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Artigo de periodico
Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection (2014)
Gutierrez, Fredy R. S; Pavanelli, Wander R; Medina, Tiago S; Silva, Grace K; Mariano, Flávia S; Guedes, Paulo M. M.; Mineo, Tiago W. P.; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Fonte: Microbes and Infection. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI, OXIGENASE, LINFÓCITOS T
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ABNT
GUTIERREZ, Fredy R. S et al. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 1, p. 28-39, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2013.10.007. Acesso em: 27 set. 2024.
APA
Gutierrez, F. R. S., Pavanelli, W. R., Medina, T. S., Silva, G. K., Mariano, F. S., Guedes, P. M. M., et al. (2014). Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection. Microbes and Infection, 16( 1), 28-39. doi:10.1016/j.micinf.2013.10.007
NLM
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Vancouver
Gutierrez FRS, Pavanelli WR, Medina TS, Silva GK, Mariano FS, Guedes PMM, Mineo TWP, Rossi MA, Cunha F de Q, Silva JS da. Haeme oxygenase activity protects the host against excessive cardiac inflammation during experimental Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 1): 28-39.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.micinf.2013.10.007
Artigo de periodico
Role of dystrophin in acute Trypanosoma cruzi infection (2014)
Malvestio, Lygia M; Celes, Mara R. N; Milanezi, Cristiane Maria; Silva, João Santana da; Jelicks, Linda A; Tanowitz, Hebert B; Rossi, Marcos Antonio; Prado, Cibele Maria
Fonte: Microbes and Infection. Unidade: FMRP
Assuntos: DOENÇA DE CHAGAS, INFLAMAÇÃO (METABOLISMO), TRYPANOSOMA CRUZI, MIÓCITOS DE MÚSCULO LISO
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MALVESTIO, Lygia M et al. Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, v. 16, n. 9, p. 768-777, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.micinf.2014.07.010. Acesso em: 27 set. 2024.
APA
Malvestio, L. M., Celes, M. R. N., Milanezi, C. M., Silva, J. S. da, Jelicks, L. A., Tanowitz, H. B., et al. (2014). Role of dystrophin in acute Trypanosoma cruzi infection. Microbes and Infection, 16( 9), 768-777. doi:10.1016/j.micinf.2014.07.010
NLM
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Vancouver
Malvestio LM, Celes MRN, Milanezi CM, Silva JS da, Jelicks LA, Tanowitz HB, Rossi MA, Prado CM. Role of dystrophin in acute Trypanosoma cruzi infection [Internet]. Microbes and Infection. 2014 ; 16( 9): 768-777.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.micinf.2014.07.010
Artigo de periodico
Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis (2013)
Czaikoski, Paula Giselle; Nascimento, Daniele Carvalho; Sônego, Fabiane; Freitas, Andressa de; Turato, Walter Miguel; Carvalho, Michel A. de; Santos, Raquel Souza; Oliveira, Gisele Pena de; Samary, Cynthia dos Santos; Tefe-Silva, Cristiane; Alves Filho, José Carlos Farias; Ferreira, Sérgio Henrique; Rossi, Marcos Antonio; Rocco, Patricia Rieken Macedo; Spiller, Fernando; Cunha, Fernando de Queiroz
Fonte: Shock. Unidades: FCF, FMRP
Assuntos: OXIGENASE, INIBIDORES DE ENZIMAS, NEUTRÓFILOS, SEPSE, PNEUMONIA
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ABNT
CZAIKOSKI, Paula Giselle et al. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, v. 39, n. 4, p. 389-396, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e31828bbcf9. Acesso em: 27 set. 2024.
APA
Czaikoski, P. G., Nascimento, D. C., Sônego, F., Freitas, A. de, Turato, W. M., Carvalho, M. A. de, et al. (2013). Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis. Shock, 39( 4), 389-396. doi:10.1097/SHK.0b013e31828bbcf9
NLM
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 set. 27 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Vancouver
Czaikoski PG, Nascimento DC, Sônego F, Freitas A de, Turato WM, Carvalho MA de, Santos RS, Oliveira GP de, Samary C dos S, Tefe-Silva C, Alves Filho JCF, Ferreira SH, Rossi MA, Rocco PRM, Spiller F, Cunha F de Q. Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis [Internet]. Shock. 2013 ; 39( 4): 389-396.[citado 2024 set. 27 ] Available from: https://doi.org/10.1097/SHK.0b013e31828bbcf9
Artigo de periodico
5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection (2013)
Tristão, Fabrine Sales Massafera; Rocha, Fernanda Agostini; Moreira, Ana Paula; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio; Silva, João Santana da
Fonte: Infection and Immunity. Unidade: FMRP
Assunto: PARACOCCIDIOIDES BRASILIENSIS (IMUNOLOGIA)
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TRISTÃO, Fabrine Sales Massafera et al. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection. Infection and Immunity, v. 81, n. 4, p. 1256-1266, 2013Tradução . . Disponível em: https://doi.org/10.1128/IAI.01209-12. Acesso em: 27 set. 2024.
APA
Tristão, F. S. M., Rocha, F. A., Moreira, A. P., Cunha, F. de Q., Rossi, M. A., & Silva, J. S. da. (2013). 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection. Infection and Immunity, 81( 4), 1256-1266. doi:10.1128/IAI.01209-12
NLM
Tristão FSM, Rocha FA, Moreira AP, Cunha F de Q, Rossi MA, Silva JS da. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2013 ; 81( 4): 1256-1266.[citado 2024 set. 27 ] Available from: https://doi.org/10.1128/IAI.01209-12
Vancouver
Tristão FSM, Rocha FA, Moreira AP, Cunha F de Q, Rossi MA, Silva JS da. 5-lipoxygenase activity increases susceptibility to experimental paracoccidioides brasiliensis infection [Internet]. Infection and Immunity. 2013 ; 81( 4): 1256-1266.[citado 2024 set. 27 ] Available from: https://doi.org/10.1128/IAI.01209-12
Artigo de periodico
Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms (2011)
Oliveira, Marcela S.; Floriano, Elaine Medeiros; Mazin, Suleimy Cristina; Martinez, Edson Zangiacomi; Vicente, Walter Villela de Andrade; Peres, Luiz Cesar; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Fonte: Cardiovascular Pathology. Unidade: FMRP
Assuntos: ISQUEMIA MIOCÁRDICA, CARDIOPATIAS CONGÊNITAS, ESTRESSE OXIDATIVO
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ABNT
OLIVEIRA, Marcela S. et al. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, v. 20, n. 1, p. e43-e52, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.carpath.2010.01.012. Acesso em: 27 set. 2024.
APA
Oliveira, M. S., Floriano, E. M., Mazin, S. C., Martinez, E. Z., Vicente, W. V. de A., Peres, L. C., et al. (2011). Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms. Cardiovascular Pathology, 20( 1), e43-e52. doi:10.1016/j.carpath.2010.01.012
NLM
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Vancouver
Oliveira MS, Floriano EM, Mazin SC, Martinez EZ, Vicente WV de A, Peres LC, Rossi MA, Ramos SG. Ischemic myocardial injuries after cardiac malformation repair in infants may be associated with oxidative stress mechanisms [Internet]. Cardiovascular Pathology. 2011 ;20( 1): e43-e52.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.carpath.2010.01.012
Artigo de periodico
A scanning electron microscopy study of diseased root surfaces condotioned with EDTA gel plus cetavlon after scaling and root planning (2011)
Martins Junior, Walter; Rossi, Andiara de; Rached, Ricardo Samih Georges Abi; Rossi, Marcos Antonio
Fonte: Journal of Electron Microscopy. Unidades: FORP, FMRP
Assuntos: MICROSCOPIA ELETRÔNICA DE VARREDURA, RAIZ DENTÁRIA
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ABNT
MARTINS JUNIOR, Walter et al. A scanning electron microscopy study of diseased root surfaces condotioned with EDTA gel plus cetavlon after scaling and root planning. Journal of Electron Microscopy, v. 60, n. 2, p. 167-175, 2011Tradução . . Disponível em: https://doi.org/10.1093/jmicro/dfq083. Acesso em: 27 set. 2024.
APA
Martins Junior, W., Rossi, A. de, Rached, R. S. G. A., & Rossi, M. A. (2011). A scanning electron microscopy study of diseased root surfaces condotioned with EDTA gel plus cetavlon after scaling and root planning. Journal of Electron Microscopy, 60( 2), 167-175. doi:10.1093/jmicro/dfq083
NLM
Martins Junior W, Rossi A de, Rached RSGA, Rossi MA. A scanning electron microscopy study of diseased root surfaces condotioned with EDTA gel plus cetavlon after scaling and root planning [Internet]. Journal of Electron Microscopy. 2011 ; 60( 2): 167-175.[citado 2024 set. 27 ] Available from: https://doi.org/10.1093/jmicro/dfq083
Vancouver
Martins Junior W, Rossi A de, Rached RSGA, Rossi MA. A scanning electron microscopy study of diseased root surfaces condotioned with EDTA gel plus cetavlon after scaling and root planning [Internet]. Journal of Electron Microscopy. 2011 ; 60( 2): 167-175.[citado 2024 set. 27 ] Available from: https://doi.org/10.1093/jmicro/dfq083
Artigo de periodico
Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model (2011)
Mata, Karina M.; Prudente, Paula Stramandinoli; Rocha, Fábio S.; Prado, Cibele Maria; Floriano, Elaine Medeiros; Elias Júnior, Jorge; Rizzi, Elen; Gerlach, Raquel Fernanda; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Fonte: International Journal of Experimental Pathology. Unidades: FMRP, FORP
Assuntos: ANEURISMA AÓRTICO, METALOPROTEINASES
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ABNT
MATA, Karina M. et al. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model. International Journal of Experimental Pathology, v. 92, n. 1, p. 26-39, 2011Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2613.2010.00746.x. Acesso em: 27 set. 2024.
APA
Mata, K. M., Prudente, P. S., Rocha, F. S., Prado, C. M., Floriano, E. M., Elias Júnior, J., et al. (2011). Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model. International Journal of Experimental Pathology, 92( 1), 26-39. doi:10.1111/j.1365-2613.2010.00746.x
NLM
Mata KM, Prudente PS, Rocha FS, Prado CM, Floriano EM, Elias Júnior J, Rizzi E, Gerlach RF, Rossi MA, Ramos SG. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model [Internet]. International Journal of Experimental Pathology. 2011 ; 92( 1): 26-39.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2613.2010.00746.x
Vancouver
Mata KM, Prudente PS, Rocha FS, Prado CM, Floriano EM, Elias Júnior J, Rizzi E, Gerlach RF, Rossi MA, Ramos SG. Combining two potential causes of metalloproteinase secretion causes abnominal aortic aneurysms in rats: a new experimental model [Internet]. International Journal of Experimental Pathology. 2011 ; 92( 1): 26-39.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2613.2010.00746.x
Artigo de periodico
Oropouche virus experimental infection in the golden hamster (Mesocrisetus auratus) (2011)
Rodrigues, Alcir Humberto; Santos, Rodrigo Ivo Marques dos; Lopes, Maria Beatriz Sampaio; Arruda, Eurico; Arisi, Gabriel Maisonnave; Bernardes, Emerson Soares; Silva, Maria Lúcia da; Rossi, Marcos Antonio
Fonte: Virus Research. Unidade: FMRP
Assuntos: ARBOVÍRUS, INFECÇÃO EXPERIMENTAL ANIMAL
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ABNT
RODRIGUES, Alcir Humberto et al. Oropouche virus experimental infection in the golden hamster (Mesocrisetus auratus). Virus Research, v. 155, n. 1, p. 35-41, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.virusres.2010.08.009. Acesso em: 27 set. 2024.
APA
Rodrigues, A. H., Santos, R. I. M. dos, Lopes, M. B. S., Arruda, E., Arisi, G. M., Bernardes, E. S., et al. (2011). Oropouche virus experimental infection in the golden hamster (Mesocrisetus auratus). Virus Research, 155( 1), 35-41. doi:10.1016/j.virusres.2010.08.009
NLM
Rodrigues AH, Santos RIM dos, Lopes MBS, Arruda E, Arisi GM, Bernardes ES, Silva ML da, Rossi MA. Oropouche virus experimental infection in the golden hamster (Mesocrisetus auratus) [Internet]. Virus Research. 2011 ; 155( 1): 35-41.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.virusres.2010.08.009
Vancouver
Rodrigues AH, Santos RIM dos, Lopes MBS, Arruda E, Arisi GM, Bernardes ES, Silva ML da, Rossi MA. Oropouche virus experimental infection in the golden hamster (Mesocrisetus auratus) [Internet]. Virus Research. 2011 ; 155( 1): 35-41.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.virusres.2010.08.009
Artigo de periodico
Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension (2010)
Rizzi, Élen; Castro, Michele M.; Prado, Cibele M.; Silva, Carlos A.; Fazan Junior, Rubens; Rossi, Marcos Antonio; Tanus-Santos, José Eduardo; Gerlach, Raquel Fernanda
Fonte: Journal of Cardiac Failure. Unidades: FMRP, FORP
Assuntos: HIPERTROFIA VENTRICULAR ESQUERDA, HIPERTENSÃO
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ABNT
RIZZI, Élen et al. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, v. 16, n. 7, p. 599-608, 2010Tradução . . Acesso em: 27 set. 2024.
APA
Rizzi, É., Castro, M. M., Prado, C. M., Silva, C. A., Fazan Junior, R., Rossi, M. A., et al. (2010). Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure, 16( 7), 599-608.
NLM
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 set. 27 ]
Vancouver
Rizzi É, Castro MM, Prado CM, Silva CA, Fazan Junior R, Rossi MA, Tanus-Santos JE, Gerlach RF. Matrix metalloproteinase inhibition improves cardiac dysfunction and remodeling in 2-kidney, 1-Clip hypertension. Journal of Cardiac Failure. 2010 ; 16( 7): 599-608.[citado 2024 set. 27 ]
Artigo de periodico
Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle (2010)
Baruffi, Marcelo Dias; Stowell, Sean R.; Song, Shuh-Chyung; Arthur, Connie M.; Cho, Moonjae; Montes, Marlise Bonetti Agostinho; Rossi, Marcos Antonio; James, Judith A.; McEver, Rodger P.; Rodrigues, Lílian Cataldi; Cummings, Richard D.
Fonte: Glycobiology. Unidades: FCFRP, ICB, FMRP
Assuntos: LEUCÓCITOS, MÚSCULOS, ANTICORPOS MONOCLONAIS
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ABNT
BARUFFI, Marcelo Dias et al. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle. Glycobiology, v. 20, n. 5, p. 507-520, 2010Tradução . . Disponível em: https://doi.org/10.1093/glycob/cwp203. Acesso em: 27 set. 2024.
APA
Baruffi, M. D., Stowell, S. R., Song, S. -C., Arthur, C. M., Cho, M., Montes, M. B. A., et al. (2010). Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle. Glycobiology, 20( 5), 507-520. doi:10.1093/glycob/cwp203
NLM
Baruffi MD, Stowell SR, Song S-C, Arthur CM, Cho M, Montes MBA, Rossi MA, James JA, McEver RP, Rodrigues LC, Cummings RD. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle [Internet]. Glycobiology. 2010 ; 20( 5): 507-520.[citado 2024 set. 27 ] Available from: https://doi.org/10.1093/glycob/cwp203
Vancouver
Baruffi MD, Stowell SR, Song S-C, Arthur CM, Cho M, Montes MBA, Rossi MA, James JA, McEver RP, Rodrigues LC, Cummings RD. Differential expression of immunomodulatory galectin-1 in peripheral leukocytes and adult tissues and its cytosolic organization in striated muscle [Internet]. Glycobiology. 2010 ; 20( 5): 507-520.[citado 2024 set. 27 ] Available from: https://doi.org/10.1093/glycob/cwp203
Artigo de periodico
Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins (2010)
Celes, Mara Rúbia Nunes; Dueñas, Diego Torres; Prado, Cibele Maria; Campos, Érica Carolina; Moreira, Jorge Eduardo; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Fonte: Shock. Unidade: FMRP
Assuntos: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
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ABNT
CELES, Mara Rúbia Nunes et al. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock, v. 33, n. 3, p. 322-331, 2010Tradução . . Acesso em: 27 set. 2024.
APA
Celes, M. R. N., Dueñas, D. T., Prado, C. M., Campos, É. C., Moreira, J. E., Cunha, F. de Q., & Rossi, M. A. (2010). Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock, 33( 3), 322-331.
NLM
Celes MRN, Dueñas DT, Prado CM, Campos ÉC, Moreira JE, Cunha F de Q, Rossi MA. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock. 2010 ; 33( 3): 322-331.[citado 2024 set. 27 ]
Vancouver
Celes MRN, Dueñas DT, Prado CM, Campos ÉC, Moreira JE, Cunha F de Q, Rossi MA. Increased sarcolemmal permeability as an early event in experimental septic cardiomyopathy: a potential role for oxidative damage to lipids and proteins. Shock. 2010 ; 33( 3): 322-331.[citado 2024 set. 27 ]
Artigo de periodico
Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling (2010)
Castro, Michele Mazzaron de; Rizzi, Elen; Prado, Cibele Maria; Rossi, Marcos Antonio; Santos, José Eduardo Tanus dos; Gerlach, Raquel Fernanda
Fonte: Matrix Biology. Unidades: FMRP, FORP
Assuntos: HIPERTENSÃO, METALOPROTEINASES
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ABNT
CASTRO, Michele Mazzaron de et al. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling. Matrix Biology, v. 29, n. 3, p. 194-201, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.matbio.2009.11.005. Acesso em: 27 set. 2024.
APA
Castro, M. M. de, Rizzi, E., Prado, C. M., Rossi, M. A., Santos, J. E. T. dos, & Gerlach, R. F. (2010). Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling. Matrix Biology, 29( 3), 194-201. doi:10.1016/j.matbio.2009.11.005
NLM
Castro MM de, Rizzi E, Prado CM, Rossi MA, Santos JET dos, Gerlach RF. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling [Internet]. Matrix Biology. 2010 ; 29( 3): 194-201.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.matbio.2009.11.005
Vancouver
Castro MM de, Rizzi E, Prado CM, Rossi MA, Santos JET dos, Gerlach RF. Imbalance between matrix metalloproteinases and tissue inhibtor of metalloproteinases in hypertensive vascular remodeling [Internet]. Matrix Biology. 2010 ; 29( 3): 194-201.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.matbio.2009.11.005
Artigo de periodico
Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis (2010)
Celes, Mara Rúbia Nunes; Malvestio, Lygia M.; Torres-Dueñas, Diego; Blefari, Valdecir; Campos, Érica Carolina; Ramos, Simone Gusmão; Prado, Cibele Maria; Cunha, Fernando de Queiroz; Rossi, Marcos Antonio
Fonte: Laboratory Investigation. Unidade: FMRP
Assuntos: SARCOLEMA, SEPSE, MIOCARDIOPATIAS
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ABNT
CELES, Mara Rúbia Nunes et al. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, v. 90, n. 4, p. 531-542, 2010Tradução . . Disponível em: https://doi.org/10.1186/cc9157. Acesso em: 27 set. 2024.
APA
Celes, M. R. N., Malvestio, L. M., Torres-Dueñas, D., Blefari, V., Campos, É. C., Ramos, S. G., et al. (2010). Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis. Laboratory Investigation, 90( 4), 531-542. doi:10.1186/cc9157
NLM
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 set. 27 ] Available from: https://doi.org/10.1186/cc9157
Vancouver
Celes MRN, Malvestio LM, Torres-Dueñas D, Blefari V, Campos ÉC, Ramos SG, Prado CM, Cunha F de Q, Rossi MA. Disruption of sarcolemmal dystrophin and 'beta'-dystroglycan may be a potential mechanism for myocardial dysfunction in severe sepsis [Internet]. Laboratory Investigation. 2010 ; 90( 4): 531-542.[citado 2024 set. 27 ] Available from: https://doi.org/10.1186/cc9157
Artigo de periodico
IL-4 regulates susceptibility to intestinal inflammation in murine food allergy (2009)
Cardoso, Cristina Ribeiro; Provinciatto, Pauline Rossetti; Godoi, Dannielle Fernandes; Ferreira, Beatriz Rossetti ; Teixeira, Gerlinde; Rossi, Marcos Antonio; Cunha, Fernando de Queiroz; Silva, João Santana da
Fonte: American Journal of Physiology. Unidades: EERP, FMRP
Assuntos: HIPERSENSIBILIDADE ALIMENTAR, INTESTINOS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARDOSO, Cristina Ribeiro et al. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, v. 296, n. 3, p. G593-G600, 2009Tradução . . Disponível em: https://doi.org/10.1152/ajpgi.90431.2008. Acesso em: 27 set. 2024.
APA
Cardoso, C. R., Provinciatto, P. R., Godoi, D. F., Ferreira, B. R., Teixeira, G., Rossi, M. A., et al. (2009). IL-4 regulates susceptibility to intestinal inflammation in murine food allergy. American Journal of Physiology, 296( 3), G593-G600. doi:10.1152/ajpgi.90431.2008
NLM
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 set. 27 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Vancouver
Cardoso CR, Provinciatto PR, Godoi DF, Ferreira BR, Teixeira G, Rossi MA, Cunha F de Q, Silva JS da. IL-4 regulates susceptibility to intestinal inflammation in murine food allergy [Internet]. American Journal of Physiology. 2009 ; 296( 3): G593-G600.[citado 2024 set. 27 ] Available from: https://doi.org/10.1152/ajpgi.90431.2008
Artigo de periodico
Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress (2009)
Faria, Gisele; Cardoso, Cristina Ribeiro Barros; Larson, Roy Edward; Silva, João Santana da; Rossi, Marcos Antonio
Fonte: Toxicology and Applied Pharmacology. Unidades: FORP, FMRP
Assuntos: APOPTOSE, FIBROBLASTOS, RETÍCULO ENDOPLASMÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FARIA, Gisele et al. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress. Toxicology and Applied Pharmacology, v. 234, n. 2, p. 256-265, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.taap.2008.10.012. Acesso em: 27 set. 2024.
APA
Faria, G., Cardoso, C. R. B., Larson, R. E., Silva, J. S. da, & Rossi, M. A. (2009). Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress. Toxicology and Applied Pharmacology, 234( 2), 256-265. doi:10.1016/j.taap.2008.10.012
NLM
Faria G, Cardoso CRB, Larson RE, Silva JS da, Rossi MA. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress [Internet]. Toxicology and Applied Pharmacology. 2009 ; 234( 2): 256-265.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.taap.2008.10.012
Vancouver
Faria G, Cardoso CRB, Larson RE, Silva JS da, Rossi MA. Chlorhexidine-induced apoptosis or necrosis in L929 fibroblasts: a role for endoplasmic reticulum stress [Internet]. Toxicology and Applied Pharmacology. 2009 ; 234( 2): 256-265.[citado 2024 set. 27 ] Available from: https://doi.org/10.1016/j.taap.2008.10.012
Artigo de periodico
Micro-positron emission tomography in the evaluation of trypanosoma cruzi-induced heart disease: comparison with other modalities (2009)
Prado, Cibele Maria; Fine, Eugene J.; Koba, Wade; Zhao, Dazhi; Rossi, Marcos Antonio; Tanowitz, Herbert B.; Jelicks, Linda A.
Fonte: American Journal of Tropical Medicine and Hygiene. Unidade: FMRP
Assuntos: TRYPANOSOMA CRUZI (EVOLUÇÃO), DOENÇAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PRADO, Cibele Maria et al. Micro-positron emission tomography in the evaluation of trypanosoma cruzi-induced heart disease: comparison with other modalities. American Journal of Tropical Medicine and Hygiene, v. 81, n. 5, p. 900-905, 2009Tradução . . Acesso em: 27 set. 2024.
APA
Prado, C. M., Fine, E. J., Koba, W., Zhao, D., Rossi, M. A., Tanowitz, H. B., & Jelicks, L. A. (2009). Micro-positron emission tomography in the evaluation of trypanosoma cruzi-induced heart disease: comparison with other modalities. American Journal of Tropical Medicine and Hygiene, 81( 5), 900-905.
NLM
Prado CM, Fine EJ, Koba W, Zhao D, Rossi MA, Tanowitz HB, Jelicks LA. Micro-positron emission tomography in the evaluation of trypanosoma cruzi-induced heart disease: comparison with other modalities. American Journal of Tropical Medicine and Hygiene. 2009 ; 81( 5): 900-905.[citado 2024 set. 27 ]
Vancouver
Prado CM, Fine EJ, Koba W, Zhao D, Rossi MA, Tanowitz HB, Jelicks LA. Micro-positron emission tomography in the evaluation of trypanosoma cruzi-induced heart disease: comparison with other modalities. American Journal of Tropical Medicine and Hygiene. 2009 ; 81( 5): 900-905.[citado 2024 set. 27 ]
Artigo de periodico
In situzymography and immunolabeling in fixed and decalcified craniofacial tissues (2009)
Porto, Isabel M.; Rocha, Lenaldo Branco; Rossi, Marcos Antonio; Gerlach, Raquel Fernanda
Fonte: Journal of Histochemistry and Cytochemistry. Unidades: FMRP, FORP
Assuntos: METABOLISMO DE PROTEÍNA, MICROSCOPIA DE FLUORESCÊNCIA, IMUNOHISTOQUÍMICA, ENZIMAS HIDROLÍTICAS, DENTINA, OSSO E OSSOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PORTO, Isabel M. et al. In situzymography and immunolabeling in fixed and decalcified craniofacial tissues. Journal of Histochemistry and Cytochemistry, v. 57, n. 7, p. 615-622, 2009Tradução . . Acesso em: 27 set. 2024.
APA
Porto, I. M., Rocha, L. B., Rossi, M. A., & Gerlach, R. F. (2009). In situzymography and immunolabeling in fixed and decalcified craniofacial tissues. Journal of Histochemistry and Cytochemistry, 57( 7), 615-622.
NLM
Porto IM, Rocha LB, Rossi MA, Gerlach RF. In situzymography and immunolabeling in fixed and decalcified craniofacial tissues. Journal of Histochemistry and Cytochemistry. 2009 ; 57( 7): 615-622.[citado 2024 set. 27 ]
Vancouver
Porto IM, Rocha LB, Rossi MA, Gerlach RF. In situzymography and immunolabeling in fixed and decalcified craniofacial tissues. Journal of Histochemistry and Cytochemistry. 2009 ; 57( 7): 615-622.[citado 2024 set. 27 ]
Artigo de periodico
Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? (2009)
Beneli, Cristina Tonin; Barbosa, Priscila de Freitas; Floriano, Elaine Medeiros; Abreu, Mônica Azevedo de; Ramalho, Fernando Silva; Elias Júnior, Jorge; Rossi, Marcos Antonio; Ramos, Simone Gusmão
Fonte: International Journal of Experimental Pathology. Unidade: FMRP
Assuntos: ARTERIOSCLEROSE, ESTENOSE DA VALVA AÓRTICA, FIBRINÓLISE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BENELI, Cristina Tonin et al. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation?. International Journal of Experimental Pathology, v. 90, n. 3, p. 295-310, 2009Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2613.2008.00630.x. Acesso em: 27 set. 2024.
APA
Beneli, C. T., Barbosa, P. de F., Floriano, E. M., Abreu, M. A. de, Ramalho, F. S., Elias Júnior, J., et al. (2009). Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? International Journal of Experimental Pathology, 90( 3), 295-310. doi:10.1111/j.1365-2613.2008.00630.x
NLM
Beneli CT, Barbosa P de F, Floriano EM, Abreu MA de, Ramalho FS, Elias Júnior J, Rossi MA, Ramos SG. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? [Internet]. International Journal of Experimental Pathology. 2009 ; 90( 3): 295-310.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00630.x
Vancouver
Beneli CT, Barbosa P de F, Floriano EM, Abreu MA de, Ramalho FS, Elias Júnior J, Rossi MA, Ramos SG. Can eccentric arterial plaques alone cause flow stagnation points and favour thrombus incorporation? [Internet]. International Journal of Experimental Pathology. 2009 ; 90( 3): 295-310.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2613.2008.00630.x
Artigo de periodico-carta/editorial
The muscle-collagen ratio in left ventricular hypertrophy and aorta remodelling in hypertension.[Carta] (2008)
Rossi, Marcos Antonio
Fonte: Acta Physiologica. Unidade: FMRP
Assuntos: HIPERTROFIA VENTRICULAR ESQUERDA, HIPERTENSÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROSSI, Marcos Antonio. The muscle-collagen ratio in left ventricular hypertrophy and aorta remodelling in hypertension.[Carta]. Acta Physiologica. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 27 set. 2024. , 2008
APA
Rossi, M. A. (2008). The muscle-collagen ratio in left ventricular hypertrophy and aorta remodelling in hypertension.[Carta]. Acta Physiologica. Oxford: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo.
NLM
Rossi MA. The muscle-collagen ratio in left ventricular hypertrophy and aorta remodelling in hypertension.[Carta]. Acta Physiologica. 2008 ; 194 1-2.[citado 2024 set. 27 ]
Vancouver
Rossi MA. The muscle-collagen ratio in left ventricular hypertrophy and aorta remodelling in hypertension.[Carta]. Acta Physiologica. 2008 ; 194 1-2.[citado 2024 set. 27 ]
Artigo de periodico
Biofilm on the apical region of roots in primary teeth with vital and necrotic pulps with or without radiographically evident apical pathosis (2008)
Rocha, C. T.; Rossi, Marcos Antonio; Leonardo, M. R.; Rocha, Lenaldo Branco; Nelson Filho, Paulo; Silva, Léa Assed Bezerra da
Fonte: International Endodontic Journal. Unidades: FORP, FMRP
Assuntos: NECROSE DA POLPA DENTÁRIA, RADIOGRAFIA PERIAPICAL
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ROCHA, C. T. et al. Biofilm on the apical region of roots in primary teeth with vital and necrotic pulps with or without radiographically evident apical pathosis. International Endodontic Journal, v. 41, n. 8, p. 664-669, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1365-2591.2008.01411.x. Acesso em: 27 set. 2024.
APA
Rocha, C. T., Rossi, M. A., Leonardo, M. R., Rocha, L. B., Nelson Filho, P., & Silva, L. A. B. da. (2008). Biofilm on the apical region of roots in primary teeth with vital and necrotic pulps with or without radiographically evident apical pathosis. International Endodontic Journal, 41( 8), 664-669. doi:10.1111/j.1365-2591.2008.01411.x
NLM
Rocha CT, Rossi MA, Leonardo MR, Rocha LB, Nelson Filho P, Silva LAB da. Biofilm on the apical region of roots in primary teeth with vital and necrotic pulps with or without radiographically evident apical pathosis [Internet]. International Endodontic Journal. 2008 ; 41( 8): 664-669.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2591.2008.01411.x
Vancouver
Rocha CT, Rossi MA, Leonardo MR, Rocha LB, Nelson Filho P, Silva LAB da. Biofilm on the apical region of roots in primary teeth with vital and necrotic pulps with or without radiographically evident apical pathosis [Internet]. International Endodontic Journal. 2008 ; 41( 8): 664-669.[citado 2024 set. 27 ] Available from: https://doi.org/10.1111/j.1365-2591.2008.01411.x

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