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Artigo de periodico
Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats (2016)
Rodrigues, Hosana Gomes; Vinolo, Marco Aurélio Ramirez; Sato, Fabio Takeo; Magdalon, Juliana; Kuhl, Carolina Meneguetti Cintra; Yamagata, Ana Sayuri; Pessoa, Ana Flávia Marçal; Moraes, Gabriélla Malheiros; Santos, Marinilce Fagundes dos ; Lima, Camila; Farsky, Sandra Helena Poliselli ; Câmara, Niels Olsen Saraiva ; Williner, Maria Rosa; Bernal, Claudio Adrián; Calder,Philip C; Curi, Rui
Source: PLOS ONE. Unidades: ICB, FCF
Subjects: ÁCIDO LINOLEICO, RATOS, CICATRIZAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RODRIGUES, Hosana Gomes et al. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, v. 11, n. 10, p. e0165115, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0165115. Acesso em: 19 jul. 2024.
APA
Rodrigues, H. G., Vinolo, M. A. R., Sato, F. T., Magdalon, J., Kuhl, C. M. C., Yamagata, A. S., et al. (2016). Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats. PLOS ONE, 11( 10), e0165115. doi:10.1371/journal.pone.0165115
NLM
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Vancouver
Rodrigues HG, Vinolo MAR, Sato FT, Magdalon J, Kuhl CMC, Yamagata AS, Pessoa AFM, Moraes GM, Santos MF dos, Lima C, Farsky SHP, Câmara NOS, Williner MR, Bernal CA, Calder PC, Curi R. Oral administration of linoleic acid induces new vessel formation and improves skin wound healing in diabetic rats [Internet]. PLOS ONE. 2016 ; 11( 10): e0165115.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0165115
Artigo de periodico
Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats (2016)
Munhoz, Ana Cláudia; Riva, Patrícia; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MUNHOZ, Ana Cláudia et al. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, v. 11, n. 6, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0158166. Acesso em: 19 jul. 2024.
APA
Munhoz, A. C., Riva, P., Simões, D., Curi, R., & Carpinelli, A. R. (2016). Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats. PLOS ONE, 11( 6), 1-17. doi:10.1371/journal.pone.0158166
NLM
Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0158166
Vancouver
Munhoz AC, Riva P, Simões D, Curi R, Carpinelli AR. Control of insulin secretion by production of reactive oxygen species:: study performed in pancreatic islets from fed and 48-Hour fasted wistar rats [Internet]. PLOS ONE. 2016 ; 11( 6): 1-17.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0158166
Artigo de periodico
NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells (2015)
Kuwabara, Wilson Mitsuo Tatagiba; Zhang, Liling; Schuiki, Irmgard; Curi, Rui; Volchuk, Allen; Loureiro, Tatiana Carolina Alba
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, ATIVAÇÃO ENZIMÁTICA, NEUTRÓFILOS, LEUCEMIA, RETICULO ENDOPLASMÁTICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KUWABARA, Wilson Mitsuo Tatagiba et al. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, v. 10, n. 2, p. 1-15, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0116410. Acesso em: 19 jul. 2024.
APA
Kuwabara, W. M. T., Zhang, L., Schuiki, I., Curi, R., Volchuk, A., & Loureiro, T. C. A. (2015). NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells. PLOS ONE, 10( 2), 1-15. doi:10.1371/journal.pone.0116410
NLM
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Vancouver
Kuwabara WMT, Zhang L, Schuiki I, Curi R, Volchuk A, Loureiro TCA. NADPH oxidase-dependent production of reactive oxygen species induces endoplasmatic reticulum stress in neutrophil-like HL60 cells [Internet]. PLOS ONE. 2015 ; 10( 2): 1-15.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0116410
Artigo de periodico
Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation (2013)
Hatanaka, Elaine; Dermargos, Alexandre; Hirata, Aparecida Emiko; Vinolo, Marco Aurélio Ramirez; Carpinelli, Angelo Rafael ; Newsholme, Philip; Armelin, Hugo Aguirre ; Curi, Rui
Source: PLOS ONE. Unidades: ICB, IQ
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HATANAKA, Elaine et al. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation. PLOS ONE, v. 8, p. 1-8, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0058626. Acesso em: 19 jul. 2024.
APA
Hatanaka, E., Dermargos, A., Hirata, A. E., Vinolo, M. A. R., Carpinelli, A. R., Newsholme, P., et al. (2013). Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation. PLOS ONE, 8, 1-8. doi:10.1371/journal.pone.0058626
NLM
Hatanaka E, Dermargos A, Hirata AE, Vinolo MAR, Carpinelli AR, Newsholme P, Armelin HA, Curi R. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation [Internet]. PLOS ONE. 2013 ; 8 1-8.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0058626
Vancouver
Hatanaka E, Dermargos A, Hirata AE, Vinolo MAR, Carpinelli AR, Newsholme P, Armelin HA, Curi R. Oleic, linoleic and linolenic acids increase ROS production by fibroblasts via NADPH oxidase activation [Internet]. PLOS ONE. 2013 ; 8 1-8.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0058626
Artigo de periodico
Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats (2013)
Vitzel, Kaio Fernando; Bikopoulos, George; Hung, Steven; Curi, Rui; Ceddia, Rolando B.
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
VITZEL, Kaio Fernando et al. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, v. 8, n. 8, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0071944. Acesso em: 19 jul. 2024.
APA
Vitzel, K. F., Bikopoulos, G., Hung, S., Curi, R., & Ceddia, R. B. (2013). Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats. PLOS ONE, 8( 8), 1-10. doi:10.1371/journal.pone.0071944
NLM
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0071944
Vancouver
Vitzel KF, Bikopoulos G, Hung S, Curi R, Ceddia RB. Loss of the anorexic response to systemic 5-aminoimidazole-4-carboxamide-1-β-D-ribofuranoside administration despite reducing hypothalamic AMP-activated protein kinase phosphorylation in insulin- deficient rats [Internet]. PLOS ONE. 2013 ; 8( 8): 1-10.[citado 2024 jul. 19 ] Available from: https://doi.org/10.1371/journal.pone.0071944

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