Filtros : "Indexado na Base de Dados Science Citation Index" "ROSSONI, LUCIANA VENTURINI" "ICB" Removidos: "Fisiologia Humana" "Center for Regenerative Medicine, USF Health Heart Institute, University of South Florida, Tampa, FL, USA" "RUSP" Limpar

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  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIComo citar
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    • ABNT

      PACHECO, Bruna P. M. et al. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, v. 29, n. 3, p. 520-528, 2011Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 13 nov. 2024.
    • APA

      Pacheco, B. P. M., Crajoinas, R. O., Couto, G. K., Davel, A. P. C., Lessa, L. M., Rossoni, L. V., & Girardi, A. C. C. (2011). Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, 29( 3), 520-528. doi:10.1097/HJH.0b013e32833ca68c
    • NLM

      Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
    • Vancouver

      Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
  • Fonte: American Journal of Physiology. Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

    Acesso à fonteDOIComo citar
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    • ABNT

      CRAJOINAS, Renato O. et al. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, v. 299, n. 4, p. F872-F881, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00654.2009. Acesso em: 13 nov. 2024.
    • APA

      Crajoinas, R. O., Lessa, L. M. A., Carraro-Lacroix, L. R., Davel, A. P. C., Pacheco, B. P. M., Rossoni, L. V., et al. (2010). Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, 299( 4), F872-F881. doi:10.1152/ajprenal.00654.2009
    • NLM

      Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
    • Vancouver

      Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
  • Fonte: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

    Acesso à fonteDOIComo citar
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    • ABNT

      GALLO, Luana C. et al. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, v. 87, n. 9-10 p. 302-308, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.005. Acesso em: 13 nov. 2024.
    • APA

      Gallo, L. C., Davel, A. P. C., Xavier, F. E., & Rossoni, L. V. (2010). Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, 87( 9-10 p. 302-308). doi:10.1016/j.lfs.2010.07.005
    • NLM

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
    • Vancouver

      Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA (HUMANO)

    Acesso à fonteDOIComo citar
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      KOBAYASI, Renata et al. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, v. 28, n. 10, p. 2111-2119, 2010Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 13 nov. 2024.
    • APA

      Kobayasi, R., Akamine, E. H., Davel, A. P. C., Rodrigues, M. A. M., Carvalho, C. R. de O., & Rossoni, L. V. (2010). Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, 28( 10), 2111-2119. doi:10.1097/HJH.0b013e32833ca68c
    • NLM

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
    • Vancouver

      Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c

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