Fonte: Neuroimmunomodulation. Unidade: FM
Assuntos: INFLAMAÇÃO, ESTRESSE PSICOLÓGICO (EFEITOS), MODELOS ANIMAIS, COBAIAS, MATRIZ EXTRACELULAR, DOENÇA CRÔNICA, RESPIRAÇÃO ARTIFICIAL, MORFOMETRIA, ASMA (EPIDEMIOLOGIA)
ABNT
MARQUES, Ricardo Henrique et al. Inducible Nitric Oxide Synthase Inhibition Attenuates Physical Stress-Induced Lung Hyper-Responsiveness and Oxidative Stress in Animals with Lung Inflammation. Neuroimmunomodulation, v. 19, n. 3, p. 158-170, 2012Tradução . . Disponível em: http://content.karger.com/ProdukteDB/produkte.asp?doi=10.1159/000331264. Acesso em: 03 nov. 2025.APA
Marques, R. H., Reis, F. G., Starling, C. M., Cabido, C., Almeida-Reis, R. de, Dolhnikoff, M., et al. (2012). Inducible Nitric Oxide Synthase Inhibition Attenuates Physical Stress-Induced Lung Hyper-Responsiveness and Oxidative Stress in Animals with Lung Inflammation. Neuroimmunomodulation, 19( 3), 158-170. doi:10.1159/000331264NLM
Marques RH, Reis FG, Starling CM, Cabido C, Almeida-Reis R de, Dolhnikoff M, Prado CM, Leick EA, Martins M de A, Tibério IFLC. Inducible Nitric Oxide Synthase Inhibition Attenuates Physical Stress-Induced Lung Hyper-Responsiveness and Oxidative Stress in Animals with Lung Inflammation [Internet]. Neuroimmunomodulation. 2012 ; 19( 3): 158-170.[citado 2025 nov. 03 ] Available from: http://content.karger.com/ProdukteDB/produkte.asp?doi=10.1159/000331264Vancouver
Marques RH, Reis FG, Starling CM, Cabido C, Almeida-Reis R de, Dolhnikoff M, Prado CM, Leick EA, Martins M de A, Tibério IFLC. Inducible Nitric Oxide Synthase Inhibition Attenuates Physical Stress-Induced Lung Hyper-Responsiveness and Oxidative Stress in Animals with Lung Inflammation [Internet]. Neuroimmunomodulation. 2012 ; 19( 3): 158-170.[citado 2025 nov. 03 ] Available from: http://content.karger.com/ProdukteDB/produkte.asp?doi=10.1159/000331264
