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Artigo de periodico
Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats (2008)
Camara, Paula R. S.; Ferraz, Gerson J. N.; Franco-Penteado, Carla F.; Sbragia-Neto, Lourenco; Meirelles, Luciana R.; Teixeira, Simone A.; Muscará, Marcelo Nicolás ; Velloso, Licio A.; Antunes, Edson; Ferraz, Jose G. P.
Fonte: European Journal of Pharmacology. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMARA, Paula R. S. et al. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats. European Journal of Pharmacology, v. 589, n. 1-3, p. 245-250, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2008.05.004. Acesso em: 08 nov. 2024.
APA
Camara, P. R. S., Ferraz, G. J. N., Franco-Penteado, C. F., Sbragia-Neto, L., Meirelles, L. R., Teixeira, S. A., et al. (2008). Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats. European Journal of Pharmacology, 589( 1-3), 245-250. doi:10.1016/j.ejphar.2008.05.004
NLM
Camara PRS, Ferraz GJN, Franco-Penteado CF, Sbragia-Neto L, Meirelles LR, Teixeira SA, Muscará MN, Velloso LA, Antunes E, Ferraz JGP. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats [Internet]. European Journal of Pharmacology. 2008 ; 589( 1-3): 245-250.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2008.05.004
Vancouver
Camara PRS, Ferraz GJN, Franco-Penteado CF, Sbragia-Neto L, Meirelles LR, Teixeira SA, Muscará MN, Velloso LA, Antunes E, Ferraz JGP. Ablation of primary afferent neurons by neonatal capsaicin treatment reduces the susceptibility of the portal hypertensive gastric mucosa to ethanol-induced injury in cirrhotic rats [Internet]. European Journal of Pharmacology. 2008 ; 589( 1-3): 245-250.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2008.05.004
Artigo de periodico
In vivo blockade of Ca+2-dependent nitric oxide synthases impairs expressions of L-selectin and PECAM-1 (2008)
Hebeda, Cristina B.; Teixeira, Simone Aparecida; Muscará, Marcelo Nicolás ; Vinolo, Marco Antonio R.; Curi, Rui; Mello, Suzana Beatriz Veríssimo de; Farsky, Sandra Helena Poliselli
Fonte: Biochemical and Biophysical Research Communications. Unidades: ICB, FM, FCF
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HEBEDA, Cristina B. et al. In vivo blockade of Ca+2-dependent nitric oxide synthases impairs expressions of L-selectin and PECAM-1. Biochemical and Biophysical Research Communications, v. 377, n. 2, p. 694-698, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.bbrc.2008.10.055. Acesso em: 08 nov. 2024.
APA
Hebeda, C. B., Teixeira, S. A., Muscará, M. N., Vinolo, M. A. R., Curi, R., Mello, S. B. V. de, & Farsky, S. H. P. (2008). In vivo blockade of Ca+2-dependent nitric oxide synthases impairs expressions of L-selectin and PECAM-1. Biochemical and Biophysical Research Communications, 377( 2), 694-698. doi:10.1016/j.bbrc.2008.10.055
NLM
Hebeda CB, Teixeira SA, Muscará MN, Vinolo MAR, Curi R, Mello SBV de, Farsky SHP. In vivo blockade of Ca+2-dependent nitric oxide synthases impairs expressions of L-selectin and PECAM-1 [Internet]. Biochemical and Biophysical Research Communications. 2008 ; 377( 2): 694-698.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.bbrc.2008.10.055
Vancouver
Hebeda CB, Teixeira SA, Muscará MN, Vinolo MAR, Curi R, Mello SBV de, Farsky SHP. In vivo blockade of Ca+2-dependent nitric oxide synthases impairs expressions of L-selectin and PECAM-1 [Internet]. Biochemical and Biophysical Research Communications. 2008 ; 377( 2): 694-698.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1016/j.bbrc.2008.10.055
Artigo de periodico
An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption (2008)
Herrera, B. S.; Ohira, T.; Gao, L.; Omori, K.; Yang, R.; Zhu, M.; Muscará, Marcelo Nicolás ; Serhan, C. N.; Van Dyke, T. E.; Gyurko, R.
Fonte: British Journal of Pharmacology. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HERRERA, B. S. et al. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption. British Journal of Pharmacology, v. 155, n. 8, p. 1214-1223, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.367. Acesso em: 08 nov. 2024.
APA
Herrera, B. S., Ohira, T., Gao, L., Omori, K., Yang, R., Zhu, M., et al. (2008). An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption. British Journal of Pharmacology, 155( 8), 1214-1223. doi:10.1038/bjp.2008.367
NLM
Herrera BS, Ohira T, Gao L, Omori K, Yang R, Zhu M, Muscará MN, Serhan CN, Van Dyke TE, Gyurko R. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption [Internet]. British Journal of Pharmacology. 2008 ; 155( 8): 1214-1223.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1038/bjp.2008.367
Vancouver
Herrera BS, Ohira T, Gao L, Omori K, Yang R, Zhu M, Muscará MN, Serhan CN, Van Dyke TE, Gyurko R. An endogenous regulator of inflammation, resolving E1, modulates osteoclast differentiation and bone resorption [Internet]. British Journal of Pharmacology. 2008 ; 155( 8): 1214-1223.[citado 2024 nov. 08 ] Available from: https://doi.org/10.1038/bjp.2008.367

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