ReP USP - Resultado da busca

Artigo de periodico
ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma (2018)
Guerra, Heydi Noriega; Cruz, Mário Costa; Ribeiro, Priscilla Ramos Lara; Strnadel, Jan; Wang, Huawei; Klemke, Richard L; Jaeger, Ruy Gastaldoni ; Freitas, Vanessa Morais
Source: Experimental Cell Research. Unidade: ICB
Subjects: FIBROSSARCOMA, PROLIFERAÇÃO CELULAR, MIGRAÇÃO, BIOLOGIA CELULAR
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ABNT
GUERRA, Heydi Noriega et al. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, v. 363, n. 2, p. 271-282, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2018.01.017. Acesso em: 05 nov. 2024.
APA
Guerra, H. N., Cruz, M. C., Ribeiro, P. R. L., Strnadel, J., Wang, H., Klemke, R. L., et al. (2018). ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, 363( 2), 271-282. doi:10.1016/j.yexcr.2018.01.017
NLM
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017
Vancouver
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017
Artigo de periodico
The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer (2017)
Smuczeka, Basilio; Santos, Emerson de S.; Siqueira, Adriane S.; Pinheiro, Joao J. V.; Freitas, Vanessa Morais ; Jaeger, Ruy Gastaldoni
Source: Experimental Cell Research. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
SMUCZEKA, Basilio et al. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, v. 358, n. 2, p. 323-334, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2017.07.005. Acesso em: 05 nov. 2024.
APA
Smuczeka, B., Santos, E. de S., Siqueira, A. S., Pinheiro, J. J. V., Freitas, V. M., & Jaeger, R. G. (2017). The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, 358( 2), 323-334. doi:10.1016/j.yexcr.2017.07.005
NLM
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Vancouver
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Artigo de periodico
Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation (2016)
Ribeiro, André Luis Ribeiro; Costa, Natacha Malu Miranda; Siqueira, Adriane Sousa de; Silva, Walessa Brasil da; Kataoka, Maria Sueli da Silva; Jaeger, Ruy Gastaldoni ; Alves Junior, Sérgio de Melo; Smith, Andrew M; Pinheiro, João de Jesus Viana
Source: Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. Unidade: ICB
Subjects: TUMORES ODONTOGÊNICOS, CISTO ODONTOGÊNICO CALCIFICANTE, MUCOSA ORAL
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ABNT
RIBEIRO, André Luis Ribeiro et al. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, v. 122, n. 4, p. 500-508, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.oooo.2016.06.004. Acesso em: 05 nov. 2024.
APA
Ribeiro, A. L. R., Costa, N. M. M., Siqueira, A. S. de, Silva, W. B. da, Kataoka, M. S. da S., Jaeger, R. G., et al. (2016). Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, 122( 4), 500-508. doi:10.1016/j.oooo.2016.06.004
NLM
Ribeiro ALR, Costa NMM, Siqueira AS de, Silva WB da, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2016 ; 122( 4): 500-508.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.oooo.2016.06.004
Vancouver
Ribeiro ALR, Costa NMM, Siqueira AS de, Silva WB da, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2016 ; 122( 4): 500-508.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.oooo.2016.06.004
Artigo de periodico
Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor (2016)
Costa, Natacha Malu Miranda da; Fialho, Amanda Dalla Vechia; Proietti, Carolina Carmine; Kataoka, Maria Sueli da Silva; Jaeger, Ruy Gastaldoni ; Alves-Júnior, Sérgio Melo; Pinheiro, João de Jesus Viana
Source: Histopathology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Natacha Malu Miranda da et al. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor. Histopathology, v. 69, n. 1, p. 99-106, 2016Tradução . . Disponível em: https://doi.org/10.1111/his.12922. Acesso em: 05 nov. 2024.
APA
Costa, N. M. M. da, Fialho, A. D. V., Proietti, C. C., Kataoka, M. S. da S., Jaeger, R. G., Alves-Júnior, S. M., & Pinheiro, J. de J. V. (2016). Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor. Histopathology, 69( 1), 99-106. doi:10.1111/his.12922
NLM
Costa NMM da, Fialho ADV, Proietti CC, Kataoka MS da S, Jaeger RG, Alves-Júnior SM, Pinheiro J de JV. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor [Internet]. Histopathology. 2016 ; 69( 1): 99-106.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/his.12922
Vancouver
Costa NMM da, Fialho ADV, Proietti CC, Kataoka MS da S, Jaeger RG, Alves-Júnior SM, Pinheiro J de JV. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor [Internet]. Histopathology. 2016 ; 69( 1): 99-106.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/his.12922

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