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Filtros : "Indexado na Base de Dados MEDLINE" "Journal of Molecular Medicine" Removidos: "2020" "Coordenação de Aperfeiçoamento de Pessoal de Nível Superior (CAPES)" Limpar

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  • Artigo de periodico

    S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone (2017)

    Takano, Ana Paula Cremasco ; Munhoz, Carolina Demarchi ; Moriscot, Anselmo Sigari ; Gupta, Sudhiranjan; Barreto-Chaves, Maria Luiza Morais

    Fonte: Journal of Molecular Medicine. Unidade: ICB

    Assunto: ANATOMIA

    Acesso à fonteDOIComo citar
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    • ABNT

      TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 09 nov. 2024.

    • APA

      Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y

    • NLM

      Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-017-1511-y

    • Vancouver

      Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-017-1511-y

  • Artigo de periodico

    New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases (2015)

    Disatnik, Marie-Hélène; Hwang, Sunhee; Ferreira, Julio Cesar Batista ; Mochly-Rosen, Daria

    Fonte: Journal of Molecular Medicine. Unidade: ICB

    Assuntos: ANATOMIA, DOENÇAS CARDIOVASCULARES, MITOCÔNDRIAS

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    • ABNT

      DISATNIK, Marie-Hélène et al. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases. Journal of Molecular Medicine, v. 93, n. 3, p. 279-287, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00109-015-1256-4. Acesso em: 09 nov. 2024.

    • APA

      Disatnik, M. -H., Hwang, S., Ferreira, J. C. B., & Mochly-Rosen, D. (2015). New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases. Journal of Molecular Medicine, 93( 3), 279-287. doi:10.1007/s00109-015-1256-4

    • NLM

      Disatnik M-H, Hwang S, Ferreira JCB, Mochly-Rosen D. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases [Internet]. Journal of Molecular Medicine. 2015 ; 93( 3): 279-287.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-015-1256-4

    • Vancouver

      Disatnik M-H, Hwang S, Ferreira JCB, Mochly-Rosen D. New therapeutics to modulate mitochondrial dynamics and mitophagy in cardiac diseases [Internet]. Journal of Molecular Medicine. 2015 ; 93( 3): 279-287.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-015-1256-4

  • Artigo de periodico

    Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)

    Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.

    Fonte: Journal of Molecular Medicine. Unidade: ICB

    Assuntos: FISIOLOGIA, HISTOLOGIA

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    • ABNT

      MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 09 nov. 2024.

    • APA

      Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2

    • NLM

      Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-013-1105-2

    • Vancouver

      Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 nov. 09 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
  • 1

1 - 3 (3 registros)

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