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Artigo de periodico
Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 (2023)
Dourado, Thales de Mileto Henrique; Assis, Victor Oliveira; Awata, Wanessa Mayumi Carvalho; Mello, Marcela Maria Blascke de ; Cárnio, Evelin Capellari ; Castro, Michele Mazzaron de ; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: EERP, FMRP
Subjects: ETANOL, ESPÉCIES REATIVAS DE OXIGÊNIO, TROMBOXANOS, HIPERTENSÃO, ENDOTÉLIO VASCULAR
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ABNT
DOURADO, Thales de Mileto Henrique et al. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, v. 949, p. 1-10, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2023.175723. Acesso em: 14 nov. 2024.
APA
Dourado, T. de M. H., Assis, V. O., Awata, W. M. C., Mello, M. M. B. de, Cárnio, E. C., Castro, M. M. de, & Tirapelli, C. R. (2023). Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2. European Journal of Pharmacology, 949, 1-10. doi:10.1016/j.ejphar.2023.175723
NLM
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Vancouver
Dourado T de MH, Assis VO, Awata WMC, Mello MMB de, Cárnio EC, Castro MM de, Tirapelli CR. Mineralocorticoid receptors contribute to ethanol-induced vascular hypercontractility through reactive oxygen species generation and up-regulation of cyclooxygenase 2 [Internet]. European Journal of Pharmacology. 2023 ; 949 1-10.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2023.175723
Artigo de periodico
Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction (2022)
Silva, Josiane Fernandes da; Bolsoni, Juliana A.; Costa, Rafael Menezes da ; Alves, Juliano Vilela ; Bressan, Alecsander Fabrício Moreira ; Silva, Luiz Eduardo Virgilio da ; Costa, Tiago Januário da ; Oliveira, Antonio E. R.; Manzato, Carla Pavan; Silva, Carlos Alberto Aguiar da; Fazan Júnior, Rubens ; Cunha, Fernando de Queiroz ; Nakaya, Helder Takashi Imoto ; Carneiro, Fernando Silva ; Tostes, Rita de Cássia Aleixo
Source: British Journal of Pharmacology. Unidades: FMRP, FCF
Subjects: GORDURAS NA DIETA, CÉLULAS ENDOTELIAIS, ENDOTÉLIO VASCULAR, OBESIDADE, HIDROCARBONETOS, VASODILATAÇÃO
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SILVA, Josiane Fernandes da et al. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, v. 179, n. 12, p. 2938-2952, 2022Tradução . . Disponível em: https://doi.org/10.1111/bph.15789. Acesso em: 14 nov. 2024.
APA
Silva, J. F. da, Bolsoni, J. A., Costa, R. M. da, Alves, J. V., Bressan, A. F. M., Silva, L. E. V. da, et al. (2022). Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction. British Journal of Pharmacology, 179( 12), 2938-2952. doi:10.1111/bph.15789
NLM
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/bph.15789
Vancouver
Silva JF da, Bolsoni JA, Costa RM da, Alves JV, Bressan AFM, Silva LEV da, Costa TJ da, Oliveira AER, Manzato CP, Silva CAA da, Fazan Júnior R, Cunha F de Q, Nakaya HTI, Carneiro FS, Tostes R de CA. Aryl hydrocarbon receptor (AhR) activation contributes to high‐fat diet‐induced vascular dysfunction [Internet]. British Journal of Pharmacology. 2022 ; 179( 12): 2938-2952.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/bph.15789
Artigo de periodico
Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients (2021)
Potje, Simone Regina ; Costa, Tiago Januário da ; Fraga-Silva, Thais Fernanda de Campos ; Martins, Ronaldo Bragança ; Benatti, Maíra Nilson; Almado, Carlos Eduardo Lopes; Sá, Keyla Santos Guedes de ; Bonato, Vânia Luiza Deperon ; Arruda, Eurico ; Louzada Júnior, Paulo ; Oliveira, Renê Donizete Ribeiro de; Zamboni, Dario Simões ; Becari, Christiane; Martins, Maria Auxiliadora ; Tostes, Rita de Cássia Aleixo
Source: Life Sciences. Unidades: FMRP, FCFRP
Subjects: ENDOTÉLIO VASCULAR, COVID-19, COAGULAÇÃO SANGUÍNEA, ESTUDOS DE CASOS E CONTROLES, TROMBOSE, INTERLEUCINA 6
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POTJE, Simone Regina et al. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients. Life Sciences, v. 276, 2021Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2021.119376. Acesso em: 14 nov. 2024.
APA
Potje, S. R., Costa, T. J. da, Fraga-Silva, T. F. de C., Martins, R. B., Benatti, M. N., Almado, C. E. L., et al. (2021). Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients. Life Sciences, 276. doi:10.1016/j.lfs.2021.119376
NLM
Potje SR, Costa TJ da, Fraga-Silva TF de C, Martins RB, Benatti MN, Almado CEL, Sá KSG de, Bonato VLD, Arruda E, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Becari C, Martins MA, Tostes R de CA. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients [Internet]. Life Sciences. 2021 ; 276[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2021.119376
Vancouver
Potje SR, Costa TJ da, Fraga-Silva TF de C, Martins RB, Benatti MN, Almado CEL, Sá KSG de, Bonato VLD, Arruda E, Louzada Júnior P, Oliveira RDR de, Zamboni DS, Becari C, Martins MA, Tostes R de CA. Heparin prevents in vitro glycocalyx shedding induced by plasma from COVID-19 patients [Internet]. Life Sciences. 2021 ; 276[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2021.119376
Artigo de periodico
Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human (2020)
Costa, Eduardo D.; Silva, Josiane Fernandes da; Garcia, Daniela C.; Wainstein, Alberto J.; Rezende, Bruno A.; Tostes, Rita de Cássia Aleixo ; Teixeira, Mauro Martins; Côrtes, Steyner F.; Lemos, Virgínia S.
Source: Nitric Oxide. Unidade: FMRP
Subjects: ENVELHECIMENTO, TABAGISMO, ENDOTÉLIO VASCULAR, METABOLISMO, INIBIDORES DE ENZIMAS, FARMACOLOGIA, MEIA IDADE, ÓXIDO NÍTRICO
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ABNT
COSTA, Eduardo D. et al. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human. Nitric Oxide, v. 98, p. 20-28, 2020Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2020.02.003. Acesso em: 14 nov. 2024.
APA
Costa, E. D., Silva, J. F. da, Garcia, D. C., Wainstein, A. J., Rezende, B. A., Tostes, R. de C. A., et al. (2020). Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human. Nitric Oxide, 98, 20-28. doi:10.1016/j.niox.2020.02.003
NLM
Costa ED, Silva JF da, Garcia DC, Wainstein AJ, Rezende BA, Tostes R de CA, Teixeira MM, Côrtes SF, Lemos VS. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human [Internet]. Nitric Oxide. 2020 ; 98 20-28.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.niox.2020.02.003
Vancouver
Costa ED, Silva JF da, Garcia DC, Wainstein AJ, Rezende BA, Tostes R de CA, Teixeira MM, Côrtes SF, Lemos VS. Decreased expression of neuronal nitric oxide synthase contributes to the endothelial dysfunction associated with cigarette smoking in human [Internet]. Nitric Oxide. 2020 ; 98 20-28.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.niox.2020.02.003
Artigo de periodico
Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation (2018)
Wynne, Brandi M.; Labazi, Hicham; Lima, Victor V.; Carneiro, Fernando Silva; Webb, R. Clinton; Tostes, Rita de Cássia Aleixo; Giachini, Fernanda Regina Casagrande
Source: Canadian Journal of Physiology and Pharmacology. Unidade: FMRP
Subjects: VASODILATAÇÃO, ENDOTÉLIO VASCULAR, ATIVAÇÃO ENZIMÁTICA, ÓXIDO NÍTRICO, ACIDENTE VASCULAR CEREBRAL
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ABNT
WYNNE, Brandi M. et al. Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation. Canadian Journal of Physiology and Pharmacology, v. 96, n. 8, p. 719-727, 2018Tradução . . Disponível em: https://doi.org/10.1139/cjpp-2017-0477. Acesso em: 14 nov. 2024.
APA
Wynne, B. M., Labazi, H., Lima, V. V., Carneiro, F. S., Webb, R. C., Tostes, R. de C. A., & Giachini, F. R. C. (2018). Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation. Canadian Journal of Physiology and Pharmacology, 96( 8), 719-727. doi:10.1139/cjpp-2017-0477
NLM
Wynne BM, Labazi H, Lima VV, Carneiro FS, Webb RC, Tostes R de CA, Giachini FRC. Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation [Internet]. Canadian Journal of Physiology and Pharmacology. 2018 ; 96( 8): 719-727.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2017-0477
Vancouver
Wynne BM, Labazi H, Lima VV, Carneiro FS, Webb RC, Tostes R de CA, Giachini FRC. Mesenteric arteries from stroke-prone spontaneously hypertensive rats exhibit an increase in nitric-oxide-dependent vasorelaxation [Internet]. Canadian Journal of Physiology and Pharmacology. 2018 ; 96( 8): 719-727.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1139/cjpp-2017-0477
Artigo de periodico
Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism (2018)
Nascimento, M.; Piran, R.; Da Costa, R. M.; Giordani, M. A.; Carneiro, Fernando Silva; Aguiar, D. H.; Dias, M. C.; Sugizaki, M. M.; Luvizotto, R. A.; Nascimento, A. F.; Bomfim, G. F.
Source: Life Sciences. Unidade: FMRP
Subjects: AORTA, FÍGADO, PRESSÃO SANGUÍNEA, ENDOTÉLIO VASCULAR, DOENÇAS VASCULARES, RATOS WISTAR
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ABNT
NASCIMENTO, M. et al. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, v. 212, p. 168-175, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.09.051. Acesso em: 14 nov. 2024.
APA
Nascimento, M., Piran, R., Da Costa, R. M., Giordani, M. A., Carneiro, F. S., Aguiar, D. H., et al. (2018). Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism. Life Sciences, 212, 168-175. doi:10.1016/j.lfs.2018.09.051
NLM
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Vancouver
Nascimento M, Piran R, Da Costa RM, Giordani MA, Carneiro FS, Aguiar DH, Dias MC, Sugizaki MM, Luvizotto RA, Nascimento AF, Bomfim GF. Hepatic injury induced by thioacetamide causes aortic endothelial dysfunction by a cyclooxygenase-dependent mechanism [Internet]. Life Sciences. 2018 ; 212 168-175.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.lfs.2018.09.051
Artigo de periodico
Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide (2018)
Gonzaga, Natália de Almeida; Awata, Wanessa M. C.; Vale, Gabriel Tavares do; Marchi, Kátia Colombo; Muniz, Jaqueline Jóice ; Santos, José Eduardo Tanus dos ; Tirapelli, Carlos Renato
Source: Vascular Pharmacology. Unidades: FMRP, EERP
Subjects: ETANOL, PERÓXIDO DE HIDROGÊNIO, TECIDO ADIPOSO, CATALASE, ENDOTÉLIO VASCULAR
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ABNT
GONZAGA, Natália de Almeida et al. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, v. 111, p. 44-53, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2018.08.010. Acesso em: 14 nov. 2024.
APA
Gonzaga, N. de A., Awata, W. M. C., Vale, G. T. do, Marchi, K. C., Muniz, J. J., Santos, J. E. T. dos, & Tirapelli, C. R. (2018). Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide. Vascular Pharmacology, 111, 44-53. doi:10.1016/j.vph.2018.08.010
NLM
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Vancouver
Gonzaga N de A, Awata WMC, Vale GT do, Marchi KC, Muniz JJ, Santos JET dos, Tirapelli CR. Perivascular adipose tissue protects against the vascular dysfunction induced by acute ethanol intake: role of hydrogen peroxide [Internet]. Vascular Pharmacology. 2018 ; 111 44-53.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.vph.2018.08.010
Artigo de periodico
Clinical and pharmacogenetic impact of endothelial nitric oxide synthase polymorphisms on cardiovascular diseases (2017)
Oliveira-Paula, Gustavo Henrique; Lacchini, Riccardo ; Tanus-Santos, José Eduardo
Source: Nitric Oxide. Unidades: EERP, FMRP
Subjects: ÓXIDO NÍTRICO, POLIMORFISMO, FARMACOGENÉTICA, ENDOTÉLIO VASCULAR
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ABNT
OLIVEIRA-PAULA, Gustavo Henrique e LACCHINI, Riccardo e TANUS-SANTOS, José Eduardo. Clinical and pharmacogenetic impact of endothelial nitric oxide synthase polymorphisms on cardiovascular diseases. Nitric Oxide, v. 63, p. 39-51, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.niox.2016.08.004. Acesso em: 14 nov. 2024.
APA
Oliveira-Paula, G. H., Lacchini, R., & Tanus-Santos, J. E. (2017). Clinical and pharmacogenetic impact of endothelial nitric oxide synthase polymorphisms on cardiovascular diseases. Nitric Oxide, 63, 39-51. doi:10.1016/j.niox.2016.08.004
NLM
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Clinical and pharmacogenetic impact of endothelial nitric oxide synthase polymorphisms on cardiovascular diseases [Internet]. Nitric Oxide. 2017 ; 63 39-51.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.niox.2016.08.004
Vancouver
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Clinical and pharmacogenetic impact of endothelial nitric oxide synthase polymorphisms on cardiovascular diseases [Internet]. Nitric Oxide. 2017 ; 63 39-51.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.niox.2016.08.004
Artigo de periodico
Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species (2017)
Côco, Hariane; Pernomian, Larissa; Pereira, Priscila C.; Gomes, Mayara Santos; Marchi, Katia C.; Lopes, Alexandre H.; Cunha, Thiago Mattar; Tirapelli, Carlos Renato ; Oliveira, Ana Maria de
Source: Journal of Pharmacy and Pharmacology. Unidades: FCFRP, FMRP, EERP
Subjects: ANGIOTENSINA II, ARTÉRIAS CARÓTIDAS, ENDOTÉLIO VASCULAR, ESTRESSE PSICOLÓGICO, ESPÉCIES REATIVAS DE OXIGÊNIO
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ABNT
CÔCO, Hariane et al. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, v. 69, n. 1, p. 52-65, 2017Tradução . . Disponível em: https://doi.org/10.1111/jphp.12659. Acesso em: 14 nov. 2024.
APA
Côco, H., Pernomian, L., Pereira, P. C., Gomes, M. S., Marchi, K. C., Lopes, A. H., et al. (2017). Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species. Journal of Pharmacy and Pharmacology, 69( 1), 52-65. doi:10.1111/jphp.12659
NLM
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/jphp.12659
Vancouver
Côco H, Pernomian L, Pereira PC, Gomes MS, Marchi KC, Lopes AH, Cunha TM, Tirapelli CR, Oliveira AM de. Chronic restraint stress increases angiotensin II potency in the rat carotid: role of cyclooxygenases and reactive oxygen species [Internet]. Journal of Pharmacy and Pharmacology. 2017 ; 69( 1): 52-65.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1111/jphp.12659
Artigo de periodico
Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries (2017)
Simplicio, Janaina A.; Vale, Gabriel T. do; Gonzaga, Natália A.; Leite, Letícia N.; Hipólito, Ulisses V.; Pereira, Camila A.; Tostes, Rita de Cássia Aleixo; Tirapelli, Carlos Renato
Source: Journal of Physiology and Biochemistry. Unidades: FMRP, EERP
Subjects: ETANOL, HIPERTENSÃO, ESTRESSE OXIDATIVO, DOENÇAS CARDIOVASCULARES, ENDOTÉLIO VASCULAR, GLICOPROTEÍNAS DA MEMBRANA
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ABNT
SIMPLICIO, Janaina A. et al. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries. Journal of Physiology and Biochemistry, v. 73, n. 1, p. 5-16, 2017Tradução . . Disponível em: https://doi.org/10.1007/s13105-016-0519-z. Acesso em: 14 nov. 2024.
APA
Simplicio, J. A., Vale, G. T. do, Gonzaga, N. A., Leite, L. N., Hipólito, U. V., Pereira, C. A., et al. (2017). Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries. Journal of Physiology and Biochemistry, 73( 1), 5-16. doi:10.1007/s13105-016-0519-z
NLM
Simplicio JA, Vale GT do, Gonzaga NA, Leite LN, Hipólito UV, Pereira CA, Tostes R de CA, Tirapelli CR. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries [Internet]. Journal of Physiology and Biochemistry. 2017 ; 73( 1): 5-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s13105-016-0519-z
Vancouver
Simplicio JA, Vale GT do, Gonzaga NA, Leite LN, Hipólito UV, Pereira CA, Tostes R de CA, Tirapelli CR. Reactive oxygen species derived from NAD(P)H oxidase play a role on ethanol-induced hypertension and endothelial dysfunction in rat resistance arteries [Internet]. Journal of Physiology and Biochemistry. 2017 ; 73( 1): 5-16.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s13105-016-0519-z
Artigo de periodico
Pharmacological characterization of the mechanisms underlying the vascular effects of succinate (2016)
Leite, Letícia N.; Gonzaga, Natália A.; Simplicio, Janaina A.; Vale, Gabriel T. do; Carballido, José M.; Alves Filho, José Carlos Farias; Tirapelli, Carlos Renato
Source: European Journal of Pharmacology. Unidades: FMRP, EERP
Subjects: AORTA TORÁCICA, ENDOTÉLIO VASCULAR, VASODILATAÇÃO
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ABNT
LEITE, Letícia N. et al. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, v. 789, p. 334-343, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.07.045. Acesso em: 14 nov. 2024.
APA
Leite, L. N., Gonzaga, N. A., Simplicio, J. A., Vale, G. T. do, Carballido, J. M., Alves Filho, J. C. F., & Tirapelli, C. R. (2016). Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, 789, 334-343. doi:10.1016/j.ejphar.2016.07.045
NLM
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Vancouver
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Artigo de periodico
Antihypertensive therapy in pre-eclampsia: effects of plasma from nonresponsive patients on endothelial gene expression (2016)
Luizon, Marcelo R.; Caldeira dias, Mayara; Deffune, Elenice; Fernandes, Karla S.; Cavalli, Ricardo de Carvalho; Tanus-Santos, José Eduardo; Sandrim, Valéria Cristina
Source: Pharmacogenomics. Unidade: FMRP
Subjects: FÁRMACOS (SISTEMA CARDIOVASCULAR), ENDOTÉLIO VASCULAR, EXPRESSÃO GÊNICA, CÉLULAS ENDOTELIAIS, PLASMA, PRÉ-ECLÂMPSIA
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ABNT
LUIZON, Marcelo R. et al. Antihypertensive therapy in pre-eclampsia: effects of plasma from nonresponsive patients on endothelial gene expression. Pharmacogenomics, v. 17, n. 10, p. 1121-1127, 2016Tradução . . Disponível em: https://doi.org/10.2217/pgs-2016-0033. Acesso em: 14 nov. 2024.
APA
Luizon, M. R., Caldeira dias, M., Deffune, E., Fernandes, K. S., Cavalli, R. de C., Tanus-Santos, J. E., & Sandrim, V. C. (2016). Antihypertensive therapy in pre-eclampsia: effects of plasma from nonresponsive patients on endothelial gene expression. Pharmacogenomics, 17( 10), 1121-1127. doi:10.2217/pgs-2016-0033
NLM
Luizon MR, Caldeira dias M, Deffune E, Fernandes KS, Cavalli R de C, Tanus-Santos JE, Sandrim VC. Antihypertensive therapy in pre-eclampsia: effects of plasma from nonresponsive patients on endothelial gene expression [Internet]. Pharmacogenomics. 2016 ; 17( 10): 1121-1127.[citado 2024 nov. 14 ] Available from: https://doi.org/10.2217/pgs-2016-0033
Vancouver
Luizon MR, Caldeira dias M, Deffune E, Fernandes KS, Cavalli R de C, Tanus-Santos JE, Sandrim VC. Antihypertensive therapy in pre-eclampsia: effects of plasma from nonresponsive patients on endothelial gene expression [Internet]. Pharmacogenomics. 2016 ; 17( 10): 1121-1127.[citado 2024 nov. 14 ] Available from: https://doi.org/10.2217/pgs-2016-0033
Artigo de periodico
Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries (2016)
Simplicio, Janaina A.; Hipólito, Ulisses Vilela; Vale, Gabriel Tavares do; Callera, Glaucia Elena; Pereira, Camila André; Touyz, Rhian M; Tostes, Rita de Cássia Aleixo; Tirapelli, Carlos Renato
Source: Arquivos Brasileiros de Cardiologia. Unidades: FMRP, EERP
Subjects: ETANOL, ESTRESSE OXIDATIVO, PROTEÍNAS, ENDOTÉLIO VASCULAR
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ABNT
SIMPLICIO, Janaina A. et al. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries. Arquivos Brasileiros de Cardiologia, v. 107, n. 5, p. 427-436, 2016Tradução . . Disponível em: https://doi.org/10.5935/abc.20160147. Acesso em: 14 nov. 2024.
APA
Simplicio, J. A., Hipólito, U. V., Vale, G. T. do, Callera, G. E., Pereira, C. A., Touyz, R. M., et al. (2016). Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries. Arquivos Brasileiros de Cardiologia, 107( 5), 427-436. doi:10.5935/abc.20160147
NLM
Simplicio JA, Hipólito UV, Vale GT do, Callera GE, Pereira CA, Touyz RM, Tostes R de CA, Tirapelli CR. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries [Internet]. Arquivos Brasileiros de Cardiologia. 2016 ; 107( 5): 427-436.[citado 2024 nov. 14 ] Available from: https://doi.org/10.5935/abc.20160147
Vancouver
Simplicio JA, Hipólito UV, Vale GT do, Callera GE, Pereira CA, Touyz RM, Tostes R de CA, Tirapelli CR. Acute ethanol intake induces NAD(P)H oxidase activation and Rhoa translocation in resistance arteries [Internet]. Arquivos Brasileiros de Cardiologia. 2016 ; 107( 5): 427-436.[citado 2024 nov. 14 ] Available from: https://doi.org/10.5935/abc.20160147
Parte de monografia/livro
Endotélio e genética (2016)
Lacchini, Riccardo ; Paula, Gustavo Henrique Oliveira de; Santos, José Eduardo Tanus dos
Source: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Volume 1. Unidades: EERP, FMRP
Subjects: ENDOTÉLIO VASCULAR, GENÉTICA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LACCHINI, Riccardo e PAULA, Gustavo Henrique Oliveira de e SANTOS, José Eduardo Tanus dos. Endotélio e genética. Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Volume 1. Tradução . Rio de Janeiro: Atheneu, 2016. . . Acesso em: 14 nov. 2024.
APA
Lacchini, R., Paula, G. H. O. de, & Santos, J. E. T. dos. (2016). Endotélio e genética. In Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Volume 1. Rio de Janeiro: Atheneu.
NLM
Lacchini R, Paula GHO de, Santos JET dos. Endotélio e genética. In: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Volume 1. Rio de Janeiro: Atheneu; 2016. [citado 2024 nov. 14 ]
Vancouver
Lacchini R, Paula GHO de, Santos JET dos. Endotélio e genética. In: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Volume 1. Rio de Janeiro: Atheneu; 2016. [citado 2024 nov. 14 ]
Artigo de periodico
Data on the mechanisms underlying succinate-induced aortic contraction (2016)
Gonzaga, Natália A.; Simplicio, Janaina A.; Leite, Letícia N.; Vale, Gabriel T.; Carballido, José M.; Alves Filho, José Carlos Farias; Tirapelli, Carlos Renato
Source: Data in Brief. Unidades: FMRP, EERP
Subjects: AORTA, ENDOTÉLIO VASCULAR, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZAGA, Natália A. et al. Data on the mechanisms underlying succinate-induced aortic contraction. Data in Brief, v. 9, p. 206-212, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.dib.2016.08.022. Acesso em: 14 nov. 2024.
APA
Gonzaga, N. A., Simplicio, J. A., Leite, L. N., Vale, G. T., Carballido, J. M., Alves Filho, J. C. F., & Tirapelli, C. R. (2016). Data on the mechanisms underlying succinate-induced aortic contraction. Data in Brief, 9, 206-212. doi:10.1016/j.dib.2016.08.022
NLM
Gonzaga NA, Simplicio JA, Leite LN, Vale GT, Carballido JM, Alves Filho JCF, Tirapelli CR. Data on the mechanisms underlying succinate-induced aortic contraction [Internet]. Data in Brief. 2016 ; 9 206-212.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.dib.2016.08.022
Vancouver
Gonzaga NA, Simplicio JA, Leite LN, Vale GT, Carballido JM, Alves Filho JCF, Tirapelli CR. Data on the mechanisms underlying succinate-induced aortic contraction [Internet]. Data in Brief. 2016 ; 9 206-212.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.dib.2016.08.022
Artigo de periodico
Omeprazole impairs vascular redox biology and causes xanthine oxidoreductase-mediated endothelial dysfunction (2016)
Pinheiro, Lucas C.; Oliveira-Paula, Gustavo H.; Portella, Rafael L.; Guimarães, Danielle A.; Angelis, Celio D. de; Tanus-Santos, José Eduardo
Source: Redox Biology. Unidade: FMRP
Subjects: ESTRESSE OXIDATIVO, FÁRMACOS (SISTEMA DIGESTÓRIO), ENDOTÉLIO VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINHEIRO, Lucas C. et al. Omeprazole impairs vascular redox biology and causes xanthine oxidoreductase-mediated endothelial dysfunction. Redox Biology, v. 9, p. 134-143, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.redox.2016.08.001. Acesso em: 14 nov. 2024.
APA
Pinheiro, L. C., Oliveira-Paula, G. H., Portella, R. L., Guimarães, D. A., Angelis, C. D. de, & Tanus-Santos, J. E. (2016). Omeprazole impairs vascular redox biology and causes xanthine oxidoreductase-mediated endothelial dysfunction. Redox Biology, 9, 134-143. doi:10.1016/j.redox.2016.08.001
NLM
Pinheiro LC, Oliveira-Paula GH, Portella RL, Guimarães DA, Angelis CD de, Tanus-Santos JE. Omeprazole impairs vascular redox biology and causes xanthine oxidoreductase-mediated endothelial dysfunction [Internet]. Redox Biology. 2016 ; 9 134-143.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.redox.2016.08.001
Vancouver
Pinheiro LC, Oliveira-Paula GH, Portella RL, Guimarães DA, Angelis CD de, Tanus-Santos JE. Omeprazole impairs vascular redox biology and causes xanthine oxidoreductase-mediated endothelial dysfunction [Internet]. Redox Biology. 2016 ; 9 134-143.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.redox.2016.08.001
Artigo de periodico
NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature (2016)
Marchi, Kátia Colombo; Ceron, Carla Speroni; Muniz, Jaqueline J.; De Martinis, Bruno Spinosa; Tanus-Santos, José Eduardo; Tirapelli, Carlos Renato
Source: Alcohol and Alcoholism. Unidades: FFCLRP, FMRP, EERP
Subjects: HIPERTENSÃO, ESTRESSE OXIDATIVO, ETANOL, ENDOTÉLIO VASCULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARCHI, Kátia Colombo et al. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature. Alcohol and Alcoholism, v. 51, n. 5, p. 522-534, 2016Tradução . . Disponível em: https://doi.org/10.1093/alcalc/agw043. Acesso em: 14 nov. 2024.
APA
Marchi, K. C., Ceron, C. S., Muniz, J. J., De Martinis, B. S., Tanus-Santos, J. E., & Tirapelli, C. R. (2016). NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature. Alcohol and Alcoholism, 51( 5), 522-534. doi:10.1093/alcalc/agw043
NLM
Marchi KC, Ceron CS, Muniz JJ, De Martinis BS, Tanus-Santos JE, Tirapelli CR. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature [Internet]. Alcohol and Alcoholism. 2016 ; 51( 5): 522-534.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/alcalc/agw043
Vancouver
Marchi KC, Ceron CS, Muniz JJ, De Martinis BS, Tanus-Santos JE, Tirapelli CR. NADPH oxidase plays a role on ethanol-induced hypertension and reactive oxygen species generation in the vasculature [Internet]. Alcohol and Alcoholism. 2016 ; 51( 5): 522-534.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1093/alcalc/agw043
Artigo de periodico
Matrix metalloproteinase (MMP)-2 decreases calponin-1 levels and contributes to arterial remodeling in early hypertension (2016)
Belo, Vanessa de Almeida; Parente, Juliana Montenegro; Tanus-Santos, José Eduardo; Castro, Michele Mazzaron de
Source: Biochemical Pharmacology. Unidade: FMRP
Subjects: HIPERTENSÃO, METALOPROTEINASES, ENDOTÉLIO VASCULAR, BIOMARCADORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BELO, Vanessa de Almeida et al. Matrix metalloproteinase (MMP)-2 decreases calponin-1 levels and contributes to arterial remodeling in early hypertension. Biochemical Pharmacology, v. 118, p. 50-58, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2016.08.012. Acesso em: 14 nov. 2024.
APA
Belo, V. de A., Parente, J. M., Tanus-Santos, J. E., & Castro, M. M. de. (2016). Matrix metalloproteinase (MMP)-2 decreases calponin-1 levels and contributes to arterial remodeling in early hypertension. Biochemical Pharmacology, 118, 50-58. doi:10.1016/j.bcp.2016.08.012
NLM
Belo V de A, Parente JM, Tanus-Santos JE, Castro MM de. Matrix metalloproteinase (MMP)-2 decreases calponin-1 levels and contributes to arterial remodeling in early hypertension [Internet]. Biochemical Pharmacology. 2016 ; 118 50-58.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bcp.2016.08.012
Vancouver
Belo V de A, Parente JM, Tanus-Santos JE, Castro MM de. Matrix metalloproteinase (MMP)-2 decreases calponin-1 levels and contributes to arterial remodeling in early hypertension [Internet]. Biochemical Pharmacology. 2016 ; 118 50-58.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1016/j.bcp.2016.08.012
Artigo de periodico-carta/editorial
In reply to: Enalapril and the VEGFA gene [Carta]: personalized medicine in hypertension therapy (2016)
Oliveira-Paula, G. H.; Lacchini, Riccardo ; Tanus-Santos, José Eduardo
Source: European Journal of Clinical Pharmacology. Unidades: EERP, FMRP
Subjects: ENDOTÉLIO VASCULAR, HIPERTENSÃO, POLIMORFISMO, VASODILATAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
OLIVEIRA-PAULA, G. H. e LACCHINI, Riccardo e TANUS-SANTOS, José Eduardo. In reply to: Enalapril and the VEGFA gene [Carta]: personalized medicine in hypertension therapy. European Journal of Clinical Pharmacology. Heidelberg: Escola de Enfermagem de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1007/s00228-015-1985-x. Acesso em: 14 nov. 2024. , 2016
APA
Oliveira-Paula, G. H., Lacchini, R., & Tanus-Santos, J. E. (2016). In reply to: Enalapril and the VEGFA gene [Carta]: personalized medicine in hypertension therapy. European Journal of Clinical Pharmacology. Heidelberg: Escola de Enfermagem de Ribeirão Preto, Universidade de São Paulo. doi:10.1007/s00228-015-1985-x
NLM
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. In reply to: Enalapril and the VEGFA gene [Carta]: personalized medicine in hypertension therapy [Internet]. European Journal of Clinical Pharmacology. 2016 ; 72( 1): 127-128.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00228-015-1985-x
Vancouver
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. In reply to: Enalapril and the VEGFA gene [Carta]: personalized medicine in hypertension therapy [Internet]. European Journal of Clinical Pharmacology. 2016 ; 72( 1): 127-128.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00228-015-1985-x
Artigo de periodico
Antihypertensive therapy in preeclampsia is not modulated by VEGF polymorphisms (2015)
Sandrim, Valeria C.; Palei, Ana C. T.; Eleuterio, Nibia; Tanus-Santos, José Eduardo; Cavalli, Ricardo de Carvalho
Source: Archives of Gynecology and Obstetrics. Unidade: FMRP
Subjects: ENDOTÉLIO VASCULAR, POLIMORFISMO, PRÉ-ECLÂMPSIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SANDRIM, Valeria C. et al. Antihypertensive therapy in preeclampsia is not modulated by VEGF polymorphisms. Archives of Gynecology and Obstetrics, v. 291, n. 4, p. 799-803, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00404-014-3475-2. Acesso em: 14 nov. 2024.
APA
Sandrim, V. C., Palei, A. C. T., Eleuterio, N., Tanus-Santos, J. E., & Cavalli, R. de C. (2015). Antihypertensive therapy in preeclampsia is not modulated by VEGF polymorphisms. Archives of Gynecology and Obstetrics, 291( 4), 799-803. doi:10.1007/s00404-014-3475-2
NLM
Sandrim VC, Palei ACT, Eleuterio N, Tanus-Santos JE, Cavalli R de C. Antihypertensive therapy in preeclampsia is not modulated by VEGF polymorphisms [Internet]. Archives of Gynecology and Obstetrics. 2015 ; 291( 4): 799-803.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00404-014-3475-2
Vancouver
Sandrim VC, Palei ACT, Eleuterio N, Tanus-Santos JE, Cavalli R de C. Antihypertensive therapy in preeclampsia is not modulated by VEGF polymorphisms [Internet]. Archives of Gynecology and Obstetrics. 2015 ; 291( 4): 799-803.[citado 2024 nov. 14 ] Available from: https://doi.org/10.1007/s00404-014-3475-2

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