ReP USP - Resultado da busca

Artigo de periodico
Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies (2015)
Lucena, Camila Ferraz; Roma, Leticia Prates; Graciano, Maria Fernanda Rodrigues; Veras, Katherine; Simões, Daniel; Curi, Rui; Carpinelli, Angelo Rafael
Fonte: Pancreas. Unidade: ICB
Assuntos: FISIOLOGIA, ILHOTAS DE LANGERHANS, SUPLEMENTOS ALIMENTARES PARA ANIMAIS, ÓLEOS DE PEIUXE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LUCENA, Camila Ferraz et al. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, v. 44, n. 2, p. 287-295, 2015Tradução . . Disponível em: https://doi.org/10.1097/MPA.0000000000000249. Acesso em: 31 out. 2024.
APA
Lucena, C. F., Roma, L. P., Graciano, M. F. R., Veras, K., Simões, D., Curi, R., & Carpinelli, A. R. (2015). Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies. Pancreas, 44( 2), 287-295. doi:10.1097/MPA.0000000000000249
NLM
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 out. 31 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Vancouver
Lucena CF, Roma LP, Graciano MFR, Veras K, Simões D, Curi R, Carpinelli AR. Omega-3 supplementation improves pancreatic islet redox status: in vivo and in vitro studies [Internet]. Pancreas. 2015 ; 44( 2): 287-295.[citado 2024 out. 31 ] Available from: https://doi.org/10.1097/MPA.0000000000000249
Artigo de periodico
Celecoxib and Ibuprofen restore the ATP content and the gluconeogenesis activity in the liver of walker-256 tumor-bearing rats (2015)
Souza, Camila Oliveira de; Kurauti, Mirian Ayumi; Silva, Flaviane de Fatima; Morais, Hely de; Curi, Rui; Hirabara, Sandro Massao; Rosa Neto, José Cesar ; Souza, Helenir Medri de
Fonte: Cellular Physiology and Biochemistry. Unidade: ICB
Assuntos: FISIOLOGIA, ERROS INATOS DO METABOLISMO, CÉLULAS CULTIVADAS DE TUMOR, TUMOR DE WALKER ANIMAL, GLICOGENOSE
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUZA, Camila Oliveira de et al. Celecoxib and Ibuprofen restore the ATP content and the gluconeogenesis activity in the liver of walker-256 tumor-bearing rats. Cellular Physiology and Biochemistry, v. 36, n. 4, p. 1659-1669, 2015Tradução . . Disponível em: https://doi.org/10.1159/000430326. Acesso em: 31 out. 2024.
APA
Souza, C. O. de, Kurauti, M. A., Silva, F. de F., Morais, H. de, Curi, R., Hirabara, S. M., et al. (2015). Celecoxib and Ibuprofen restore the ATP content and the gluconeogenesis activity in the liver of walker-256 tumor-bearing rats. Cellular Physiology and Biochemistry, 36( 4), 1659-1669. doi:10.1159/000430326
NLM
Souza CO de, Kurauti MA, Silva F de F, Morais H de, Curi R, Hirabara SM, Rosa Neto JC, Souza HM de. Celecoxib and Ibuprofen restore the ATP content and the gluconeogenesis activity in the liver of walker-256 tumor-bearing rats [Internet]. Cellular Physiology and Biochemistry. 2015 ; 36( 4): 1659-1669.[citado 2024 out. 31 ] Available from: https://doi.org/10.1159/000430326
Vancouver
Souza CO de, Kurauti MA, Silva F de F, Morais H de, Curi R, Hirabara SM, Rosa Neto JC, Souza HM de. Celecoxib and Ibuprofen restore the ATP content and the gluconeogenesis activity in the liver of walker-256 tumor-bearing rats [Internet]. Cellular Physiology and Biochemistry. 2015 ; 36( 4): 1659-1669.[citado 2024 out. 31 ] Available from: https://doi.org/10.1159/000430326
Artigo de periodico
The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management (2015)
Krause, Mauricio; Heck, Thiago Gomes; Bittencourt, Aline; Scomazzon, Sofia Pizzato; Newsholme, Philip; Curi, Rui; Bittencourt Junior, Paulo Ivo Homem de
Fonte: Mediators of Inflammation. Unidade: ICB
Assuntos: FISIOLOGIA, INSULINA, EQUILIBRIO MUSCULOSQUELÉTICO, INFLAMAÇÃO, PROTEINAS DO CHOQUE TÉRMICO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KRAUSE, Mauricio et al. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management. Mediators of Inflammation, v. 2015, 2015Tradução . . Disponível em: https://doi.org/10.1155/2015/249205. Acesso em: 31 out. 2024.
APA
Krause, M., Heck, T. G., Bittencourt, A., Scomazzon, S. P., Newsholme, P., Curi, R., & Bittencourt Junior, P. I. H. de. (2015). The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management. Mediators of Inflammation, 2015. doi:10.1155/2015/249205
NLM
Krause M, Heck TG, Bittencourt A, Scomazzon SP, Newsholme P, Curi R, Bittencourt Junior PIH de. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 out. 31 ] Available from: https://doi.org/10.1155/2015/249205
Vancouver
Krause M, Heck TG, Bittencourt A, Scomazzon SP, Newsholme P, Curi R, Bittencourt Junior PIH de. The chaperone balance hypothesis: the importance of the extracellular to intracellular HSP70 ratio to inflammation-driven type 2 diabetes, the effect of exercise, and the implications for clinical management [Internet]. Mediators of Inflammation. 2015 ; 2015[citado 2024 out. 31 ] Available from: https://doi.org/10.1155/2015/249205
Artigo de periodico
Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells (2014)
Martín-Rufián, Mercedes; Nascimento-Gomes, Renata; Higuero, Ana; Crisma, Amanda R.; Campos-Sandoval, José A.; Gómez-Garcia, María C.; Cardona, Carolina; Cheng, Tzuling; Lobo, Carolina; Segura, Juan A.; Alonso, Francisco J.; Szeliga, Monika; Albrecht, Jan; Curi, Rui; Márquez, Javier; Colquhoun, Alison ; DeBerardinis, Ralph J.; Matés, José M.
Fonte: Journal of Molecular Medicine. Unidade: ICB
Assuntos: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARTÍN-RUFIÁN, Mercedes et al. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, v. 92, n. 3, p. 277-290, 2014Tradução . . Disponível em: https://doi.org/10.1007/s00109-013-1105-2. Acesso em: 31 out. 2024.
APA
Martín-Rufián, M., Nascimento-Gomes, R., Higuero, A., Crisma, A. R., Campos-Sandoval, J. A., Gómez-Garcia, M. C., et al. (2014). Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells. Journal of Molecular Medicine, 92( 3), 277-290. doi:10.1007/s00109-013-1105-2
NLM
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Vancouver
Martín-Rufián M, Nascimento-Gomes R, Higuero A, Crisma AR, Campos-Sandoval JA, Gómez-Garcia MC, Cardona C, Cheng T, Lobo C, Segura JA, Alonso FJ, Szeliga M, Albrecht J, Curi R, Márquez J, Colquhoun A, DeBerardinis RJ, Matés JM. Both GLS silencing and GLS2 overexpression synergize with oxidative stress against proliferation of glioma cells [Internet]. Journal of Molecular Medicine. 2014 ; 92( 3): 277-290.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00109-013-1105-2
Artigo de periodico
Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells (2013)
Lima, Larissa de Sá; Kawamoto, Elisa Mitiko ; Munhoz, Carolina Demarchi ; Kinoshita, Paula Fernanda; Orellana, Ana Maria Marques; Curi, Rui; Rossoni, Luciana Venturini ; Avellar, M. C. W.; Scavone, Cristoforo
Fonte: Neuropharmacology. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 31 out. 2024.
APA
Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
NLM
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Vancouver
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Artigo de periodico
High-fat diet blunts activation of the nuclear factor-κB signaling pathway in lipopolysaccharide-stimulated peritoneal macrophages of Wistar rats (2013)
Borges, Maria Carolina; Vinolo, Marco Aurélio Ramirez; Crisma, Amanda Rabello; Fock, Ricardo Ambrósio ; Borelli, Primavera ; Tirapegui, Júlio; Curi, Rui; Rogero, Marcelo Macedo
Fonte: Nutrition. Unidades: FCF, ICB, FSP
Assuntos: LIPOPOLISSACARÍDEOS, MACRÓFAGOS, INFLAMAÇÃO
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BORGES, Maria Carolina et al. High-fat diet blunts activation of the nuclear factor-κB signaling pathway in lipopolysaccharide-stimulated peritoneal macrophages of Wistar rats. Nutrition, v. 29, n. 2, p. 443-449, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.nut.2012.06.008. Acesso em: 31 out. 2024.
APA
Borges, M. C., Vinolo, M. A. R., Crisma, A. R., Fock, R. A., Borelli, P., Tirapegui, J., et al. (2013). High-fat diet blunts activation of the nuclear factor-κB signaling pathway in lipopolysaccharide-stimulated peritoneal macrophages of Wistar rats. Nutrition, 29( 2), 443-449. doi:10.1016/j.nut.2012.06.008
NLM
Borges MC, Vinolo MAR, Crisma AR, Fock RA, Borelli P, Tirapegui J, Curi R, Rogero MM. High-fat diet blunts activation of the nuclear factor-κB signaling pathway in lipopolysaccharide-stimulated peritoneal macrophages of Wistar rats [Internet]. Nutrition. 2013 ; 29( 2): 443-449.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.nut.2012.06.008
Vancouver
Borges MC, Vinolo MAR, Crisma AR, Fock RA, Borelli P, Tirapegui J, Curi R, Rogero MM. High-fat diet blunts activation of the nuclear factor-κB signaling pathway in lipopolysaccharide-stimulated peritoneal macrophages of Wistar rats [Internet]. Nutrition. 2013 ; 29( 2): 443-449.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.nut.2012.06.008
Artigo de periodico
Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro (2013)
Paschoal, Vivian Almeida; Vinolo, M. A. R.; Crisma, A. R.; Magdalon, Juliana; Curi, Rui
Fonte: Lipids. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PASCHOAL, Vivian Almeida et al. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro. Lipids, v. 48, n. 2, p. 93-103, 2013Tradução . . Disponível em: https://doi.org/10.1007/s11745-012-3726-6. Acesso em: 31 out. 2024.
APA
Paschoal, V. A., Vinolo, M. A. R., Crisma, A. R., Magdalon, J., & Curi, R. (2013). Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro. Lipids, 48( 2), 93-103. doi:10.1007/s11745-012-3726-6
NLM
Paschoal VA, Vinolo MAR, Crisma AR, Magdalon J, Curi R. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro [Internet]. Lipids. 2013 ; 48( 2): 93-103.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s11745-012-3726-6
Vancouver
Paschoal VA, Vinolo MAR, Crisma AR, Magdalon J, Curi R. Eicosapentaenoic (EPA) and Docosahexaenoic (DHA) acid differentially modulate rat neutrophil function in vitro [Internet]. Lipids. 2013 ; 48( 2): 93-103.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s11745-012-3726-6
Artigo de periodico
Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration (2013)
Marçal, Anderson Carlos; Leonelli, Mauro; Fiamoncini, Jarlei ; Deschamps, Francisco C.; Rodrigues, Maria A. M.; Curi, Rui; Carpinelli, Angelo Rafael ; Britto, Luiz Roberto Giorgetti de ; Carvalho, Carla Roberta de Oliveira
Fonte: Cell Biochemistry and Function. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARÇAL, Anderson Carlos et al. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration. Cell Biochemistry and Function, v. 31, n. 1, p. 65-74, 2013Tradução . . Disponível em: https://doi.org/10.1002/cbf.2861. Acesso em: 31 out. 2024.
APA
Marçal, A. C., Leonelli, M., Fiamoncini, J., Deschamps, F. C., Rodrigues, M. A. M., Curi, R., et al. (2013). Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration. Cell Biochemistry and Function, 31( 1), 65-74. doi:10.1002/cbf.2861
NLM
Marçal AC, Leonelli M, Fiamoncini J, Deschamps FC, Rodrigues MAM, Curi R, Carpinelli AR, Britto LRG de, Carvalho CR de O. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration [Internet]. Cell Biochemistry and Function. 2013 ; 31( 1): 65-74.[citado 2024 out. 31 ] Available from: https://doi.org/10.1002/cbf.2861
Vancouver
Marçal AC, Leonelli M, Fiamoncini J, Deschamps FC, Rodrigues MAM, Curi R, Carpinelli AR, Britto LRG de, Carvalho CR de O. Diet-induced obesity impairs AKT signalling in the retina and causes retinal degeneration [Internet]. Cell Biochemistry and Function. 2013 ; 31( 1): 65-74.[citado 2024 out. 31 ] Available from: https://doi.org/10.1002/cbf.2861
Artigo de periodico
Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets (2013)
Marçal, Anderson Carlos; Camporez, João Paulo Gabriel; Lima-Salgado, Thais M.; Cintra, D. E.; Akamine, Eliana Hiromi ; Ribeiro, L. M.; Almeida, F. N.; Zanuto, R. P.; Curi, Rui; Boldrini, Silvia de Campos; Liberti, Edson Aparecido; Fiamoncini, Jarlei ; Hirabara, S. M.; Deschamps, F. C.; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Fonte: British Journal of Nutrition. Unidade: ICB
Assuntos: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARÇAL, Anderson Carlos et al. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, v. 109, n. 12, p. 2154-2165, 2013Tradução . . Disponível em: https://doi.org/10.1017/S0007114512004576. Acesso em: 31 out. 2024.
APA
Marçal, A. C., Camporez, J. P. G., Lima-Salgado, T. M., Cintra, D. E., Akamine, E. H., Ribeiro, L. M., et al. (2013). Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, 109( 12), 2154-2165. doi:10.1017/S0007114512004576
NLM
Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 out. 31 ] Available from: https://doi.org/10.1017/S0007114512004576
Vancouver
Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 out. 31 ] Available from: https://doi.org/10.1017/S0007114512004576
Artigo de periodico
Fish oil supplementation for two generations increases insulin sensitivity in rats (2013)
Hirabara, Sandro M.; Folador, Alesandra; Fiamoncini, Jarlei ; Lambertucci, Rafael H.; Rodrigues Jr, Carlos Flores; Rocha, Marlene Santos da; Aikawa, Julia; Yamazaki, Ricardo K.; Martins, Amanda R.; Rodrigues, Alice Cristina ; Carpinelli, Angelo Rafael ; Pithon-Curi, Tania C.; Fernandes, Luiz C.; Gorjão, Renata; Curi, Rui
Fonte: Journal of Nutritional Biochemistry. Unidade: ICB
Assuntos: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
HIRABARA, Sandro M. et al. Fish oil supplementation for two generations increases insulin sensitivity in rats. Journal of Nutritional Biochemistry, v. 24, n. 6, p. 1136-1145, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2012.08.014. Acesso em: 31 out. 2024.
APA
Hirabara, S. M., Folador, A., Fiamoncini, J., Lambertucci, R. H., Rodrigues Jr, C. F., Rocha, M. S. da, et al. (2013). Fish oil supplementation for two generations increases insulin sensitivity in rats. Journal of Nutritional Biochemistry, 24( 6), 1136-1145. doi:10.1016/j.jnutbio.2012.08.014
NLM
Hirabara SM, Folador A, Fiamoncini J, Lambertucci RH, Rodrigues Jr CF, Rocha MS da, Aikawa J, Yamazaki RK, Martins AR, Rodrigues AC, Carpinelli AR, Pithon-Curi TC, Fernandes LC, Gorjão R, Curi R. Fish oil supplementation for two generations increases insulin sensitivity in rats [Internet]. Journal of Nutritional Biochemistry. 2013 ; 24( 6): 1136-1145.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.jnutbio.2012.08.014
Vancouver
Hirabara SM, Folador A, Fiamoncini J, Lambertucci RH, Rodrigues Jr CF, Rocha MS da, Aikawa J, Yamazaki RK, Martins AR, Rodrigues AC, Carpinelli AR, Pithon-Curi TC, Fernandes LC, Gorjão R, Curi R. Fish oil supplementation for two generations increases insulin sensitivity in rats [Internet]. Journal of Nutritional Biochemistry. 2013 ; 24( 6): 1136-1145.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.jnutbio.2012.08.014
Artigo de periodico
A catalytically-inactivesnakevenomLys49phospholipaseA2 homolog induces expression of cyclooxygenase-2 and production of prostaglandins through selected signaling pathways in macrophages (2013)
Moreira, Vanessa; Souto, Pollyana Cristina Maggio de Castro; Vinolo, Marco Aurélio; Lomonte, Bruno; Gutiérrez, José María; Curi, Rui; Teixeira, Catarina
Fonte: European Journal of Pharmacology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Vanessa et al. A catalytically-inactivesnakevenomLys49phospholipaseA2 homolog induces expression of cyclooxygenase-2 and production of prostaglandins through selected signaling pathways in macrophages. European Journal of Pharmacology, v. 708, n. 1-3, p. 68-79, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2013.01.061. Acesso em: 31 out. 2024.
APA
Moreira, V., Souto, P. C. M. de C., Vinolo, M. A., Lomonte, B., Gutiérrez, J. M., Curi, R., & Teixeira, C. (2013). A catalytically-inactivesnakevenomLys49phospholipaseA2 homolog induces expression of cyclooxygenase-2 and production of prostaglandins through selected signaling pathways in macrophages. European Journal of Pharmacology, 708( 1-3), 68-79. doi:10.1016/j.ejphar.2013.01.061
NLM
Moreira V, Souto PCM de C, Vinolo MA, Lomonte B, Gutiérrez JM, Curi R, Teixeira C. A catalytically-inactivesnakevenomLys49phospholipaseA2 homolog induces expression of cyclooxygenase-2 and production of prostaglandins through selected signaling pathways in macrophages [Internet]. European Journal of Pharmacology. 2013 ; 708( 1-3): 68-79.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2013.01.061
Vancouver
Moreira V, Souto PCM de C, Vinolo MA, Lomonte B, Gutiérrez JM, Curi R, Teixeira C. A catalytically-inactivesnakevenomLys49phospholipaseA2 homolog induces expression of cyclooxygenase-2 and production of prostaglandins through selected signaling pathways in macrophages [Internet]. European Journal of Pharmacology. 2013 ; 708( 1-3): 68-79.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.ejphar.2013.01.061
Artigo de periodico
Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis (2012)
Cappi, Sylas Bezerra; Noritomi, Danilo Teixeira; Velasco, Irineu Tadeu ; Curi, Rui ; Loureiro, Tatiana Carolina Alba ; Soriano, Francisco Garcia
Fonte: Intensive Care Medicine. Unidades: ICB, FM
Assuntos: FISIOLOGIA, LIPÍDEOS, GORDURAS, SEPSE, GLICEMIA, ESTUDOS PROSPECTIVOS, ESTUDOS RANDOMIZADOS, LIPOPROTEÍNAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAPPI, Sylas Bezerra et al. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, v. 38, p. 634-641, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00134-011-2458-z. Acesso em: 31 out. 2024.
APA
Cappi, S. B., Noritomi, D. T., Velasco, I. T., Curi, R., Loureiro, T. C. A., & Soriano, F. G. (2012). Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis. Intensive Care Medicine, 38, 634-641. doi:10.1007/s00134-011-2458-z
NLM
Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
Vancouver
Cappi SB, Noritomi DT, Velasco IT, Curi R, Loureiro TCA, Soriano FG. Dyslipidemia: a prospective controlled randomized trial of intensive glycemic control in sepsis [Internet]. Intensive Care Medicine. 2012 ; 38 634-641.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00134-011-2458-z
Artigo de periodico
Effects of moderate electrical stimulation on reactive species production by primary rat skeletal muscle cells: cross talk between superoxide and nitric oxide production (2012)
Lambertucci, Rafael Herling; Silveira, Leonardo dos Reis; Hirabara, Sandro Massao; Curi, Rui; Sweeney, Gary; Pithon-Curi, Tania Cristina
Fonte: Journal of Cellular Physiology. Unidades: ICB, EEFERP
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LAMBERTUCCI, Rafael Herling et al. Effects of moderate electrical stimulation on reactive species production by primary rat skeletal muscle cells: cross talk between superoxide and nitric oxide production. Journal of Cellular Physiology, v. 227, n. 6, p. 2511-2518, 2012Tradução . . Disponível em: https://doi.org/10.1002/jcp.22989. Acesso em: 31 out. 2024.
APA
Lambertucci, R. H., Silveira, L. dos R., Hirabara, S. M., Curi, R., Sweeney, G., & Pithon-Curi, T. C. (2012). Effects of moderate electrical stimulation on reactive species production by primary rat skeletal muscle cells: cross talk between superoxide and nitric oxide production. Journal of Cellular Physiology, 227( 6), 2511-2518. doi:10.1002/jcp.22989
NLM
Lambertucci RH, Silveira L dos R, Hirabara SM, Curi R, Sweeney G, Pithon-Curi TC. Effects of moderate electrical stimulation on reactive species production by primary rat skeletal muscle cells: cross talk between superoxide and nitric oxide production [Internet]. Journal of Cellular Physiology. 2012 ; 227( 6): 2511-2518.[citado 2024 out. 31 ] Available from: https://doi.org/10.1002/jcp.22989
Vancouver
Lambertucci RH, Silveira L dos R, Hirabara SM, Curi R, Sweeney G, Pithon-Curi TC. Effects of moderate electrical stimulation on reactive species production by primary rat skeletal muscle cells: cross talk between superoxide and nitric oxide production [Internet]. Journal of Cellular Physiology. 2012 ; 227( 6): 2511-2518.[citado 2024 out. 31 ] Available from: https://doi.org/10.1002/jcp.22989
Artigo de periodico
Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages (2012)
Magdalon, Juliana; Vinolo, Marco Aurélio Ramirez; Rodrigues, Hosana Gomes; Paschoal, Vivian Almeida; Torres, Rosângela Pavan; Mancini-Filho, Jorge ; Calder, Philip C; Hatanaka, Elaine; Curi, Rui
Fonte: Lipids. Unidades: FCF, ICB
Assuntos: ÁCIDO LINOLEICO, ÁCIDOS GRAXOS, EICOSANOIDES, CITOCINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MAGDALON, Juliana et al. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages. Lipids, v. 47, n. 8, p. 803-812, 2012Tradução . . Disponível em: https://doi.org/10.1007/s11745-012-3687-9. Acesso em: 31 out. 2024.
APA
Magdalon, J., Vinolo, M. A. R., Rodrigues, H. G., Paschoal, V. A., Torres, R. P., Mancini-Filho, J., et al. (2012). Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages. Lipids, 47( 8), 803-812. doi:10.1007/s11745-012-3687-9
NLM
Magdalon J, Vinolo MAR, Rodrigues HG, Paschoal VA, Torres RP, Mancini-Filho J, Calder PC, Hatanaka E, Curi R. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages [Internet]. Lipids. 2012 ; 47( 8): 803-812.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s11745-012-3687-9
Vancouver
Magdalon J, Vinolo MAR, Rodrigues HG, Paschoal VA, Torres RP, Mancini-Filho J, Calder PC, Hatanaka E, Curi R. Oral administration of oleic or linoleic acids modulates the production of inflammatory mediators by rat macrophages [Internet]. Lipids. 2012 ; 47( 8): 803-812.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s11745-012-3687-9
Artigo de periodico
Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle (2012)
Pinheiro, Carlos Hermano da Justa; Gerlinger-Romero, Frederico; Guimarães-Ferreira, Lucas; Souza-Jr, Alcione Lescano de; Vitzel, Kaio Fernando; Nachbar, Renato Tadeu; Nunes, Maria Tereza ; Curi, Rui
Fonte: European Journal of Applied Physiology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PINHEIRO, Carlos Hermano da Justa et al. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle. European Journal of Applied Physiology, v. 112, n. 7, p. 2531-2537, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00421-011-2224-5. Acesso em: 31 out. 2024.
APA
Pinheiro, C. H. da J., Gerlinger-Romero, F., Guimarães-Ferreira, L., Souza-Jr, A. L. de, Vitzel, K. F., Nachbar, R. T., et al. (2012). Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle. European Journal of Applied Physiology, 112( 7), 2531-2537. doi:10.1007/s00421-011-2224-5
NLM
Pinheiro CH da J, Gerlinger-Romero F, Guimarães-Ferreira L, Souza-Jr AL de, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 7): 2531-2537.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00421-011-2224-5
Vancouver
Pinheiro CH da J, Gerlinger-Romero F, Guimarães-Ferreira L, Souza-Jr AL de, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Metabolic and functional effects of beta-hydroxy-betamethylbutyrate (HMB) supplementation in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 7): 2531-2537.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00421-011-2224-5
Artigo de periodico
Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice (2012)
Carvalho Filho, M. A.; Carvalho, B. M.; Oliveira, A. G.; Guadagnini, D.; Ueno, M.; Dias M. M.; Tsukumo, D. M.; Hirabata, Sandro Massao; Reis, L. F.; Curi, Rui; Carvalheira, J. B. C.; Saad, Mario J. A.
Fonte: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CARVALHO FILHO, M. A. et al. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, v. 153, n. 11, p. 5261-5274, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1400. Acesso em: 31 out. 2024.
APA
Carvalho Filho, M. A., Carvalho, B. M., Oliveira, A. G., Guadagnini, D., Ueno, M., Dias M. M.,, et al. (2012). Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice. Endocrinology, 153( 11), 5261-5274. doi:10.1210/en.2012-1400
NLM
Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 out. 31 ] Available from: https://doi.org/10.1210/en.2012-1400
Vancouver
Carvalho Filho MA, Carvalho BM, Oliveira AG, Guadagnini D, Ueno M, Dias M. M., Tsukumo DM, Hirabata SM, Reis LF, Curi R, Carvalheira JBC, Saad MJA. Double-stranded RNA-sctivated protein kinase is a key modulator of insulin sensitivity in physiological conditions and in obesity in mice [Internet]. Endocrinology. 2012 ; 153( 11): 5261-5274.[citado 2024 out. 31 ] Available from: https://doi.org/10.1210/en.2012-1400
Artigo de periodico
Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle (2012)
Guimarães-Ferreira, Lucas; Pinheiro, Carlos Hermano da Justa; Gerlinger-Romero, Frederico; Vitzel, Kaio Fernando; Nachbar, Renato Tadeu; Nunes, Maria Tereza ; Curi, Rui
Fonte: European Journal of Applied Physiology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GUIMARÃES-FERREIRA, Lucas et al. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle. European Journal of Applied Physiology, v. 112, n. 11, p. 3905-3911, 2012Tradução . . Disponível em: https://doi.org/10.1007/s00421-012-2378-9. Acesso em: 31 out. 2024.
APA
Guimarães-Ferreira, L., Pinheiro, C. H. da J., Gerlinger-Romero, F., Vitzel, K. F., Nachbar, R. T., Nunes, M. T., & Curi, R. (2012). Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle. European Journal of Applied Physiology, 112( 11), 3905-3911. doi:10.1007/s00421-012-2378-9
NLM
Guimarães-Ferreira L, Pinheiro CH da J, Gerlinger-Romero F, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 11): 3905-3911.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00421-012-2378-9
Vancouver
Guimarães-Ferreira L, Pinheiro CH da J, Gerlinger-Romero F, Vitzel KF, Nachbar RT, Nunes MT, Curi R. Short-term creatine supplementation decreases reactive oxygen species content with no changes in expression and activity of antioxidant enzymes in skeletal muscle [Internet]. European Journal of Applied Physiology. 2012 ; 112( 11): 3905-3911.[citado 2024 out. 31 ] Available from: https://doi.org/10.1007/s00421-012-2378-9
Artigo de periodico
Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia (2010)
Arruda, Ana Paula; Milanski, Marciane; Romanatto, Talita; Solon, Carina; Coope, Andressa; Festuccia, William Tadeu Lara ; Velloso, Licio Augusto; Hirabara, Sandro Massao; Ropelle, Eduardo; Cavalheira, José B.; Vercesi, Anibal Eugenio; Curi, Rui
Fonte: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARRUDA, Ana Paula et al. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology, v. 151, n. 2, p. 683-694, 2010Tradução . . Acesso em: 31 out. 2024.
APA
Arruda, A. P., Milanski, M., Romanatto, T., Solon, C., Coope, A., Festuccia, W. T. L., et al. (2010). Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology, 151( 2), 683-694.
NLM
Arruda AP, Milanski M, Romanatto T, Solon C, Coope A, Festuccia WTL, Velloso LA, Hirabara SM, Ropelle E, Cavalheira JB, Vercesi AE, Curi R. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology. 2010 ; 151( 2): 683-694.[citado 2024 out. 31 ]
Vancouver
Arruda AP, Milanski M, Romanatto T, Solon C, Coope A, Festuccia WTL, Velloso LA, Hirabara SM, Ropelle E, Cavalheira JB, Vercesi AE, Curi R. Hypothalamic actions of tumor necrosis factor α provide the thermogenic core for the wastage syndrome in cachexia. Endocrinology. 2010 ; 151( 2): 683-694.[citado 2024 out. 31 ]

Unidades USP

ReP

Filtros

Departamento

Autores

Autores USP

Assuntos

Título da fonte

País de publicação

Indexado em

Afiliação dos autores externos normalizada

Afiliação dos autores externos não normalizada