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Artigo de periodico
Clathrin adaptor AP-1–mediated Golgi export of amyloid precursor protein is crucial for the production of neurotoxic amyloid fragments (2022)
Januário, Yunan Costa; Eden, Jessica; Oliveira, Luan S. de; De Pace, Raffaella; Tavares, Lucas A.; Silva-Januário, Mara E. da; Apolloni, Vinícius B.; Wilby, Elise L.; Altmeyer, Randolf; Burgos, Patricia V.; Corrêa, Sonia A. L.; Gershlick, David C.; Silva, Luis Lamberti Pinto da
Source: Journal of Biological Chemistry. Unidade: FMRP
Subjects: DOENÇA DE ALZHEIMER, PROTEÍNAS, PEPTÍDEOS, AMILOIDOSE, COMPLEXO DE GOLGI, FATOR DE TRANSCRIÇÃO AP-1
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ABNT
JANUÁRIO, Yunan Costa et al. Clathrin adaptor AP-1–mediated Golgi export of amyloid precursor protein is crucial for the production of neurotoxic amyloid fragments. Journal of Biological Chemistry, v. 298, n. 8, 2022Tradução . . Disponível em: https://doi.org/10.1016/j.jbc.2022.102172. Acesso em: 05 nov. 2024.
APA
Januário, Y. C., Eden, J., Oliveira, L. S. de, De Pace, R., Tavares, L. A., Silva-Januário, M. E. da, et al. (2022). Clathrin adaptor AP-1–mediated Golgi export of amyloid precursor protein is crucial for the production of neurotoxic amyloid fragments. Journal of Biological Chemistry, 298( 8). doi:10.1016/j.jbc.2022.102172
NLM
Januário YC, Eden J, Oliveira LS de, De Pace R, Tavares LA, Silva-Januário ME da, Apolloni VB, Wilby EL, Altmeyer R, Burgos PV, Corrêa SAL, Gershlick DC, Silva LLP da. Clathrin adaptor AP-1–mediated Golgi export of amyloid precursor protein is crucial for the production of neurotoxic amyloid fragments [Internet]. Journal of Biological Chemistry. 2022 ; 298( 8):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.jbc.2022.102172
Vancouver
Januário YC, Eden J, Oliveira LS de, De Pace R, Tavares LA, Silva-Januário ME da, Apolloni VB, Wilby EL, Altmeyer R, Burgos PV, Corrêa SAL, Gershlick DC, Silva LLP da. Clathrin adaptor AP-1–mediated Golgi export of amyloid precursor protein is crucial for the production of neurotoxic amyloid fragments [Internet]. Journal of Biological Chemistry. 2022 ; 298( 8):[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.jbc.2022.102172
Artigo de periodico
A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance (2022)
Alves, Suélen Santos ; Silva Júnior, Rui Milton Patrício da; Delfino-Pereira, Polianna; Pereira, Marília Gabriella Alves Goulart; Vasconcelos, Israel Costa; Schwaemmle, Hanna; Mazzei, Rodrigo Focosi; Carlos, Maiko Luiz ; Espreáfico, Enilza Maria; Tedesco, Antônio Cláudio ; Sebollela, Adriano Silva ; Almeida, Sebastião de Sousa ; Oliveira, José Antonio Cortes de; Garcia-Cairasco, Norberto
Source: Molecular Neurobiology. Unidades: FFCLRP, FMRP
Subjects: EPILEPSIA, DOENÇA DE ALZHEIMER, RESISTÊNCIA À INSULINA, MEMÓRIA
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ABNT
ALVES, Suélen Santos et al. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, v. 59, n. 6, p. 3721-3737, 2022Tradução . . Disponível em: https://doi.org/10.1007/s12035-022-02810-w. Acesso em: 05 nov. 2024.
APA
Alves, S. S., Silva Júnior, R. M. P. da, Delfino-Pereira, P., Pereira, M. G. A. G., Vasconcelos, I. C., Schwaemmle, H., et al. (2022). A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance. Molecular Neurobiology, 59( 6), 3721-3737. doi:10.1007/s12035-022-02810-w
NLM
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Vancouver
Alves SS, Silva Júnior RMP da, Delfino-Pereira P, Pereira MGAG, Vasconcelos IC, Schwaemmle H, Mazzei RF, Carlos ML, Espreáfico EM, Tedesco AC, Sebollela AS, Almeida S de S, Oliveira JAC de, Garcia-Cairasco N. A genetic model of epilepsy with a partial Alzheimer’s disease-like phenotype and central insulin resistance [Internet]. Molecular Neurobiology. 2022 ; 59( 6): 3721-3737.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1007/s12035-022-02810-w
Artigo de periodico
Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein via the MVB/lysosomal pathway (2017)
González, Alexis E.; Muñoz, Vanessa C.; Cavieres, Viviana A.; Bustamante, Hianara A.; Cornejo, Víctor-Hugo; Januário, Yunan C.; González, Ibeth; Hetz, Claudio; Silva, Luis Lamberti Pinto da; Rojas-Fernández, Alejandro; Hay, Ronald T.; Mardones, Gonzalo A.; Burgos, Patricia V.
Source: The FASEB Journal. Unidade: FMRP
Subjects: DOENÇA DE ALZHEIMER, FATORES DE TRANSCRIÇÃO, CÉLULAS CULTIVADAS DE TUMOR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GONZÁLEZ, Alexis E. et al. Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein via the MVB/lysosomal pathway. The FASEB Journal, v. 31, n. 6, p. 2446-2459, 2017Tradução . . Disponível em: https://doi.org/10.1096/fj.201600713r. Acesso em: 05 nov. 2024.
APA
González, A. E., Muñoz, V. C., Cavieres, V. A., Bustamante, H. A., Cornejo, V. -H., Januário, Y. C., et al. (2017). Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein via the MVB/lysosomal pathway. The FASEB Journal, 31( 6), 2446-2459. doi:10.1096/fj.201600713r
NLM
González AE, Muñoz VC, Cavieres VA, Bustamante HA, Cornejo V-H, Januário YC, González I, Hetz C, Silva LLP da, Rojas-Fernández A, Hay RT, Mardones GA, Burgos PV. Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein via the MVB/lysosomal pathway [Internet]. The FASEB Journal. 2017 ; 31( 6): 2446-2459.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1096/fj.201600713r
Vancouver
González AE, Muñoz VC, Cavieres VA, Bustamante HA, Cornejo V-H, Januário YC, González I, Hetz C, Silva LLP da, Rojas-Fernández A, Hay RT, Mardones GA, Burgos PV. Autophagosomes cooperate in the degradation of intracellular C-terminal fragments of the amyloid precursor protein via the MVB/lysosomal pathway [Internet]. The FASEB Journal. 2017 ; 31( 6): 2446-2459.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1096/fj.201600713r
Artigo de periodico
Bloking effects of human tau on squid giant synapse transmission and its prevention by T-817 MA (2011)
Moreno, Herman; Choi, Soonwook; Yu, Eunah; Brusco, Janaína; Avila, Jesus; Moreira, Jorge Eduardo; Sugimori, Mutsuyuki; Llinás, Rodolfo R.
Source: Frontiers in Synaptic Neuroscience. Unidade: FMRP
Assunto: DOENÇA DE ALZHEIMER
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ABNT
MORENO, Herman et al. Bloking effects of human tau on squid giant synapse transmission and its prevention by T-817 MA. Frontiers in Synaptic Neuroscience, v. 3, 2011Tradução . . Acesso em: 05 nov. 2024.
APA
Moreno, H., Choi, S., Yu, E., Brusco, J., Avila, J., Moreira, J. E., et al. (2011). Bloking effects of human tau on squid giant synapse transmission and its prevention by T-817 MA. Frontiers in Synaptic Neuroscience, 3.
NLM
Moreno H, Choi S, Yu E, Brusco J, Avila J, Moreira JE, Sugimori M, Llinás RR. Bloking effects of human tau on squid giant synapse transmission and its prevention by T-817 MA. Frontiers in Synaptic Neuroscience. 2011 ; 3[citado 2024 nov. 05 ]
Vancouver
Moreno H, Choi S, Yu E, Brusco J, Avila J, Moreira JE, Sugimori M, Llinás RR. Bloking effects of human tau on squid giant synapse transmission and its prevention by T-817 MA. Frontiers in Synaptic Neuroscience. 2011 ; 3[citado 2024 nov. 05 ]
Artigo de periodico
Sorting of the Alzheimer's disease amyloid precursor protein mediated by the AP-4 complex (2010)
Burgos, Patricia V.; Mardones, Gonzalo A.; Rojas, Adriana L.; Silva, Luis Lamberti Pinto da; Prabhu, Yogikala; Hurley, James H.; Bonifacino, Juan S.
Source: Developmental Cell. Unidade: FMRP
Subjects: DOENÇA DE ALZHEIMER, PROTEÍNAS, TRIAGEM
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BURGOS, Patricia V. et al. Sorting of the Alzheimer's disease amyloid precursor protein mediated by the AP-4 complex. Developmental Cell, v. 18, n. 3, p. 425-436, 2010Tradução . . Acesso em: 05 nov. 2024.
APA
Burgos, P. V., Mardones, G. A., Rojas, A. L., Silva, L. L. P. da, Prabhu, Y., Hurley, J. H., & Bonifacino, J. S. (2010). Sorting of the Alzheimer's disease amyloid precursor protein mediated by the AP-4 complex. Developmental Cell, 18( 3), 425-436.
NLM
Burgos PV, Mardones GA, Rojas AL, Silva LLP da, Prabhu Y, Hurley JH, Bonifacino JS. Sorting of the Alzheimer's disease amyloid precursor protein mediated by the AP-4 complex. Developmental Cell. 2010 ; 18( 3): 425-436.[citado 2024 nov. 05 ]
Vancouver
Burgos PV, Mardones GA, Rojas AL, Silva LLP da, Prabhu Y, Hurley JH, Bonifacino JS. Sorting of the Alzheimer's disease amyloid precursor protein mediated by the AP-4 complex. Developmental Cell. 2010 ; 18( 3): 425-436.[citado 2024 nov. 05 ]
Artigo de periodico
Synaptic transmission block presynaptic injection of oligomeric amyloid beta (2009)
Moreno, Herman; Yu, Eunah; Pigino, Gustavo; Hernandez, Alejandro I.; Kim, Natalia; Moreira, Jorge Eduardo; Sugimori, Mutsuyuki; Llinás, Rodolfo Riascos
Source: Proceedings of the National Academy of Sciences of the United States of America. Unidade: FMRP
Subjects: DOENÇA DE ALZHEIMER, MICROSCOPIA DE FLUORESCÊNCIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MORENO, Herman et al. Synaptic transmission block presynaptic injection of oligomeric amyloid beta. Proceedings of the National Academy of Sciences of the United States of America, v. 106, n. 14, p. 5901-5906, 2009Tradução . . Disponível em: https://doi.org/10.1073/pnas.0900944106. Acesso em: 05 nov. 2024.
APA
Moreno, H., Yu, E., Pigino, G., Hernandez, A. I., Kim, N., Moreira, J. E., et al. (2009). Synaptic transmission block presynaptic injection of oligomeric amyloid beta. Proceedings of the National Academy of Sciences of the United States of America, 106( 14), 5901-5906. doi:10.1073/pnas.0900944106
NLM
Moreno H, Yu E, Pigino G, Hernandez AI, Kim N, Moreira JE, Sugimori M, Llinás RR. Synaptic transmission block presynaptic injection of oligomeric amyloid beta [Internet]. Proceedings of the National Academy of Sciences of the United States of America. 2009 ; 106( 14): 5901-5906.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1073/pnas.0900944106
Vancouver
Moreno H, Yu E, Pigino G, Hernandez AI, Kim N, Moreira JE, Sugimori M, Llinás RR. Synaptic transmission block presynaptic injection of oligomeric amyloid beta [Internet]. Proceedings of the National Academy of Sciences of the United States of America. 2009 ; 106( 14): 5901-5906.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1073/pnas.0900944106

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