ReP USP - Resultado da busca

Artigo de periodico
Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) (2015)
Echem, Cinthya; Bomfim, Gisele Facholi; Ceravolo, Graziela Scalianti; Oliveira, Maria Aparecida; Eichler, Rosangela Aparecida Santos; Bechara, Luiz Roberto; Veras, Mariana Matera; Saldiva, Paulo Hilário Nascimento ; Ferreira, Julio Cesar Batista ; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli
Fonte: International Journal of Cardiology. Unidades: ICB, FM
Assuntos: ANATOMIA, FARMACOLOGIA, HIPERTENSÃO, RATOS, PRESSÃO SANGUÍNEA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ECHEM, Cinthya et al. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, v. 187, p. 243-245, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2015.03.190. Acesso em: 25 set. 2024.
APA
Echem, C., Bomfim, G. F., Ceravolo, G. S., Oliveira, M. A., Eichler, R. A. S., Bechara, L. R., et al. (2015). Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, 187, 243-245. doi:10.1016/j.ijcard.2015.03.190
NLM
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Vancouver
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Artigo de periodico
Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells (2014)
Queiroz, Eveline, A. I. F.; Puukila, Stephanie; Eichler, Rosangela; Sampaio, Sandra C.; Forsyth, Heidi L.; Lees, Simon J.; Barbosa, Aneli M.; Dekker, Robert F. H.; Fortes, Zuleica Bruno; Khaper, Neelam
Fonte: PLOS ONE. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
QUEIROZ, Eveline et al. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells. PLOS ONE, v. 9, n. 5, p. 1-18, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0098207. Acesso em: 25 set. 2024.
APA
Queiroz, E., Puukila, S., Eichler, R., Sampaio, S. C., Forsyth, H. L., Lees, S. J., et al. (2014). Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells. PLOS ONE, 9( 5), 1-18. doi:10.1371/journal.pone.0098207
NLM
Queiroz E, Puukila S, Eichler R, Sampaio SC, Forsyth HL, Lees SJ, Barbosa AM, Dekker RFH, Fortes ZB, Khaper N. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells [Internet]. PLOS ONE. 2014 ; 9( 5): 1-18.[citado 2024 set. 25 ] Available from: https://doi.org/10.1371/journal.pone.0098207
Vancouver
Queiroz E, Puukila S, Eichler R, Sampaio SC, Forsyth HL, Lees SJ, Barbosa AM, Dekker RFH, Fortes ZB, Khaper N. Metformin induces apoptosis and cell cycle arrest mediated by oOxidative stress, AMPK and FOXO3a in MCF-7 breast cancer cells [Internet]. PLOS ONE. 2014 ; 9( 5): 1-18.[citado 2024 set. 25 ] Available from: https://doi.org/10.1371/journal.pone.0098207
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Fonte: PLOS ONE. Unidades: ICB, FMRP
Assuntos: ANATOMIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 25 set. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 set. 25 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 set. 25 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats (2013)
Sartoretto, Simone Marcieli; Santos-Eichler, Rosângela; Tostes, Rita de Cássia Aleixo; Carvalho, Maria Helena Catelli de; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno
Fonte: Journal of Diabetes. Unidades: ICB, FMRP
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SARTORETTO, Simone Marcieli et al. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, v. 5, n. 2, p. 197-206, 2013Tradução . . Acesso em: 25 set. 2024.
APA
Sartoretto, S. M., Santos-Eichler, R., Tostes, R. de C. A., Carvalho, M. H. C. de, Akamine, E. H., & Fortes, Z. B. (2013). Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes, 5( 2), 197-206.
NLM
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 set. 25 ]
Vancouver
Sartoretto SM, Santos-Eichler R, Tostes R de CA, Carvalho MHC de, Akamine EH, Fortes ZB. Role of nitric oxide and endothelin in endothelial maintenance of vasoconstrictor responses in aortas of diabetic female rats. Journal of Diabetes. 2013 ; 5( 2): 197-206.[citado 2024 set. 25 ]
Artigo de periodico
Vascular disease in diabetic women: why do they miss the female protection? (2012)
Dantas, Ana Paula Villela; Fortes, Zuleica Bruno; Carvalho, Maria Helena Catelli de
Fonte: Experimental Diabetes Research. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DANTAS, Ana Paula Villela e FORTES, Zuleica Bruno e CARVALHO, Maria Helena Catelli de. Vascular disease in diabetic women: why do they miss the female protection?. Experimental Diabetes Research, v. 2012, p. 1-10, 2012Tradução . . Disponível em: https://doi.org/10.1155/2012/570598. Acesso em: 25 set. 2024.
APA
Dantas, A. P. V., Fortes, Z. B., & Carvalho, M. H. C. de. (2012). Vascular disease in diabetic women: why do they miss the female protection? Experimental Diabetes Research, 2012, 1-10. doi:10.1155/2012/570598
NLM
Dantas APV, Fortes ZB, Carvalho MHC de. Vascular disease in diabetic women: why do they miss the female protection? [Internet]. Experimental Diabetes Research. 2012 ; 2012 1-10.[citado 2024 set. 25 ] Available from: https://doi.org/10.1155/2012/570598
Vancouver
Dantas APV, Fortes ZB, Carvalho MHC de. Vascular disease in diabetic women: why do they miss the female protection? [Internet]. Experimental Diabetes Research. 2012 ; 2012 1-10.[citado 2024 set. 25 ] Available from: https://doi.org/10.1155/2012/570598
Artigo de periodico
Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring (2012)
Ponzio, Beatriz Felice; Carvalho, Maria Helena Catelli; Fortes, Zuleica Bruno; Franco, Maria do Carmo
Fonte: Life Sciences. Unidade: ICB
Assunto: FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
PONZIO, Beatriz Felice et al. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring. Life Sciences, v. 90, n. 15-16, p. 571-577, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.01.017. Acesso em: 25 set. 2024.
APA
Ponzio, B. F., Carvalho, M. H. C., Fortes, Z. B., & Franco, M. do C. (2012). Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring. Life Sciences, 90( 15-16), 571-577. doi:10.1016/j.lfs.2012.01.017
NLM
Ponzio BF, Carvalho MHC, Fortes ZB, Franco M do C. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring [Internet]. Life Sciences. 2012 ; 90( 15-16): 571-577.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017
Vancouver
Ponzio BF, Carvalho MHC, Fortes ZB, Franco M do C. Implications of maternal nutrient restriction in transgenerational programming of hypertension and endothelial dysfunction across F1–F3 offspring [Internet]. Life Sciences. 2012 ; 90( 15-16): 571-577.[citado 2024 set. 25 ] Available from: https://doi.org/10.1016/j.lfs.2012.01.017

Unidades USP

ReP

Filtros

Autores

Autores USP

Indexado em

Afiliação dos autores externos não normalizada