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Artigo de periodico
Atrial fibrillation promotion in a rat model of heart failure induced by left ventricle radiofrequency ablation (2018)
Santos, Luis Felipe Neves dos; Antonio, Ednei Luiz; Serra, Andrey Jorge; Yoshizaki, Amanda; Seibt, Larissa Emília; Silva, Flávio André; Couto, Gisele Kruger; Rossoni, Luciana Venturini ; Tucci, Paulo José Ferreira; Paola, Angelo Amato Vincenzo de; Costa, Guilherme Drummond Fenelon
Fonte: International Journal of Cardiology - Heart & Vasculature. Unidade: ICB
Assuntos: FIBRILAÇÃO ATRIAL, FIBRILAÇÃO VENTRICULAR, RATOS, INSUFICIÊNCIA CARDÍACA ANIMAL, VENTRÍCULO CARDÍACO DE ANIMAL, ECOCARDIOGRAFIA, WESTERN BLOTTING
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ABNT
SANTOS, Luis Felipe Neves dos et al. Atrial fibrillation promotion in a rat model of heart failure induced by left ventricle radiofrequency ablation. International Journal of Cardiology - Heart & Vasculature, v. 21, p. 22-28, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ijcha.2018.09.003. Acesso em: 13 nov. 2024.
APA
Santos, L. F. N. dos, Antonio, E. L., Serra, A. J., Yoshizaki, A., Seibt, L. E., Silva, F. A., et al. (2018). Atrial fibrillation promotion in a rat model of heart failure induced by left ventricle radiofrequency ablation. International Journal of Cardiology - Heart & Vasculature, 21, 22-28. doi:10.1016/j.ijcha.2018.09.003
NLM
Santos LFN dos, Antonio EL, Serra AJ, Yoshizaki A, Seibt LE, Silva FA, Couto GK, Rossoni LV, Tucci PJF, Paola AAV de, Costa GDF. Atrial fibrillation promotion in a rat model of heart failure induced by left ventricle radiofrequency ablation [Internet]. International Journal of Cardiology - Heart & Vasculature. 2018 ; 21 22-28.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ijcha.2018.09.003
Vancouver
Santos LFN dos, Antonio EL, Serra AJ, Yoshizaki A, Seibt LE, Silva FA, Couto GK, Rossoni LV, Tucci PJF, Paola AAV de, Costa GDF. Atrial fibrillation promotion in a rat model of heart failure induced by left ventricle radiofrequency ablation [Internet]. International Journal of Cardiology - Heart & Vasculature. 2018 ; 21 22-28.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.ijcha.2018.09.003
Artigo de periodico
Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues (2016)
Victório, Jamaira A.; Fontes, Milene T.; Rossoni, Luciana Venturini ; Davel, Ana P.
Fonte: Frontiers in Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
VICTÓRIO, Jamaira A. et al. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, v. 7, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.3389/fphys.2016.00295. Acesso em: 13 nov. 2024.
APA
Victório, J. A., Fontes, M. T., Rossoni, L. V., & Davel, A. P. (2016). Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, 7, 1-10. doi:10.3389/fphys.2016.00295
NLM
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Vancouver
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 nov. 13 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Artigo de periodico
Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR (2010)
Crajoinas, Renato O.; Lessa, Lucília M. A.; Carraro-Lacroix, Luciene R.; Davel, Ana Paula Couto; Pacheco, Bruna P. M.; Rossoni, Luciana Venturini ; Malnic, Gerhald; Girardi, Adriana Castello Costa
Fonte: American Journal of Physiology. Renal Physiology. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
CRAJOINAS, Renato O. et al. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, v. 299, n. 4, p. F872-F881, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00654.2009. Acesso em: 13 nov. 2024.
APA
Crajoinas, R. O., Lessa, L. M. A., Carraro-Lacroix, L. R., Davel, A. P. C., Pacheco, B. P. M., Rossoni, L. V., et al. (2010). Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, 299( 4), F872-F881. doi:10.1152/ajprenal.00654.2009
NLM
Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
Vancouver
Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
Artigo de periodico
Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C (2010)
Gallo, Luana C.; Davel, Ana Paula Couto; Xavier, Fabiano E.; Rossoni, Luciana Venturini
Fonte: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
GALLO, Luana C. et al. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, v. 87, n. 9-10 p. 302-308, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.005. Acesso em: 13 nov. 2024.
APA
Gallo, L. C., Davel, A. P. C., Xavier, F. E., & Rossoni, L. V. (2010). Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, 87( 9-10 p. 302-308). doi:10.1016/j.lfs.2010.07.005
NLM
Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
Vancouver
Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
Artigo de periodico
Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice (2010)
Kobayasi, Renata; Akamine, Eliana Hiromi ; Davel, Ana Paula Couto; Rodrigues, Maria A. M.; Carvalho, Carla Roberta de Oliveira ; Rossoni, Luciana Venturini
Fonte: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
KOBAYASI, Renata et al. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, v. 28, n. 10, p. 2111-2119, 2010Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 13 nov. 2024.
APA
Kobayasi, R., Akamine, E. H., Davel, A. P. C., Rodrigues, M. A. M., Carvalho, C. R. de O., & Rossoni, L. V. (2010). Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, 28( 10), 2111-2119. doi:10.1097/HJH.0b013e32833ca68c
NLM
Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
Vancouver
Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c

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