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Artigo de periodico
Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats (2011)
Camporez, João Paulo Gabriel; Akamine, Eliana Hiromi ; Davel, Ana Paula Couto; Franci, Celso Rodrigues; Rossoni, Luciana Venturini ; Carvalho, Carla Roberta de Oliveira
Fonte: Journal of Physiology. Unidades: ICB, FMRP
Assuntos: FISIOLOGIA, FARMACOLOGIA
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ABNT
CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 10 out. 2024.
APA
Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
NLM
Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 out. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
Vancouver
Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 out. 10 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
Artigo de periodico
Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update (2011)
Davel, A. P.; Wenceslau, Camilla Ferreira; Akamine, Eliana Hiromi ; Xavier, F. E.; Couto, Gisele Kruger; Oliveira, H. T.; Rossoni, Luciana Venturini
Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
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ABNT
DAVEL, A. P. et al. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 920-932, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500104. Acesso em: 10 out. 2024.
APA
Davel, A. P., Wenceslau, C. F., Akamine, E. H., Xavier, F. E., Couto, G. K., Oliveira, H. T., & Rossoni, L. V. (2011). Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, 44( 9), 920-932. doi:10.1590/S0100-879X2011007500104
NLM
Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 out. 10 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
Vancouver
Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 out. 10 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
Artigo de periodico
Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats (2011)
Delbin, Maria Andréia; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Antunes, Edson; Zanesco, Angelina
Fonte: Inflammation. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
DELBIN, Maria Andréia et al. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, v. 34, n. 5, p. 319-325, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10753-010-9237-9. Acesso em: 10 out. 2024.
APA
Delbin, M. A., Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2011). Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, 34( 5), 319-325. doi:10.1007/s10753-010-9237-9
NLM
Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
Vancouver
Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
Artigo de periodico
Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries (2011)
Rossoni, Luciana Venturini ; Wareing, Mark; Wenceslau, Camilla Ferreira; Al-Abri, Mahmood; Cobb, Chris; Austin, Clare
Fonte: Clinical Science. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ROSSONI, Luciana Venturini et al. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, v. 121, n. 10, p. 449-458, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20110259. Acesso em: 10 out. 2024.
APA
Rossoni, L. V., Wareing, M., Wenceslau, C. F., Al-Abri, M., Cobb, C., & Austin, C. (2011). Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, 121( 10), 449-458. doi:10.1042/CS20110259
NLM
Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 out. 10 ] Available from: https://doi.org/10.1042/CS20110259
Vancouver
Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 out. 10 ] Available from: https://doi.org/10.1042/CS20110259
Artigo de periodico
Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms (2009)
Xavier, Fabiano E.; Davel, Ana Paula C.; Fukuda, Lívia E.; Rossoni, Luciana Venturini
Fonte: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
XAVIER, Fabiano E. et al. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, v. 27, n. 6, p. 1233-1242, 2009Tradução . . Disponível em: https://doi.org/10.1097/hjh.0b013e32832a391f. Acesso em: 10 out. 2024.
APA
Xavier, F. E., Davel, A. P. C., Fukuda, L. E., & Rossoni, L. V. (2009). Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, 27( 6), 1233-1242. doi:10.1097/hjh.0b013e32832a391f
NLM
Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 out. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
Vancouver
Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 out. 10 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
Artigo de periodico
Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation (2008)
Aras-López, R.; Blanco-Rivero, J.; Hernanz, R.; Briones, A. M.; Rossoni, Luciana Venturini ; Ferrer, M.; Salaices, M.; Balfagón, G.
Fonte: Journal of Physiology and Biochemistry. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
ARAS-LÓPEZ, R. et al. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation. Journal of Physiology and Biochemistry, v. 64, n. 2, p. 115-126, 2008Tradução . . Disponível em: https://doi.org/10.1007/bf03168239. Acesso em: 10 out. 2024.
APA
Aras-López, R., Blanco-Rivero, J., Hernanz, R., Briones, A. M., Rossoni, L. V., Ferrer, M., et al. (2008). Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation. Journal of Physiology and Biochemistry, 64( 2), 115-126. doi:10.1007/bf03168239
NLM
Aras-López R, Blanco-Rivero J, Hernanz R, Briones AM, Rossoni LV, Ferrer M, Salaices M, Balfagón G. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation [Internet]. Journal of Physiology and Biochemistry. 2008 ; 64( 2): 115-126.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/bf03168239
Vancouver
Aras-López R, Blanco-Rivero J, Hernanz R, Briones AM, Rossoni LV, Ferrer M, Salaices M, Balfagón G. Chronic ouabain treatment increases the contribution of nitric oxide to endotheliumdependent relaxation [Internet]. Journal of Physiology and Biochemistry. 2008 ; 64( 2): 115-126.[citado 2024 out. 10 ] Available from: https://doi.org/10.1007/bf03168239
Artigo de periodico
Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule (2008)
Girardi, Adriana Castello Costa; Fukuda, Lívia Emy; Rossoni, Luciana Venturini ; Malnic, Gerhard; Rebouças, Nancy Amaral
Fonte: American Journal of Physiology: Renal Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
GIRARDI, Adriana Castello Costa et al. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, v. 294, n. 2, p. F414-F422, 2008Tradução . . Acesso em: 10 out. 2024.
APA
Girardi, A. C. C., Fukuda, L. E., Rossoni, L. V., Malnic, G., & Rebouças, N. A. (2008). Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, 294( 2), F414-F422.
NLM
Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 out. 10 ]
Vancouver
Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 out. 10 ]
Artigo de periodico
Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? (2008)
Coimbra, Rosemeire; Sanchez, Lylian S.; Potenza, Janaina M.; Rossoni, Luciana Venturini ; Amaral, Sandra Lia; Michelini, Lisete Compagno
Fonte: Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
COIMBRA, Rosemeire et al. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats?. Hypertension, v. 52, n. 3, p. 514-521, 2008Tradução . . Acesso em: 10 out. 2024.
APA
Coimbra, R., Sanchez, L. S., Potenza, J. M., Rossoni, L. V., Amaral, S. L., & Michelini, L. C. (2008). Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension, 52( 3), 514-521.
NLM
Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 out. 10 ]
Vancouver
Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 out. 10 ]
Artigo de periodico
Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta (2008)
Davel, Ana Paula C.; Fukuda, Lívia E.; Sá, Larissa Lima de; Munhoz, Carolina Demarchi ; Scavone, Cristoforo ; Sanz-Rosa, David; Cachofeiro, Victoria; Lahera, Vicente; Rossoni, Luciana Venturini
Fonte: American Journal of Physiology - Heart Aand Circulatory Physiology. Unidade: ICB
Assuntos: FARMACOLOGIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAVEL, Ana Paula C. et al. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta. American Journal of Physiology - Heart Aand Circulatory Physiology, v. 295, n. 1, p. H211-H219, 2008Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.00581.2007. Acesso em: 10 out. 2024.
APA
Davel, A. P. C., Fukuda, L. E., Sá, L. L. de, Munhoz, C. D., Scavone, C., Sanz-Rosa, D., et al. (2008). Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta. American Journal of Physiology - Heart Aand Circulatory Physiology, 295( 1), H211-H219. doi:10.1152/ajpheart.00581.2007
NLM
Davel APC, Fukuda LE, Sá LL de, Munhoz CD, Scavone C, Sanz-Rosa D, Cachofeiro V, Lahera V, Rossoni LV. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta [Internet]. American Journal of Physiology - Heart Aand Circulatory Physiology. 2008 ; 295( 1): H211-H219.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.00581.2007
Vancouver
Davel APC, Fukuda LE, Sá LL de, Munhoz CD, Scavone C, Sanz-Rosa D, Cachofeiro V, Lahera V, Rossoni LV. Effects of isoproterenol treatment for 7 days on inflammatory mediators in the rat aorta [Internet]. American Journal of Physiology - Heart Aand Circulatory Physiology. 2008 ; 295( 1): H211-H219.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.00581.2007
Artigo de periodico
Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats (2008)
Moraes, Camila de ; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Antunes, Edson; Zanesco, Angelina
Fonte: BMC Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
MORAES, Camila de et al. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, v. 8, 2008Tradução . . Disponível em: https://doi.org/10.1186/1472-6793-8-12. Acesso em: 10 out. 2024.
APA
Moraes, C. de, Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2008). Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, 8. doi:10.1186/1472-6793-8-12
NLM
Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 out. 10 ] Available from: https://doi.org/10.1186/1472-6793-8-12
Vancouver
Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 out. 10 ] Available from: https://doi.org/10.1186/1472-6793-8-12
Artigo de periodico
Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats (2008)
Baldo, Marcelo P.; Davel, Ana Paula Couto; Nicoletti-Carvalho, José Edgar; Bordin, Silvana ; Rossoni, Luciana Venturini ; Mill, José G.
Fonte: Journal of Cardiovascular Pharmacology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BALDO, Marcelo P. et al. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, v. 52, n. 4, p. 375-380, 2008Tradução . . Acesso em: 10 out. 2024.
APA
Baldo, M. P., Davel, A. P. C., Nicoletti-Carvalho, J. E., Bordin, S., Rossoni, L. V., & Mill, J. G. (2008). Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, 52( 4), 375-380.
NLM
Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 out. 10 ]
Vancouver
Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 out. 10 ]
Artigo de periodico
Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats (2007)
Rocha, F. L.; Carmo, E. C.; Roque, F. R.; Hashimoto, N. Y.; Rossoni, Luciana Venturini ; Frimm, C.; Anéas, I.; Negrão, C. E.; Krieger, José Eduardo; Oliveira, E. M.
Fonte: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FM, EEFE
Assunto: FISIOLOGIA
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ABNT
ROCHA, F. L. et al. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, v. 293, n. 6, p. H3575-H3583, 2007Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.01251.2006. Acesso em: 10 out. 2024.
APA
Rocha, F. L., Carmo, E. C., Roque, F. R., Hashimoto, N. Y., Rossoni, L. V., Frimm, C., et al. (2007). Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, 293( 6), H3575-H3583. doi:10.1152/ajpheart.01251.2006
NLM
Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006
Vancouver
Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 out. 10 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006

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