Filtros : "Indexado na Base de Dados Index Medicus" "Kawamoto, Elisa Mitiko" "ICB" Removidos: "Fisisologia Humana" "Indexado na Base de dados Scopus" "Harvard University (HU)" "Ferro, Emer Suavinho" "bb" "Áustria" "2023" "FCF" Limpar

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  • Fonte: Ageing Research Reviews. Unidade: ICB

    Assunto: FARMACOLOGIA

    Acesso à fonteDOIComo citar
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    • ABNT

      CARARO-LOPES, Marina Minto et al. The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, v. 36, p. 137-148, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.arr.2017.03.003. Acesso em: 18 nov. 2024.
    • APA

      Cararo-Lopes, M. M., Mazucanti, C. H. Y., Scavone, C., Kawamoto, E. M., & Berwick, D. C. (2017). The relevance of α-KLOTHO to the central nervous system: some key questions. Ageing Research Reviews, 36, 137-148. doi:10.1016/j.arr.2017.03.003
    • NLM

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
    • Vancouver

      Cararo-Lopes MM, Mazucanti CHY, Scavone C, Kawamoto EM, Berwick DC. The relevance of α-KLOTHO to the central nervous system: some key questions [Internet]. Ageing Research Reviews. 2017 ; 36 137-148.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.arr.2017.03.003
  • Fonte: Current Topics in Medicinal Chemistry. Unidade: ICB

    Assuntos: FARMACOLOGIA, ENVELHECIMENTO, INSULINA, DEGENERAÇÃO NEURAL

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    • ABNT

      MAZUCANTI, Caio Henrique et al. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, v. 15, n. 21, p. 2116-2138, 2015Tradução . . Disponível em: https://doi.org/10.2174/1568026615666150610125715. Acesso em: 18 nov. 2024.
    • APA

      Mazucanti, C. H., Cabral-Costa, J. V., Vasconcelos, A. R., Andreotti, D. Z., Scavone, C., & Kawamoto, E. M. (2015). Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration. Current Topics in Medicinal Chemistry, 15( 21), 2116-2138. doi:10.2174/1568026615666150610125715
    • NLM

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2174/1568026615666150610125715
    • Vancouver

      Mazucanti CH, Cabral-Costa JV, Vasconcelos AR, Andreotti DZ, Scavone C, Kawamoto EM. Longevity pathways (mTOR, SIRT, Insulin/IGF-1) as key modulatory targets on aging and neurodegeneration [Internet]. Current Topics in Medicinal Chemistry. 2015 ; 15( 21): 2116-2138.[citado 2024 nov. 18 ] Available from: https://doi.org/10.2174/1568026615666150610125715
  • Fonte: Journal of Neuroinflammation. Unidades: ICB, EACH

    Assunto: FARMACOLOGIA

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    • ABNT

      VASCONCELOS, Andrea Rodrigues et al. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, v. 11, n. 85, p. 1-14, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-85. Acesso em: 18 nov. 2024.
    • APA

      Vasconcelos, A. R., Yshii, L. M., Viel, T. A., Buck, H. S., Mattson, M. P., Scavone, C., & Kawamoto, E. M. (2014). Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment. Journal of Neuroinflammation, 11( 85), 1-14. doi:10.1186/1742-2094-11-85
    • NLM

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/1742-2094-11-85
    • Vancouver

      Vasconcelos AR, Yshii LM, Viel TA, Buck HS, Mattson MP, Scavone C, Kawamoto EM. Intermittent fasting attenuates lipopolysaccharideinduced neuroinflammation and memory impairment [Internet]. Journal of Neuroinflammation. 2014 ; 11( 85): 1-14.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/1742-2094-11-85
  • Fonte: Journal of Neuroinflammation. Unidade: ICB

    Assuntos: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO

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    • ABNT

      KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 18 nov. 2024.
    • APA

      Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
    • NLM

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
    • Vancouver

      Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
  • Fonte: Neuropharmacology. Unidade: ICB

    Assuntos: FISIOLOGIA, FARMACOLOGIA

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    • ABNT

      LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 18 nov. 2024.
    • APA

      Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
    • NLM

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
    • Vancouver

      Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
  • Fonte: Neurosignals. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, v. 21, n. 1-2, p. 75-88, 2013Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 18 nov. 2024.
    • APA

      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2013). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neurosignals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neurosignals. 2013 ; 21( 1-2): 75-88.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1159/000336074
  • Fonte: Neuro-Signals. Unidade: ICB

    Assunto: FARMACOLOGIA

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    • ABNT

      KAWAMOTO, Elisa Mitiko et al. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, v. 21, n. 1-2, p. 75-88, 2012Tradução . . Disponível em: https://doi.org/10.1159/000336074. Acesso em: 18 nov. 2024.
    • APA

      Kawamoto, E. M., Scavone, C., Mattson, M. P., & Camandola, S. (2012). Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide. Neuro-Signals, 21( 1-2), 75-88. doi:10.1159/000336074
    • NLM

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1159/000336074
    • Vancouver

      Kawamoto EM, Scavone C, Mattson MP, Camandola S. Curcumin requires tumor necrosis factor α signaling to alleviate cognitive impairment elicited by lipopolysaccharide [Internet]. Neuro-Signals. 2012 ; 21( 1-2): 75-88.[citado 2024 nov. 18 ] Available from: https://doi.org/10.1159/000336074

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