Filtros : "Indexado na Base de Dados Index Medicus" "ICB-BMF" "2014" Removidos: "Carlotti Júnior, Carlos Gilberto" "FM-SD" Limpar

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  • Source: Journal of Neuroinflammation. Unidades: ICB, FMRP

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      HERNANDES, Marina S. et al. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, v. 11, n. 01, p. 01-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-36. Acesso em: 20 ago. 2024.
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      Hernandes, M. S., D'Ávila, J. C., Trevelin, S. C., Reis, P. A., Kinjo, E. R., Lopes, L. R., et al. (2014). The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, 11( 01), 01-12. doi:10.1186/1742-2094-11-36
    • NLM

      Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/1742-2094-11-36
    • Vancouver

      Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/1742-2094-11-36
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FARMACOLOGIA

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      DASGUPTA, Sayani et al. Proteasome inhibitors alter levels of intracellular peptides in HEK293T and SH-SY5Y cells. PLOS ONE, v. 9, n. 7, p. 1-15, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0103604. Acesso em: 20 ago. 2024.
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      Dasgupta, S., Castro, L. M. de, Dulman, R., Yang, C., Schmidt, M., Ferro, E. S., & Fricker, L. D. (2014). Proteasome inhibitors alter levels of intracellular peptides in HEK293T and SH-SY5Y cells. PLOS ONE, 9( 7), 1-15. doi:10.1371/journal.pone.0103604
    • NLM

      Dasgupta S, Castro LM de, Dulman R, Yang C, Schmidt M, Ferro ES, Fricker LD. Proteasome inhibitors alter levels of intracellular peptides in HEK293T and SH-SY5Y cells [Internet]. PLOS ONE. 2014 ; 9( 7): 1-15.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0103604
    • Vancouver

      Dasgupta S, Castro LM de, Dulman R, Yang C, Schmidt M, Ferro ES, Fricker LD. Proteasome inhibitors alter levels of intracellular peptides in HEK293T and SH-SY5Y cells [Internet]. PLOS ONE. 2014 ; 9( 7): 1-15.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0103604
  • Source: PLOS ONE. Unidade: ICB

    Assunto: FARMACOLOGIA

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      QUEIROZ, Eveline et al. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells. PLOS ONE, v. 9, n. 5, p. 1-18, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0098207. Acesso em: 20 ago. 2024.
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      Queiroz, E., Puukila, S., Eichler, R., Sampaio, S. C., Forsyth, H. L., Lees, S. J., et al. (2014). Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells. PLOS ONE, 9( 5), 1-18. doi:10.1371/journal.pone.0098207
    • NLM

      Queiroz E, Puukila S, Eichler R, Sampaio SC, Forsyth HL, Lees SJ, Barbosa AM, Dekker RFH, Fortes ZB, Khaper N. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells [Internet]. PLOS ONE. 2014 ; 9( 5): 1-18.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0098207
    • Vancouver

      Queiroz E, Puukila S, Eichler R, Sampaio SC, Forsyth HL, Lees SJ, Barbosa AM, Dekker RFH, Fortes ZB, Khaper N. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells [Internet]. PLOS ONE. 2014 ; 9( 5): 1-18.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0098207
  • Source: Developmental Psychobiology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CARRARA-NASCIMENTO, Priscila Fernandes e OLIVE, M. Foster e CAMARINI, Rosana. Ethanol pre-exposure during adolescence or adulthood increases ethanol intake but ethanol-induced conditioned place preference is enhanced only when pre-exposure occurs in adolescence. Developmental Psychobiology, v. 56, n. 01, p. 36-48, 2014Tradução . . Disponível em: https://doi.org/10.1002/dev.21089. Acesso em: 20 ago. 2024.
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      Carrara-Nascimento, P. F., Olive, M. F., & Camarini, R. (2014). Ethanol pre-exposure during adolescence or adulthood increases ethanol intake but ethanol-induced conditioned place preference is enhanced only when pre-exposure occurs in adolescence. Developmental Psychobiology, 56( 01), 36-48. doi:10.1002/dev.21089
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      Carrara-Nascimento PF, Olive MF, Camarini R. Ethanol pre-exposure during adolescence or adulthood increases ethanol intake but ethanol-induced conditioned place preference is enhanced only when pre-exposure occurs in adolescence [Internet]. Developmental Psychobiology. 2014 ; 56( 01): 36-48.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1002/dev.21089
    • Vancouver

      Carrara-Nascimento PF, Olive MF, Camarini R. Ethanol pre-exposure during adolescence or adulthood increases ethanol intake but ethanol-induced conditioned place preference is enhanced only when pre-exposure occurs in adolescence [Internet]. Developmental Psychobiology. 2014 ; 56( 01): 36-48.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1002/dev.21089
  • Source: Rheumatology International. Unidade: ICB

    Assunto: FARMACOLOGIA

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      BJORDAL, Jan M. et al. Methodological shortcomings make conclusion highly sensitive to relevant changes in review protocol. Rheumatology International. Heidelberg: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1007/s00296-013-2940-8. Acesso em: 20 ago. 2024. , 2014
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      Bjordal, J. M., Chow, R. T., Lopes-Martins, R. Á. B., & Johnson, M. I. (2014). Methodological shortcomings make conclusion highly sensitive to relevant changes in review protocol. Rheumatology International. Heidelberg: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1007/s00296-013-2940-8
    • NLM

      Bjordal JM, Chow RT, Lopes-Martins RÁB, Johnson MI. Methodological shortcomings make conclusion highly sensitive to relevant changes in review protocol [Internet]. Rheumatology International. 2014 ; 34( 8): 1181-1183.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1007/s00296-013-2940-8
    • Vancouver

      Bjordal JM, Chow RT, Lopes-Martins RÁB, Johnson MI. Methodological shortcomings make conclusion highly sensitive to relevant changes in review protocol [Internet]. Rheumatology International. 2014 ; 34( 8): 1181-1183.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1007/s00296-013-2940-8
  • Source: Journal of Neuroinflammation. Unidades: ICB, EACH

    Assunto: FARMACOLOGIA

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      VASCONCELOS, Andrea Rodrigues et al. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, v. 11, n. 85, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-85. Acesso em: 20 ago. 2024.
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      Vasconcelos, A. R., Yshii, L. M., Viel, T. A., Buck, H. S., Mattson, M. P., Scavone, C., & Kawamoto, E. M. (2014). Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, 11( 85), 1-14. doi:10.1186/1742-2094-11-85
    • NLM

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/1742-2094-11-85
    • Vancouver

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/1742-2094-11-85
  • Source: Journal of Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      ROBERTO SOBRINHO, Cleyton et al. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, v. 15, p. 1309-1323, 2014Tradução . . Disponível em: https://doi.org/10.1113/jphysiol.2013.268490. Acesso em: 20 ago. 2024.
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      Roberto Sobrinho, C., Wenker, I. C., Poss, E. M., Takakura, A. C., Moreira, T. dos S., & Mulkey, D. K. (2014). Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe. Journal of Physiology, 15, 1309-1323. doi:10.1113/jphysiol.2013.268490
    • NLM

      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
    • Vancouver

      Roberto Sobrinho C, Wenker IC, Poss EM, Takakura AC, Moreira T dos S, Mulkey DK. Purinergic signalling contributes to chemoreception in the retrotrapezoid nucleus but not the nucleus of the solitary tract or medullary raphe [Internet]. Journal of Physiology. 2014 ; 15 1309-1323.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1113/jphysiol.2013.268490
  • Source: International Journal of Environmental Research and Public Health. Unidade: ICB

    Assunto: FARMACOLOGIA

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      WANG, Yuan-Pang e GORENSTEIN, Clarice. Attitude and impact of perceived depression in the workplace. International Journal of Environmental Research and Public Health, v. 11, n. 6, p. 6021-6036, 2014Tradução . . Disponível em: https://doi.org/10.3390/ijerph110606021. Acesso em: 20 ago. 2024.
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      Wang, Y. -P., & Gorenstein, C. (2014). Attitude and impact of perceived depression in the workplace. International Journal of Environmental Research and Public Health, 11( 6), 6021-6036. doi:10.3390/ijerph110606021
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      Wang Y-P, Gorenstein C. Attitude and impact of perceived depression in the workplace [Internet]. International Journal of Environmental Research and Public Health. 2014 ; 11( 6): 6021-6036.[citado 2024 ago. 20 ] Available from: https://doi.org/10.3390/ijerph110606021
    • Vancouver

      Wang Y-P, Gorenstein C. Attitude and impact of perceived depression in the workplace [Internet]. International Journal of Environmental Research and Public Health. 2014 ; 11( 6): 6021-6036.[citado 2024 ago. 20 ] Available from: https://doi.org/10.3390/ijerph110606021
  • Source: Experimental Gerontology. Unidade: ICB

    Subjects: FARMACOLOGIA, CAMUNDONGO, VASCULARIZAÇÃO EM ANIMAL

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      DANTAS, Ana Paula et al. Western diet consumption promotes vascular remodeling in non-senescent mice consistent with accelerated senescence, but does not modify vascular morphology in senescent ones. Experimental Gerontology, v. 55, p. 1-11, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.exger.2014.03.004. Acesso em: 20 ago. 2024.
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      Dantas, A. P., Onetti, Y., Oliveira, M. A., Carvalho, M. H. C., Heras, M., Vila, E., & Jiménez-Altayó, F. (2014). Western diet consumption promotes vascular remodeling in non-senescent mice consistent with accelerated senescence, but does not modify vascular morphology in senescent ones. Experimental Gerontology, 55, 1-11. doi:10.1016/j.exger.2014.03.004
    • NLM

      Dantas AP, Onetti Y, Oliveira MA, Carvalho MHC, Heras M, Vila E, Jiménez-Altayó F. Western diet consumption promotes vascular remodeling in non-senescent mice consistent with accelerated senescence, but does not modify vascular morphology in senescent ones [Internet]. Experimental Gerontology. 2014 ; 55 1-11.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1016/j.exger.2014.03.004
    • Vancouver

      Dantas AP, Onetti Y, Oliveira MA, Carvalho MHC, Heras M, Vila E, Jiménez-Altayó F. Western diet consumption promotes vascular remodeling in non-senescent mice consistent with accelerated senescence, but does not modify vascular morphology in senescent ones [Internet]. Experimental Gerontology. 2014 ; 55 1-11.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1016/j.exger.2014.03.004
  • Source: Antioxidants & Redox Signaling. Unidade: ICB

    Assunto: FARMACOLOGIA

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      SANTOS, Celio X. C. et al. Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension. Antioxidants & Redox Signaling, v. 20, n. 1, p. 121-134, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.004. Acesso em: 20 ago. 2024.
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      Santos, C. X. C., Nabeebaccus, A. A., Shah, A. M., Camargo, L. de L., Filho, S. V., & Lopes, L. R. (2014). Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension. Antioxidants & Redox Signaling, 20( 1), 121-134. doi:10.1016/j.lfs.2013.11.004
    • NLM

      Santos CXC, Nabeebaccus AA, Shah AM, Camargo L de L, Filho SV, Lopes LR. Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension [Internet]. Antioxidants & Redox Signaling. 2014 ; 20( 1): 121-134.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
    • Vancouver

      Santos CXC, Nabeebaccus AA, Shah AM, Camargo L de L, Filho SV, Lopes LR. Endoplasmic reticulum stress and Nox-mediated reactive oxygen species signaling in the peripheral vasculature: potential role in hypertension [Internet]. Antioxidants & Redox Signaling. 2014 ; 20( 1): 121-134.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
  • Source: American Academy Journal of Periodontology. Unidade: ICB

    Assunto: FARMACOLOGIA

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      HERRERA, Bruno S. et al. Peripheral blood mononuclear phagocytes from patients with chronic periodontitis are primed for osteoclast formation. American Academy Journal of Periodontology, v. 85, n. 4, p. e72-e81, 2014Tradução . . Disponível em: https://doi.org/10.1902/jop.2013.130280. Acesso em: 20 ago. 2024.
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      Herrera, B. S., Bastos, A. S., Coimbra, L. S., Teixeira, S. A., Rossa Junior, C., Dyke, T. E. V., et al. (2014). Peripheral blood mononuclear phagocytes from patients with chronic periodontitis are primed for osteoclast formation. American Academy Journal of Periodontology, 85( 4), e72-e81. doi:10.1902/jop.2013.130280
    • NLM

      Herrera BS, Bastos AS, Coimbra LS, Teixeira SA, Rossa Junior C, Dyke TEV, Muscará MN, Spolidorio LC. Peripheral blood mononuclear phagocytes from patients with chronic periodontitis are primed for osteoclast formation [Internet]. American Academy Journal of Periodontology. 2014 ; 85( 4): e72-e81.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1902/jop.2013.130280
    • Vancouver

      Herrera BS, Bastos AS, Coimbra LS, Teixeira SA, Rossa Junior C, Dyke TEV, Muscará MN, Spolidorio LC. Peripheral blood mononuclear phagocytes from patients with chronic periodontitis are primed for osteoclast formation [Internet]. American Academy Journal of Periodontology. 2014 ; 85( 4): e72-e81.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1902/jop.2013.130280
  • Source: Experimental Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      TAKAKURA, Ana Carolina et al. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats. Experimental Physiology, v. 99, n. 3, p. 571-585, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.076752. Acesso em: 20 ago. 2024.
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      Takakura, A. C., Falquetto, B., Cruz, J. C., Colombari, E., & Moreira, T. dos S. (2014). Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats. Experimental Physiology, 99( 3), 571-585. doi:10.1113/expphysiol.2013.076752
    • NLM

      Takakura AC, Falquetto B, Cruz JC, Colombari E, Moreira T dos S. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats [Internet]. Experimental Physiology. 2014 ; 99( 3): 571-585.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1113/expphysiol.2013.076752
    • Vancouver

      Takakura AC, Falquetto B, Cruz JC, Colombari E, Moreira T dos S. Phox2b-expressing retrotrapezoid neurons and the integration of central and peripheral chemosensory control of breathing in conscious rats [Internet]. Experimental Physiology. 2014 ; 99( 3): 571-585.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1113/expphysiol.2013.076752
  • Source: American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA, FISIOLOGIA RESPIRATÓRIA E CIRCULATÓRIA, RESPIRAÇÃO

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      DAMASCENO, Rosélia S. e TAKAKURA, Ana Carolina e MOREIRA, Thiago dos Santos. Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, v. 307, n. 1, p. R57-67, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpregu.00024.2014. Acesso em: 20 ago. 2024.
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      Damasceno, R. S., Takakura, A. C., & Moreira, T. dos S. (2014). Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 307( 1), R57-67. doi:10.1152/ajpregu.00024.2014
    • NLM

      Damasceno RS, Takakura AC, Moreira T dos S. Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 1): R57-67.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1152/ajpregu.00024.2014
    • Vancouver

      Damasceno RS, Takakura AC, Moreira T dos S. Regulation of the chemosensory control of breathing by Kölliker-Fuse neurons [Internet]. American Journal of Physiology. Regulatory, Integrative and Comparative Physiology. 2014 ; 307( 1): R57-67.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1152/ajpregu.00024.2014
  • Source: European Journal of Pain. Unidade: ICB

    Assunto: FARMACOLOGIA

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      CAMARGO, E. A. et al. Inhibition of inducible nitric oxide synthase-derived nitric oxide as a therapeutical target for acute pancreatitis induced by secretory phospholipase A2. European Journal of Pain, v. 18, n. 5, p. 691-700, 2014Tradução . . Disponível em: https://doi.org/10.1002/j.1532-2149.2013.00414.x. Acesso em: 20 ago. 2024.
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      Camargo, E. A., Santana, D. G., Silva, C. I., Teixeira, S. A., Toyama, M. H., Cotrim, C., et al. (2014). Inhibition of inducible nitric oxide synthase-derived nitric oxide as a therapeutical target for acute pancreatitis induced by secretory phospholipase A2. European Journal of Pain, 18( 5), 691-700. doi:10.1002/j.1532-2149.2013.00414.x
    • NLM

      Camargo EA, Santana DG, Silva CI, Teixeira SA, Toyama MH, Cotrim C, Landucci ECT, Antunes E, Muscará MN, Costa SKP. Inhibition of inducible nitric oxide synthase-derived nitric oxide as a therapeutical target for acute pancreatitis induced by secretory phospholipase A2 [Internet]. European Journal of Pain. 2014 ; 18( 5): 691-700.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1002/j.1532-2149.2013.00414.x
    • Vancouver

      Camargo EA, Santana DG, Silva CI, Teixeira SA, Toyama MH, Cotrim C, Landucci ECT, Antunes E, Muscará MN, Costa SKP. Inhibition of inducible nitric oxide synthase-derived nitric oxide as a therapeutical target for acute pancreatitis induced by secretory phospholipase A2 [Internet]. European Journal of Pain. 2014 ; 18( 5): 691-700.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1002/j.1532-2149.2013.00414.x
  • Source: Lasers in Medical Science. Unidade: ICB

    Assunto: FARMACOLOGIA

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      ALMEIDA, Patricia de et al. What is the best treatment to decrease pro-inflammatory cytokine release in acute skeletal muscle injury induced by trauma in rats: low-level laser therapy, diclofenac, or cryotherapy?. Lasers in Medical Science, v. 29, n. 2, p. 653-658, 2014Tradução . . Disponível em: https://doi.org/10.1007/s10103-013-1377-3. Acesso em: 20 ago. 2024.
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      Almeida, P. de, Tomazoni, S. S., Frigo, L., Carvalho, P. de T. C. de, Vanin, A. A., Santos, L. A., et al. (2014). What is the best treatment to decrease pro-inflammatory cytokine release in acute skeletal muscle injury induced by trauma in rats: low-level laser therapy, diclofenac, or cryotherapy? Lasers in Medical Science, 29( 2), 653-658. doi:10.1007/s10103-013-1377-3
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      Almeida P de, Tomazoni SS, Frigo L, Carvalho P de TC de, Vanin AA, Santos LA, Albuquerque-Pontes GM, De Marchi T, Tairova O, Marcos RL, Lopes-Martins RÁB, Leal-Junior ECP. What is the best treatment to decrease pro-inflammatory cytokine release in acute skeletal muscle injury induced by trauma in rats: low-level laser therapy, diclofenac, or cryotherapy? [Internet]. Lasers in Medical Science. 2014 ; 29( 2): 653-658.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1007/s10103-013-1377-3
    • Vancouver

      Almeida P de, Tomazoni SS, Frigo L, Carvalho P de TC de, Vanin AA, Santos LA, Albuquerque-Pontes GM, De Marchi T, Tairova O, Marcos RL, Lopes-Martins RÁB, Leal-Junior ECP. What is the best treatment to decrease pro-inflammatory cytokine release in acute skeletal muscle injury induced by trauma in rats: low-level laser therapy, diclofenac, or cryotherapy? [Internet]. Lasers in Medical Science. 2014 ; 29( 2): 653-658.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1007/s10103-013-1377-3
  • Source: PLOS ONE. Unidade: ICB

    Subjects: FISIOLOGIA, FARMACOLOGIA

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      MASSON, Gustavo Santos et al. Time-dependent effects of training on cardiovascular control in spontaneously hypertensive rats: role for brain oxidative stress and inflammation and baroreflex sensitivity. PLOS ONE, v. 9, n. 5, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0094927. Acesso em: 20 ago. 2024.
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      Masson, G. S., Costa, T. S. R. da, Yshii, L., Fernandes, D. C., Soares, P. P. S., Laurindo, F. R., et al. (2014). Time-dependent effects of training on cardiovascular control in spontaneously hypertensive rats: role for brain oxidative stress and inflammation and baroreflex sensitivity. PLOS ONE, 9( 5), 1-10. doi:10.1371/journal.pone.0094927
    • NLM

      Masson GS, Costa TSR da, Yshii L, Fernandes DC, Soares PPS, Laurindo FR, Scavone C, Michelini LC. Time-dependent effects of training on cardiovascular control in spontaneously hypertensive rats: role for brain oxidative stress and inflammation and baroreflex sensitivity [Internet]. PLOS ONE. 2014 ; 9( 5): 1-10.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0094927
    • Vancouver

      Masson GS, Costa TSR da, Yshii L, Fernandes DC, Soares PPS, Laurindo FR, Scavone C, Michelini LC. Time-dependent effects of training on cardiovascular control in spontaneously hypertensive rats: role for brain oxidative stress and inflammation and baroreflex sensitivity [Internet]. PLOS ONE. 2014 ; 9( 5): 1-10.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone.0094927
  • Source: Journal of Biological Chemistry. Unidades: ICB, IQ

    Subjects: FENÓTIPOS, OLIGOPEPTÍDEOS

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    • ABNT

      CAVALCANTI, Diogo Manuel Lopes de Paiva et al. Neurolysin knockout mice generation and initial phenotype characterization. Journal of Biological Chemistry, v. 289, n. 22, p. 15426-15440, 2014Tradução . . Disponível em: https://doi.org/10.1074/jbc.M113.539148. Acesso em: 20 ago. 2024.
    • APA

      Cavalcanti, D. M. L. de P., Castro, L. M. de, Rosa Neto, J. C., Seelaender, M. C. L., Neves, R. X. das, Oliveira, V., et al. (2014). Neurolysin knockout mice generation and initial phenotype characterization. Journal of Biological Chemistry, 289( 22), 15426-15440. doi:10.1074/jbc.M113.539148
    • NLM

      Cavalcanti DML de P, Castro LM de, Rosa Neto JC, Seelaender MCL, Neves RX das, Oliveira V, Forti FL, Iwai LK, Gozzo FC, Todiras M, Schadock I, Barros CC, Bader M, Ferro ES. Neurolysin knockout mice generation and initial phenotype characterization [Internet]. Journal of Biological Chemistry. 2014 ; 289( 22): 15426-15440.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1074/jbc.M113.539148
    • Vancouver

      Cavalcanti DML de P, Castro LM de, Rosa Neto JC, Seelaender MCL, Neves RX das, Oliveira V, Forti FL, Iwai LK, Gozzo FC, Todiras M, Schadock I, Barros CC, Bader M, Ferro ES. Neurolysin knockout mice generation and initial phenotype characterization [Internet]. Journal of Biological Chemistry. 2014 ; 289( 22): 15426-15440.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1074/jbc.M113.539148
  • Source: Journal of Neuroinflammation. Unidade: ICB

    Subjects: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

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      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 20 ago. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Source: PLOS ONE. Unidade: ICB

    Subjects: FARMACOLOGIA, ANGIOTENSINA II, OBESIDADE, RATOS

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    • ABNT

      HAGIHARA, Graziela Neves et al. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats. PLOS ONE, v. 9, n. 8, p. 1-10, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone. Acesso em: 20 ago. 2024.
    • APA

      Hagihara, G. N., Lobato, N. S., Filgueira, F. P., Akamine, E. H., Aragão, D. S., Casarini, D. E., et al. (2014). Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats. PLOS ONE, 9( 8), 1-10. doi:10.1371/journal.pone
    • NLM

      Hagihara GN, Lobato NS, Filgueira FP, Akamine EH, Aragão DS, Casarini DE, Carvalho MHC de, Fortes ZB. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats [Internet]. PLOS ONE. 2014 ; 9( 8): 1-10.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone
    • Vancouver

      Hagihara GN, Lobato NS, Filgueira FP, Akamine EH, Aragão DS, Casarini DE, Carvalho MHC de, Fortes ZB. Upregulation of ERK1/2-eNOS via AT2 receptors decreases the contractile response to angiotensin II in resistance mesenteric arteries from obese rats [Internet]. PLOS ONE. 2014 ; 9( 8): 1-10.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1371/journal.pone
  • Source: Journal of Periodontology. Unidades: FO, ICB

    Subjects: BIOMARCADORES, PERIODONTITE CRÔNICA, EXPRESSÃO GÊNICA, CICATRIZAÇÃO

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    • ABNT

      SILVA, Henrique Aparecido Bueno da et al. Expression of protease activated receptor-1 in chronic periodontitis. Journal of Periodontology, v. 85, n. 12, p. 1763-1769, 2014Tradução . . Disponível em: https://doi.org/10.1902/jop.2014.140172. Acesso em: 20 ago. 2024.
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      Silva, H. A. B. da, Alves, V. T. E., Spolidório, L. C., César Neto, J. B., Eichler, R. S., Carvalho, M. H. C. de, & Holzhausen, M. (2014). Expression of protease activated receptor-1 in chronic periodontitis. Journal of Periodontology, 85( 12), 1763-1769. doi:10.1902/jop.2014.140172
    • NLM

      Silva HAB da, Alves VTE, Spolidório LC, César Neto JB, Eichler RS, Carvalho MHC de, Holzhausen M. Expression of protease activated receptor-1 in chronic periodontitis [Internet]. Journal of Periodontology. 2014 ; 85( 12): 1763-1769.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1902/jop.2014.140172
    • Vancouver

      Silva HAB da, Alves VTE, Spolidório LC, César Neto JB, Eichler RS, Carvalho MHC de, Holzhausen M. Expression of protease activated receptor-1 in chronic periodontitis [Internet]. Journal of Periodontology. 2014 ; 85( 12): 1763-1769.[citado 2024 ago. 20 ] Available from: https://doi.org/10.1902/jop.2014.140172

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