Filtros : "Indexado na Base de dados PubMed" "ROSSONI, LUCIANA VENTURINI" "ICB" Removidos: "Espanha" "Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)" Limpar

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  • Fonte: Journal of Physiology. Unidades: ICB, FMRP

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      CAMPOREZ, João Paulo Gabriel et al. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, v. 589, n. 10, p. 2585-2596, 2011Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2011.206078. Acesso em: 01 nov. 2024.
    • APA

      Camporez, J. P. G., Akamine, E. H., Davel, A. P. C., Franci, C. R., Rossoni, L. V., & Carvalho, C. R. de O. (2011). Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats. Journal of Physiology, 589( 10), 2585-2596. doi:10.1113/jphysiol.2011.206078
    • NLM

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
    • Vancouver

      Camporez JPG, Akamine EH, Davel APC, Franci CR, Rossoni LV, Carvalho CR de O. Dehydroepiandrosterone protects against oxidative stress-induced endothelial dysfunction in ovariectomized rats [Internet]. Journal of Physiology. 2011 ; 589( 10): 2585-2596.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1113/jphysiol.2011.206078
  • Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      DAVEL, A. P. et al. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, v. 44, n. 9, p. 920-932, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500104. Acesso em: 01 nov. 2024.
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      Davel, A. P., Wenceslau, C. F., Akamine, E. H., Xavier, F. E., Couto, G. K., Oliveira, H. T., & Rossoni, L. V. (2011). Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update. Brazilian Journal of Medical and Biological Research, 44( 9), 920-932. doi:10.1590/S0100-879X2011007500104
    • NLM

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
    • Vancouver

      Davel AP, Wenceslau CF, Akamine EH, Xavier FE, Couto GK, Oliveira HT, Rossoni LV. Endothelial dysfunction in cardiovascular and endocrine-metabolic diseases: an update [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 9): 920-932.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1590/S0100-879X2011007500104
  • Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      BESSA, K. L. et al. Drag reduction by polyethylene glycol in the tail arterial bed of normotensive and hypertensive rats. Brazilian Journal of Medical and Biological Research, v. 44, n. 8, p. 767-777, 2011Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2011007500071. Acesso em: 01 nov. 2024.
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      Bessa, K. L., Belletati, J. F., Santos, L. dos, Rossoni, L. V., & Ortiz, J. P. (2011). Drag reduction by polyethylene glycol in the tail arterial bed of normotensive and hypertensive rats. Brazilian Journal of Medical and Biological Research, 44( 8), 767-777. doi:10.1590/S0100-879X2011007500071
    • NLM

      Bessa KL, Belletati JF, Santos L dos, Rossoni LV, Ortiz JP. Drag reduction by polyethylene glycol in the tail arterial bed of normotensive and hypertensive rats [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 8): 767-777.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1590/S0100-879X2011007500071
    • Vancouver

      Bessa KL, Belletati JF, Santos L dos, Rossoni LV, Ortiz JP. Drag reduction by polyethylene glycol in the tail arterial bed of normotensive and hypertensive rats [Internet]. Brazilian Journal of Medical and Biological Research. 2011 ; 44( 8): 767-777.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1590/S0100-879X2011007500071
  • Fonte: Inflammation. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      DELBIN, Maria Andréia et al. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, v. 34, n. 5, p. 319-325, 2011Tradução . . Disponível em: https://doi.org/10.1007/s10753-010-9237-9. Acesso em: 01 nov. 2024.
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      Delbin, M. A., Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2011). Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats. Inflammation, 34( 5), 319-325. doi:10.1007/s10753-010-9237-9
    • NLM

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
    • Vancouver

      Delbin MA, Davel APC, Rossoni LV, Antunes E, Zanesco A. Beneficial effects of physical training on the cardio-inflammatory disorder induced by lung ischemia/reperfusion in rats [Internet]. Inflammation. 2011 ; 34( 5): 319-325.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1007/s10753-010-9237-9
  • Fonte: Clinical Science. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      ROSSONI, Luciana Venturini et al. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, v. 121, n. 10, p. 449-458, 2011Tradução . . Disponível em: https://doi.org/10.1042/CS20110259. Acesso em: 01 nov. 2024.
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      Rossoni, L. V., Wareing, M., Wenceslau, C. F., Al-Abri, M., Cobb, C., & Austin, C. (2011). Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries. Clinical Science, 121( 10), 449-458. doi:10.1042/CS20110259
    • NLM

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1042/CS20110259
    • Vancouver

      Rossoni LV, Wareing M, Wenceslau CF, Al-Abri M, Cobb C, Austin C. Acute simvastatin increases endothelial nitric oxide synthase phosphorylation via AMP-activated protein kinase and reduces contractility of isolated rat mesenteric resistance arteries [Internet]. Clinical Science. 2011 ; 121( 10): 449-458.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1042/CS20110259
  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      XAVIER, Fabiano E. et al. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, v. 27, n. 6, p. 1233-1242, 2009Tradução . . Disponível em: https://doi.org/10.1097/hjh.0b013e32832a391f. Acesso em: 01 nov. 2024.
    • APA

      Xavier, F. E., Davel, A. P. C., Fukuda, L. E., & Rossoni, L. V. (2009). Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms. Journal of Hypertension, 27( 6), 1233-1242. doi:10.1097/hjh.0b013e32832a391f
    • NLM

      Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
    • Vancouver

      Xavier FE, Davel APC, Fukuda LE, Rossoni LV. Chronic ouabain treatment exacerbates blood pressure elevation in spontaneously hypertensive rats: the role of vascular mechanisms [Internet]. Journal of Hypertension. 2009 ; 27( 6): 1233-1242.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1097/hjh.0b013e32832a391f
  • Fonte: Journal of Cardiovascular Pharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      FUKUDA, Lívia Emy et al. Fenofibrate and pioglitazone do not ameliorate the altered vascular reactivity in aorta of isoproterenol-treated rats. Journal of Cardiovascular Pharmacology, v. 55, n. 5, p. 413-421, 2008Tradução . . Acesso em: 01 nov. 2024.
    • APA

      Fukuda, L. E., Davel, A. P. C., Verissimo-Filho, S., Lopes, L. R., Cachofeiro, V., Lahera, V., & Rossoni, L. V. (2008). Fenofibrate and pioglitazone do not ameliorate the altered vascular reactivity in aorta of isoproterenol-treated rats. Journal of Cardiovascular Pharmacology, 55( 5), 413-421.
    • NLM

      Fukuda LE, Davel APC, Verissimo-Filho S, Lopes LR, Cachofeiro V, Lahera V, Rossoni LV. Fenofibrate and pioglitazone do not ameliorate the altered vascular reactivity in aorta of isoproterenol-treated rats. Journal of Cardiovascular Pharmacology. 2008 ; 55( 5): 413-421.[citado 2024 nov. 01 ]
    • Vancouver

      Fukuda LE, Davel APC, Verissimo-Filho S, Lopes LR, Cachofeiro V, Lahera V, Rossoni LV. Fenofibrate and pioglitazone do not ameliorate the altered vascular reactivity in aorta of isoproterenol-treated rats. Journal of Cardiovascular Pharmacology. 2008 ; 55( 5): 413-421.[citado 2024 nov. 01 ]
  • Fonte: American Journal of Physiology: Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      GIRARDI, Adriana Castello Costa et al. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, v. 294, n. 2, p. F414-F422, 2008Tradução . . Acesso em: 01 nov. 2024.
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      Girardi, A. C. C., Fukuda, L. E., Rossoni, L. V., Malnic, G., & Rebouças, N. A. (2008). Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology, 294( 2), F414-F422.
    • NLM

      Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 nov. 01 ]
    • Vancouver

      Girardi ACC, Fukuda LE, Rossoni LV, Malnic G, Rebouças NA. Dipeptidyl peptidase IV inhibition downregulates Na -H + exchanger NHE3 in rat renal proximal tubule. American Journal of Physiology: Renal Physiology. 2008 ; 294( 2): F414-F422.[citado 2024 nov. 01 ]
  • Fonte: British Journal of Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      XAVIER, F. E. et al. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, v. 154, n. 6, p. 1225-1235, 2008Tradução . . Disponível em: https://doi.org/10.1038/bjp.2008.200. Acesso em: 01 nov. 2024.
    • APA

      Xavier, F. E., Aras-López, R., Arroyo-Villa, I., del Campo, L., Salaices, M., Rossoni, L. V., et al. (2008). Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin. British Journal of Pharmacology, 154( 6), 1225-1235. doi:10.1038/bjp.2008.200
    • NLM

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/bjp.2008.200
    • Vancouver

      Xavier FE, Aras-López R, Arroyo-Villa I, del Campo L, Salaices M, Rossoni LV, Ferrer M, Balfagón G. Aldosterone induces endothelial dysfunction in resistance arteries from normotensive and hypertensive rats by increasing thromboxane A2 and prostacyclin [Internet]. British Journal of Pharmacology. 2008 ; 154( 6): 1225-1235.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1038/bjp.2008.200
  • Fonte: Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      COIMBRA, Rosemeire et al. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats?. Hypertension, v. 52, n. 3, p. 514-521, 2008Tradução . . Acesso em: 01 nov. 2024.
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      Coimbra, R., Sanchez, L. S., Potenza, J. M., Rossoni, L. V., Amaral, S. L., & Michelini, L. C. (2008). Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension, 52( 3), 514-521.
    • NLM

      Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 nov. 01 ]
    • Vancouver

      Coimbra R, Sanchez LS, Potenza JM, Rossoni LV, Amaral SL, Michelini LC. Is gender crucial for cardiovascular adjustments induced by exercise training in female spontaneously hypertensive rats? Hypertension. 2008 ; 52( 3): 514-521.[citado 2024 nov. 01 ]
  • Fonte: BMC Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      MORAES, Camila de et al. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, v. 8, 2008Tradução . . Disponível em: https://doi.org/10.1186/1472-6793-8-12. Acesso em: 01 nov. 2024.
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      Moraes, C. de, Davel, A. P. C., Rossoni, L. V., Antunes, E., & Zanesco, A. (2008). Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats. BMC Physiology, 8. doi:10.1186/1472-6793-8-12
    • NLM

      Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/1472-6793-8-12
    • Vancouver

      Moraes C de, Davel APC, Rossoni LV, Antunes E, Zanesco A. Exercise training improves relaxation response and SOD-1 expression in aortic and mesenteric rings from high caloric diet-fed rats [Internet]. BMC Physiology. 2008 ; 8[citado 2024 nov. 01 ] Available from: https://doi.org/10.1186/1472-6793-8-12
  • Fonte: Journal of Cardiovascular Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

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      BALDO, Marcelo P. et al. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, v. 52, n. 4, p. 375-380, 2008Tradução . . Acesso em: 01 nov. 2024.
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      Baldo, M. P., Davel, A. P. C., Nicoletti-Carvalho, J. E., Bordin, S., Rossoni, L. V., & Mill, J. G. (2008). Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology, 52( 4), 375-380.
    • NLM

      Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 nov. 01 ]
    • Vancouver

      Baldo MP, Davel APC, Nicoletti-Carvalho JE, Bordin S, Rossoni LV, Mill JG. Granulocyte colony-stimulating factor reduces mortality by suppressing ventricular arrhythmias in acute phase of myocardial infarction in rats. Journal of Cardiovascular Pharmacology. 2008 ; 52( 4): 375-380.[citado 2024 nov. 01 ]
  • Fonte: American Journal of Physiology: Heart and Circulatory Physiology. Unidades: ICB, FM, EEFE

    Assunto: FISIOLOGIA

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    • ABNT

      ROCHA, F. L. et al. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, v. 293, n. 6, p. H3575-H3583, 2007Tradução . . Disponível em: https://doi.org/10.1152/ajpheart.01251.2006. Acesso em: 01 nov. 2024.
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      Rocha, F. L., Carmo, E. C., Roque, F. R., Hashimoto, N. Y., Rossoni, L. V., Frimm, C., et al. (2007). Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats. American Journal of Physiology: Heart and Circulatory Physiology, 293( 6), H3575-H3583. doi:10.1152/ajpheart.01251.2006
    • NLM

      Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006
    • Vancouver

      Rocha FL, Carmo EC, Roque FR, Hashimoto NY, Rossoni LV, Frimm C, Anéas I, Negrão CE, Krieger JE, Oliveira EM. Anabolic steroids induce cardiac renin-angiotensin system and impair the beneficial effects of aerobic training in rats [Internet]. American Journal of Physiology: Heart and Circulatory Physiology. 2007 ; 293( 6): H3575-H3583.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpheart.01251.2006
  • Fonte: Life Sciences. Unidades: ICB, EEFE

    Assunto: FISIOLOGIA

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      SANTOS, Leonardo dos et al. Effects of high sodium intake diet on the vascular reactivity to phenylephrine on rat isolated caudal and renal vascular beds: Endothelial modulation. Life Sciences, v. 78, n. 19, p. 2272-2279, 2006Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2005.09.028. Acesso em: 01 nov. 2024.
    • APA

      Santos, L. dos, Gonçalves, M. V., Vassallo, D. V., Oliveira, E. M., & Rossoni, L. V. (2006). Effects of high sodium intake diet on the vascular reactivity to phenylephrine on rat isolated caudal and renal vascular beds: Endothelial modulation. Life Sciences, 78( 19), 2272-2279. doi:10.1016/j.lfs.2005.09.028
    • NLM

      Santos L dos, Gonçalves MV, Vassallo DV, Oliveira EM, Rossoni LV. Effects of high sodium intake diet on the vascular reactivity to phenylephrine on rat isolated caudal and renal vascular beds: Endothelial modulation [Internet]. Life Sciences. 2006 ; 78( 19): 2272-2279.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.lfs.2005.09.028
    • Vancouver

      Santos L dos, Gonçalves MV, Vassallo DV, Oliveira EM, Rossoni LV. Effects of high sodium intake diet on the vascular reactivity to phenylephrine on rat isolated caudal and renal vascular beds: Endothelial modulation [Internet]. Life Sciences. 2006 ; 78( 19): 2272-2279.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.lfs.2005.09.028

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