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Artigo de periodico
Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells (2020)
Zogbi, Camila; Oliveira, Nathalia C; Levy, Debora; Bydlowski, Sérgio Paulo ; Bassaneze, Vinicius; Neri, Elida A; Krieger, Jose Eduardo
Source: Scientific reports. Unidade: FM
Subjects: ANIMAIS, CÉLULAS ENDOTELIAIS, INTERLEUCINA 4, INTERLEUCINA 6, MACRÓFAGOS, MIOCÁRDIO, RATOS
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ABNT
ZOGBI, Camila et al. Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells. Scientific reports, v. 10, n. 1, 2020Tradução . . Disponível em: https://doi.org/10.1038/s41598-020-69413-0. Acesso em: 09 nov. 2024.
APA
Zogbi, C., Oliveira, N. C., Levy, D., Bydlowski, S. P., Bassaneze, V., Neri, E. A., & Krieger, J. E. (2020). Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells. Scientific reports, 10( 1). doi:10.1038/s41598-020-69413-0
NLM
Zogbi C, Oliveira NC, Levy D, Bydlowski SP, Bassaneze V, Neri EA, Krieger JE. Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells [Internet]. Scientific reports. 2020 ; 10( 1):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-020-69413-0
Vancouver
Zogbi C, Oliveira NC, Levy D, Bydlowski SP, Bassaneze V, Neri EA, Krieger JE. Beneficial effects of IL-4 and IL-6 on rat neonatal target cardiac cells [Internet]. Scientific reports. 2020 ; 10( 1):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1038/s41598-020-69413-0
Artigo de periodico
Integrated proteomics and metabolomics analysis reveals differential lipid metabolism in human umbilical vein endothelial cells under high and low shear stress (2019)
Venturini, Gabriela; Malagrino, Pamella Araujo; Padilha, Kallyandra; Tanaka, Leonardo Yuji; Laurindo, Francisco Rafael; Dariolli, Rafael; Carvalho, Valdemir Melechco; Cardozo, Karina Helena Morais; Krieger, José Eduardo ; Pereira, Alexandre da Costa
Source: American journal of physiology-cell physiology. Unidade: FM
Subjects: PROTEÔMICA, METABOLÔMICA, CÉLULAS ENDOTELIAIS, ARTÉRIAS UMBILICAIS, ESTRESSE, METABOLISMO DE GORDURA
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VENTURINI, Gabriela et al. Integrated proteomics and metabolomics analysis reveals differential lipid metabolism in human umbilical vein endothelial cells under high and low shear stress. American journal of physiology-cell physiology, v. 317, n. 2, p. C326-C338, 2019Tradução . . Disponível em: https://doi.org/10.1152/ajpcell.00128.2018. Acesso em: 09 nov. 2024.
APA
Venturini, G., Malagrino, P. A., Padilha, K., Tanaka, L. Y., Laurindo, F. R., Dariolli, R., et al. (2019). Integrated proteomics and metabolomics analysis reveals differential lipid metabolism in human umbilical vein endothelial cells under high and low shear stress. American journal of physiology-cell physiology, 317( 2), C326-C338. doi:10.1152/ajpcell.00128.2018
NLM
Venturini G, Malagrino PA, Padilha K, Tanaka LY, Laurindo FR, Dariolli R, Carvalho VM, Cardozo KHM, Krieger JE, Pereira A da C. Integrated proteomics and metabolomics analysis reveals differential lipid metabolism in human umbilical vein endothelial cells under high and low shear stress [Internet]. American journal of physiology-cell physiology. 2019 ; 317( 2): C326-C338.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpcell.00128.2018
Vancouver
Venturini G, Malagrino PA, Padilha K, Tanaka LY, Laurindo FR, Dariolli R, Carvalho VM, Cardozo KHM, Krieger JE, Pereira A da C. Integrated proteomics and metabolomics analysis reveals differential lipid metabolism in human umbilical vein endothelial cells under high and low shear stress [Internet]. American journal of physiology-cell physiology. 2019 ; 317( 2): C326-C338.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/ajpcell.00128.2018
Artigo de periodico
Beta-arrestin is critical for early shear stress-induced Akt/eNOS activation in human vascular endothelial cells (2017)
Carneiro, Ana Paula; Alaniz, Miriam Helena Fonseca; Dallan, Luís Alberto Oliveira; Miyakawa, Ayumi Aurea; Krieger, José Eduardo
Source: Biochemical and biophysical research communications. Unidade: FM
Subjects: PROTEÍNAS G, ESTRESSE OXIDATIVO, CÉLULAS ENDOTELIAIS, ÓXIDO NÍTRICO
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CARNEIRO, Ana Paula et al. Beta-arrestin is critical for early shear stress-induced Akt/eNOS activation in human vascular endothelial cells. Biochemical and biophysical research communications, v. 483, n. 01, p. 71-80, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2017.01.003. Acesso em: 09 nov. 2024.
APA
Carneiro, A. P., Alaniz, M. H. F., Dallan, L. A. O., Miyakawa, A. A., & Krieger, J. E. (2017). Beta-arrestin is critical for early shear stress-induced Akt/eNOS activation in human vascular endothelial cells. Biochemical and biophysical research communications, 483( 01), 71-80. doi:10.1016/j.bbrc.2017.01.003
NLM
Carneiro AP, Alaniz MHF, Dallan LAO, Miyakawa AA, Krieger JE. Beta-arrestin is critical for early shear stress-induced Akt/eNOS activation in human vascular endothelial cells [Internet]. Biochemical and biophysical research communications. 2017 ; 483( 01): 71-80.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2017.01.003
Vancouver
Carneiro AP, Alaniz MHF, Dallan LAO, Miyakawa AA, Krieger JE. Beta-arrestin is critical for early shear stress-induced Akt/eNOS activation in human vascular endothelial cells [Internet]. Biochemical and biophysical research communications. 2017 ; 483( 01): 71-80.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2017.01.003
Parte de monografia/livro
Fatores de crescimento vascular, células progenitoras e angiogênese (2016)
Gowdak, Luís Henrique Wolff; Krieger, José Eduardo
Source: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Unidade: FM
Subjects: FATORES DE CRESCIMENTO, TERAPIA BIOLÓGICA, CÉLULAS, CÉLULAS ENDOTELIAIS
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GOWDAK, Luís Henrique Wolff e KRIEGER, José Eduardo. Fatores de crescimento vascular, células progenitoras e angiogênese. Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Tradução . Rio de Janeiro: Atheneu, 2016. . . Acesso em: 09 nov. 2024.
APA
Gowdak, L. H. W., & Krieger, J. E. (2016). Fatores de crescimento vascular, células progenitoras e angiogênese. In Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Rio de Janeiro: Atheneu.
NLM
Gowdak LHW, Krieger JE. Fatores de crescimento vascular, células progenitoras e angiogênese. In: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Rio de Janeiro: Atheneu; 2016. [citado 2024 nov. 09 ]
Vancouver
Gowdak LHW, Krieger JE. Fatores de crescimento vascular, células progenitoras e angiogênese. In: Endotélio e doenças cardiovasculares : biologia vascular e síndromes clínicas. Rio de Janeiro: Atheneu; 2016. [citado 2024 nov. 09 ]
Artigo de periodico
AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177 (2013)
Barauna, Valério G.; Mantuan, Pâmela R.; Magalhães, Flávio C.; Campos, Luciene C. G.; Krieger, José E.
Source: Biochemical and Biophysical Research Communications. Unidade: FM
Subjects: ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS, RECEPTORES DE ANGIOTENSINA, CISALHAMENTO
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ABNT
BARAUNA, Valério G. et al. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177. Biochemical and Biophysical Research Communications, v. 441, n. 4, p. 713-719, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2013.10.108. Acesso em: 09 nov. 2024.
APA
Barauna, V. G., Mantuan, P. R., Magalhães, F. C., Campos, L. C. G., & Krieger, J. E. (2013). AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177. Biochemical and Biophysical Research Communications, 441( 4), 713-719. doi:10.1016/j.bbrc.2013.10.108
NLM
Barauna VG, Mantuan PR, Magalhães FC, Campos LCG, Krieger JE. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177 [Internet]. Biochemical and Biophysical Research Communications. 2013 ; 441( 4): 713-719.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2013.10.108
Vancouver
Barauna VG, Mantuan PR, Magalhães FC, Campos LCG, Krieger JE. AT1 receptor blocker potentiates shear-stress induced nitric oxide production via modulation of eNOS phosphorylation of residues Thr495 and Ser1177 [Internet]. Biochemical and Biophysical Research Communications. 2013 ; 441( 4): 713-719.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1016/j.bbrc.2013.10.108
Artigo de periodico
ACE as a Mechanosensor to Shear Stress Influences the Control of Its Own Regulation via Phosphorylation of Cytoplasmic Ser1270 (2011)
Barauna, Valerio Garrone; Campos, Luciene Cristina Gastalho; Miyakawa, Ayumi Aurea; Krieger, Jose Eduardo
Source: PLOS ONE. Unidade: FM
Subjects: WESTERN BLOTTING, PROLIFERAÇÃO CELULAR, SISTEMA RENINA-ANGIOTENSINA, MATRIZ EXTRACELULAR, CITOPLASMA, APOPTOSE, CÉLULAS ENDOTELIAIS
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ABNT
BARAUNA, Valerio Garrone et al. ACE as a Mechanosensor to Shear Stress Influences the Control of Its Own Regulation via Phosphorylation of Cytoplasmic Ser1270. PLOS ONE, v. 6, n. 81, 2011Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0022803. Acesso em: 09 nov. 2024.
APA
Barauna, V. G., Campos, L. C. G., Miyakawa, A. A., & Krieger, J. E. (2011). ACE as a Mechanosensor to Shear Stress Influences the Control of Its Own Regulation via Phosphorylation of Cytoplasmic Ser1270. PLOS ONE, 6( 81). doi:10.1371/journal.pone.0022803
NLM
Barauna VG, Campos LCG, Miyakawa AA, Krieger JE. ACE as a Mechanosensor to Shear Stress Influences the Control of Its Own Regulation via Phosphorylation of Cytoplasmic Ser1270 [Internet]. PLOS ONE. 2011 ; 6( 81):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0022803
Vancouver
Barauna VG, Campos LCG, Miyakawa AA, Krieger JE. ACE as a Mechanosensor to Shear Stress Influences the Control of Its Own Regulation via Phosphorylation of Cytoplasmic Ser1270 [Internet]. PLOS ONE. 2011 ; 6( 81):[citado 2024 nov. 09 ] Available from: https://doi.org/10.1371/journal.pone.0022803
Artigo de periodico
Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation (2009)
Dias, Rodrigo G.; Alves, Maira-Janieira N. N.; Pereira, Alexandre C.; Rondon, Maria Urbana P.B.; Santos, Marcelo R. dos; Krieger, José Eduardo; Krieger, Marta H.; Negrão, C. E.
Source: Physiological genomics. Unidades: FM, EEFE
Subjects: POLIMORFISMO, VASODILATAÇÃO, ÓXIDO NÍTRICO, EXERCÍCIO FÍSICO, CÉLULAS ENDOTELIAIS
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DIAS, Rodrigo G. et al. Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation. Physiological genomics, v. 37, n. 2, p. 99-107, 2009Tradução . . Disponível em: https://doi.org/10.1152/physiolgenomics.90368.2008. Acesso em: 09 nov. 2024.
APA
Dias, R. G., Alves, M. -J. N. N., Pereira, A. C., Rondon, M. U. P. B., Santos, M. R. dos, Krieger, J. E., et al. (2009). Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation. Physiological genomics, 37( 2), 99-107. doi:10.1152/physiolgenomics.90368.2008
NLM
Dias RG, Alves M-JNN, Pereira AC, Rondon MUPB, Santos MR dos, Krieger JE, Krieger MH, Negrão CE. Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation [Internet]. Physiological genomics. 2009 ; 37( 2): 99-107.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/physiolgenomics.90368.2008
Vancouver
Dias RG, Alves M-JNN, Pereira AC, Rondon MUPB, Santos MR dos, Krieger JE, Krieger MH, Negrão CE. Glu298Asp eNOS gene polymorphism causes attenuation in nonexercising muscle vasodilatation [Internet]. Physiological genomics. 2009 ; 37( 2): 99-107.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1152/physiolgenomics.90368.2008
Artigo de periodico
Nitric Oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress (2003)
Pertrini, C. M.; Miyakawa, A. A.; Laurindo, F. R. M.; Krieger, José Eduardo
Source: Brazilian Journal of Medical and Biological Research. Unidade: FM
Subjects: ANGIOTENSINAS, ÓXIDO NÍTRICO, CÉLULAS ENDOTELIAIS
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ABNT
PERTRINI, C. M. et al. Nitric Oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress. Brazilian Journal of Medical and Biological Research, v. 36, n. 9, p. 1175-78, 2003Tradução . . Disponível em: https://doi.org/10.1590/s0100-879x2003000900005. Acesso em: 09 nov. 2024.
APA
Pertrini, C. M., Miyakawa, A. A., Laurindo, F. R. M., & Krieger, J. E. (2003). Nitric Oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress. Brazilian Journal of Medical and Biological Research, 36( 9), 1175-78. doi:10.1590/s0100-879x2003000900005
NLM
Pertrini CM, Miyakawa AA, Laurindo FRM, Krieger JE. Nitric Oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress [Internet]. Brazilian Journal of Medical and Biological Research. 2003 ; 36( 9): 1175-78.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/s0100-879x2003000900005
Vancouver
Pertrini CM, Miyakawa AA, Laurindo FRM, Krieger JE. Nitric Oxide regulates angiotensin-I converting enzyme under static conditions but not under shear stress [Internet]. Brazilian Journal of Medical and Biological Research. 2003 ; 36( 9): 1175-78.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1590/s0100-879x2003000900005

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