Source: Journal of Hypertension. Unidade: ICB
Subjects: ANATOMIA, CÉLULAS ENDOTELIAIS, ÓXIDO NÍTRICO, VASODILATAÇÃO, PERÓXIDO DE HIDROGÊNIO, HIPERTENSÃO, CAMUNDONGOS, HOMEOSTASE, MODELOS ANIMAIS DE DOENÇAS, CANAIS IÔNICOS, CÁLCIO, REAÇÃO EM CADEIA POR POLIMERASE
ABNT
ALVES-LOPESA, Rheure et al. Vasoprotective effects of NOX4 are mediated via polymerase and transient receptor potential melastatin 2 cation channels in endothelial cells. Journal of Hypertension, v. 41, n. 9, p. 1389-1400, 2023Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000003478. Acesso em: 03 set. 2024.APA
Alves-Lopesa, R., Neves, K. B., Harvey, A., Montezano, A. C., Touyz, R. M., & Lacchini, S. (2023). Vasoprotective effects of NOX4 are mediated via polymerase and transient receptor potential melastatin 2 cation channels in endothelial cells. Journal of Hypertension, 41( 9), 1389-1400. doi:10.1097/HJH.0000000000003478NLM
Alves-Lopesa R, Neves KB, Harvey A, Montezano AC, Touyz RM, Lacchini S. Vasoprotective effects of NOX4 are mediated via polymerase and transient receptor potential melastatin 2 cation channels in endothelial cells [Internet]. Journal of Hypertension. 2023 ; 41( 9): 1389-1400.[citado 2024 set. 03 ] Available from: https://doi.org/10.1097/HJH.0000000000003478Vancouver
Alves-Lopesa R, Neves KB, Harvey A, Montezano AC, Touyz RM, Lacchini S. Vasoprotective effects of NOX4 are mediated via polymerase and transient receptor potential melastatin 2 cation channels in endothelial cells [Internet]. Journal of Hypertension. 2023 ; 41( 9): 1389-1400.[citado 2024 set. 03 ] Available from: https://doi.org/10.1097/HJH.0000000000003478