Filtros : "Indexado no Current Abstracts" "CHAMMAS, ROGER" Removidos: "BERTI FILHO, EVONEO" "MPC digital" Limpar

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  • Source: Cancer Letters. Unidade: FM

    Subjects: METÁSTASE NEOPLÁSICA, MELANOMA ANIMAL, ANTICORPOS MONOCLONAIS

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    • ABNT

      AGUIAR, Rodrigo Barbosa de et al. Blocking FGF2 with a new specific monoclonal antibody impairs angiogenesis and experimental metastatic melanoma, suggesting a potential role in adjuvant settings. Cancer Letters, v. 371, n. 2, p. 151–160, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.canlet.2015.11.030. Acesso em: 02 out. 2024.
    • APA

      Aguiar, R. B. de, Parise, C. B., Souza, C. R. T., Braggion, C., Quintilio, W., Moro, A. M., et al. (2016). Blocking FGF2 with a new specific monoclonal antibody impairs angiogenesis and experimental metastatic melanoma, suggesting a potential role in adjuvant settings. Cancer Letters, 371( 2), 151–160. doi:10.1016/j.canlet.2015.11.030
    • NLM

      Aguiar RB de, Parise CB, Souza CRT, Braggion C, Quintilio W, Moro AM, Marques FLN, Buchpiguel CA, Chammas R, Moraes JZ de. Blocking FGF2 with a new specific monoclonal antibody impairs angiogenesis and experimental metastatic melanoma, suggesting a potential role in adjuvant settings [Internet]. Cancer Letters. 2016 ; 371( 2): 151–160.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2015.11.030
    • Vancouver

      Aguiar RB de, Parise CB, Souza CRT, Braggion C, Quintilio W, Moro AM, Marques FLN, Buchpiguel CA, Chammas R, Moraes JZ de. Blocking FGF2 with a new specific monoclonal antibody impairs angiogenesis and experimental metastatic melanoma, suggesting a potential role in adjuvant settings [Internet]. Cancer Letters. 2016 ; 371( 2): 151–160.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2015.11.030
  • Source: FEBS Letters. Unidade: FM

    Subjects: NEOPLASIAS CUTÂNEAS (TERAPIA), MELANOMA, METÁSTASE NEOPLÁSICA

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      SAITO, Renata de Freitas et al. Emerging targets for combination therapy in melanomas. FEBS Letters, v. 589, n. 22, p. 3438-3448, 2015Tradução . . Disponível em: http://onlinelibrary.wiley.com/doi/10.1016/j.febslet.2015.09.022/epdf. Acesso em: 02 out. 2024.
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      Saito, R. de F., Tortelli, T. C., Jacomassi, M. D. 'A., Otake, A. H., & Chammas, R. (2015). Emerging targets for combination therapy in melanomas. FEBS Letters, 589( 22), 3438-3448. doi:10.1016/j.febslet.2015.09.022
    • NLM

      Saito R de F, Tortelli TC, Jacomassi MD'A, Otake AH, Chammas R. Emerging targets for combination therapy in melanomas [Internet]. FEBS Letters. 2015 ; 589( 22): 3438-3448.[citado 2024 out. 02 ] Available from: http://onlinelibrary.wiley.com/doi/10.1016/j.febslet.2015.09.022/epdf
    • Vancouver

      Saito R de F, Tortelli TC, Jacomassi MD'A, Otake AH, Chammas R. Emerging targets for combination therapy in melanomas [Internet]. FEBS Letters. 2015 ; 589( 22): 3438-3448.[citado 2024 out. 02 ] Available from: http://onlinelibrary.wiley.com/doi/10.1016/j.febslet.2015.09.022/epdf
  • Source: Cancer Letters. Unidade: FM

    Subjects: NEOPLASIAS, SISTEMA RENINA-ANGIOTENSINA

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      COSTA, Patrícia L. N. da et al. The role of kinin receptors in cancer and therapeutic opportunities. Cancer Letters, v. 345, n. 1, p. 27-38, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.canlet.2013.12.009. Acesso em: 02 out. 2024.
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      Costa, P. L. N. da, Sirois, P., Tannock, I. F., & Chammas, R. (2014). The role of kinin receptors in cancer and therapeutic opportunities. Cancer Letters, 345( 1), 27-38. doi:10.1016/j.canlet.2013.12.009
    • NLM

      Costa PLN da, Sirois P, Tannock IF, Chammas R. The role of kinin receptors in cancer and therapeutic opportunities [Internet]. Cancer Letters. 2014 ; 345( 1): 27-38.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2013.12.009
    • Vancouver

      Costa PLN da, Sirois P, Tannock IF, Chammas R. The role of kinin receptors in cancer and therapeutic opportunities [Internet]. Cancer Letters. 2014 ; 345( 1): 27-38.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2013.12.009
  • Source: Histology and Histopathology. Unidade: FM

    Subjects: SCHISTOSOMA MANSONI (PATOLOGIA), ESQUISTOSSOMOSE MANSONI (PATOLOGIA), CAMUNDONGOS, DOENÇA CRÔNICA, DOENÇAS INFECCIOSAS (PARASITOLOGIA)

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      BRAND, Camila et al. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice. Histology and Histopathology, v. 27, n. 8, p. 1109-1120, 2012Tradução . . Disponível em: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf. Acesso em: 02 out. 2024.
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      Brand, C., Oliveira, F. L., Takiya, C. M., Palumbo Jr, A., Hsu, D. K., Liu, F. -T., et al. (2012). The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice. Histology and Histopathology, 27( 8), 1109-1120. Recuperado de http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
    • NLM

      Brand C, Oliveira FL, Takiya CM, Palumbo Jr A, Hsu DK, Liu F-T, Borojevic R, Chammas R, El-Cheikh MC. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice [Internet]. Histology and Histopathology. 2012 ; 27( 8): 1109-1120.[citado 2024 out. 02 ] Available from: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
    • Vancouver

      Brand C, Oliveira FL, Takiya CM, Palumbo Jr A, Hsu DK, Liu F-T, Borojevic R, Chammas R, El-Cheikh MC. The involvement of the spleen during chronic phase of Schistosoma mansoni infection in galectin-3-/- mice [Internet]. Histology and Histopathology. 2012 ; 27( 8): 1109-1120.[citado 2024 out. 02 ] Available from: http://www.hh.um.es/pdf/Vol_27/27_8/Brand-27-1109-1120-2012.pdf
  • Source: Journal of Dermatological Science. Unidade: FM

    Subjects: POLIMORFISMO, RNA, GENES, MELANONA, ESTUDO DE CASO, NEOPLASIAS CUTÂNEAS

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    • ABNT

      GONÇALVES, Fernanda T. et al. European ancestry and polymorphisms in DNA repair genes modify the risk of melanoma: A case–control study in a high UV index region in Brazil. Journal of Dermatological Science, v. 64, n. 1, p. 59-66, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.jdermsci.2011.06.003. Acesso em: 02 out. 2024.
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      Gonçalves, F. T., Eluf Neto, J., Francisco, G., Souza, S. P. de, Luiz, O. C., Festa Neto, C., et al. (2011). European ancestry and polymorphisms in DNA repair genes modify the risk of melanoma: A case–control study in a high UV index region in Brazil. Journal of Dermatological Science, 64( 1), 59-66. doi:10.1016/j.jdermsci.2011.06.003
    • NLM

      Gonçalves FT, Eluf Neto J, Francisco G, Souza SP de, Luiz OC, Festa Neto C, Sanches JA, Chammas R, Gattas GJF. European ancestry and polymorphisms in DNA repair genes modify the risk of melanoma: A case–control study in a high UV index region in Brazil [Internet]. Journal of Dermatological Science. 2011 ; 64( 1): 59-66.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.jdermsci.2011.06.003
    • Vancouver

      Gonçalves FT, Eluf Neto J, Francisco G, Souza SP de, Luiz OC, Festa Neto C, Sanches JA, Chammas R, Gattas GJF. European ancestry and polymorphisms in DNA repair genes modify the risk of melanoma: A case–control study in a high UV index region in Brazil [Internet]. Journal of Dermatological Science. 2011 ; 64( 1): 59-66.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.jdermsci.2011.06.003
  • Source: PLOS ONE. Unidades: FMRP, FCF, FM

    Subjects: MELANOMA, BIOINFORMÁTICA

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    • ABNT

      SOUSA, Josane de Freitas et al. Novel primate-specific genes, RMEL 1, 2 and 3, with highly restricted expression in melanoma, assessed by new data mining tool. PLOS ONE, v. 5, n. 10, p. e13510-1- e13510-13, 2010Tradução . . Disponível em: https://repositorio.usp.br/directbitstream/a794b396-a376-4b37-8aa1-64dbc51541aa/002132082.pdf. Acesso em: 02 out. 2024.
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      Sousa, J. de F., Torrieri, R., Silva, R. R., Pereira, C. G., Valente, V., Torrieri, É., et al. (2010). Novel primate-specific genes, RMEL 1, 2 and 3, with highly restricted expression in melanoma, assessed by new data mining tool. PLOS ONE, 5( 10), e13510-1- e13510-13. Recuperado de https://repositorio.usp.br/directbitstream/a794b396-a376-4b37-8aa1-64dbc51541aa/002132082.pdf
    • NLM

      Sousa J de F, Torrieri R, Silva RR, Pereira CG, Valente V, Torrieri É, Peronni KC, Martins W, Muto N, Francisco G, Brohem CA, Carlotti Júnior CG, Maria-Engler SS, Chammas R, Espreafico EM. Novel primate-specific genes, RMEL 1, 2 and 3, with highly restricted expression in melanoma, assessed by new data mining tool [Internet]. PLOS ONE. 2010 ; 5( 10): e13510-1- e13510-13.[citado 2024 out. 02 ] Available from: https://repositorio.usp.br/directbitstream/a794b396-a376-4b37-8aa1-64dbc51541aa/002132082.pdf
    • Vancouver

      Sousa J de F, Torrieri R, Silva RR, Pereira CG, Valente V, Torrieri É, Peronni KC, Martins W, Muto N, Francisco G, Brohem CA, Carlotti Júnior CG, Maria-Engler SS, Chammas R, Espreafico EM. Novel primate-specific genes, RMEL 1, 2 and 3, with highly restricted expression in melanoma, assessed by new data mining tool [Internet]. PLOS ONE. 2010 ; 5( 10): e13510-1- e13510-13.[citado 2024 out. 02 ] Available from: https://repositorio.usp.br/directbitstream/a794b396-a376-4b37-8aa1-64dbc51541aa/002132082.pdf
  • Source: Proteome Science. Unidades: FMRP, FM

    Subjects: NEUTRÓFILOS, PROTEÍNAS

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      TOMAZELLA, Gisele Guiçardi et al. Proteomic analysis of total cellular proteins of human neutrophils. Proteome Science, v. 7, 2009Tradução . . Disponível em: https://doi.org/10.1186/1477-5956-7-32. Acesso em: 02 out. 2024.
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      Tomazella, G. G., Silva, I. da, Laure, H. J., Rosa, J. C., Chammas, R., Wiker, H. G., et al. (2009). Proteomic analysis of total cellular proteins of human neutrophils. Proteome Science, 7. doi:10.1186/1477-5956-7-32
    • NLM

      Tomazella GG, Silva I da, Laure HJ, Rosa JC, Chammas R, Wiker HG, Souza GA de, Greene LJ. Proteomic analysis of total cellular proteins of human neutrophils [Internet]. Proteome Science. 2009 ; 7[citado 2024 out. 02 ] Available from: https://doi.org/10.1186/1477-5956-7-32
    • Vancouver

      Tomazella GG, Silva I da, Laure HJ, Rosa JC, Chammas R, Wiker HG, Souza GA de, Greene LJ. Proteomic analysis of total cellular proteins of human neutrophils [Internet]. Proteome Science. 2009 ; 7[citado 2024 out. 02 ] Available from: https://doi.org/10.1186/1477-5956-7-32
  • Source: Cancer Letters. Unidades: FM, ICB

    Subjects: MELANOMA, CULTURA DE CÉLULAS

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      LIMA, Luize G. et al. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner. Cancer Letters, v. 283, n. 2, p. 168-175, 2009Tradução . . Disponível em: https://doi.org/10.1016/j.canlet.2009.03.041. Acesso em: 02 out. 2024.
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      Lima, L. G., Chammas, R., Monteiro, R. Q., Moreira, M. E. C., & Barcinski, M. A. (2009). Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner. Cancer Letters, 283( 2), 168-175. doi:10.1016/j.canlet.2009.03.041
    • NLM

      Lima LG, Chammas R, Monteiro RQ, Moreira MEC, Barcinski MA. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner [Internet]. Cancer Letters. 2009 ; 283( 2): 168-175.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2009.03.041
    • Vancouver

      Lima LG, Chammas R, Monteiro RQ, Moreira MEC, Barcinski MA. Tumor-derived microvesicles modulate the establishment of metastatic melanoma in a phosphatidylserine-dependent manner [Internet]. Cancer Letters. 2009 ; 283( 2): 168-175.[citado 2024 out. 02 ] Available from: https://doi.org/10.1016/j.canlet.2009.03.041

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