Filtros : "Indexado na Base de Dados MEDLINE" "Kinote, Andrezza" "2013" Removidos: "DEGRADAÇÃO ELETROQUÍMICA" "MSDA Animal Health. São Paulo, SP" "ICMC/UFSCar" Limpar

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  • Source: American Journal of Physiology, Endocrinology and Methabolism. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      FARIA, Juliana A. et al. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, v. 305, n. 2, p. E230-E242, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2013. Acesso em: 02 jul. 2024.
    • APA

      Faria, J. A., Kinote, A., Ignacio-Souza, L. M., Araújo, T. M. de, Razolli, D. S., Doneda, D. L., et al. (2013). Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, 305( 2), E230-E242. doi:10.1152/ajpendo.00094.2013
    • NLM

      Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
    • Vancouver

      Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
  • Source: European Journal Pharmacology. Unidade: ICB

    Assunto: FISIOLOGIA

    Acesso à fonteDOIHow to cite
    A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
    • ABNT

      GAZZOTO FILHO, Ademir et al. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats. European Journal Pharmacology, v. 700, n. 1-3, p. 201-209, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2012.11.059. Acesso em: 02 jul. 2024.
    • APA

      Gazzoto Filho, A., Kinote, A., Pereira, D. J., Rennó, A., Santos, R. C. dos, Ferreira-Melo, S. E., et al. (2013). Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats. European Journal Pharmacology, 700( 1-3), 201-209. doi:10.1016/j.ejphar.2012.11.059
    • NLM

      Gazzoto Filho A, Kinote A, Pereira DJ, Rennó A, Santos RC dos, Ferreira-Melo SE, Velloso LA, Bordin S, Anhê GF, Moreno Junior H. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats [Internet]. European Journal Pharmacology. 2013 ; 700( 1-3): 201-209.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2012.11.059
    • Vancouver

      Gazzoto Filho A, Kinote A, Pereira DJ, Rennó A, Santos RC dos, Ferreira-Melo SE, Velloso LA, Bordin S, Anhê GF, Moreno Junior H. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats [Internet]. European Journal Pharmacology. 2013 ; 700( 1-3): 201-209.[citado 2024 jul. 02 ] Available from: https://doi.org/10.1016/j.ejphar.2012.11.059

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