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Artigo de periodico
The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes (2021)
Santos, Renato Elias Rodrigues de Souza ; Silva Júnior, Waldir P. da; Harrison, Simone; Skaar, Eric P.; Chazin, Walter J.; Silva Neto, José Freire da
Source: Infection and Immunity. Unidade: FMRP
Subjects: BIOFILMES, COMPLEXO ANTÍGENO-ANTICORPO, LEUCÓCITOS, NEUTRÓFILOS, VIRULÊNCIA, ZINCO
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SANTOS, Renato Elias Rodrigues de Souza et al. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, v. 89, n. 11, 2021Tradução . . Disponível em: https://doi.org/10.1128/IAI.00311-21. Acesso em: 08 jul. 2024.
APA
Santos, R. E. R. de S., Silva Júnior, W. P. da, Harrison, S., Skaar, E. P., Chazin, W. J., & Silva Neto, J. F. da. (2021). The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes. Infection and Immunity, 89( 11). doi:10.1128/IAI.00311-21
NLM
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1128/IAI.00311-21
Vancouver
Santos RER de S, Silva Júnior WP da, Harrison S, Skaar EP, Chazin WJ, Silva Neto JF da. The zinc transporter ZnuABC is critical for the virulence of Chromobacterium violaceum and contributes to diverse zinc-dependent physiological processes [Internet]. Infection and Immunity. 2021 ; 89( 11):[citado 2024 jul. 08 ] Available from: https://doi.org/10.1128/IAI.00311-21
Artigo de periodico
Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation (2018)
Kanashiro, Alexandre; Franchin, Marcelo; Bassi, Gabriel Shimizu; Santana, Dênis Augusto Reis; Cunha, Thiago Mattar; Cunha, Fernando de Queiroz; Ulloa, Luis; Rodrigues, Gerson Jonathan
Source: Fundamental and Clinical Pharmacology. Unidade: FMRP
Subjects: ARTRITE, RECEPTORES ANDROGÊNICOS, SISTEMA NERVOSO PARASSIMPÁTICO, NEUTRÓFILOS
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KANASHIRO, Alexandre et al. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, v. 32, n. 2, p. 155-162, 2018Tradução . . Disponível em: https://doi.org/10.1111/fcp.12338. Acesso em: 08 jul. 2024.
APA
Kanashiro, A., Franchin, M., Bassi, G. S., Santana, D. A. R., Cunha, T. M., Cunha, F. de Q., et al. (2018). Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation. Fundamental and Clinical Pharmacology, 32( 2), 155-162. doi:10.1111/fcp.12338
NLM
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/fcp.12338
Vancouver
Kanashiro A, Franchin M, Bassi GS, Santana DAR, Cunha TM, Cunha F de Q, Ulloa L, Rodrigues GJ. Inhibition of spinal p38 MAPK prevents articular neutrophil infiltration in experimental arthritis via sympathetic activation [Internet]. Fundamental and Clinical Pharmacology. 2018 ; 32( 2): 155-162.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1111/fcp.12338
Artigo de periodico
Analysis of chemokines and reactive oxygen species formation by rat and human neutrophils induced by microcystin-LA, -YR and -LR (2008)
Kujbida, Paula; Hatanaka, Elaine; Campa, Ana ; Curi, Rui; Farsky, Sandra Helena Poliselli ; Pinto, Ernani
Source: Toxicon. Unidades: FCF, ICB
Subjects: NEUTRÓFILOS, BIOQUÍMICA
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KUJBIDA, Paula et al. Analysis of chemokines and reactive oxygen species formation by rat and human neutrophils induced by microcystin-LA, -YR and -LR. Toxicon, v. 51, n. 7, p. 1274-1280, 2008Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2008.02.013. Acesso em: 08 jul. 2024.
APA
Kujbida, P., Hatanaka, E., Campa, A., Curi, R., Farsky, S. H. P., & Pinto, E. (2008). Analysis of chemokines and reactive oxygen species formation by rat and human neutrophils induced by microcystin-LA, -YR and -LR. Toxicon, 51( 7), 1274-1280. doi:10.1016/j.toxicon.2008.02.013
NLM
Kujbida P, Hatanaka E, Campa A, Curi R, Farsky SHP, Pinto E. Analysis of chemokines and reactive oxygen species formation by rat and human neutrophils induced by microcystin-LA, -YR and -LR [Internet]. Toxicon. 2008 ;51( 7): 1274-1280.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.toxicon.2008.02.013
Vancouver
Kujbida P, Hatanaka E, Campa A, Curi R, Farsky SHP, Pinto E. Analysis of chemokines and reactive oxygen species formation by rat and human neutrophils induced by microcystin-LA, -YR and -LR [Internet]. Toxicon. 2008 ;51( 7): 1274-1280.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.toxicon.2008.02.013
Artigo de periodico
Interaction between serum amyloid A and leukocytes - A possible role in the progression of vascular complications in diabetes (2007)
Hatanaka, Elaine; Monteagudo, Patrícia Teófilo; Marrocos, Mauro Sérgio Martins; Campa, Ana
Source: Immunology Letters. Unidade: FCF
Subjects: DIABETES MELLITUS, BIOQUÍMICA CLÍNICA, CITOCINAS, NEUTRÓFILOS, MONÓCITOS
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HATANAKA, Elaine et al. Interaction between serum amyloid A and leukocytes - A possible role in the progression of vascular complications in diabetes. Immunology Letters, v. 108, n. 2, p. 160-166, 2007Tradução . . Disponível em: https://doi.org/10.1016/j.imlet.2006.12.005. Acesso em: 08 jul. 2024.
APA
Hatanaka, E., Monteagudo, P. T., Marrocos, M. S. M., & Campa, A. (2007). Interaction between serum amyloid A and leukocytes - A possible role in the progression of vascular complications in diabetes. Immunology Letters, 108( 2), 160-166. doi:10.1016/j.imlet.2006.12.005
NLM
Hatanaka E, Monteagudo PT, Marrocos MSM, Campa A. Interaction between serum amyloid A and leukocytes - A possible role in the progression of vascular complications in diabetes [Internet]. Immunology Letters. 2007 ; 108( 2): 160-166.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.imlet.2006.12.005
Vancouver
Hatanaka E, Monteagudo PT, Marrocos MSM, Campa A. Interaction between serum amyloid A and leukocytes - A possible role in the progression of vascular complications in diabetes [Internet]. Immunology Letters. 2007 ; 108( 2): 160-166.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.imlet.2006.12.005
Artigo de periodico
Endogenous glucocorticoids control neutrophil mobilization from bone marrow to blood and tissues in non-inflammatory conditions (2007)
Cavalcanti, Danielle Maia de Holanda; Lotufo, Celina Monteiro da Cruz; Borelli, Primavera ; Ferreira, Zulma S ; Markus, Regina P ; Farsky, Sandra Helena Poliselli
Source: British Journal of Pharmacology. Unidades: FCF, IB
Subjects: ADRENALECTOMIA ANIMAL, NEUTRÓFILOS, CÉLULAS ENDOTELIAIS
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ABNT
CAVALCANTI, Danielle Maia de Holanda et al. Endogenous glucocorticoids control neutrophil mobilization from bone marrow to blood and tissues in non-inflammatory conditions. British Journal of Pharmacology, v. 152, n. 8, p. 1291-1300, 2007Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0707512. Acesso em: 08 jul. 2024.
APA
Cavalcanti, D. M. de H., Lotufo, C. M. da C., Borelli, P., Ferreira, Z. S., Markus, R. P., & Farsky, S. H. P. (2007). Endogenous glucocorticoids control neutrophil mobilization from bone marrow to blood and tissues in non-inflammatory conditions. British Journal of Pharmacology, 152( 8), 1291-1300. doi:10.1038/sj.bjp.0707512
NLM
Cavalcanti DM de H, Lotufo CM da C, Borelli P, Ferreira ZS, Markus RP, Farsky SHP. Endogenous glucocorticoids control neutrophil mobilization from bone marrow to blood and tissues in non-inflammatory conditions [Internet]. British Journal of Pharmacology. 2007 ; 152( 8): 1291-1300.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1038/sj.bjp.0707512
Vancouver
Cavalcanti DM de H, Lotufo CM da C, Borelli P, Ferreira ZS, Markus RP, Farsky SHP. Endogenous glucocorticoids control neutrophil mobilization from bone marrow to blood and tissues in non-inflammatory conditions [Internet]. British Journal of Pharmacology. 2007 ; 152( 8): 1291-1300.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1038/sj.bjp.0707512
Artigo de periodico
Effect of in vivo phenol or hydroquinone exposure on events related to neutrophil delivery during an inflammatory response (2006)
Macedo, Sandra Manoela Dias; Lourenço, Emerson Luiz Botelho; Borelli, Primavera ; Fock, Ricardo Ambrósio ; Ferreira Jr., J. M.; Farsky, Sandra Helena Poliselli
Source: Toxicology. Unidade: FCF
Subjects: INFLAMAÇÃO, NEUTRÓFILOS, LEUCÓCITOS
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ABNT
MACEDO, Sandra Manoela Dias et al. Effect of in vivo phenol or hydroquinone exposure on events related to neutrophil delivery during an inflammatory response. Toxicology, v. 220, n. 2-3, p. 126-135, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.tox.2005.12.008. Acesso em: 08 jul. 2024.
APA
Macedo, S. M. D., Lourenço, E. L. B., Borelli, P., Fock, R. A., Ferreira Jr., J. M., & Farsky, S. H. P. (2006). Effect of in vivo phenol or hydroquinone exposure on events related to neutrophil delivery during an inflammatory response. Toxicology, 220( 2-3), 126-135. doi:10.1016/j.tox.2005.12.008
NLM
Macedo SMD, Lourenço ELB, Borelli P, Fock RA, Ferreira Jr. JM, Farsky SHP. Effect of in vivo phenol or hydroquinone exposure on events related to neutrophil delivery during an inflammatory response [Internet]. Toxicology. 2006 ; 220( 2-3): 126-135.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.tox.2005.12.008
Vancouver
Macedo SMD, Lourenço ELB, Borelli P, Fock RA, Ferreira Jr. JM, Farsky SHP. Effect of in vivo phenol or hydroquinone exposure on events related to neutrophil delivery during an inflammatory response [Internet]. Toxicology. 2006 ; 220( 2-3): 126-135.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.tox.2005.12.008
Artigo de periodico
Melatonin effect on endothelial cells reduces vascular permeability increase induced by leukotriene 'B IND. 4' (2006)
Lotufo, Celina Monteiro da Cruz; Yamashita, Cintia Etsuko; Farsky, Sandra Helena Poliselli ; Markus, Regina P
Source: European Journal of Pharmacology. Unidades: FCF, IB
Subjects: MELATONINA (EFEITOS), INFLAMAÇÃO, CÉLULAS ENDOTELIAIS, NEUTRÓFILOS
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ABNT
LOTUFO, Celina Monteiro da Cruz et al. Melatonin effect on endothelial cells reduces vascular permeability increase induced by leukotriene 'B IND. 4'. European Journal of Pharmacology, v. 534, n. 1-3, p. 258-263, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2006.01.050. Acesso em: 08 jul. 2024.
APA
Lotufo, C. M. da C., Yamashita, C. E., Farsky, S. H. P., & Markus, R. P. (2006). Melatonin effect on endothelial cells reduces vascular permeability increase induced by leukotriene 'B IND. 4'. European Journal of Pharmacology, 534( 1-3), 258-263. doi:10.1016/j.ejphar.2006.01.050
NLM
Lotufo CM da C, Yamashita CE, Farsky SHP, Markus RP. Melatonin effect on endothelial cells reduces vascular permeability increase induced by leukotriene 'B IND. 4' [Internet]. European Journal of Pharmacology. 2006 ; 534( 1-3): 258-263.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2006.01.050
Vancouver
Lotufo CM da C, Yamashita CE, Farsky SHP, Markus RP. Melatonin effect on endothelial cells reduces vascular permeability increase induced by leukotriene 'B IND. 4' [Internet]. European Journal of Pharmacology. 2006 ; 534( 1-3): 258-263.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2006.01.050
Artigo de periodico
Adrenal deficiency alters mechanisms of neutrophil mobilization (2006)
Cavalcanti, Danielle Maia de Holanda; Lotufo, Celina Monteiro da Cruz; Borelli, Primavera ; Tavassi, Ana Maria C.; Pereira, Agostinho Luiz Maia; Markus, Regina P ; Farsky, Sandra Helena Poliselli
Source: Molecular and Cellular Endocrinology. Unidades: FCF, IB
Subjects: IMUNOLOGIA CELULAR, NEUTRÓFILOS, HORMÔNIOS, INTERAÇÃO CELULAR
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ABNT
CAVALCANTI, Danielle Maia de Holanda et al. Adrenal deficiency alters mechanisms of neutrophil mobilization. Molecular and Cellular Endocrinology, v. 249, n. 1-2, p. 32-39, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2006.01.007. Acesso em: 08 jul. 2024.
APA
Cavalcanti, D. M. de H., Lotufo, C. M. da C., Borelli, P., Tavassi, A. M. C., Pereira, A. L. M., Markus, R. P., & Farsky, S. H. P. (2006). Adrenal deficiency alters mechanisms of neutrophil mobilization. Molecular and Cellular Endocrinology, 249( 1-2), 32-39. doi:10.1016/j.mce.2006.01.007
NLM
Cavalcanti DM de H, Lotufo CM da C, Borelli P, Tavassi AMC, Pereira ALM, Markus RP, Farsky SHP. Adrenal deficiency alters mechanisms of neutrophil mobilization [Internet]. Molecular and Cellular Endocrinology. 2006 ; 249( 1-2): 32-39.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.mce.2006.01.007
Vancouver
Cavalcanti DM de H, Lotufo CM da C, Borelli P, Tavassi AMC, Pereira ALM, Markus RP, Farsky SHP. Adrenal deficiency alters mechanisms of neutrophil mobilization [Internet]. Molecular and Cellular Endocrinology. 2006 ; 249( 1-2): 32-39.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.mce.2006.01.007
Artigo de periodico
Hyperresponsiveness of neutrophils from gp 91(phox) deficient patients to lipopolysaccharide and serum amyloid A (2004)
Hatanaka, Elaine; Costa-Carvalho, Beatriz Tavares; Condino-Neto, Antonio; Campa, Ana
Source: Immunology Letters. Unidade: FCF
Subjects: NEUTRÓFILOS, INTERLEUCINA 8, MONÓCITOS, CITOCINAS
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ABNT
HATANAKA, Elaine et al. Hyperresponsiveness of neutrophils from gp 91(phox) deficient patients to lipopolysaccharide and serum amyloid A. Immunology Letters, v. 94, n. 1-2, p. 43-46, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.imlet.2004.04.016. Acesso em: 08 jul. 2024.
APA
Hatanaka, E., Costa-Carvalho, B. T., Condino-Neto, A., & Campa, A. (2004). Hyperresponsiveness of neutrophils from gp 91(phox) deficient patients to lipopolysaccharide and serum amyloid A. Immunology Letters, 94( 1-2), 43-46. doi:10.1016/j.imlet.2004.04.016
NLM
Hatanaka E, Costa-Carvalho BT, Condino-Neto A, Campa A. Hyperresponsiveness of neutrophils from gp 91(phox) deficient patients to lipopolysaccharide and serum amyloid A [Internet]. Immunology Letters. 2004 ; 94( 1-2): 43-46.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.imlet.2004.04.016
Vancouver
Hatanaka E, Costa-Carvalho BT, Condino-Neto A, Campa A. Hyperresponsiveness of neutrophils from gp 91(phox) deficient patients to lipopolysaccharide and serum amyloid A [Internet]. Immunology Letters. 2004 ; 94( 1-2): 43-46.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.imlet.2004.04.016
Artigo de periodico
Neutrophils as a specific target for melatonin and kynuramines: effects on cytokine release (2004)
Silva, Sueli de Oliveira; Rodrigues, Maria Rita; Ximenes, Valdecir Farias; Bueno-da-Silva, A. E. B.; Amarante-Mendes, João Gustavo Pessini ; Campa, Ana
Source: Journal of Neuroimmunology. Unidades: FCF, ICB
Subjects: NEUTRÓFILOS, CITOCINAS, MONÓCITOS
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ABNT
SILVA, Sueli de Oliveira et al. Neutrophils as a specific target for melatonin and kynuramines: effects on cytokine release. Journal of Neuroimmunology, v. 156, n. 1-2, p. 146-152, 2004Tradução . . Disponível em: https://doi.org/10.1016/j.jneuroim.2004.07.015. Acesso em: 08 jul. 2024.
APA
Silva, S. de O., Rodrigues, M. R., Ximenes, V. F., Bueno-da-Silva, A. E. B., Amarante-Mendes, J. G. P., & Campa, A. (2004). Neutrophils as a specific target for melatonin and kynuramines: effects on cytokine release. Journal of Neuroimmunology, 156( 1-2), 146-152. doi:10.1016/j.jneuroim.2004.07.015
NLM
Silva S de O, Rodrigues MR, Ximenes VF, Bueno-da-Silva AEB, Amarante-Mendes JGP, Campa A. Neutrophils as a specific target for melatonin and kynuramines: effects on cytokine release [Internet]. Journal of Neuroimmunology. 2004 ; 156( 1-2): 146-152.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.jneuroim.2004.07.015
Vancouver
Silva S de O, Rodrigues MR, Ximenes VF, Bueno-da-Silva AEB, Amarante-Mendes JGP, Campa A. Neutrophils as a specific target for melatonin and kynuramines: effects on cytokine release [Internet]. Journal of Neuroimmunology. 2004 ; 156( 1-2): 146-152.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.jneuroim.2004.07.015
Artigo de periodico
The acute phase protein serum amyloid A primes neutrophils (2003)
Hatanaka, Elaine; Ribeiro, Fernanda Pereira; Campa, Ana
Source: FEMS Immunology and Medical Microbiology. Unidade: FCF
Subjects: BIOQUÍMICA, NEUTRÓFILOS
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ABNT
HATANAKA, Elaine e RIBEIRO, Fernanda Pereira e CAMPA, Ana. The acute phase protein serum amyloid A primes neutrophils. FEMS Immunology and Medical Microbiology, v. 38, n. 1, p. 81-84, 2003Tradução . . Disponível em: https://doi.org/10.1016/s0928-8244(03)00112-3. Acesso em: 08 jul. 2024.
APA
Hatanaka, E., Ribeiro, F. P., & Campa, A. (2003). The acute phase protein serum amyloid A primes neutrophils. FEMS Immunology and Medical Microbiology, 38( 1), 81-84. doi:10.1016/s0928-8244(03)00112-3
NLM
Hatanaka E, Ribeiro FP, Campa A. The acute phase protein serum amyloid A primes neutrophils [Internet]. FEMS Immunology and Medical Microbiology. 2003 ; 38( 1): 81-84.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/s0928-8244(03)00112-3
Vancouver
Hatanaka E, Ribeiro FP, Campa A. The acute phase protein serum amyloid A primes neutrophils [Internet]. FEMS Immunology and Medical Microbiology. 2003 ; 38( 1): 81-84.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/s0928-8244(03)00112-3
Artigo de periodico
Effects of acute and long-term diazepam administrations on neutrophil activity: a flow cytometric study (2003)
Silva, Fábio Ribeiro da; Lazzarini, Ricardo; Sá-Rocha, Luiz Carlos de ; Morgulis, Maria Silvia Furquim de Azevedo; Massoco, Cristina de Oliveira; Palermo-Neto, João
Source: European Journal of Pharmacology. Unidade: FMVZ
Subjects: CITOMETRIA DE FLUXO, NEUTRÓFILOS, RECEPTORES DE BENZODIAZEPINA, TOLERÂNCIA (DESENVOLVIMENTO)
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ABNT
SILVA, Fábio Ribeiro da et al. Effects of acute and long-term diazepam administrations on neutrophil activity: a flow cytometric study. European Journal of Pharmacology, v. 478, n. 2, p. 97-104, 2003Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2003.08.046. Acesso em: 08 jul. 2024.
APA
Silva, F. R. da, Lazzarini, R., Sá-Rocha, L. C. de, Morgulis, M. S. F. de A., Massoco, C. de O., & Palermo-Neto, J. (2003). Effects of acute and long-term diazepam administrations on neutrophil activity: a flow cytometric study. European Journal of Pharmacology, 478( 2), 97-104. doi:10.1016/j.ejphar.2003.08.046
NLM
Silva FR da, Lazzarini R, Sá-Rocha LC de, Morgulis MSF de A, Massoco C de O, Palermo-Neto J. Effects of acute and long-term diazepam administrations on neutrophil activity: a flow cytometric study [Internet]. European Journal of Pharmacology. 2003 ; 478( 2): 97-104.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2003.08.046
Vancouver
Silva FR da, Lazzarini R, Sá-Rocha LC de, Morgulis MSF de A, Massoco C de O, Palermo-Neto J. Effects of acute and long-term diazepam administrations on neutrophil activity: a flow cytometric study [Internet]. European Journal of Pharmacology. 2003 ; 478( 2): 97-104.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/j.ejphar.2003.08.046
Artigo de periodico
Effects of midazolam on equine innate immune response: a flow cytometric study (2003)
Massoco, Cristina de Oliveira; Palermo-Neto, João
Source: Veterinary Immunology and Immunopathology. Unidade: FMVZ
Subjects: EQUINOS, NEUTRÓFILOS, FÁRMACOS (SISTEMA NERVOSO CENTRAL), SEDATIVOS, CITOMETRIA DE FLUXO, TÉCNICAS INVESTIGATIVAS, FAGOCITOSE
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ABNT
MASSOCO, Cristina de Oliveira e PALERMO-NETO, João. Effects of midazolam on equine innate immune response: a flow cytometric study. Veterinary Immunology and Immunopathology, n. 1-2, p. 11-19, 2003Tradução . . Disponível em: https://doi.org/10.1016/S0165-2427(03)00097-7. Acesso em: 08 jul. 2024.
APA
Massoco, C. de O., & Palermo-Neto, J. (2003). Effects of midazolam on equine innate immune response: a flow cytometric study. Veterinary Immunology and Immunopathology, ( 1-2), 11-19. doi:10.1016/S0165-2427(03)00097-7
NLM
Massoco C de O, Palermo-Neto J. Effects of midazolam on equine innate immune response: a flow cytometric study [Internet]. Veterinary Immunology and Immunopathology. 2003 ;( 1-2): 11-19.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/S0165-2427(03)00097-7
Vancouver
Massoco C de O, Palermo-Neto J. Effects of midazolam on equine innate immune response: a flow cytometric study [Internet]. Veterinary Immunology and Immunopathology. 2003 ;( 1-2): 11-19.[citado 2024 jul. 08 ] Available from: https://doi.org/10.1016/S0165-2427(03)00097-7

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