Filtros : "Indexado na Base de dados Excerpta Medica" "Indexado na Base de dados Index Medicus" "CARPINELLI, ANGELO RAFAEL" "ICB" Removidos: "ANTIDEPRESSIVOS" "Castro-Faria-Neto, H. C." Limpar

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  • Source: Journal of Pineal Research. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      PICINATO, M. C. et al. Activation of insulin and IGF-1 signaling pathways by melatonin through MT1 receptor in isolated rat pancreatic islets. Journal of Pineal Research, v. 44, n. 1, p. 88-94, 2008Tradução . . Disponível em: https://doi.org/10.1111/j.1600-079x.2007.00493.x. Acesso em: 28 out. 2024.
    • APA

      Picinato, M. C., Hirata, A. E., Cipolla Neto, J., Curi, R., Carvalho, C. R. de O., Anhê, G. F., & Carpinelli, A. R. (2008). Activation of insulin and IGF-1 signaling pathways by melatonin through MT1 receptor in isolated rat pancreatic islets. Journal of Pineal Research, 44( 1), 88-94. doi:10.1111/j.1600-079x.2007.00493.x
    • NLM

      Picinato MC, Hirata AE, Cipolla Neto J, Curi R, Carvalho CR de O, Anhê GF, Carpinelli AR. Activation of insulin and IGF-1 signaling pathways by melatonin through MT1 receptor in isolated rat pancreatic islets [Internet]. Journal of Pineal Research. 2008 ; 44( 1): 88-94.[citado 2024 out. 28 ] Available from: https://doi.org/10.1111/j.1600-079x.2007.00493.x
    • Vancouver

      Picinato MC, Hirata AE, Cipolla Neto J, Curi R, Carvalho CR de O, Anhê GF, Carpinelli AR. Activation of insulin and IGF-1 signaling pathways by melatonin through MT1 receptor in isolated rat pancreatic islets [Internet]. Journal of Pineal Research. 2008 ; 44( 1): 88-94.[citado 2024 out. 28 ] Available from: https://doi.org/10.1111/j.1600-079x.2007.00493.x
  • Source: Journal of Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      NEWSHOLLME, P. et al. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity. Journal of Physiology, v. 583, n. 1, p. 9-24, 2007Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2007.135871. Acesso em: 28 out. 2024.
    • APA

      Newshollme, P., Haber, E. P., Hirabara, S. M., Rebelato, E. L. O., Procópio, J., Morgan, D., et al. (2007). Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity. Journal of Physiology, 583( 1), 9-24. doi:10.1113/jphysiol.2007.135871
    • NLM

      Newshollme P, Haber EP, Hirabara SM, Rebelato ELO, Procópio J, Morgan D, Oliveira-Emilio HC, Carpinelli AR, Curi R. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity [Internet]. Journal of Physiology. 2007 ; 583( 1): 9-24.[citado 2024 out. 28 ] Available from: https://doi.org/10.1113/jphysiol.2007.135871
    • Vancouver

      Newshollme P, Haber EP, Hirabara SM, Rebelato ELO, Procópio J, Morgan D, Oliveira-Emilio HC, Carpinelli AR, Curi R. Diabetes associated cell stress and dysfunction: role f mitochondrial and non-mitochondrial ROS production and activity [Internet]. Journal of Physiology. 2007 ; 583( 1): 9-24.[citado 2024 out. 28 ] Available from: https://doi.org/10.1113/jphysiol.2007.135871
  • Source: Cell Biochemistry and Function. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
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    • ABNT

      XIMENES, Helena M. A. et al. Propionate inhibits glucose-induced insulin secretion in isolated rat pancreatic islets. Cell Biochemistry and Function, v. 25, p. 173-178, 2007Tradução . . Disponível em: https://doi.org/10.1002/cbf.1297. Acesso em: 28 out. 2024.
    • APA

      Ximenes, H. M. A., Hirata, A. E., Rocha, M. S., Curi, R., & Carpinelli, A. R. (2007). Propionate inhibits glucose-induced insulin secretion in isolated rat pancreatic islets. Cell Biochemistry and Function, 25, 173-178. doi:10.1002/cbf.1297
    • NLM

      Ximenes HMA, Hirata AE, Rocha MS, Curi R, Carpinelli AR. Propionate inhibits glucose-induced insulin secretion in isolated rat pancreatic islets [Internet]. Cell Biochemistry and Function. 2007 ; 25 173-178.[citado 2024 out. 28 ] Available from: https://doi.org/10.1002/cbf.1297
    • Vancouver

      Ximenes HMA, Hirata AE, Rocha MS, Curi R, Carpinelli AR. Propionate inhibits glucose-induced insulin secretion in isolated rat pancreatic islets [Internet]. Cell Biochemistry and Function. 2007 ; 25 173-178.[citado 2024 out. 28 ] Available from: https://doi.org/10.1002/cbf.1297
  • Source: Diabetologia. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      MORGAN, Daniela et al. Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line. Diabetologia, v. 50, n. 2, p. 359-369, 2007Tradução . . Disponível em: https://doi.org/10.1007/s00125-006-0462-6. Acesso em: 28 out. 2024.
    • APA

      Morgan, D., Oliveira-Emilio, H. R. de, Keane, D., Hirata, A. E., Santos da Rocha, M., Bordin, S., et al. (2007). Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line. Diabetologia, 50( 2), 359-369. doi:10.1007/s00125-006-0462-6
    • NLM

      Morgan D, Oliveira-Emilio HR de, Keane D, Hirata AE, Santos da Rocha M, Bordin S, Curi R, Newsholme P, Carpinelli AR. Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line [Internet]. Diabetologia. 2007 ; 50( 2): 359-369.[citado 2024 out. 28 ] Available from: https://doi.org/10.1007/s00125-006-0462-6
    • Vancouver

      Morgan D, Oliveira-Emilio HR de, Keane D, Hirata AE, Santos da Rocha M, Bordin S, Curi R, Newsholme P, Carpinelli AR. Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line [Internet]. Diabetologia. 2007 ; 50( 2): 359-369.[citado 2024 out. 28 ] Available from: https://doi.org/10.1007/s00125-006-0462-6

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