Filtros : "Indexado na Base de dados Science Citation Index" "ROSSONI, LUCIANA VENTURINI" "ICB" Removidos: "Fisiologia Humana" "RUSP" Limpar

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  • Fonte: Journal of Physiology. Unidades: ICB, FMRP

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 10 out. 2024.
    • APA

      Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
    • NLM

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 out. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
    • Vancouver

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 out. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
  • Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      DAVEL, A. P. et al. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 920-932, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500104. Acesso em: 10 out. 2024.
    • APA

      Davel, A. P., Wenceslau, C. F., Akamine, E. H., Xavier, F. E., Couto, G. K., Oliveira, H. T., & Rossoni, L. V. (2011). Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, 44( 9), 920-932. doi:10.1590/S0100-879X2011007500104
    • NLM

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 out. 10 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
    • Vancouver

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 out. 10 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
  • Fonte: Inflammation. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      DELBIN, Maria Andréia et al. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, v. 34, n. 5, p. 319-325, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10753-010-9237-9. Acesso em: 10 out. 2024.
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      Delbin, M. A., Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2011). Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, 34( 5), 319-325. doi:10.1007/s10753-010-9237-9
    • NLM

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
    • Vancouver

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
  • Fonte: Clinical Science. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      ROSSONI, Luciana Venturini et al. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, v. 121, n. 10, p. 449-458, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20110259. Acesso em: 10 out. 2024.
    • APA

      Rossoni, L. V., Wareing, M., Wenceslau, C. F., Al-Abri, M., Cobb, C., & Austin, C. (2011). Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, 121( 10), 449-458. doi:10.1042/CS20110259
    • NLM

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 out. 10 ] Available from: https://doi.org/10.1042/CS20110259
    • Vancouver

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 out. 10 ] Available from: https://doi.org/10.1042/CS20110259
  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      XAVIER, Fabiano E. et al. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, v. 27, n. 6, p. 1233-1242, 2009Tradução . . Disponível em: https://doi.org/10.1097/hjh.0b013e32832a391f. Acesso em: 10 out. 2024.
    • APA

      Xavier, F. E., Davel, A. P. C., Fukuda, L. E., & Rossoni, L. V. (2009). Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, 27( 6), 1233-1242. doi:10.1097/hjh.0b013e32832a391f
    • NLM

      Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 out. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
    • Vancouver

      Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 out. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
  • Fonte: Journal of Physiology and Biochemistry. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      ARAS-LÓPEZ, R. et al. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation. Journal of Physiology and Biochemistry, v. 64, n. 2, p. 115-126, 2008Tradução . . Disponível em: https://doi.org/10.1007/bf03168239. Acesso em: 10 out. 2024.
    • APA

      Aras-López, R., Blanco-Rivero, J., Hernanz, R., Briones, A. M., Rossoni, L. V., Ferrer, M., et al. (2008). Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation. Journal of Physiology and Biochemistry, 64( 2), 115-126. doi:10.1007/bf03168239
    • NLM

      Aras-López R, Blanco-Rivero J, Hernanz R, Briones AM, Rossoni LV, Ferrer M, Salaices M, Balfagón G. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation [Internet]. Journal of Physiology and Biochemistry. 2008 ; 64( 2): 115-126.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/bf03168239
    • Vancouver

      Aras-López R, Blanco-Rivero J, Hernanz R, Briones AM, Rossoni LV, Ferrer M, Salaices M, Balfagón G. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation [Internet]. Journal of Physiology and Biochemistry. 2008 ; 64( 2): 115-126.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/bf03168239
  • Fonte: American Journal of Physiology: Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      GIRARDI, Adriana Castello Costa et al. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, v. 294, n. 2, p. F414-F422, 2008Tradução . . Acesso em: 10 out. 2024.
    • APA

      Girardi, A. C. C., Fukuda, L. E., Rossoni, L. V., Malnic, G., & Rebouças, N. A. (2008). Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, 294( 2), F414-F422.
    • NLM

      Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 out. 10 ]
    • Vancouver

      Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 out. 10 ]
  • Fonte: British Journal of Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      XAVIER, F. E. et al. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, v. 154, n. 6, p. 1225-1235, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.200. Acesso em: 10 out. 2024.
    • APA

      Xavier, F. E., Aras-López, R., Arroyo-Villa, I., del Campo, L., Salaices, M., Rossoni, L. V., et al. (2008). Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, 154( 6), 1225-1235. doi:10.1038/bjp.2008.200
    • NLM

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/bjp.2008.200
    • Vancouver

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/bjp.2008.200
  • Fonte: Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      COIMBRA, Rosemeire et al. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats?. Hypertension, v. 52, n. 3, p. 514-521, 2008Tradução . . Acesso em: 10 out. 2024.
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      Coimbra, R., Sanchez, L. S., Potenza, J. M., Rossoni, L. V., Amaral, S. L., & Michelini, L. C. (2008). Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension, 52( 3), 514-521.
    • NLM

      Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 out. 10 ]
    • Vancouver

      Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 out. 10 ]
  • Fonte: American Journal of Physiology - Heart Aand Circulatory Physiology. Unidade: ICB

    Assuntos: FARMACOLOGIA, FISIOLOGIA

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    • ABNT

      DAVEL, Ana Paula C. et al. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta. American Journal of Physiology - Heart Aand Circulatory Physiology, v. 295, n. 1, p. H211-H219, 2008Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00581.2007. Acesso em: 10 out. 2024.
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      Davel, A. P. C., Fukuda, L. E., Sá, L. L. de, Munhoz, C. D., Scavone, C., Sanz-Rosa, D., et al. (2008). Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta. American Journal of Physiology - Heart Aand Circulatory Physiology, 295( 1), H211-H219. doi:10.1152/ajpheart.00581.2007
    • NLM

      Davel APC, Fukuda LE, Sá LL de, Munhoz CD, Scavone C, Sanz-Rosa D, Cachofeiro V, Lahera V, Rossoni LV. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta [Internet]. American Journal of Physiology - Heart Aand Circulatory Physiology. 2008 ; 295( 1): H211-H219.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.00581.2007
    • Vancouver

      Davel APC, Fukuda LE, Sá LL de, Munhoz CD, Scavone C, Sanz-Rosa D, Cachofeiro V, Lahera V, Rossoni LV. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta [Internet]. American Journal of Physiology - Heart Aand Circulatory Physiology. 2008 ; 295( 1): H211-H219.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.00581.2007
  • Fonte: BMC Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      MORAES, Camila de et al. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, v. 8, 2008Tradução . . Disponível em: https://doi.org/10.1186/1472-6793-8-12. Acesso em: 10 out. 2024.
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      Moraes, C. de, Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2008). Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, 8. doi:10.1186/1472-6793-8-12
    • NLM

      Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 out. 10 ] Available from: https://doi.org/10.1186/1472-6793-8-12
    • Vancouver

      Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 out. 10 ] Available from: https://doi.org/10.1186/1472-6793-8-12
  • Fonte: Journal of Cardiovascular Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BALDO, Marcelo P. et al. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, v. 52, n. 4, p. 375-380, 2008Tradução . . Acesso em: 10 out. 2024.
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      Baldo, M. P., Davel, A. P. C., Nicoletti-Carvalho, J. E., Bordin, S., Rossoni, L. V., & Mill, J. G. (2008). Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, 52( 4), 375-380.
    • NLM

      Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 out. 10 ]
    • Vancouver

      Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 out. 10 ]
  • Fonte: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FM, EEFE

    Assunto: FISIOLOGIA

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      ROCHA, F. L. et al. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, v. 293, n. 6, p. H3575-H3583, 2007Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.01251.2006. Acesso em: 10 out. 2024.
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      Rocha, F. L., Carmo, E. C., Roque, F. R., Hashimoto, N. Y., Rossoni, L. V., Frimm, C., et al. (2007). Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, 293( 6), H3575-H3583. doi:10.1152/ajpheart.01251.2006
    • NLM

      Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006
    • Vancouver

      Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006
  • Fonte: British Journal of Pharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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      DAVEL, Ana Paula Couto et al. Changes in vascular reactivity following administration of isoproterenol for 1 week: a role for endothelial modulation. British Journal of Pharmacology, v. 148, p. 629-639, 2006Tradução . . Disponível em: https://doi.org/10.1038/sj.bjp.0706749. Acesso em: 10 out. 2024.
    • APA

      Davel, A. P. C., Kawamoto, E. M., Scavone, C., Vassallo, D. V., & Rossoni, L. V. (2006). Changes in vascular reactivity following administration of isoproterenol for 1 week: a role for endothelial modulation. British Journal of Pharmacology, 148, 629-639. doi:10.1038/sj.bjp.0706749
    • NLM

      Davel APC, Kawamoto EM, Scavone C, Vassallo DV, Rossoni LV. Changes in vascular reactivity following administration of isoproterenol for 1 week: a role for endothelial modulation [Internet]. British Journal of Pharmacology. 2006 ; 148 629-639.[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706749
    • Vancouver

      Davel APC, Kawamoto EM, Scavone C, Vassallo DV, Rossoni LV. Changes in vascular reactivity following administration of isoproterenol for 1 week: a role for endothelial modulation [Internet]. British Journal of Pharmacology. 2006 ; 148 629-639.[citado 2024 out. 10 ] Available from: https://doi.org/10.1038/sj.bjp.0706749

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