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Artigo de periodico
Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway (2015)
Davel, Ana P.; Victorio, Jamaira A.; Delbin, Maria Andréia; Fukuda, Livia Emy; Rossoni, Luciana Venturini
Fonte: Life Sciences. Unidade: ICB
Assuntos: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS
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ABNT
DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 13 nov. 2024.
APA
Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
NLM
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Vancouver
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Artigo de periodico
Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway (2014)
Wenceslau, Camilla F.; Rossoni, Luciana Venturini
Fonte: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
WENCESLAU, Camilla F. e ROSSONI, Luciana Venturini. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, v. 32, n. 3, p. 542-555, 2014Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000000059. Acesso em: 13 nov. 2024.
APA
Wenceslau, C. F., & Rossoni, L. V. (2014). Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, 32( 3), 542-555. doi:10.1097/HJH.0000000000000059
NLM
Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
Vancouver
Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
Artigo de periodico
Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus (2014)
Kinoshita, Paula Fernanda; Yshii, Lidia Mitiko; Vasconcelos, Andrea Rodrigues; Orellana, Ana Maria Marques; Lima, Larissa de Sá; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Kawamoto, Elisa Mitiko ; Scavone, Cristoforo
Fonte: Journal of Neuroinflammation. Unidade: ICB
Assuntos: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO
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ABNT
KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 13 nov. 2024.
APA
Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
NLM
Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
Vancouver
Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
Artigo de periodico
Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats (2011)
Pacheco, Bruna P. M.; Crajoinas, Renato O.; Couto, Gisele Kruger; Davel, Ana Paula Couto; Lessa, Lucília M.; Rossoni, Luciana Venturini ; Girardi, Adriana C. C.
Fonte: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
PACHECO, Bruna P. M. et al. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, v. 29, n. 3, p. 520-528, 2011Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 13 nov. 2024.
APA
Pacheco, B. P. M., Crajoinas, R. O., Couto, G. K., Davel, A. P. C., Lessa, L. M., Rossoni, L. V., & Girardi, A. C. C. (2011). Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats. Journal of Hypertension, 29( 3), 520-528. doi:10.1097/HJH.0b013e32833ca68c
NLM
Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
Vancouver
Pacheco BPM, Crajoinas RO, Couto GK, Davel APC, Lessa LM, Rossoni LV, Girardi ACC. Dipeptidyl peptidase IV inhibition attenuates blood pressure rising in young spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2011 ; 29( 3): 520-528.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
Artigo de periodico
Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR (2010)
Crajoinas, Renato O.; Lessa, Lucília M. A.; Carraro-Lacroix, Luciene R.; Davel, Ana Paula Couto; Pacheco, Bruna P. M.; Rossoni, Luciana Venturini ; Malnic, Gerhald; Girardi, Adriana Castello Costa
Fonte: American Journal of Physiology. Renal Physiology. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
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ABNT
CRAJOINAS, Renato O. et al. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, v. 299, n. 4, p. F872-F881, 2010Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00654.2009. Acesso em: 13 nov. 2024.
APA
Crajoinas, R. O., Lessa, L. M. A., Carraro-Lacroix, L. R., Davel, A. P. C., Pacheco, B. P. M., Rossoni, L. V., et al. (2010). Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR. American Journal of Physiology. Renal Physiology, 299( 4), F872-F881. doi:10.1152/ajprenal.00654.2009
NLM
Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
Vancouver
Crajoinas RO, Lessa LMA, Carraro-Lacroix LR, Davel APC, Pacheco BPM, Rossoni LV, Malnic G, Girardi ACC. Posttranslational mechanisms associated with reduced NHE3 activity in adult vs. young prehypertensive SHR [Internet]. American Journal of Physiology. Renal Physiology. 2010 ; 299( 4): F872-F881.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1152/ajprenal.00654.2009
Artigo de periodico
Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C (2010)
Gallo, Luana C.; Davel, Ana Paula Couto; Xavier, Fabiano E.; Rossoni, Luciana Venturini
Fonte: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
GALLO, Luana C. et al. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, v. 87, n. 9-10 p. 302-308, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2010.07.005. Acesso em: 13 nov. 2024.
APA
Gallo, L. C., Davel, A. P. C., Xavier, F. E., & Rossoni, L. V. (2010). Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C. Life Sciences, 87( 9-10 p. 302-308). doi:10.1016/j.lfs.2010.07.005
NLM
Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
Vancouver
Gallo LC, Davel APC, Xavier FE, Rossoni LV. Time-dependent increases in ouabain-sensitive Na T’, K.+’-ATPase activity in aortas from diabetic rats: the role of prostanoids and protein kinase C [Internet]. Life Sciences. 2010 ; 87( 9-10 p. 302-308):[citado 2024 nov. 13 ] Available from: https://doi.org/10.1016/j.lfs.2010.07.005
Artigo de periodico
Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice (2010)
Kobayasi, Renata; Akamine, Eliana Hiromi ; Davel, Ana Paula Couto; Rodrigues, Maria A. M.; Carvalho, Carla Roberta de Oliveira ; Rossoni, Luciana Venturini
Fonte: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA (HUMANO)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KOBAYASI, Renata et al. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, v. 28, n. 10, p. 2111-2119, 2010Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32833ca68c. Acesso em: 13 nov. 2024.
APA
Kobayasi, R., Akamine, E. H., Davel, A. P. C., Rodrigues, M. A. M., Carvalho, C. R. de O., & Rossoni, L. V. (2010). Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice. Journal of Hypertension, 28( 10), 2111-2119. doi:10.1097/HJH.0b013e32833ca68c
NLM
Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c
Vancouver
Kobayasi R, Akamine EH, Davel APC, Rodrigues MAM, Carvalho CR de O, Rossoni LV. Oxidative stress and inflammatory mediators contribute to endothelial dysfunction in high-fat diet-induced obesity in mice [Internet]. Journal of Hypertension. 2010 ; 28( 10): 2111-2119.[citado 2024 nov. 13 ] Available from: https://doi.org/10.1097/HJH.0b013e32833ca68c

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