Filtros : "IQ005" "RECEPTORES" "Meotti, Flavia Carla" Removidos: "CARVALHO, DIRCEU TORNAVOI DE" "SBBq" Limpar

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  • Source: Biochemical Pharmacology. Unidade: IQ

    Subjects: RECEPTORES, CININAS

    Acesso à fonteDOIHow to cite
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    • ABNT

      MEOTTI, Flavia Carla et al. The transient receptor potential ankyrin-1 mediates mechanical hyperalgesia induced by the activation of 'B IND. 1' receptor in mice. Biochemical Pharmacology, v. 125, n. 2017, p. 75-83, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.bcp.2016.11.003. Acesso em: 31 out. 2024.
    • APA

      Meotti, F. C., Figueiredo, C. P., Manjavachi, M., & Calixto, J. B. (2017). The transient receptor potential ankyrin-1 mediates mechanical hyperalgesia induced by the activation of 'B IND. 1' receptor in mice. Biochemical Pharmacology, 125( 2017), 75-83. doi:10.1016/j.bcp.2016.11.003
    • NLM

      Meotti FC, Figueiredo CP, Manjavachi M, Calixto JB. The transient receptor potential ankyrin-1 mediates mechanical hyperalgesia induced by the activation of 'B IND. 1' receptor in mice [Internet]. Biochemical Pharmacology. 2017 ; 125( 2017): 75-83.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.bcp.2016.11.003
    • Vancouver

      Meotti FC, Figueiredo CP, Manjavachi M, Calixto JB. The transient receptor potential ankyrin-1 mediates mechanical hyperalgesia induced by the activation of 'B IND. 1' receptor in mice [Internet]. Biochemical Pharmacology. 2017 ; 125( 2017): 75-83.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.bcp.2016.11.003
  • Source: Brazilian Journal of Pharmaceutical Sciences. Unidade: IQ

    Assunto: RECEPTORES

    How to cite
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    • ABNT

      MEOTTI, Flavia Carla. Resenha sem título próprio. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Instituto de Química, Universidade de São Paulo. . Acesso em: 31 out. 2024. , 2014
    • APA

      Meotti, F. C. (2014). Resenha sem título próprio. Brazilian Journal of Pharmaceutical Sciences. São Paulo: Instituto de Química, Universidade de São Paulo.
    • NLM

      Meotti FC. Resenha sem título próprio. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50( 1): 228.[citado 2024 out. 31 ]
    • Vancouver

      Meotti FC. Resenha sem título próprio. Brazilian Journal of Pharmaceutical Sciences. 2014 ; 50( 1): 228.[citado 2024 out. 31 ]
  • Source: Neuroscience. Unidade: IQ

    Subjects: RECEPTORES, GLUTAMATOS

    Acesso à fonteDOIHow to cite
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    • ABNT

      BICCA, M. A et al. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, v. 192, n. 29, p. 631-641, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2011.06.038. Acesso em: 31 out. 2024.
    • APA

      Bicca, M. A., Figueiredo, C. P., Piermartiri, T. C., Meotti, F. C., Bouzon, Z. L., Tasca, C. I., et al. (2011). The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice. Neuroscience, 192( 29), 631-641. doi:10.1016/j.neuroscience.2011.06.038
    • NLM

      Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038
    • Vancouver

      Bicca MA, Figueiredo CP, Piermartiri TC, Meotti FC, Bouzon ZL, Tasca CI, Medeiros R, Calixto JB. The selective and competitive N-methyl-D-aspartate receptor antogonist, (-)-6-phosphonomethyl-deca-hydroisoquinoline-3-carboxylic acid, prevents synaptic toxicity induced by amyloid-β in mice [Internet]. Neuroscience. 2011 ; 192( 29): 631-641.[citado 2024 out. 31 ] Available from: https://doi.org/10.1016/j.neuroscience.2011.06.038

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