ReP USP - Resultado da busca

Artigo de periodico
ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma (2018)
Guerra, Heydi Noriega; Cruz, Mário Costa; Ribeiro, Priscilla Ramos Lara; Strnadel, Jan; Wang, Huawei; Klemke, Richard L; Jaeger, Ruy Gastaldoni ; Freitas, Vanessa Morais
Source: Experimental Cell Research. Unidade: ICB
Subjects: FIBROSSARCOMA, PROLIFERAÇÃO CELULAR, MIGRAÇÃO, BIOLOGIA CELULAR
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUERRA, Heydi Noriega et al. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, v. 363, n. 2, p. 271-282, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2018.01.017. Acesso em: 05 nov. 2024.
APA
Guerra, H. N., Cruz, M. C., Ribeiro, P. R. L., Strnadel, J., Wang, H., Klemke, R. L., et al. (2018). ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma. Experimental Cell Research, 363( 2), 271-282. doi:10.1016/j.yexcr.2018.01.017
NLM
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017
Vancouver
Guerra HN, Cruz MC, Ribeiro PRL, Strnadel J, Wang H, Klemke RL, Jaeger RG, Freitas VM. ADAMTS-1 disrupts HGF/c-MET signaling and HGF-stimulated cellular processes in fibrosarcoma [Internet]. Experimental Cell Research. 2018 ; 363( 2): 271-282.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2018.01.017
Artigo de periodico
The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer (2017)
Smuczeka, Basilio; Santos, Emerson de S.; Siqueira, Adriane S.; Pinheiro, Joao J. V.; Freitas, Vanessa Morais ; Jaeger, Ruy Gastaldoni
Source: Experimental Cell Research. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SMUCZEKA, Basilio et al. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, v. 358, n. 2, p. 323-334, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2017.07.005. Acesso em: 05 nov. 2024.
APA
Smuczeka, B., Santos, E. de S., Siqueira, A. S., Pinheiro, J. J. V., Freitas, V. M., & Jaeger, R. G. (2017). The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, 358( 2), 323-334. doi:10.1016/j.yexcr.2017.07.005
NLM
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Vancouver
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Artigo de periodico
Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor (2016)
Costa, Natacha Malu Miranda da; Fialho, Amanda Dalla Vechia; Proietti, Carolina Carmine; Kataoka, Maria Sueli da Silva; Jaeger, Ruy Gastaldoni ; Alves-Júnior, Sérgio Melo; Pinheiro, João de Jesus Viana
Source: Histopathology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, Natacha Malu Miranda da et al. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor. Histopathology, v. 69, n. 1, p. 99-106, 2016Tradução . . Disponível em: https://doi.org/10.1111/his.12922. Acesso em: 05 nov. 2024.
APA
Costa, N. M. M. da, Fialho, A. D. V., Proietti, C. C., Kataoka, M. S. da S., Jaeger, R. G., Alves-Júnior, S. M., & Pinheiro, J. de J. V. (2016). Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor. Histopathology, 69( 1), 99-106. doi:10.1111/his.12922
NLM
Costa NMM da, Fialho ADV, Proietti CC, Kataoka MS da S, Jaeger RG, Alves-Júnior SM, Pinheiro J de JV. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor [Internet]. Histopathology. 2016 ; 69( 1): 99-106.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/his.12922
Vancouver
Costa NMM da, Fialho ADV, Proietti CC, Kataoka MS da S, Jaeger RG, Alves-Júnior SM, Pinheiro J de JV. Role of hypoxia-related proteins in invasion of ameloblastoma cells: crosstalk between NOTCH1, hypoxia-inducible factor 1α, a disintegrin and metalloproteinase 12, and heparin-binding epidermal growth factor [Internet]. Histopathology. 2016 ; 69( 1): 99-106.[citado 2024 nov. 05 ] Available from: https://doi.org/10.1111/his.12922

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