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Artigo de periodico
Influence of the anteromedial thalamus on social defeat-associated contextual fear memory (2018)
Rangel Júnior, Miguel José; Baldo, Marcus Vinícius Chrysóstomo ; Canteras, Newton Sabino
Fonte: Behavioural Brain Research. Unidade: ICB
Assuntos: MEDO, MEMÓRIA, NÚCLEOS TALÂMICOS, NEUROANATOMIA, TÁLAMO
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ABNT
RANGEL JÚNIOR, Miguel José e BALDO, Marcus Vinícius Chrysóstomo e CANTERAS, Newton Sabino. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory. Behavioural Brain Research, v. 339, p. 269-277, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2017.10.038. Acesso em: 06 nov. 2024.
APA
Rangel Júnior, M. J., Baldo, M. V. C., & Canteras, N. S. (2018). Influence of the anteromedial thalamus on social defeat-associated contextual fear memory. Behavioural Brain Research, 339, 269-277. doi:10.1016/j.bbr.2017.10.038
NLM
Rangel Júnior MJ, Baldo MVC, Canteras NS. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory [Internet]. Behavioural Brain Research. 2018 ; 339 269-277.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2017.10.038
Vancouver
Rangel Júnior MJ, Baldo MVC, Canteras NS. Influence of the anteromedial thalamus on social defeat-associated contextual fear memory [Internet]. Behavioural Brain Research. 2018 ; 339 269-277.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2017.10.038
Artigo de periodico
Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context (2018)
Bindi, Ricardo Passoni; Baldo, Marcus Vinícius Chrysóstomo ; Canteras, Newton Sabino
Fonte: Behavioural Brain Research. Unidade: ICB
Assuntos: ANIMAIS PREDADORES, MEDO, MEMÓRIA, NEUROANATOMIA, AMÍGDALA DO CEREBELO, HIPOTÁLAMO
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ABNT
BINDI, Ricardo Passoni e BALDO, Marcus Vinícius Chrysóstomo e CANTERAS, Newton Sabino. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context. Behavioural Brain Research, v. 342, p. 51-56, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2018.01.016. Acesso em: 06 nov. 2024.
APA
Bindi, R. P., Baldo, M. V. C., & Canteras, N. S. (2018). Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context. Behavioural Brain Research, 342, 51-56. doi:10.1016/j.bbr.2018.01.016
NLM
Bindi RP, Baldo MVC, Canteras NS. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context [Internet]. Behavioural Brain Research. 2018 ; 342 51-56.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2018.01.016
Vancouver
Bindi RP, Baldo MVC, Canteras NS. Roles of the anterior basolateral amygdalar nucleus during exposure to a live predator and to a predator-associated context [Internet]. Behavioural Brain Research. 2018 ; 342 51-56.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2018.01.016
Artigo de periodico
Electrical stimulation of the insular cortex as a novel target for the relief of refractory pain: an experimental approach in rodents (2018)
Dimov, Luiz Fabio ; Toniolo, Elaine Flamia; Matielo, Heloísa Alonso ; Andrade, Daniel Ciampi de ; Larrea, Luis Garcia ; Ballester, Gerson ; Teixeira, Manoel Jacobsen ; Dale, Camila Squarzoni
Fonte: Behavioural Brain Research. Unidades: FM, ICB
Assuntos: ESTIMULAÇÃO CEREBRAL, DOR, RATOS, SUBSTÂNCIA CINZENTA PERIAQUEDUTAL, DOR, ANATOMIA, CÓRTEX CEREBRAL DE ANIMAL
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DIMOV, Luiz Fabio et al. Electrical stimulation of the insular cortex as a novel target for the relief of refractory pain: an experimental approach in rodents. Behavioural Brain Research, v. 346, p. 86-95, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2017.11.036. Acesso em: 06 nov. 2024.
APA
Dimov, L. F., Toniolo, E. F., Matielo, H. A., Andrade, D. C. de, Larrea, L. G., Ballester, G., et al. (2018). Electrical stimulation of the insular cortex as a novel target for the relief of refractory pain: an experimental approach in rodents. Behavioural Brain Research, 346, 86-95. doi:10.1016/j.bbr.2017.11.036
NLM
Dimov LF, Toniolo EF, Matielo HA, Andrade DC de, Larrea LG, Ballester G, Teixeira MJ, Dale CS. Electrical stimulation of the insular cortex as a novel target for the relief of refractory pain: an experimental approach in rodents [Internet]. Behavioural Brain Research. 2018 ; 346 86-95.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2017.11.036
Vancouver
Dimov LF, Toniolo EF, Matielo HA, Andrade DC de, Larrea LG, Ballester G, Teixeira MJ, Dale CS. Electrical stimulation of the insular cortex as a novel target for the relief of refractory pain: an experimental approach in rodents [Internet]. Behavioural Brain Research. 2018 ; 346 86-95.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.bbr.2017.11.036
Artigo de periodico
Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats (2018)
Flores, Rafael Appel; Silva, Eduardo Simão da; Ribas, Anderson Savaris; Taschetto, Ana Paula Dambros; Zampieri, Thais Tessari; Donato Junior, José ; Paschoalini, Marta Aparecida
Fonte: Brain Research. Unidade: ICB
Assuntos: ADRENALINA, SEROTONINA, INGESTÃO, ALIMENTOS, NEURÔNIOS, RATOS
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FLORES, Rafael Appel et al. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats. Brain Research, v. 1678, p. 153-163, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2017.10.021. Acesso em: 06 nov. 2024.
APA
Flores, R. A., Silva, E. S. da, Ribas, A. S., Taschetto, A. P. D., Zampieri, T. T., Donato Junior, J., & Paschoalini, M. A. (2018). Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats. Brain Research, 1678, 153-163. doi:10.1016/j.brainres.2017.10.021
NLM
Flores RA, Silva ES da, Ribas AS, Taschetto APD, Zampieri TT, Donato Junior J, Paschoalini MA. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats [Internet]. Brain Research. 2018 ; 1678 153-163.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2017.10.021
Vancouver
Flores RA, Silva ES da, Ribas AS, Taschetto APD, Zampieri TT, Donato Junior J, Paschoalini MA. Evaluation of food intake and Fos expression in serotonergic neurons of raphe nuclei after intracerebroventricular injection of adrenaline in free-feeding rats [Internet]. Brain Research. 2018 ; 1678 153-163.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2017.10.021
Artigo de periodico
Melatonin decreases neuronal excitability in a sub-population of dorsal root ganglion neurons (2018)
Abreu, Klausen Oliveira ; Silva, Francisco Walber Ferreira da ; Alves, Kerly Shamyra da Silva ; Santos, Nathalia Maria Silva dos ; Teixeira, Ana Carolina Cardoso ; Amaral, Fernanda Gaspar do; Cipolla Neto, José ; Cardoso, José Henrique Leal
Fonte: Brain Research. Unidade: ICB
Assuntos: MELATONINA, ANTIOXIDANTES, RATOS, REAÇÃO EM CADEIA POR POLIMERASE, ANTAGONISTAS, GÂNGLIOS
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ABNT
ABREU, Klausen Oliveira et al. Melatonin decreases neuronal excitability in a sub-population of dorsal root ganglion neurons. Brain Research, v. 1692, p. 1-8, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2018.04.027. Acesso em: 06 nov. 2024.
APA
Abreu, K. O., Silva, F. W. F. da, Alves, K. S. da S., Santos, N. M. S. dos, Teixeira, A. C. C., Amaral, F. G. do, et al. (2018). Melatonin decreases neuronal excitability in a sub-population of dorsal root ganglion neurons. Brain Research, 1692, 1-8. doi:10.1016/j.brainres.2018.04.027
NLM
Abreu KO, Silva FWF da, Alves KS da S, Santos NMS dos, Teixeira ACC, Amaral FG do, Cipolla Neto J, Cardoso JHL. Melatonin decreases neuronal excitability in a sub-population of dorsal root ganglion neurons [Internet]. Brain Research. 2018 ; 1692 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2018.04.027
Vancouver
Abreu KO, Silva FWF da, Alves KS da S, Santos NMS dos, Teixeira ACC, Amaral FG do, Cipolla Neto J, Cardoso JHL. Melatonin decreases neuronal excitability in a sub-population of dorsal root ganglion neurons [Internet]. Brain Research. 2018 ; 1692 1-8.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2018.04.027
Artigo de periodico
Perillyl alcohol exhibits in vitro inhibitory activity against Plasmodium falciparum and protects against experimental cerebral malaria (2018)
Rodriguez, Adriana A. Marin; Carvalho, Leonardo José de Moura ; Ichikawa, Emilia Akemi Shiraishi Kimura; Katzin, Alejandro Miguel
Fonte: International Journal of Antimicrobial Agents. Unidade: ICB
Assuntos: PARASITOLOGIA, PLASMODIUM FALCIPARUM, MALÁRIA
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ABNT
RODRIGUEZ, Adriana A. Marin et al. Perillyl alcohol exhibits in vitro inhibitory activity against Plasmodium falciparum and protects against experimental cerebral malaria. International Journal of Antimicrobial Agents, v. 51, n. 3, p. 370-377, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.ijantimicag.2017.08.025. Acesso em: 06 nov. 2024.
APA
Rodriguez, A. A. M., Carvalho, L. J. de M., Ichikawa, E. A. S. K., & Katzin, A. M. (2018). Perillyl alcohol exhibits in vitro inhibitory activity against Plasmodium falciparum and protects against experimental cerebral malaria. International Journal of Antimicrobial Agents, 51( 3), 370-377. doi:10.1016/j.ijantimicag.2017.08.025
NLM
Rodriguez AAM, Carvalho LJ de M, Ichikawa EASK, Katzin AM. Perillyl alcohol exhibits in vitro inhibitory activity against Plasmodium falciparum and protects against experimental cerebral malaria [Internet]. International Journal of Antimicrobial Agents. 2018 ; 51( 3): 370-377.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijantimicag.2017.08.025
Vancouver
Rodriguez AAM, Carvalho LJ de M, Ichikawa EASK, Katzin AM. Perillyl alcohol exhibits in vitro inhibitory activity against Plasmodium falciparum and protects against experimental cerebral malaria [Internet]. International Journal of Antimicrobial Agents. 2018 ; 51( 3): 370-377.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijantimicag.2017.08.025
Artigo de periodico
NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance (2018)
Crunfli, Fernanda ; Mazucanti, Caio Henrique Yokoyama ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Rodrigues, Alice Cristina ; Scavone, Cristoforo ; Torrão, Andréa da Silva
Fonte: Journal of Alzheimer's Disease. Unidade: ICB
Assuntos: INSULINA, DOENÇA DE ALZHEIMER, NEURÔNIOS, PROTEÍNAS QUINASES, METABOLISMO ENERGÉTICO, RECEPTORES DE INSULINA
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CRUNFLI, Fernanda et al. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, v. 65, n. 4, p. 1427-1443, 2018Tradução . . Disponível em: https://doi.org/10.3233/JAD-180284. Acesso em: 06 nov. 2024.
APA
Crunfli, F., Mazucanti, C. H. Y., Moraes, R. C. M. de, Costa, A. P., Rodrigues, A. C., Scavone, C., & Torrão, A. da S. (2018). NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, 65( 4), 1427-1443. doi:10.3233/JAD-180284
NLM
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3233/JAD-180284
Vancouver
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 nov. 06 ] Available from: https://doi.org/10.3233/JAD-180284
Artigo de periodico
Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model (2018)
Santos, Fabio Martinez dos; Silva, Joyce Teixeira; Rocha, Igor Rafael Correia; Martins, Daniel Oliveira; Chacur, Marucia
Fonte: Brain Research. Unidade: ICB
Assuntos: NERVO CIÁTICO, SUBSTÂNCIA P, DOR, DOENÇAS DO SISTEMA NERVOSO PERIFÉRICO, NEUROPEPTÍDEOS, FISIOTERAPIA, GÂNGLIOS, WESTERN BLOTTING
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ABNT
SANTOS, Fabio Martinez dos et al. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model. Brain Research, v. 1687, p. 60-65, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.brainres.2018.02.034. Acesso em: 06 nov. 2024.
APA
Santos, F. M. dos, Silva, J. T., Rocha, I. R. C., Martins, D. O., & Chacur, M. (2018). Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model. Brain Research, 1687, 60-65. doi:10.1016/j.brainres.2018.02.034
NLM
Santos FM dos, Silva JT, Rocha IRC, Martins DO, Chacur M. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model [Internet]. Brain Research. 2018 ; 1687 60-65.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2018.02.034
Vancouver
Santos FM dos, Silva JT, Rocha IRC, Martins DO, Chacur M. Non-pharmacological treatment affects neuropeptide expression in neuropathic pain model [Internet]. Brain Research. 2018 ; 1687 60-65.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.brainres.2018.02.034
Artigo de periodico
Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus (2016)
Ferreira, Ivan Gonçalves de Castro; Rodrigues, Mirian Molnar; Silva Neto, José Freire da; Mazzon, Ricardo Ruiz; Marques, Marilis do Valle
Fonte: Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. Unidades: FMRP, ICB
Assuntos: HOMEOSTASE, FERRO, ESTRESSE OXIDATIVO, PROTEÍNAS
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ABNT
FERREIRA, Ivan Gonçalves de Castro et al. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine, v. 29, n. 5, p. 851-862, 2016Tradução . . Disponível em: https://doi.org/10.1007/s10534-016-9956-y. Acesso em: 06 nov. 2024.
APA
Ferreira, I. G. de C., Rodrigues, M. M., Silva Neto, J. F. da, Mazzon, R. R., & Marques, M. do V. (2016). Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine, 29( 5), 851-862. doi:10.1007/s10534-016-9956-y
NLM
Ferreira IG de C, Rodrigues MM, Silva Neto JF da, Mazzon RR, Marques M do V. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus [Internet]. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. 2016 ; 29( 5): 851-862.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s10534-016-9956-y
Vancouver
Ferreira IG de C, Rodrigues MM, Silva Neto JF da, Mazzon RR, Marques M do V. Role and regulation of ferritin-like proteins in iron homeostasis and oxidative stress survival of Caulobacter crescentus [Internet]. Biometals : an international journal on the role of metal ions in biology, biochemistry, and medicine. 2016 ; 29( 5): 851-862.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1007/s10534-016-9956-y
Artigo de periodico
Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation (2016)
Ribeiro, André Luis Ribeiro; Costa, Natacha Malu Miranda; Siqueira, Adriane Sousa de; Silva, Walessa Brasil da; Kataoka, Maria Sueli da Silva; Jaeger, Ruy Gastaldoni ; Alves Junior, Sérgio de Melo; Smith, Andrew M; Pinheiro, João de Jesus Viana
Fonte: Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. Unidade: ICB
Assuntos: TUMORES ODONTOGÊNICOS, CISTO ODONTOGÊNICO CALCIFICANTE, MUCOSA ORAL
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ABNT
RIBEIRO, André Luis Ribeiro et al. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, v. 122, n. 4, p. 500-508, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.oooo.2016.06.004. Acesso em: 06 nov. 2024.
APA
Ribeiro, A. L. R., Costa, N. M. M., Siqueira, A. S. de, Silva, W. B. da, Kataoka, M. S. da S., Jaeger, R. G., et al. (2016). Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology, 122( 4), 500-508. doi:10.1016/j.oooo.2016.06.004
NLM
Ribeiro ALR, Costa NMM, Siqueira AS de, Silva WB da, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2016 ; 122( 4): 500-508.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.oooo.2016.06.004
Vancouver
Ribeiro ALR, Costa NMM, Siqueira AS de, Silva WB da, Kataoka MS da S, Jaeger RG, Alves Junior S de M, Smith AM, Pinheiro J de JV. Keratocystic odontogenic tumor overexpresses invadopodia-related proteins, suggesting invadopodia formation [Internet]. Oral Surgery, Oral Medicine, Oral Pathology and Oral Radiology. 2016 ; 122( 4): 500-508.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.oooo.2016.06.004
Artigo de periodico
Resveratrol inhibits Trypanosoma cruzi arginine kinase and exerts a trypanocidal activity (2016)
Vera, Edward A. Valera; Sayé, Melisa; Reigada, Chantal; Damasceno, Damasceno; Silber, Ariel Mariano ; Miranda, Mariana R.; Pereira, Claudio A.
Fonte: International Journal of Biological Macromolecules. Unidade: ICB
Assuntos: PARASITOLOGIA, DOENÇA DE CHAGAS, INIBIDORES QUÍMICOS, TRYPANOSOMA CRUZI
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ABNT
VERA, Edward A. Valera et al. Resveratrol inhibits Trypanosoma cruzi arginine kinase and exerts a trypanocidal activity. International Journal of Biological Macromolecules, v. 87, p. 498-503, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijbiomac.2016.03.014. Acesso em: 06 nov. 2024.
APA
Vera, E. A. V., Sayé, M., Reigada, C., Damasceno, D., Silber, A. M., Miranda, M. R., & Pereira, C. A. (2016). Resveratrol inhibits Trypanosoma cruzi arginine kinase and exerts a trypanocidal activity. International Journal of Biological Macromolecules, 87, 498-503. doi:10.1016/j.ijbiomac.2016.03.014
NLM
Vera EAV, Sayé M, Reigada C, Damasceno D, Silber AM, Miranda MR, Pereira CA. Resveratrol inhibits Trypanosoma cruzi arginine kinase and exerts a trypanocidal activity [Internet]. International Journal of Biological Macromolecules. 2016 ; 87 498-503.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.03.014
Vancouver
Vera EAV, Sayé M, Reigada C, Damasceno D, Silber AM, Miranda MR, Pereira CA. Resveratrol inhibits Trypanosoma cruzi arginine kinase and exerts a trypanocidal activity [Internet]. International Journal of Biological Macromolecules. 2016 ; 87 498-503.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijbiomac.2016.03.014
Artigo de periodico
Genetic diversity of bats coronaviruses in the Atlantic Forest hotspot biome, Brazil (2016)
Góes, Luiz Gustavo Bentim; Campos, Angélica Cristine de Almeida; Carvalho, Cristiano de; Ambar, Guilherme; Queiroz, Luzia Helena; Cruz Neto, Ariovaldo Pereira; Munir, Muhammad; Durigon, Edison Luiz
Fonte: Infection, Genetics and Evolution. Unidade: ICB
Assuntos: DIVERSIDADE GENÉTICA, CORONAVIRUS, MORCEGOS, SAÚDE PÚBLICA
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ABNT
GÓES, Luiz Gustavo Bentim et al. Genetic diversity of bats coronaviruses in the Atlantic Forest hotspot biome, Brazil. Infection, Genetics and Evolution, v. 44, p. 510-513, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.meegid.2016.07.034. Acesso em: 06 nov. 2024.
APA
Góes, L. G. B., Campos, A. C. de A., Carvalho, C. de, Ambar, G., Queiroz, L. H., Cruz Neto, A. P., et al. (2016). Genetic diversity of bats coronaviruses in the Atlantic Forest hotspot biome, Brazil. Infection, Genetics and Evolution, 44, 510-513. doi:10.1016/j.meegid.2016.07.034
NLM
Góes LGB, Campos AC de A, Carvalho C de, Ambar G, Queiroz LH, Cruz Neto AP, Munir M, Durigon EL. Genetic diversity of bats coronaviruses in the Atlantic Forest hotspot biome, Brazil [Internet]. Infection, Genetics and Evolution. 2016 ; 44 510-513.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.meegid.2016.07.034
Vancouver
Góes LGB, Campos AC de A, Carvalho C de, Ambar G, Queiroz LH, Cruz Neto AP, Munir M, Durigon EL. Genetic diversity of bats coronaviruses in the Atlantic Forest hotspot biome, Brazil [Internet]. Infection, Genetics and Evolution. 2016 ; 44 510-513.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.meegid.2016.07.034
Artigo de periodico
Microenvironment and autophagy cross-talk: implications in cancer therapy (2016)
Gomes, Luciana R.; Vessoni, Alexandre T.; Menck, Carlos Frederico Martins
Fonte: Pharmacological Research. Unidade: ICB
Assunto: MICROBIOLOGIA
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ABNT
GOMES, Luciana R. e VESSONI, Alexandre T. e MENCK, Carlos Frederico Martins. Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, v. 107, p. 300-307, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2016.03.031. Acesso em: 06 nov. 2024.
APA
Gomes, L. R., Vessoni, A. T., & Menck, C. F. M. (2016). Microenvironment and autophagy cross-talk: implications in cancer therapy. Pharmacological Research, 107, 300-307. doi:10.1016/j.phrs.2016.03.031
NLM
Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
Vancouver
Gomes LR, Vessoni AT, Menck CFM. Microenvironment and autophagy cross-talk: implications in cancer therapy [Internet]. Pharmacological Research. 2016 ; 107 300-307.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.phrs.2016.03.031
Artigo de periodico
Molecular mechanisms of membrane impermeability in clinical isolates of Enterobacteriaceae exposed to imipenem selective pressure (2016)
Pavez, Mónica; Vieira, Camila; Araújo, Maria Rita de; Cerda, Alvaro; Almeida, Lara Mendes de; Lincopan, Nilton ; Mamizuka, Elsa Masae
Fonte: International Journal of Antimicrobial Agents. Unidades: FCF, ICB
Assuntos: ENTEROBACTERIACEAE, RESISTÊNCIA MICROBIANA ÀS DROGAS
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ABNT
PAVEZ, Mónica et al. Molecular mechanisms of membrane impermeability in clinical isolates of Enterobacteriaceae exposed to imipenem selective pressure. International Journal of Antimicrobial Agents, v. 48, n. 1, p. 78-85, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ijantimicag.2016.04.016. Acesso em: 06 nov. 2024.
APA
Pavez, M., Vieira, C., Araújo, M. R. de, Cerda, A., Almeida, L. M. de, Lincopan, N., & Mamizuka, E. M. (2016). Molecular mechanisms of membrane impermeability in clinical isolates of Enterobacteriaceae exposed to imipenem selective pressure. International Journal of Antimicrobial Agents, 48( 1), 78-85. doi:10.1016/j.ijantimicag.2016.04.016
NLM
Pavez M, Vieira C, Araújo MR de, Cerda A, Almeida LM de, Lincopan N, Mamizuka EM. Molecular mechanisms of membrane impermeability in clinical isolates of Enterobacteriaceae exposed to imipenem selective pressure [Internet]. International Journal of Antimicrobial Agents. 2016 ; 48( 1): 78-85.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijantimicag.2016.04.016
Vancouver
Pavez M, Vieira C, Araújo MR de, Cerda A, Almeida LM de, Lincopan N, Mamizuka EM. Molecular mechanisms of membrane impermeability in clinical isolates of Enterobacteriaceae exposed to imipenem selective pressure [Internet]. International Journal of Antimicrobial Agents. 2016 ; 48( 1): 78-85.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijantimicag.2016.04.016
Artigo de periodico
Bacterial spores as particulate carriers for gene gun delivery of plasmid DNA (2016)
Aps, Luana R. M. M.; Tavares, Milene B.; Rozenfeld, Julio H. K.; Diniz, Mariana O; Ferreira, Luís Carlos de Souza ; Lamy, Maria Teresa Moura
Fonte: Journal of Biotechnology. Unidades: ICB, IF
Assuntos: IMUNIZAÇÃO, DNA, MICROBIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
APS, Luana R. M. M. et al. Bacterial spores as particulate carriers for gene gun delivery of plasmid DNA. Journal of Biotechnology, v. 228, n. ju 2016, p. 58-66, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.jbiotec.2016.04.027. Acesso em: 06 nov. 2024.
APA
Aps, L. R. M. M., Tavares, M. B., Rozenfeld, J. H. K., Diniz, M. O., Ferreira, L. C. de S., & Lamy, M. T. M. (2016). Bacterial spores as particulate carriers for gene gun delivery of plasmid DNA. Journal of Biotechnology, 228( ju 2016), 58-66. doi:10.1016/j.jbiotec.2016.04.027
NLM
Aps LRMM, Tavares MB, Rozenfeld JHK, Diniz MO, Ferreira LC de S, Lamy MTM. Bacterial spores as particulate carriers for gene gun delivery of plasmid DNA [Internet]. Journal of Biotechnology. 2016 ; 228( ju 2016): 58-66.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jbiotec.2016.04.027
Vancouver
Aps LRMM, Tavares MB, Rozenfeld JHK, Diniz MO, Ferreira LC de S, Lamy MTM. Bacterial spores as particulate carriers for gene gun delivery of plasmid DNA [Internet]. Journal of Biotechnology. 2016 ; 228( ju 2016): 58-66.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jbiotec.2016.04.027
Artigo de periodico
Quantification of nerolidol in mouse plasma using gas chromatography-mass spectrometry (2015)
Saito, Alexandre Yukio; Sussmann, Rodrigo Antonio Ceschini; Kimura, Emilia Akemi; Cassera, Maria Belen; Katzin, Alejandro Miguel
Fonte: Journal of Pharmaceutical and Biomedical Analysis. Unidade: ICB
Assuntos: CAMUNDONGO, PLASMA DE ANIMAL, CROMATOGRAFIA A GAS, ESPECTROMETRIA DE MASSAS, ÓLEOS ANIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SAITO, Alexandre Yukio et al. Quantification of nerolidol in mouse plasma using gas chromatography-mass spectrometry. Journal of Pharmaceutical and Biomedical Analysis, v. 111, p. 100-103, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.jpba.2015.03.030. Acesso em: 06 nov. 2024.
APA
Saito, A. Y., Sussmann, R. A. C., Kimura, E. A., Cassera, M. B., & Katzin, A. M. (2015). Quantification of nerolidol in mouse plasma using gas chromatography-mass spectrometry. Journal of Pharmaceutical and Biomedical Analysis, 111, 100-103. doi:10.1016/j.jpba.2015.03.030
NLM
Saito AY, Sussmann RAC, Kimura EA, Cassera MB, Katzin AM. Quantification of nerolidol in mouse plasma using gas chromatography-mass spectrometry [Internet]. Journal of Pharmaceutical and Biomedical Analysis. 2015 ; 111 100-103.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jpba.2015.03.030
Vancouver
Saito AY, Sussmann RAC, Kimura EA, Cassera MB, Katzin AM. Quantification of nerolidol in mouse plasma using gas chromatography-mass spectrometry [Internet]. Journal of Pharmaceutical and Biomedical Analysis. 2015 ; 111 100-103.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jpba.2015.03.030
Artigo de periodico
Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats (2015)
Alves-Wagner, Ana Barbara; Mori, Rosana Cristina; Silva-Sabino, Robinson; Fatima, Luciana Alves; Alves, Adilson da Silva; Britto, Luiz Roberto Giorgetti de ; Schaan, Beatriz D'Agord; Machado, Ubiratan Fabres
Fonte: Autonomic Neuroscience: Basic and Clinical. Unidade: ICB
Assuntos: INSULINA, RATOS WISTAR, GLICEMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ALVES-WAGNER, Ana Barbara et al. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats. Autonomic Neuroscience: Basic and Clinical, v. 193, p. 108-116, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.autneu.2015.10.003. Acesso em: 06 nov. 2024.
APA
Alves-Wagner, A. B., Mori, R. C., Silva-Sabino, R., Fatima, L. A., Alves, A. da S., Britto, L. R. G. de, et al. (2015). Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats. Autonomic Neuroscience: Basic and Clinical, 193, 108-116. doi:10.1016/j.autneu.2015.10.003
NLM
Alves-Wagner AB, Mori RC, Silva-Sabino R, Fatima LA, Alves A da S, Britto LRG de, Schaan BD'A, Machado UF. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2015 ; 193 108-116.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.autneu.2015.10.003
Vancouver
Alves-Wagner AB, Mori RC, Silva-Sabino R, Fatima LA, Alves A da S, Britto LRG de, Schaan BD'A, Machado UF. Beta-adrenergic blockade increases GLUT4 and improves glycemic control in insulin-treated diabetic Wistar rats [Internet]. Autonomic Neuroscience: Basic and Clinical. 2015 ; 193 108-116.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.autneu.2015.10.003
Artigo de periodico
Quantitative proteomics analysis of platelet-derived microparticles reveals distinct protein signatures when stimulated by different physiological agonists (2015)
Milioli, Marco; Ibáñez-Vea, Maria; Sidoli, Simone; Palmisano, Giuseppe ; Careri, Maria; Larsen, Martin Rossel
Fonte: Journal of Proteomics. Unidade: ICB
Assuntos: PARASITOLOGIA, PROTEINAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MILIOLI, Marco et al. Quantitative proteomics analysis of platelet-derived microparticles reveals distinct protein signatures when stimulated by different physiological agonists. Journal of Proteomics, v. 121, p. 56-66, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.jprot.2015.03.01. Acesso em: 06 nov. 2024.
APA
Milioli, M., Ibáñez-Vea, M., Sidoli, S., Palmisano, G., Careri, M., & Larsen, M. R. (2015). Quantitative proteomics analysis of platelet-derived microparticles reveals distinct protein signatures when stimulated by different physiological agonists. Journal of Proteomics, 121, 56-66. doi:10.1016/j.jprot.2015.03.01
NLM
Milioli M, Ibáñez-Vea M, Sidoli S, Palmisano G, Careri M, Larsen MR. Quantitative proteomics analysis of platelet-derived microparticles reveals distinct protein signatures when stimulated by different physiological agonists [Internet]. Journal of Proteomics. 2015 ; 121 56-66.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jprot.2015.03.01
Vancouver
Milioli M, Ibáñez-Vea M, Sidoli S, Palmisano G, Careri M, Larsen MR. Quantitative proteomics analysis of platelet-derived microparticles reveals distinct protein signatures when stimulated by different physiological agonists [Internet]. Journal of Proteomics. 2015 ; 121 56-66.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.jprot.2015.03.01
Artigo de periodico
Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) (2015)
Echem, Cinthya; Bomfim, Gisele Facholi; Ceravolo, Graziela Scalianti; Oliveira, Maria Aparecida; Eichler, Rosangela Aparecida Santos; Bechara, Luiz Roberto; Veras, Mariana Matera; Saldiva, Paulo Hilário Nascimento ; Ferreira, Julio Cesar Batista ; Akamine, Eliana Hiromi ; Fortes, Zuleica Bruno; Dantas, Ana Paula; Carvalho, Maria Helena Catelli
Fonte: International Journal of Cardiology. Unidades: ICB, FM
Assuntos: ANATOMIA, FARMACOLOGIA, HIPERTENSÃO, RATOS, PRESSÃO SANGUÍNEA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ECHEM, Cinthya et al. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, v. 187, p. 243-245, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.ijcard.2015.03.190. Acesso em: 06 nov. 2024.
APA
Echem, C., Bomfim, G. F., Ceravolo, G. S., Oliveira, M. A., Eichler, R. A. S., Bechara, L. R., et al. (2015). Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR). International Journal of Cardiology, 187, 243-245. doi:10.1016/j.ijcard.2015.03.190
NLM
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Vancouver
Echem C, Bomfim GF, Ceravolo GS, Oliveira MA, Eichler RAS, Bechara LR, Veras MM, Saldiva PHN, Ferreira JCB, Akamine EH, Fortes ZB, Dantas AP, Carvalho MHC. Anti-toll like receptor 4 (TLR4) therapy diminishes cardiac remodeling regardless of changes in blood pressure in spontaneously hypertensive rats (SHR) [Internet]. International Journal of Cardiology. 2015 ; 187 243-245.[citado 2024 nov. 06 ] Available from: https://doi.org/10.1016/j.ijcard.2015.03.190
Artigo de periodico
Potential of peptide-based enhancers for transdermal delivery (2015)
Lopes, Luciana Biagini ; Carvalho, Vanessa Freitas Mendonça de; Lemos, Débora P. de
Fonte: Current Pharmaceutical Design. Unidade: ICB
Assuntos: FARMACOLOGIA, PEPTIDEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LOPES, Luciana Biagini e CARVALHO, Vanessa Freitas Mendonça de e LEMOS, Débora P. de. Potential of peptide-based enhancers for transdermal delivery. Current Pharmaceutical Design, v. 21, n. 20, p. 2814-2822, 2015Tradução . . Disponível em: https://doi.org/10.2174/1381612821666150428143243. Acesso em: 06 nov. 2024.
APA
Lopes, L. B., Carvalho, V. F. M. de, & Lemos, D. P. de. (2015). Potential of peptide-based enhancers for transdermal delivery. Current Pharmaceutical Design, 21( 20), 2814-2822. doi:10.2174/1381612821666150428143243
NLM
Lopes LB, Carvalho VFM de, Lemos DP de. Potential of peptide-based enhancers for transdermal delivery [Internet]. Current Pharmaceutical Design. 2015 ; 21( 20): 2814-2822.[citado 2024 nov. 06 ] Available from: https://doi.org/10.2174/1381612821666150428143243
Vancouver
Lopes LB, Carvalho VFM de, Lemos DP de. Potential of peptide-based enhancers for transdermal delivery [Internet]. Current Pharmaceutical Design. 2015 ; 21( 20): 2814-2822.[citado 2024 nov. 06 ] Available from: https://doi.org/10.2174/1381612821666150428143243

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