Filtros : "Indexado na Base de Dados Index Medicus" "ROSSONI, LUCIANA VENTURINI" "ICB" Removidos: "Fisiologia Humana" "2002" "RUSP" Limpar

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  • Fonte: American Journal of Physiology. Renal Physiology. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      CASARE, Fernando Augusto Malavazzi et al. Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, v. 310, n. 11, p. F1295-F1307, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00471.2015. Acesso em: 07 set. 2024.
    • APA

      Casare, F. A. M., Thieme, K., Costa-Pessoa, J. M., Rossoni, L. V., Couto, G. K., Fernandes, F. B., et al. (2016). Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, 310( 11), F1295-F1307. doi:10.1152/ajprenal.00471.2015
    • NLM

      Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
    • Vancouver

      Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 set. 07 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
  • Fonte: Journal of Molecular and Cellular Cardiology. Unidade: ICB

    Assuntos: FISIOLOGIA, VASOS CORONÁRIOS, ÓXIDO NÍTRICO, RATOS, INFARTO NO MIOCÁRDIO

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    • ABNT

      COUTO, Gisele Kruger et al. Enhanced nitric oxide bioavailability in coronary arteries prevents the onset of heart failure in rats with myocardial infarction. Journal of Molecular and Cellular Cardiology, v. 86, p. 110-120, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.yjmcc.2015.07.017. Acesso em: 07 set. 2024.
    • APA

      Couto, G. K., Britto, L. R. G. de, Mill, J. G., & Rossoni, L. V. (2015). Enhanced nitric oxide bioavailability in coronary arteries prevents the onset of heart failure in rats with myocardial infarction. Journal of Molecular and Cellular Cardiology, 86, 110-120. doi:10.1016/j.yjmcc.2015.07.017
    • NLM

      Couto GK, Britto LRG de, Mill JG, Rossoni LV. Enhanced nitric oxide bioavailability in coronary arteries prevents the onset of heart failure in rats with myocardial infarction [Internet]. Journal of Molecular and Cellular Cardiology. 2015 ; 86 110-120.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.yjmcc.2015.07.017
    • Vancouver

      Couto GK, Britto LRG de, Mill JG, Rossoni LV. Enhanced nitric oxide bioavailability in coronary arteries prevents the onset of heart failure in rats with myocardial infarction [Internet]. Journal of Molecular and Cellular Cardiology. 2015 ; 86 110-120.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.yjmcc.2015.07.017
  • Fonte: Hypertension. Unidade: ICB

    Assuntos: FISIOLOGIA, FISIOLOGIA CARDIOVASCULAR, HIPERTENSÃO, HIPERTENSÃO, INIBIDORES QUÍMICOS

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    • ABNT

      MARTÍNEZ-MARTÍNEZ, Ernesto et al. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, v. 66, n. 4, p. 767-775, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876. Acesso em: 07 set. 2024.
    • APA

      Martínez-Martínez, E., Calvier, L., Fernández-Celis, A., Rousseau, E., Jurado-López, R., Rossoni, L. V., et al. (2015). Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, 66( 4), 767-775. doi:10.1161/HYPERTENSIONAHA.115.05876
    • NLM

      Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 set. 07 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
    • Vancouver

      Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 set. 07 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
  • Fonte: British Journal of Pharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, CÉLULAS ENDOTELIAIS, ÓLEOS ESSENCIAIS

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    • ABNT

      PEIXOTO-NEVES, Dieniffer et al. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels. British Journal of Pharmacology, v. 172, n. 14, p. 3484-3494, 2015Tradução . . Disponível em: https://doi.org/10.1111/bph.13156. Acesso em: 07 set. 2024.
    • APA

      Peixoto-Neves, D., Wang, Q., Leal-Cardoso, J. H., Rossoni, L. V., & Jaggar, J. H. (2015). Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels. British Journal of Pharmacology, 172( 14), 3484-3494. doi:10.1111/bph.13156
    • NLM

      Peixoto-Neves D, Wang Q, Leal-Cardoso JH, Rossoni LV, Jaggar JH. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels [Internet]. British Journal of Pharmacology. 2015 ; 172( 14): 3484-3494.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/bph.13156
    • Vancouver

      Peixoto-Neves D, Wang Q, Leal-Cardoso JH, Rossoni LV, Jaggar JH. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels [Internet]. British Journal of Pharmacology. 2015 ; 172( 14): 3484-3494.[citado 2024 set. 07 ] Available from: https://doi.org/10.1111/bph.13156
  • Fonte: PLOS ONE. Unidade: ICB

    Assuntos: FISIOLOGIA, EXERCICIO FÍSICO, ÓXIDO NITRICO, MENOPAUSA, ENDOTÉLIO

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    • ABNT

      BRAGA, Viviane A. V. N. et al. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, v. 10, n. 4, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0125388. Acesso em: 07 set. 2024.
    • APA

      Braga, V. A. V. N., Couto, G. K., Lazzarin, M. C., Rossoni, L. V., & Medeiros, A. (2015). Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, 10( 4), 1-13. doi:10.1371/journal.pone.0125388
    • NLM

      Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0125388
    • Vancouver

      Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 set. 07 ] Available from: https://doi.org/10.1371/journal.pone.0125388
  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      WENCESLAU, Camilla F. e ROSSONI, Luciana Venturini. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, v. 32, n. 3, p. 542-555, 2014Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000000059. Acesso em: 07 set. 2024.
    • APA

      Wenceslau, C. F., & Rossoni, L. V. (2014). Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, 32( 3), 542-555. doi:10.1097/HJH.0000000000000059
    • NLM

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
    • Vancouver

      Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
  • Fonte: Life Sciences. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      FONTES, M. T. et al. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, v. 94, n. 1, p. 24-29, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.017. Acesso em: 07 set. 2024.
    • APA

      Fontes, M. T., Silva, T. L. B. T., Mota, M. M., Barreto, A. S., Rossoni, L. V., & Santos, M. R. V. (2014). Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, 94( 1), 24-29. doi:10.1016/j.lfs.2013.11.017
    • NLM

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
    • Vancouver

      Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
  • Fonte: Journal of Neuroinflammation. Unidade: ICB

    Assuntos: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

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    • ABNT

      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 07 set. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 set. 07 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Fonte: Neuropharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 07 set. 2024.
    • APA

      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 set. 07 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Fonte: Journal of Hypertension. Unidade: ICB

    Assunto: FISIOLOGIA

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    • ABNT

      BLANCO-RIVERO, Javier et al. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, v. 31, n. 5, p. 916-926, 2013Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32835f749c. Acesso em: 07 set. 2024.
    • APA

      Blanco-Rivero, J., Roque, F. R., Sastre, E., Caracuel, L., Couto, G. K., Avendaño, M. S., et al. (2013). Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, 31( 5), 916-926. doi:10.1097/HJH.0b013e32835f749c
    • NLM

      Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
    • Vancouver

      Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
  • Fonte: Shock. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      BREITHAUPT-FALOPPA, A. C. et al. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide. Shock, v. 40, n. 3, p. 203-209, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e3182a01e24. Acesso em: 07 set. 2024.
    • APA

      Breithaupt-Faloppa, A. C., Fantozzi, E. T., Assis-Ramos, M. M., Vitoretti, L. B., Couto, G. K., Rossoni, L. V., et al. (2013). Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide. Shock, 40( 3), 203-209. doi:10.1097/SHK.0b013e3182a01e24
    • NLM

      Breithaupt-Faloppa AC, Fantozzi ET, Assis-Ramos MM, Vitoretti LB, Couto GK, Rossoni LV, Oliveira-Filho RM, Vargaftig BB, Lima WT de. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide [Internet]. Shock. 2013 ; 40( 3): 203-209.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/SHK.0b013e3182a01e24
    • Vancouver

      Breithaupt-Faloppa AC, Fantozzi ET, Assis-Ramos MM, Vitoretti LB, Couto GK, Rossoni LV, Oliveira-Filho RM, Vargaftig BB, Lima WT de. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide [Internet]. Shock. 2013 ; 40( 3): 203-209.[citado 2024 set. 07 ] Available from: https://doi.org/10.1097/SHK.0b013e3182a01e24

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