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Artigo de periodico
Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting (2024)
Rizzi, Liara; Grinberg, Lea Tenenholz
Fonte: Acta neuropathologica communications. Unidade: FM
Assuntos: DOENÇAS NEURODEGENERATIVAS, DOENÇA DE ALZHEIMER, BIOMARCADORES
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RIZZI, Liara e GRINBERG, Lea Tenenholz. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting. Acta neuropathologica communications, v. 12, n. 1, 2024Tradução . . Disponível em: https://observatorio.fm.usp.br/handle/OPI/59005. Acesso em: 10 nov. 2024.
APA
Rizzi, L., & Grinberg, L. T. (2024). Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting. Acta neuropathologica communications, 12( 1). doi:10.1186/s40478-024-01744-9
NLM
Rizzi L, Grinberg LT. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting [Internet]. Acta neuropathologica communications. 2024 ; 12( 1):[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/59005
Vancouver
Rizzi L, Grinberg LT. Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting [Internet]. Acta neuropathologica communications. 2024 ; 12( 1):[citado 2024 nov. 10 ] Available from: https://observatorio.fm.usp.br/handle/OPI/59005
Artigo de periodico
Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease (2024)
Singulani, Monique Patricio ; Ferreira, Ana Flávia Fernandes ; Figueroa, Paulina Belén Sepulveda ; Cuyul-Vásquez, Iván; Talib, Leda Leme ; Forlenza, Orestes Vicente ; Britto, Luiz Roberto Giorgetti de
Fonte: Ageing Research Reviews. Unidades: ICB, FM
Assuntos: FISIOLOGIA, DOENÇA DE ALZHEIMER, DOENÇA DE PARKINSON, LÍTIO, DOENÇAS NEURODEGENERATIVAS, REVISÃO SISTEMÁTICA
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SINGULANI, Monique Patricio et al. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, v. 95, p. 20 , 2024Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2024.102231. Acesso em: 10 nov. 2024.
APA
Singulani, M. P., Ferreira, A. F. F., Figueroa, P. B. S., Cuyul-Vásquez, I., Talib, L. L., Forlenza, O. V., & Britto, L. R. G. de. (2024). Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease. Ageing Research Reviews, 95, 20 . doi:10.1016/j.arr.2024.102231
NLM
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Vancouver
Singulani MP, Ferreira AFF, Figueroa PBS, Cuyul-Vásquez I, Talib LL, Forlenza OV, Britto LRG de. Lithium and disease modification: a systematic review and meta-analysis in Alzheimer's and Parkinson’s disease [Internet]. Ageing Research Reviews. 2024 ; 95 20 .[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.arr.2024.102231
Artigo de periodico
Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients (2023)
Ravagnania, Felipe G; Valerio, Hellen Paula ; Maués, Jersey H. S ; Oliveira, Arthur N. de; Puga, Renato David ; Oliveira, Karina Griesi ; Picosse, Fabíola Rosa ; Ferraz, Henrique Ballalai ; Catharino, Rodrigo Ramos ; Ronsein, Graziella Eliza ; Aguiar, Patricia Maria de Carvalho
Fonte: Parkinsonism and Related Disorders. Unidade: IQ
Assuntos: ASTRÓCITOS, DOENÇAS NEURODEGENERATIVAS
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RAVAGNANIA, Felipe G et al. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients. Parkinsonism and Related Disorders, v. 116, p. 1-12, 2023Tradução . . Disponível em: https://doi.org/10.1016/j.parkreldis.2023.105847. Acesso em: 10 nov. 2024.
APA
Ravagnania, F. G., Valerio, H. P., Maués, J. H. S., Oliveira, A. N. de, Puga, R. D., Oliveira, K. G., et al. (2023). Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients. Parkinsonism and Related Disorders, 116, 1-12. doi:10.1016/j.parkreldis.2023.105847
NLM
Ravagnania FG, Valerio HP, Maués JHS, Oliveira AN de, Puga RD, Oliveira KG, Picosse FR, Ferraz HB, Catharino RR, Ronsein GE, Aguiar PM de C. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients [Internet]. Parkinsonism and Related Disorders. 2023 ; 116 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.parkreldis.2023.105847
Vancouver
Ravagnania FG, Valerio HP, Maués JHS, Oliveira AN de, Puga RD, Oliveira KG, Picosse FR, Ferraz HB, Catharino RR, Ronsein GE, Aguiar PM de C. Omics profile of iPSC-derived astrocytes from Progressive Supranuclear Palsy (PSP) patients [Internet]. Parkinsonism and Related Disorders. 2023 ; 116 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.parkreldis.2023.105847
Artigo de periodico
Cochlear re-implantation with the use of multi-mode grounding associated with anodic monophasic pulses to manage abnormal facial nerve stimulation (2023)
Hyppolito, Miguel Angelo; Reis, Ana Cláudia Mirândola Barbosa; Danieli, Fabiana; Hussain, Raabid; Le Goff, Nicolas
Fonte: Cochlear Implants International. Unidade: FMRP
Assuntos: IMPLANTE DA CÓCLEA, SURDEZ, PERDA AUDITIVA, DOENÇAS NEURODEGENERATIVAS
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HYPPOLITO, Miguel Angelo et al. Cochlear re-implantation with the use of multi-mode grounding associated with anodic monophasic pulses to manage abnormal facial nerve stimulation. Cochlear Implants International, v. 24, n. 2, p. 55-64, 2023Tradução . . Disponível em: https://doi.org/10.1080/14670100.2022.2157077. Acesso em: 10 nov. 2024.
APA
Hyppolito, M. A., Reis, A. C. M. B., Danieli, F., Hussain, R., & Le Goff, N. (2023). Cochlear re-implantation with the use of multi-mode grounding associated with anodic monophasic pulses to manage abnormal facial nerve stimulation. Cochlear Implants International, 24( 2), 55-64. doi:10.1080/14670100.2022.2157077
NLM
Hyppolito MA, Reis ACMB, Danieli F, Hussain R, Le Goff N. Cochlear re-implantation with the use of multi-mode grounding associated with anodic monophasic pulses to manage abnormal facial nerve stimulation [Internet]. Cochlear Implants International. 2023 ; 24( 2): 55-64.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/14670100.2022.2157077
Vancouver
Hyppolito MA, Reis ACMB, Danieli F, Hussain R, Le Goff N. Cochlear re-implantation with the use of multi-mode grounding associated with anodic monophasic pulses to manage abnormal facial nerve stimulation [Internet]. Cochlear Implants International. 2023 ; 24( 2): 55-64.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/14670100.2022.2157077
Artigo de periodico
Neuroprotective action of α-Klotho against LPS-activated glia conditioned medium in primary neuronal culture (2022)
Nakao, Vinicius Wanatable ; Mazucanti, Caio Henrique Yokowama ; Lima, Larissa de Sá ; Mello, Paloma Segura de ; Port’s, Natacha Medeiros de Souza ; Kinoshita, Paula Fernanda ; Leite, Jacqueline Alves ; Kawamoto, Elisa Mitiko ; Scavone, Cristoforo
Fonte: Scientific Reports. Unidade: ICB
Assuntos: FARMACOLOGIA, MODELOS ANIMAIS DE DOENÇAS, CAMUNDONGOS, DOENÇAS NEURODEGENERATIVAS, MORTE CEREBRAL, CITOCINAS, ASTRÓCITOS, NEURÓGLIA, LIPOPOLISSACARÍDEOS, INTERLEUCINA 6, MEIOS DE CULTURA, INFLAMAÇÃO
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NAKAO, Vinicius Wanatable et al. Neuroprotective action of α-Klotho against LPS-activated glia conditioned medium in primary neuronal culture. Scientific Reports, v. 12, p. 1-12, 2022Tradução . . Disponível em: https://doi.org/10.1038/s41598-022-21132-4. Acesso em: 10 nov. 2024.
APA
Nakao, V. W., Mazucanti, C. H. Y., Lima, L. de S., Mello, P. S. de, Port’s, N. M. de S., Kinoshita, P. F., et al. (2022). Neuroprotective action of α-Klotho against LPS-activated glia conditioned medium in primary neuronal culture. Scientific Reports, 12, 1-12. doi:10.1038/s41598-022-21132-4
NLM
Nakao VW, Mazucanti CHY, Lima L de S, Mello PS de, Port’s NM de S, Kinoshita PF, Leite JA, Kawamoto EM, Scavone C. Neuroprotective action of α-Klotho against LPS-activated glia conditioned medium in primary neuronal culture [Internet]. Scientific Reports. 2022 ; 12 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-022-21132-4
Vancouver
Nakao VW, Mazucanti CHY, Lima L de S, Mello PS de, Port’s NM de S, Kinoshita PF, Leite JA, Kawamoto EM, Scavone C. Neuroprotective action of α-Klotho against LPS-activated glia conditioned medium in primary neuronal culture [Internet]. Scientific Reports. 2022 ; 12 1-12.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-022-21132-4
Artigo de periodico
Cardiac autonomic modulation in subjects with amyotrophic lateral sclerosis (ALS) during an upper limb virtual reality task: a prospective control trial (2022)
Silveira, Ana Clara ; Moraes, Íbis Ariana Peña de ; Vidigal, Giovanna de Paula ; Simcsik, Amanda Orasmo ; Rosa, Renata Martins ; Favero, Francis Meire ; Fernandes, Susi Mary de Souza ; Garner, David M. ; Araújo, Luciano Vieira de ; Massa, Marcelo ; Vanderlei, Luiz Carlos Marques ; Silva, Talita Dias da ; Monteiro, Carlos Bandeira de Mello
Fonte: BioMed Research International. Unidades: EACH, FM
Assuntos: ESCLEROSE AMIOTRÓFICA LATERAL, REALIDADE VIRTUAL, SISTEMA NERVOSO SIMPÁTICO, FREQUÊNCIA CARDÍACA, MEMBROS SUPERIORES, DOENÇAS NEURODEGENERATIVAS
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SILVEIRA, Ana Clara et al. Cardiac autonomic modulation in subjects with amyotrophic lateral sclerosis (ALS) during an upper limb virtual reality task: a prospective control trial. BioMed Research International, v. 2022, p. 01-11, 2022Tradução . . Disponível em: https://doi.org/10.1155/2022/4439681. Acesso em: 10 nov. 2024.
APA
Silveira, A. C., Moraes, Í. A. P. de, Vidigal, G. de P., Simcsik, A. O., Rosa, R. M., Favero, F. M., et al. (2022). Cardiac autonomic modulation in subjects with amyotrophic lateral sclerosis (ALS) during an upper limb virtual reality task: a prospective control trial. BioMed Research International, 2022, 01-11. doi:10.1155/2022/4439681
NLM
Silveira AC, Moraes ÍAP de, Vidigal G de P, Simcsik AO, Rosa RM, Favero FM, Fernandes SM de S, Garner DM, Araújo LV de, Massa M, Vanderlei LCM, Silva TD da, Monteiro CB de M. Cardiac autonomic modulation in subjects with amyotrophic lateral sclerosis (ALS) during an upper limb virtual reality task: a prospective control trial [Internet]. BioMed Research International. 2022 ; 2022 01-11.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1155/2022/4439681
Vancouver
Silveira AC, Moraes ÍAP de, Vidigal G de P, Simcsik AO, Rosa RM, Favero FM, Fernandes SM de S, Garner DM, Araújo LV de, Massa M, Vanderlei LCM, Silva TD da, Monteiro CB de M. Cardiac autonomic modulation in subjects with amyotrophic lateral sclerosis (ALS) during an upper limb virtual reality task: a prospective control trial [Internet]. BioMed Research International. 2022 ; 2022 01-11.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1155/2022/4439681
Artigo de periodico
Oxidative stress inhibition via Apocynin prevents contributes to medullary respiratory neurodegeneration and respiratory pattern dysfunction in 6-hydroxydopamine animal model of Parkinson’s disease (2022)
Nascimento, André Luiz Ferreira; Medeiros, Pamela Ojuorun Santos ; Pedrão, Luiz Fernando de Araújo Trindade ; Queiroz, Vitoria C.; Santos, Luiz Marcelo Oliveira ; Novaes, Leonardo Santana ; Caetano, Ariadiny de Lima ; Munhoz, Carolina Demarchi ; Takakura, Ana Carolina ; Falquetto, Bárbara
Fonte: Neuroscience. Unidade: ICB
Assuntos: FARMACOLOGIA, MODELOS ANIMAIS DE DOENÇAS, DOENÇA DE PARKINSON, ESTRESSE OXIDATIVO, RESPIRAÇÃO ANIMAL, INIBIDORES DE ENZIMAS, DOENÇAS NEURODEGENERATIVAS
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NASCIMENTO, André Luiz Ferreira et al. Oxidative stress inhibition via Apocynin prevents contributes to medullary respiratory neurodegeneration and respiratory pattern dysfunction in 6-hydroxydopamine animal model of Parkinson’s disease. Neuroscience, p. 1-15, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2022.07.034. Acesso em: 10 nov. 2024.
APA
Nascimento, A. L. F., Medeiros, P. O. S., Pedrão, L. F. de A. T., Queiroz, V. C., Santos, L. M. O., Novaes, L. S., et al. (2022). Oxidative stress inhibition via Apocynin prevents contributes to medullary respiratory neurodegeneration and respiratory pattern dysfunction in 6-hydroxydopamine animal model of Parkinson’s disease. Neuroscience, 1-15. doi:10.1016/j.neuroscience.2022.07.034
NLM
Nascimento ALF, Medeiros POS, Pedrão LF de AT, Queiroz VC, Santos LMO, Novaes LS, Caetano A de L, Munhoz CD, Takakura AC, Falquetto B. Oxidative stress inhibition via Apocynin prevents contributes to medullary respiratory neurodegeneration and respiratory pattern dysfunction in 6-hydroxydopamine animal model of Parkinson’s disease [Internet]. Neuroscience. 2022 ; 1-15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2022.07.034
Vancouver
Nascimento ALF, Medeiros POS, Pedrão LF de AT, Queiroz VC, Santos LMO, Novaes LS, Caetano A de L, Munhoz CD, Takakura AC, Falquetto B. Oxidative stress inhibition via Apocynin prevents contributes to medullary respiratory neurodegeneration and respiratory pattern dysfunction in 6-hydroxydopamine animal model of Parkinson’s disease [Internet]. Neuroscience. 2022 ; 1-15.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.neuroscience.2022.07.034
Artigo de periodico
Focused goodness of fit tests for gene set analyses (2022)
Zhang, Mengqi; Gelfman, Sahar; Moreno, Cristiane Araujo Martins ; Mccarthy, Janice M.; Harms, Matthew B.; Goldstein, David B.; Allen, Andrew S.
Fonte: Briefings in bioinformatics. Unidade: FM
Assuntos: ESCLEROSE AMIOTRÓFICA LATERAL, MUTAÇÃO GENÉTICA, DOENÇAS NEURODEGENERATIVAS
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ZHANG, Mengqi et al. Focused goodness of fit tests for gene set analyses. Briefings in bioinformatics, v. 23, n. 1, 2022Tradução . . Disponível em: https://doi.org/10.1093/bib/bbab472. Acesso em: 10 nov. 2024.
APA
Zhang, M., Gelfman, S., Moreno, C. A. M., Mccarthy, J. M., Harms, M. B., Goldstein, D. B., & Allen, A. S. (2022). Focused goodness of fit tests for gene set analyses. Briefings in bioinformatics, 23( 1). doi:10.1093/bib/bbab472
NLM
Zhang M, Gelfman S, Moreno CAM, Mccarthy JM, Harms MB, Goldstein DB, Allen AS. Focused goodness of fit tests for gene set analyses [Internet]. Briefings in bioinformatics. 2022 ; 23( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/bib/bbab472
Vancouver
Zhang M, Gelfman S, Moreno CAM, Mccarthy JM, Harms MB, Goldstein DB, Allen AS. Focused goodness of fit tests for gene set analyses [Internet]. Briefings in bioinformatics. 2022 ; 23( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1093/bib/bbab472
Artigo de periodico-carta/editorial
Building bridges in neuropharmacology: new therapeutic approaches for psychiatric and neurodegenerative disorders (2022)
Bailey, Alexis ; Berwick, Daniel C.; Camarini, Rosana ; Scavone, Cristoforo
Fonte: British Journal of Pharmacology. Unidade: ICB
Assuntos: FARMACOLOGIA, NEUROFARMACOLOGIA, TRANSTORNOS MENTAIS, DOENÇAS NEURODEGENERATIVAS
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BAILEY, Alexis et al. Building bridges in neuropharmacology: new therapeutic approaches for psychiatric and neurodegenerative disorders. British Journal of Pharmacology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1111/bph.15711. Acesso em: 10 nov. 2024. , 2022
APA
Bailey, A., Berwick, D. C., Camarini, R., & Scavone, C. (2022). Building bridges in neuropharmacology: new therapeutic approaches for psychiatric and neurodegenerative disorders. British Journal of Pharmacology. Oxford: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1111/bph.15711
NLM
Bailey A, Berwick DC, Camarini R, Scavone C. Building bridges in neuropharmacology: new therapeutic approaches for psychiatric and neurodegenerative disorders [Internet]. British Journal of Pharmacology. 2022 ; 179( 8): 1475-1477.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15711
Vancouver
Bailey A, Berwick DC, Camarini R, Scavone C. Building bridges in neuropharmacology: new therapeutic approaches for psychiatric and neurodegenerative disorders [Internet]. British Journal of Pharmacology. 2022 ; 179( 8): 1475-1477.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15711
Artigo de periodico
Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats (2022)
Moraes, Ruan Carlos Macêdo de ; Lima, Gabriely Cristina Alves; Cardinali, Camila Aparecida Errerias Fernandes ; Gonçalves, Alisson Carvalho de ; Portari, Guilherme Vannucchi ; Guerra-Shinohara, Elvira Maria ; Leboucher, Antoine ; Donato Junior, José ; Kleinridders, André ; Torrão, Andréa da Silva
Fonte: Life Sciences. Unidades: FCF, ICB, FM
Assuntos: DOENÇA DE ALZHEIMER, HIPOTÁLAMO, FISIOLOGIA, VITAMINAS, DEMÊNCIA, INSULINA, DOENÇAS METABÓLICAS, METABOLISMO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, DOENÇAS NEURODEGENERATIVAS
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MORAES, Ruan Carlos Macêdo de et al. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, v. 306, p. 1-13, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2022.120841. Acesso em: 10 nov. 2024.
APA
Moraes, R. C. M. de, Lima, G. C. A., Cardinali, C. A. E. F., Gonçalves, A. C. de, Portari, G. V., Guerra-Shinohara, E. M., et al. (2022). Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats. Life Sciences, 306, 1-13. doi:10.1016/j.lfs.2022.120841
NLM
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Vancouver
Moraes RCM de, Lima GCA, Cardinali CAEF, Gonçalves AC de, Portari GV, Guerra-Shinohara EM, Leboucher A, Donato Junior J, Kleinridders A, Torrão A da S. Benfotiamine protects against hypothalamic dysfunction in a STZ-induced model of neurodegeneration in rats [Internet]. Life Sciences. 2022 ; 306 1-13.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1016/j.lfs.2022.120841
Artigo de periodico
From purines to purinergic signalling: molecular functions and human diseases (2021)
Huang, Zhao; Xie, Na; Illes, Peter; Virgilio, Francesco Di ; Ulrich, Henning ; Semyanov, Alexey ; Verkhratsky, Alexei; Sperlagh, Beata; Yu, Shu-Guang; Huang, Canhua ; Tang, Yong
Fonte: Signal Transduction and Targeted Therapy. Unidade: IQ
Assuntos: PURINAS, DOENÇAS NEURODEGENERATIVAS
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ABNT
HUANG, Zhao et al. From purines to purinergic signalling: molecular functions and human diseases. Signal Transduction and Targeted Therapy, v. 6, p. 1-20 art. 162, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41392-021-00553-z. Acesso em: 10 nov. 2024.
APA
Huang, Z., Xie, N., Illes, P., Virgilio, F. D., Ulrich, H., Semyanov, A., et al. (2021). From purines to purinergic signalling: molecular functions and human diseases. Signal Transduction and Targeted Therapy, 6, 1-20 art. 162. doi:10.1038/s41392-021-00553-z
NLM
Huang Z, Xie N, Illes P, Virgilio FD, Ulrich H, Semyanov A, Verkhratsky A, Sperlagh B, Yu S-G, Huang C, Tang Y. From purines to purinergic signalling: molecular functions and human diseases [Internet]. Signal Transduction and Targeted Therapy. 2021 ; 6 1-20 art. 162.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41392-021-00553-z
Vancouver
Huang Z, Xie N, Illes P, Virgilio FD, Ulrich H, Semyanov A, Verkhratsky A, Sperlagh B, Yu S-G, Huang C, Tang Y. From purines to purinergic signalling: molecular functions and human diseases [Internet]. Signal Transduction and Targeted Therapy. 2021 ; 6 1-20 art. 162.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41392-021-00553-z
Artigo de periodico
Cannabidiol induces autophagy via ERK1/2 activation in neural cells (2021)
Vrechi, Talita Aparecida de Moraes; Leão, Anderson H. F. F.; Morais, Ingrid B. M.; Abilio, Vanessa Costhek; Zuardi, Antonio Waldo; Hallak, Jaime Eduardo Cecilio ; Crippa, José Alexandre de Souza ; Bincoletto, Cláudia; Ureshino, Rodrigo P.; Smaili, Soraya Soubhi; Pereira, Gustavo José da Silva
Fonte: Scientific Reports. Unidade: FMRP
Assuntos: CANABINOIDES, LINHAGEM CELULAR, DOENÇAS NEURODEGENERATIVAS, NEUROBLASTOMA, FARMACOTERAPIA
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VRECHI, Talita Aparecida de Moraes et al. Cannabidiol induces autophagy via ERK1/2 activation in neural cells. Scientific Reports, v. 11, n. 1, 2021Tradução . . Disponível em: https://doi.org/10.1038/s41598-021-84879-2. Acesso em: 10 nov. 2024.
APA
Vrechi, T. A. de M., Leão, A. H. F. F., Morais, I. B. M., Abilio, V. C., Zuardi, A. W., Hallak, J. E. C., et al. (2021). Cannabidiol induces autophagy via ERK1/2 activation in neural cells. Scientific Reports, 11( 1). doi:10.1038/s41598-021-84879-2
NLM
Vrechi TA de M, Leão AHFF, Morais IBM, Abilio VC, Zuardi AW, Hallak JEC, Crippa JA de S, Bincoletto C, Ureshino RP, Smaili SS, Pereira GJ da S. Cannabidiol induces autophagy via ERK1/2 activation in neural cells [Internet]. Scientific Reports. 2021 ; 11( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-021-84879-2
Vancouver
Vrechi TA de M, Leão AHFF, Morais IBM, Abilio VC, Zuardi AW, Hallak JEC, Crippa JA de S, Bincoletto C, Ureshino RP, Smaili SS, Pereira GJ da S. Cannabidiol induces autophagy via ERK1/2 activation in neural cells [Internet]. Scientific Reports. 2021 ; 11( 1):[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-021-84879-2
Artigo de periodico
The Janus face of ouabain in Na+/K+‐ATPase and calcium signalling in neurons (2021)
Kinoshita, Paula Fernanda ; Orellana, Ana Maria Marques ; Nakao, Vinicius Watanabe ; Port's, Natacha Medeiros de Souza ; Quintas, Luis Eduardo Menezes ; Kawamoto, Elisa Mitiko ; Scavone, Cristoforo
Fonte: British Journal of Pharmacology. Unidade: ICB
Assuntos: FARMACOLOGIA, ESTEROIDES, NEURÔNIOS, CÉREBRO, CÁLCIO, PROTEÍNAS DA MEMBRANA, MEMBRANA PLASMÁTICA, HOMEOSTASE, DOENÇAS NEURODEGENERATIVAS
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ABNT
KINOSHITA, Paula Fernanda et al. The Janus face of ouabain in Na+/K+‐ATPase and calcium signalling in neurons. British Journal of Pharmacology, p. 1-13, 2021Tradução . . Disponível em: https://doi.org/10.1111/bph.15419. Acesso em: 10 nov. 2024.
APA
Kinoshita, P. F., Orellana, A. M. M., Nakao, V. W., Port's, N. M. de S., Quintas, L. E. M., Kawamoto, E. M., & Scavone, C. (2021). The Janus face of ouabain in Na+/K+‐ATPase and calcium signalling in neurons. British Journal of Pharmacology, 1-13. doi:10.1111/bph.15419
NLM
Kinoshita PF, Orellana AMM, Nakao VW, Port's NM de S, Quintas LEM, Kawamoto EM, Scavone C. The Janus face of ouabain in Na+/K+‐ATPase and calcium signalling in neurons [Internet]. British Journal of Pharmacology. 2021 ; 1-13.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15419
Vancouver
Kinoshita PF, Orellana AMM, Nakao VW, Port's NM de S, Quintas LEM, Kawamoto EM, Scavone C. The Janus face of ouabain in Na+/K+‐ATPase and calcium signalling in neurons [Internet]. British Journal of Pharmacology. 2021 ; 1-13.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15419
Artigo de periodico
Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders (2021)
Leite, Jefferson Antonio ; Nunes, Bruno Ghirotto ; Targhetta, Vitor Pedro ; Lima, Jean de ; Câmara, Niels Olsen Saraiva
Fonte: British Journal of Pharmacology. Unidades: ICB, FMRP
Assuntos: IMUNOLOGIA, DOENÇA DE ALZHEIMER, TRANSTORNO AUTÍSTICO, TRANSTORNOS DO HUMOR, ESCLEROSE MÚLTIPLA, ESQUIZOFRENIA, DOENÇAS NEURODEGENERATIVAS
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ABNT
LEITE, Jefferson Antonio et al. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders. British Journal of Pharmacology, v. 179, n. 8, p. 1496-1511, 2021Tradução . . Disponível em: https://doi.org/10.1111/bph.15570. Acesso em: 10 nov. 2024.
APA
Leite, J. A., Nunes, B. G., Targhetta, V. P., Lima, J. de, & Câmara, N. O. S. (2021). Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders. British Journal of Pharmacology, 179( 8), 1496-1511. doi:10.1111/bph.15570
NLM
Leite JA, Nunes BG, Targhetta VP, Lima J de, Câmara NOS. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders [Internet]. British Journal of Pharmacology. 2021 ; 179( 8): 1496-1511.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15570
Vancouver
Leite JA, Nunes BG, Targhetta VP, Lima J de, Câmara NOS. Sirtuins as pharmacological targets in neurodegenerative and neuropsychiatric disorders [Internet]. British Journal of Pharmacology. 2021 ; 179( 8): 1496-1511.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.15570
Artigo de periodico
Neuroinflammation at single cell level: what is new? (2020)
Brandão, Wesley Nogueira ; Oliveira, M. G. de; Andreoni, R. T.; Nakaya, Helder Takashi Imoto ; Farias, A. S.; Peron, Jean Pierre Schatzmann
Fonte: Journal of Leukocyte Biology. Unidades: FCF, ICB
Assuntos: IMUNOLOGIA, DOENÇAS DO SISTEMA NERVOSO CENTRAL, EPITOPOS, DOENÇAS NEURODEGENERATIVAS, ATROFIA DE MÚLTIPLOS SISTEMAS, ENCEFALOMIELITE ANIMAL, MIELINA DE ANIMAL, CAMUNDONGOS, LINFÓCITOS T, MEDULA ESPINHAL, PLASTICIDADE NEURONAL
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ABNT
BRANDÃO, Wesley Nogueira et al. Neuroinflammation at single cell level: what is new?. Journal of Leukocyte Biology, v. 108, n. 4, p. 1129-1137, 2020Tradução . . Disponível em: https://doi.org/10.1002/JLB.3MR0620-035R. Acesso em: 10 nov. 2024.
APA
Brandão, W. N., Oliveira, M. G. de, Andreoni, R. T., Nakaya, H. T. I., Farias, A. S., & Peron, J. P. S. (2020). Neuroinflammation at single cell level: what is new? Journal of Leukocyte Biology, 108( 4), 1129-1137. doi:10.1002/JLB.3MR0620-035R
NLM
Brandão WN, Oliveira MG de, Andreoni RT, Nakaya HTI, Farias AS, Peron JPS. Neuroinflammation at single cell level: what is new? [Internet]. Journal of Leukocyte Biology. 2020 ; 108( 4): 1129-1137.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/JLB.3MR0620-035R
Vancouver
Brandão WN, Oliveira MG de, Andreoni RT, Nakaya HTI, Farias AS, Peron JPS. Neuroinflammation at single cell level: what is new? [Internet]. Journal of Leukocyte Biology. 2020 ; 108( 4): 1129-1137.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1002/JLB.3MR0620-035R
Artigo de periodico
Oxidative stress in the medullary respiratory neurons contributes to the respiratory dysfunction in the 6‐OHDA model of Parkinson's disease (2020)
Falquetto, Bárbara ; Thieme, Karina ; Malta, Marilia Brinatti; Rocha, Karina Cunha e ; Potje, Simone Regina ; Antonialli, Cristina ; Rodrigues, Alice Cristina ; Munhoz, Carolina Demarchi ; Moreira, Thiago dos Santos ; Takakura, Ana Carolina
Fonte: Journal of Physiology. Unidades: ICB, FCFRP
Assuntos: FISIOLOGIA, FARMACOLOGIA, ESTRESSE OXIDATIVO, APOPTOSE, RESPIRAÇÃO ANIMAL, RATOS, MODELOS ANIMAIS DE DOENÇAS, BARREIRA HEMATO ENCEFÁLICA, DOENÇA DE PARKINSON, RECEPTORES DOPAMINÉRGICOS, INSUFICIÊNCIA RESPIRATÓRIA, DOENÇAS NEURODEGENERATIVAS
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ABNT
FALQUETTO, Bárbara et al. Oxidative stress in the medullary respiratory neurons contributes to the respiratory dysfunction in the 6‐OHDA model of Parkinson's disease. Journal of Physiology, v. 598, n. 22, p. 5271-5293, 2020Tradução . . Disponível em: https://doi.org/10.1113/JP279791. Acesso em: 10 nov. 2024.
APA
Falquetto, B., Thieme, K., Malta, M. B., Rocha, K. C. e, Potje, S. R., Antonialli, C., et al. (2020). Oxidative stress in the medullary respiratory neurons contributes to the respiratory dysfunction in the 6‐OHDA model of Parkinson's disease. Journal of Physiology, 598( 22), 5271-5293. doi:10.1113/JP279791
NLM
Falquetto B, Thieme K, Malta MB, Rocha KC e, Potje SR, Antonialli C, Rodrigues AC, Munhoz CD, Moreira T dos S, Takakura AC. Oxidative stress in the medullary respiratory neurons contributes to the respiratory dysfunction in the 6‐OHDA model of Parkinson's disease [Internet]. Journal of Physiology. 2020 ; 598( 22): 5271-5293.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1113/JP279791
Vancouver
Falquetto B, Thieme K, Malta MB, Rocha KC e, Potje SR, Antonialli C, Rodrigues AC, Munhoz CD, Moreira T dos S, Takakura AC. Oxidative stress in the medullary respiratory neurons contributes to the respiratory dysfunction in the 6‐OHDA model of Parkinson's disease [Internet]. Journal of Physiology. 2020 ; 598( 22): 5271-5293.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1113/JP279791
Artigo de periodico
α7 nicotinic ACh receptors are necessary for memory recovery and neuroprotection promoted by attention training in amyloid‐β‐infused mice (2019)
Telles‐Longui, Milena; Mourelle, Danilo; Schöwe, Natalia Mendes; Cipolli, Gabriela Cabett; Malerba, Helena Nascimento; Buck, Hudson Sousa; Viel, Tania Araujo
Fonte: British Journal of Pharmacology. Unidades: EACH, ICB
Assuntos: MEMÓRIA, RECEPTORES COLINÉRGICOS, DOENÇAS NEURODEGENERATIVAS
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ABNT
TELLES‐LONGUI, Milena et al. α7 nicotinic ACh receptors are necessary for memory recovery and neuroprotection promoted by attention training in amyloid‐β‐infused mice. British Journal of Pharmacology, v. 17, p. 3193-3205, 2019Tradução . . Disponível em: https://doi.org/10.1111/bph.14744. Acesso em: 10 nov. 2024.
APA
Telles‐Longui, M., Mourelle, D., Schöwe, N. M., Cipolli, G. C., Malerba, H. N., Buck, H. S., & Viel, T. A. (2019). α7 nicotinic ACh receptors are necessary for memory recovery and neuroprotection promoted by attention training in amyloid‐β‐infused mice. British Journal of Pharmacology, 17, 3193-3205. doi:10.1111/bph.14744
NLM
Telles‐Longui M, Mourelle D, Schöwe NM, Cipolli GC, Malerba HN, Buck HS, Viel TA. α7 nicotinic ACh receptors are necessary for memory recovery and neuroprotection promoted by attention training in amyloid‐β‐infused mice [Internet]. British Journal of Pharmacology. 2019 ; 17 3193-3205.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.14744
Vancouver
Telles‐Longui M, Mourelle D, Schöwe NM, Cipolli GC, Malerba HN, Buck HS, Viel TA. α7 nicotinic ACh receptors are necessary for memory recovery and neuroprotection promoted by attention training in amyloid‐β‐infused mice [Internet]. British Journal of Pharmacology. 2019 ; 17 3193-3205.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1111/bph.14744
Artigo de periodico
Alterations in lipid metabolism of spinal cord linked to amyotrophic lateral sclerosis (2019)
Chaves Filho, Adriano de Britto; Pinto, Isabella Fernanda Dantas; Dantas, Lucas Souza; Xavier, Andre Machado; Inague, Alex; Faria, Rodrigo Lucas de; Medeiros, Marisa Helena Gennari de ; Glezer, Isaias ; Yoshinaga, Marcos Yukio; Miyamoto, Sayuri
Fonte: Scientific Reports. Unidades: IQ, ICB
Assuntos: ESCLEROSE AMIOTRÓFICA LATERAL, LÍQUEN PLANO BUCAL, DOENÇAS NEURODEGENERATIVAS
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ABNT
CHAVES FILHO, Adriano de Britto et al. Alterations in lipid metabolism of spinal cord linked to amyotrophic lateral sclerosis. Scientific Reports, v. 9, n. 1, p. 1-14 art. 11642, 2019Tradução . . Disponível em: https://doi.org/10.1038/s41598-019-48059-7. Acesso em: 10 nov. 2024.
APA
Chaves Filho, A. de B., Pinto, I. F. D., Dantas, L. S., Xavier, A. M., Inague, A., Faria, R. L. de, et al. (2019). Alterations in lipid metabolism of spinal cord linked to amyotrophic lateral sclerosis. Scientific Reports, 9( 1), 1-14 art. 11642. doi:10.1038/s41598-019-48059-7
NLM
Chaves Filho A de B, Pinto IFD, Dantas LS, Xavier AM, Inague A, Faria RL de, Medeiros MHG de, Glezer I, Yoshinaga MY, Miyamoto S. Alterations in lipid metabolism of spinal cord linked to amyotrophic lateral sclerosis [Internet]. Scientific Reports. 2019 ; 9( 1): 1-14 art. 11642.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-019-48059-7
Vancouver
Chaves Filho A de B, Pinto IFD, Dantas LS, Xavier AM, Inague A, Faria RL de, Medeiros MHG de, Glezer I, Yoshinaga MY, Miyamoto S. Alterations in lipid metabolism of spinal cord linked to amyotrophic lateral sclerosis [Internet]. Scientific Reports. 2019 ; 9( 1): 1-14 art. 11642.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1038/s41598-019-48059-7
Artigo de periodico
Motor impairment in a rare form of spastic paraplegia (Spoan syndrome): a 10-year follow-up (2019)
Galvão, Cláudia R. C; Cavalcante, Priscilla M. A; Olinda, Ricardo; Graciani, Zodja; Zatz, Mayana ; Kok, Fernando; Santos, Silvana ; Lancman, Selma
Fonte: BMC Neurology. Unidades: IB, FM
Assuntos: DOENÇAS NEURODEGENERATIVAS, DOENÇAS GENÉTICAS, DOENÇAS RARAS
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ABNT
GALVÃO, Cláudia R. C et al. Motor impairment in a rare form of spastic paraplegia (Spoan syndrome): a 10-year follow-up. BMC Neurology, v. 19, 2019Tradução . . Disponível em: https://doi.org/10.1186/s12883-019-1465-5. Acesso em: 10 nov. 2024.
APA
Galvão, C. R. C., Cavalcante, P. M. A., Olinda, R., Graciani, Z., Zatz, M., Kok, F., et al. (2019). Motor impairment in a rare form of spastic paraplegia (Spoan syndrome): a 10-year follow-up. BMC Neurology, 19. doi:10.1186/s12883-019-1465-5
NLM
Galvão CRC, Cavalcante PMA, Olinda R, Graciani Z, Zatz M, Kok F, Santos S, Lancman S. Motor impairment in a rare form of spastic paraplegia (Spoan syndrome): a 10-year follow-up [Internet]. BMC Neurology. 2019 ; 19[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s12883-019-1465-5
Vancouver
Galvão CRC, Cavalcante PMA, Olinda R, Graciani Z, Zatz M, Kok F, Santos S, Lancman S. Motor impairment in a rare form of spastic paraplegia (Spoan syndrome): a 10-year follow-up [Internet]. BMC Neurology. 2019 ; 19[citado 2024 nov. 10 ] Available from: https://doi.org/10.1186/s12883-019-1465-5
Artigo de periodico
Effects of mild running on substantia nigra during early neurodegeneration (2018)
Almeida, Michael F; Silva, Carolliny M; Chaves, Rodrigo S; Lima, Nathan C. R; Almeida, Renato S; Melo, Karla P; Demasi, Marilene; Fernandes, Tiago ; Oliveira, Edilamar Menezes de; Netto, Luis Eduardo Soares ; Cardoso, Sandra M; Ferrari, Merari de Fátima Ramires
Fonte: Journal of sports sciences. Unidades: EEFE, IB
Assuntos: MITOCÔNDRIAS, MESENCÉFALO, ESTRESSE OXIDATIVO, DOENÇAS NEURODEGENERATIVAS, ATIVIDADE FÍSICA, TREINAMENTO AERÓBIO, TRANSPORTE BIOLÓGICO
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ABNT
ALMEIDA, Michael F et al. Effects of mild running on substantia nigra during early neurodegeneration. Journal of sports sciences, v. 36, n. 12, p. 1363-1370, 2018Tradução . . Disponível em: https://doi.org/10.1080/02640414.2017.1378494. Acesso em: 10 nov. 2024.
APA
Almeida, M. F., Silva, C. M., Chaves, R. S., Lima, N. C. R., Almeida, R. S., Melo, K. P., et al. (2018). Effects of mild running on substantia nigra during early neurodegeneration. Journal of sports sciences, 36( 12), 1363-1370. doi:10.1080/02640414.2017.1378494
NLM
Almeida MF, Silva CM, Chaves RS, Lima NCR, Almeida RS, Melo KP, Demasi M, Fernandes T, Oliveira EM de, Netto LES, Cardoso SM, Ferrari M de FR. Effects of mild running on substantia nigra during early neurodegeneration [Internet]. Journal of sports sciences. 2018 ; 36( 12): 1363-1370.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/02640414.2017.1378494
Vancouver
Almeida MF, Silva CM, Chaves RS, Lima NCR, Almeida RS, Melo KP, Demasi M, Fernandes T, Oliveira EM de, Netto LES, Cardoso SM, Ferrari M de FR. Effects of mild running on substantia nigra during early neurodegeneration [Internet]. Journal of sports sciences. 2018 ; 36( 12): 1363-1370.[citado 2024 nov. 10 ] Available from: https://doi.org/10.1080/02640414.2017.1378494

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