Filtros : "ENDOTÉLIO" "ROSSONI, LUCIANA VENTURINI" "ICB" Removidos: "Fisiologia Humana" "RUSP" Limpar

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  • Fonte: PLOS ONE. Unidade: ICB

    Assuntos: FISIOLOGIA, EXERCICIO FÍSICO, ÓXIDO NITRICO, MENOPAUSA, ENDOTÉLIO

    Acesso à fonteDOIComo citar
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    • ABNT

      BRAGA, Viviane A. V. N. et al. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, v. 10, n. 4, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0125388. Acesso em: 13 nov. 2024.
    • APA

      Braga, V. A. V. N., Couto, G. K., Lazzarin, M. C., Rossoni, L. V., & Medeiros, A. (2015). Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, 10( 4), 1-13. doi:10.1371/journal.pone.0125388
    • NLM

      Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pone.0125388
    • Vancouver

      Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1371/journal.pone.0125388
  • Fonte: Life Sciences. Unidade: ICB

    Assuntos: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS

    Acesso à fonteDOIComo citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 13 nov. 2024.
    • APA

      Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
    • NLM

      Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
    • Vancouver

      Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018

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