. 9, n. 6, p. 18, 2019Tradução . . Disponível em: https://doi.org/10.3390/biom9060207. Acesso em: 11 nov. 2024.
APA
Dasgupta, S., Fishman, M. A., Castro, L. M., Tashima, A. K., Ferro, E. S., & Fricker, L. D. (2019). Effect of protein denaturation and enzyme inhibitors on proteasomal-mediated production of peptides in human embryonic kidney cells. Biomolecules, 9( 6), 18. doi:10.3390/biom9060207
NLM
Dasgupta S, Fishman MA, Castro LM, Tashima AK, Ferro ES, Fricker LD. Effect of protein denaturation and enzyme inhibitors on proteasomal-mediated production of peptides in human embryonic kidney cells [Internet]. Biomolecules. 2019 ; 9( 6): 18.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/biom9060207
Vancouver
Dasgupta S, Fishman MA, Castro LM, Tashima AK, Ferro ES, Fricker LD. Effect of protein denaturation and enzyme inhibitors on proteasomal-mediated production of peptides in human embryonic kidney cells [Internet]. Biomolecules. 2019 ; 9( 6): 18.[citado 2024 nov. 11 ] Available from: https://doi.org/10.3390/biom9060207
Artigo de periodico
Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells (2019)
Gimenez, Marcela; Verissimo Filho, Sidney; Wittig, Ilka ; Schickling, Brandon M; Hahner, Fabian ; Schürmann, Christoph ; Netto, Luis Eduardo Soares ; Rosa, José César ; Brandes, Ralf P ; Sartoretto, Simone Marcieli; Camargo, Livia de Lucca ; Abdulkader, Fernando Rodrigues de Moraes ; Miller Junior, Francis J ; Lopes, Lucia Rossetti
Source: Arteriosclerosis, Thrombosis, and Vascular Biology. Unidades: FMRP, ICB, IB
Subjects: FISIOLOGIA, FARMACOLOGIA, ESPECTROMETRIA DE MASSAS, PROTEÍNAS RECOMBINANTES, ATIVAÇÃO ENZIMÁTICA, MÚSCULO LISO VASCULAR, CATALISADORES, FATORES DE CRESCIMENTO, CÉLULAS MUSCULARES
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ABNT
GIMENEZ, Marcela et al. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells. Arteriosclerosis, Thrombosis, and Vascular Biology, v. 39, n. 2, p. 224-236, 2019Tradução . . Disponível em: https://doi.org/10.1161/ATVBAHA.118.311038. Acesso em: 11 nov. 2024.
APA
Gimenez, M., Verissimo Filho, S., Wittig, I., Schickling, B. M., Hahner, F., Schürmann, C., et al. (2019). Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells. Arteriosclerosis, Thrombosis, and Vascular Biology, 39( 2), 224-236. doi:10.1161/ATVBAHA.118.311038
NLM
Gimenez M, Verissimo Filho S, Wittig I, Schickling BM, Hahner F, Schürmann C, Netto LES, Rosa JC, Brandes RP, Sartoretto SM, Camargo L de L, Abdulkader FR de M, Miller Junior FJ, Lopes LR. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2019 ; 39( 2): 224-236.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1161/ATVBAHA.118.311038
Vancouver
Gimenez M, Verissimo Filho S, Wittig I, Schickling BM, Hahner F, Schürmann C, Netto LES, Rosa JC, Brandes RP, Sartoretto SM, Camargo L de L, Abdulkader FR de M, Miller Junior FJ, Lopes LR. Redox activation of nox1 (NADPH Oxidase 1) involves an intermolecular disulfide bond between protein disulfide iomerase and p47phox in vascular smooth muscle cells [Internet]. Arteriosclerosis, Thrombosis, and Vascular Biology. 2019 ; 39( 2): 224-236.[citado 2024 nov. 11 ] Available from: https://doi.org/10.1161/ATVBAHA.118.311038
Artigo de periodico
Factors promoting vulnerability to dysregulated stress reactivity and stress‐related disease (2018)
Russell, Ashley L. ; Tasker, Jeffrey G. ; Lucion, Aldo Bolten ; Temer, Jenny Fiedler ; Munhoz, Carolina Demarchi ; Wu, Tao-yiao John ; Deak, Terrence
Source: Journal of Neuroendocrinology. Unidade: ICB
Subjects: FARMACOLOGIA, ESTRESSE, ANSIEDADE, INFLAMAÇÃO, OCITOCINA, HORMÔNIOS GLICOCORTICOIDES, TRAUMATISMOS CRANIOCEREBRAIS
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ABNT
RUSSELL, Ashley L. et al. Factors promoting vulnerability to dysregulated stress reactivity and stress‐related disease. Journal of Neuroendocrinology, v. 30, n. 10, p. 13 , 2018Tradução . . Disponível em: https://doi.org/10.1111/jne.12641. Acesso em: 11 nov. 2024.
APA
Russell, A. L., Tasker, J. G., Lucion, A. B., Temer, J. F., Munhoz, C. D., Wu, T. -yiao J., & Deak, T. (2018). Factors promoting vulnerability to dysregulated stress reactivity and stress‐related disease. Journal of Neuroendocrinology, 30( 10), 13 . doi:10.1111/jne.12641
NLM
Russell AL, Tasker JG, Lucion AB, Temer JF, Munhoz CD, Wu T-yiao J, Deak T. Factors promoting vulnerability to dysregulated stress reactivity and stress‐related disease [Internet]. Journal of Neuroendocrinology. 2018 ; 30( 10): 13 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1111/jne.12641
Vancouver
Russell AL, Tasker JG, Lucion AB, Temer JF, Munhoz CD, Wu T-yiao J, Deak T. Factors promoting vulnerability to dysregulated stress reactivity and stress‐related disease [Internet]. Journal of Neuroendocrinology. 2018 ; 30( 10): 13 .[citado 2024 nov. 11 ] Available from: https://doi.org/10.1111/jne.12641
Artigo de periodico
A novel peptide that improves metabolic parameters without adverse central nervous system effects (2017)
Reckziegel, Patricia ; Festuccia, William Tadeu Lara ; Britto, Luiz Roberto Giorgetti de ; Heimann, Joel Cláudio ; Guimarães, Francisco Silveira ; Eichler, Rosangela Aparecida dos Santos ; Ferro, Emer Suavinho
Source: Scientific reports. Unidades: ICB, FM, FMRP
Subjects: PEPTÍDEOS, METABOLISMO, SISTEMA NERVOSO CENTRAL, FARMACOLOGIA, FISIOLOGIA
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ABNT
RECKZIEGEL, Patricia et al. A novel peptide that improves metabolic parameters without adverse central nervous system effects. Scientific reports, v. 7, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-13690-9. Acesso em: 11 nov. 2024.
APA
Reckziegel, P., Festuccia, W. T. L., Britto, L. R. G. de, Heimann, J. C., Guimarães, F. S., Eichler, R. A. dos S., & Ferro, E. S. (2017). A novel peptide that improves metabolic parameters without adverse central nervous system effects. Scientific reports, 7. doi:10.1038/s41598-017-13690-9
NLM
Reckziegel P, Festuccia WTL, Britto LRG de, Heimann JC, Guimarães FS, Eichler RA dos S, Ferro ES. A novel peptide that improves metabolic parameters without adverse central nervous system effects [Internet]. Scientific reports. 2017 ; 7[citado 2024 nov. 11 ] Available from: https://doi.org/10.1038/s41598-017-13690-9
Vancouver
Reckziegel P, Festuccia WTL, Britto LRG de, Heimann JC, Guimarães FS, Eichler RA dos S, Ferro ES. A novel peptide that improves metabolic parameters without adverse central nervous system effects [Internet]. Scientific reports. 2017 ; 7[citado 2024 nov. 11 ] Available from: https://doi.org/10.1038/s41598-017-13690-9

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