Source: Journal of Clinical Endocrinology and Metabolism. Unidade: FMRP
Subjects: SÍNDROME DE KALLMANN, ENDOCRINOLOGIA, METABOLISMO
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ABNT
ABREU, Ana Paula et al. Loss-of-function mutations in the genes encoding prokineticin-2 or prokineticin receptor-2 cause autosomal recessive Kallmann syndrome. Journal of Clinical Endocrinology and Metabolism, v. 93, n. 10, p. 4113-4118, 2008Tradução . . Acesso em: 21 ago. 2024.APA
Abreu, A. P., Trarbach, E. B., Castro, M. de, Costa, E. M. F., Versiani, B., Baptista, M. T. M., et al. (2008). Loss-of-function mutations in the genes encoding prokineticin-2 or prokineticin receptor-2 cause autosomal recessive Kallmann syndrome. Journal of Clinical Endocrinology and Metabolism, 93( 10), 4113-4118.NLM
Abreu AP, Trarbach EB, Castro M de, Costa EMF, Versiani B, Baptista MTM, Garmes HM, Mendonça BB, Latronico AC. Loss-of-function mutations in the genes encoding prokineticin-2 or prokineticin receptor-2 cause autosomal recessive Kallmann syndrome. Journal of Clinical Endocrinology and Metabolism. 2008 ; 93( 10): 4113-4118.[citado 2024 ago. 21 ]Vancouver
Abreu AP, Trarbach EB, Castro M de, Costa EMF, Versiani B, Baptista MTM, Garmes HM, Mendonça BB, Latronico AC. Loss-of-function mutations in the genes encoding prokineticin-2 or prokineticin receptor-2 cause autosomal recessive Kallmann syndrome. Journal of Clinical Endocrinology and Metabolism. 2008 ; 93( 10): 4113-4118.[citado 2024 ago. 21 ]