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Artigo de periodico
Behavior of the biological control agent Bacillus thuringiensis subsp. aizawai ABTS-1857 and Salmonella enterica on spinach plants and cut leaves (2021)
Zhao, Xingchen; Silva, Marcelo Belchior Rosendo da ; Linden, Inge Van der; Franco, Bernadette Dora Gombossy de Melo ; Uyttendaele, Mieke
Source: Frontiers in Microbiology. Unidade: FCF
Subjects: CONTROLE BIOLÓGICO, SALMONELLA, ESPINAFRE, CÉLULAS VEGETAIS
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ZHAO, Xingchen et al. Behavior of the biological control agent Bacillus thuringiensis subsp. aizawai ABTS-1857 and Salmonella enterica on spinach plants and cut leaves. Frontiers in Microbiology, v. 12, p. 1-14 art. 626029, 2021Tradução . . Disponível em: https://doi.org/10.3389/fmicb.2021.626029. Acesso em: 03 nov. 2024.
APA
Zhao, X., Silva, M. B. R. da, Linden, I. V. der, Franco, B. D. G. de M., & Uyttendaele, M. (2021). Behavior of the biological control agent Bacillus thuringiensis subsp. aizawai ABTS-1857 and Salmonella enterica on spinach plants and cut leaves. Frontiers in Microbiology, 12, 1-14 art. 626029. doi:10.3389/fmicb.2021.626029
NLM
Zhao X, Silva MBR da, Linden IV der, Franco BDG de M, Uyttendaele M. Behavior of the biological control agent Bacillus thuringiensis subsp. aizawai ABTS-1857 and Salmonella enterica on spinach plants and cut leaves [Internet]. Frontiers in Microbiology. 2021 ; 12 1-14 art. 626029.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2021.626029
Vancouver
Zhao X, Silva MBR da, Linden IV der, Franco BDG de M, Uyttendaele M. Behavior of the biological control agent Bacillus thuringiensis subsp. aizawai ABTS-1857 and Salmonella enterica on spinach plants and cut leaves [Internet]. Frontiers in Microbiology. 2021 ; 12 1-14 art. 626029.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3389/fmicb.2021.626029
Artigo de periodico
Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis (2020)
Romualdo, Guilherme Ribeiro; Silva, Elizangela dos Anjos; Silva, Tereza Cristina da; Aloiav, Thiago Pinheiro Arrais ; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Vinken, Mathieu ; Barbisan, Luis Fernando ; Cogliati, Bruno
Source: Environmental Toxicology. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, CARCINOGÊNESE ANIMAL, CHÁ, PLANTAS MEDICINAIS
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ROMUALDO, Guilherme Ribeiro et al. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis. Environmental Toxicology, v. 35, n. 4, p. 518-527, 2020Tradução . . Disponível em: https://doi.org/10.1002/tox.22887. Acesso em: 03 nov. 2024.
APA
Romualdo, G. R., Silva, E. dos A., Silva, T. C. da, Aloiav, T. P. A., Nogueira, M. S., Castro, I. A. de, et al. (2020). Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis. Environmental Toxicology, 35( 4), 518-527. doi:10.1002/tox.22887
NLM
Romualdo GR, Silva E dos A, Silva TC da, Aloiav TPA, Nogueira MS, Castro IA de, Vinken M, Barbisan LF, Cogliati B. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis [Internet]. Environmental Toxicology. 2020 ; 35( 4): 518-527.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1002/tox.22887
Vancouver
Romualdo GR, Silva E dos A, Silva TC da, Aloiav TPA, Nogueira MS, Castro IA de, Vinken M, Barbisan LF, Cogliati B. Burdock (Arctium lappa L.) root attenuates preneoplastic lesion development in a diet and thioacetamide-induced model of steatohepatitis-associated hepatocarcinogenesis [Internet]. Environmental Toxicology. 2020 ; 35( 4): 518-527.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1002/tox.22887
Artigo de periodico
Drinking for protection? Epidemiological and experimental evidence on the beneficial effects of coffee or major coffee compounds against gastrointestinal and liver carcinogenesis (2019)
Romualdo, Guilherme Ribeiro; Rocha, Ariane Bartolomeu; Vinken, Mathieu; Cogliati, Bruno ; Moreno, Fernando Salvador ; Chaves, María Angel García; Barbisan, Luis Fernando
Source: Food Research International. Unidades: FMVZ, FCF
Subjects: CAFÉ TRATADO, CAFEÍNA, NEOPLASIAS GASTROINTESTINAIS, CARCINOGÊNESE
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ROMUALDO, Guilherme Ribeiro et al. Drinking for protection? Epidemiological and experimental evidence on the beneficial effects of coffee or major coffee compounds against gastrointestinal and liver carcinogenesis. Food Research International, v. 123, p. 567-589, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.foodres.2019.05.029. Acesso em: 03 nov. 2024.
APA
Romualdo, G. R., Rocha, A. B., Vinken, M., Cogliati, B., Moreno, F. S., Chaves, M. A. G., & Barbisan, L. F. (2019). Drinking for protection? Epidemiological and experimental evidence on the beneficial effects of coffee or major coffee compounds against gastrointestinal and liver carcinogenesis. Food Research International, 123, 567-589. doi:10.1016/j.foodres.2019.05.029
NLM
Romualdo GR, Rocha AB, Vinken M, Cogliati B, Moreno FS, Chaves MAG, Barbisan LF. Drinking for protection? Epidemiological and experimental evidence on the beneficial effects of coffee or major coffee compounds against gastrointestinal and liver carcinogenesis [Internet]. Food Research International. 2019 ; 123 567-589.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.foodres.2019.05.029
Vancouver
Romualdo GR, Rocha AB, Vinken M, Cogliati B, Moreno FS, Chaves MAG, Barbisan LF. Drinking for protection? Epidemiological and experimental evidence on the beneficial effects of coffee or major coffee compounds against gastrointestinal and liver carcinogenesis [Internet]. Food Research International. 2019 ; 123 567-589.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.foodres.2019.05.029
Artigo de periodico
Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model (2018)
Yanguas, Sara Crespo; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Maes, Michaël; Willebrords, Joost; Shestopalov, Valery I; Goes, Bruna Marcela; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Romualdo, Guilherme Ribeiro; Barbisan, Luis Fernando; Gijbels, Eva; Vinken, Mathieu; Cogliati, Bruno
Source: Archives of Toxicology. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, BIOMARCADORES, PROTEÍNAS DA MEMBRANA, MODELOS ANIMAIS DE DOENÇAS
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YANGUAS, Sara Crespo et al. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model. Archives of Toxicology, v. 92, n. 8, p. 2607-2627, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00204-018-2255-3. Acesso em: 03 nov. 2024.
APA
Yanguas, S. C., Silva, T. C. da, Pereira, I. V. A., Maes, M., Willebrords, J., Shestopalov, V. I., et al. (2018). Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model. Archives of Toxicology, 92( 8), 2607-2627. doi:10.1007/s00204-018-2255-3
NLM
Yanguas SC, Silva TC da, Pereira IVA, Maes M, Willebrords J, Shestopalov VI, Goes BM, Nogueira MS, Castro IA de, Romualdo GR, Barbisan LF, Gijbels E, Vinken M, Cogliati B. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model [Internet]. Archives of Toxicology. 2018 ; 92( 8): 2607-2627.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1007/s00204-018-2255-3
Vancouver
Yanguas SC, Silva TC da, Pereira IVA, Maes M, Willebrords J, Shestopalov VI, Goes BM, Nogueira MS, Castro IA de, Romualdo GR, Barbisan LF, Gijbels E, Vinken M, Cogliati B. Genetic ablation of pannexin1 counteracts liver fibrosis in a chemical, but not in a surgical mouse model [Internet]. Archives of Toxicology. 2018 ; 92( 8): 2607-2627.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1007/s00204-018-2255-3
Artigo de periodico
Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease (2018)
Willebrords, Joost; Maes, Michaël; Pereira, Isabel Veloso Alves; Silva, Tereza Cristina da; Govoni, Verônica Mollica; Lopes, Valéria Veras; Yanguas, Sara Crespo; Shestopalov, Valery I; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Farhood, Anwar; Mannaerts, Inge; Van Grunsven, Leo; Akakpo, Jephte; Lebofsky, Margitta; Jaeschke, Hartmut; Cogliati, Bruno ; Vinken, Mathieu
Source: Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. Unidades: FCF, FMVZ
Subjects: HEPATOPATIAS, PROTEÍNAS DA MEMBRANA, PATOGENIA ANIMAL
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WILLEBRORDS, Joost et al. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, v. 1864, n. 3, p. 819-830, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.bbadis.2017.12.013. Acesso em: 03 nov. 2024.
APA
Willebrords, J., Maes, M., Pereira, I. V. A., Silva, T. C. da, Govoni, V. M., Lopes, V. V., et al. (2018). Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 1864( 3), 819-830. doi:10.1016/j.bbadis.2017.12.013
NLM
Willebrords J, Maes M, Pereira IVA, Silva TC da, Govoni VM, Lopes VV, Yanguas SC, Shestopalov VI, Nogueira MS, Castro IA de, Farhood A, Mannaerts I, Van Grunsven L, Akakpo J, Lebofsky M, Jaeschke H, Cogliati B, Vinken M. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2018 ; 1864( 3): 819-830.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.bbadis.2017.12.013
Vancouver
Willebrords J, Maes M, Pereira IVA, Silva TC da, Govoni VM, Lopes VV, Yanguas SC, Shestopalov VI, Nogueira MS, Castro IA de, Farhood A, Mannaerts I, Van Grunsven L, Akakpo J, Lebofsky M, Jaeschke H, Cogliati B, Vinken M. Protective effect of genetic deletion of pannexin1 in experimental mouse models of acute and chronic liver disease [Internet]. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 2018 ; 1864( 3): 819-830.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1016/j.bbadis.2017.12.013
Artigo de periodico
TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice (2018)
Yanguas, Sara Crespo; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Willebrords, Joost; Maes, Michaël; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Leclercq, Isabelle; Romualdo, Guilherme Ribeiro; Barbisan, Luis Fernando; Leybaert, Luc; Cogliati, Bruno ; Vinken, Mathieu
Source: International Journal of Molecular Sciences. Unidades: FMVZ, FCF
Subjects: HEPATOPATIAS, CONEXINAS, FIBROSE CÍSTICA
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YANGUAS, Sara Crespo et al. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice. International Journal of Molecular Sciences, v. 19, n. 3, p. 1-17, 2018Tradução . . Disponível em: https://doi.org/10.3390/ijms19030817. Acesso em: 03 nov. 2024.
APA
Yanguas, S. C., Silva, T. C. da, Pereira, I. V. A., Willebrords, J., Maes, M., Nogueira, M. S., et al. (2018). TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice. International Journal of Molecular Sciences, 19( 3), 1-17. doi:10.3390/ijms19030817
NLM
Yanguas SC, Silva TC da, Pereira IVA, Willebrords J, Maes M, Nogueira MS, Castro IA de, Leclercq I, Romualdo GR, Barbisan LF, Leybaert L, Cogliati B, Vinken M. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 3): 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3390/ijms19030817
Vancouver
Yanguas SC, Silva TC da, Pereira IVA, Willebrords J, Maes M, Nogueira MS, Castro IA de, Leclercq I, Romualdo GR, Barbisan LF, Leybaert L, Cogliati B, Vinken M. TAT-Gap19 and carbenoxolone alleviate liver fibrosis in mice [Internet]. International Journal of Molecular Sciences. 2018 ; 19( 3): 1-17.[citado 2024 nov. 03 ] Available from: https://doi.org/10.3390/ijms19030817
Artigo de periodico
Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis (2017)
Tiburcio, Taynã Cristina; Willebrords, Joost; Silva, Tereza Cristina da; Pereira, Isabel Veloso Alves; Nogueira, Marina Sayuri; Yanguas, Sara Crespo; Maes, Michaël; Silva, Elisangela dos Anjos; Dagli, Maria Lúcia Zaidan ; Castro, Inar Alves de ; Oliveira, Cláudia Pinto Marques Souza de; Vinken, Mathieu; Cogliati, Bruno
Source: Clinical and Experimental Pharmacology and Physiology. Unidades: FMVZ, FCF, FM
Subjects: CONEXINAS, HEPATOPATIAS, ESTRESSE OXIDATIVO
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TIBURCIO, Taynã Cristina et al. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, v. 44, n. 2, p. 197-206, 2017Tradução . . Disponível em: https://doi.org/10.1111/1440-1681.12701. Acesso em: 03 nov. 2024.
APA
Tiburcio, T. C., Willebrords, J., Silva, T. C. da, Pereira, I. V. A., Nogueira, M. S., Yanguas, S. C., et al. (2017). Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis. Clinical and Experimental Pharmacology and Physiology, 44( 2), 197-206. doi:10.1111/1440-1681.12701
NLM
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/1440-1681.12701
Vancouver
Tiburcio TC, Willebrords J, Silva TC da, Pereira IVA, Nogueira MS, Yanguas SC, Maes M, Silva E dos A, Dagli MLZ, Castro IA de, Oliveira CPMS de, Vinken M, Cogliati B. Connexin32 deficiency is associated with liver injury, inflammation and oxidative stress in experimental non-alcoholic steatohepatitis [Internet]. Clinical and Experimental Pharmacology and Physiology. 2017 ; 44( 2): 197-206.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1111/1440-1681.12701
Artigo de periodico
Inhibition of connexin hemichannels alleviates non-alcoholic steatohepatitis in mice (2017)
Willebrords, Joost; Cogliati, Bruno ; Pereira, Isabel Veloso Alves; Silva, Tereza Cristina da; Yanguas, Sara Crespo; Maes, Michaël; Govoni, Verônica Mollica; Lima, Andressa; Felisbino, Daniele Aparecida; Decrock, Elke; Nogueira, Marina Sayuri; Castro, Inar Alves de ; Leclercq, Isabelle; Leybaert, Luc; Rodrigues, Robim Marcelino; Vinken, Mathieu
Source: Scientific Reports. Unidades: FMVZ, FCF
Subjects: CONEXINAS, HEPATITE, FÁRMACOS (SISTEMA DIGESTÓRIO)
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ABNT
WILLEBRORDS, Joost et al. Inhibition of connexin hemichannels alleviates non-alcoholic steatohepatitis in mice. Scientific Reports, v. 7, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-08583-w. Acesso em: 03 nov. 2024.
APA
Willebrords, J., Cogliati, B., Pereira, I. V. A., Silva, T. C. da, Yanguas, S. C., Maes, M., et al. (2017). Inhibition of connexin hemichannels alleviates non-alcoholic steatohepatitis in mice. Scientific Reports, 7, 1-11. doi:10.1038/s41598-017-08583-w
NLM
Willebrords J, Cogliati B, Pereira IVA, Silva TC da, Yanguas SC, Maes M, Govoni VM, Lima A, Felisbino DA, Decrock E, Nogueira MS, Castro IA de, Leclercq I, Leybaert L, Rodrigues RM, Vinken M. Inhibition of connexin hemichannels alleviates non-alcoholic steatohepatitis in mice [Internet]. Scientific Reports. 2017 ; 7 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-08583-w
Vancouver
Willebrords J, Cogliati B, Pereira IVA, Silva TC da, Yanguas SC, Maes M, Govoni VM, Lima A, Felisbino DA, Decrock E, Nogueira MS, Castro IA de, Leclercq I, Leybaert L, Rodrigues RM, Vinken M. Inhibition of connexin hemichannels alleviates non-alcoholic steatohepatitis in mice [Internet]. Scientific Reports. 2017 ; 7 1-11.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1038/s41598-017-08583-w
Artigo de periodico
Connexin32 deficiency exacerbates carbon tetrachloride-induced hepatocellular injury and liver fibrosis in mice (2016)
Cogliati, Bruno ; Yanguas, Sara Crespo; Silva, Tereza Cristina da; Aloia, Thiago Pinheiro Arrais; Nogueira, Marina Sayuri; Lima, Mirela Aline Real de; Chaible, Lucas Martins; Sanches, Daniel Soares; Willebrords, Joost; Maes, Michaël; Pereira, Isabel Veloso Alves; Castro, Inar Alves de ; Vinken, Mathieu; Dagli, Maria Lúcia Zaidan
Source: Toxicology Mechanisms and Methods. Unidades: FMVZ, FCF
Subjects: APOPTOSE, CONEXINAS, ESTRESSE OXIDATIVO
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ABNT
COGLIATI, Bruno et al. Connexin32 deficiency exacerbates carbon tetrachloride-induced hepatocellular injury and liver fibrosis in mice. Toxicology Mechanisms and Methods, v. 26, n. 5, p. 362-370, 2016Tradução . . Disponível em: https://doi.org/10.1080/15376516.2016.1190991. Acesso em: 03 nov. 2024.
APA
Cogliati, B., Yanguas, S. C., Silva, T. C. da, Aloia, T. P. A., Nogueira, M. S., Lima, M. A. R. de, et al. (2016). Connexin32 deficiency exacerbates carbon tetrachloride-induced hepatocellular injury and liver fibrosis in mice. Toxicology Mechanisms and Methods, 26( 5), 362-370. doi:10.1080/15376516.2016.1190991
NLM
Cogliati B, Yanguas SC, Silva TC da, Aloia TPA, Nogueira MS, Lima MAR de, Chaible LM, Sanches DS, Willebrords J, Maes M, Pereira IVA, Castro IA de, Vinken M, Dagli MLZ. Connexin32 deficiency exacerbates carbon tetrachloride-induced hepatocellular injury and liver fibrosis in mice [Internet]. Toxicology Mechanisms and Methods. 2016 ; 26( 5): 362-370.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1080/15376516.2016.1190991
Vancouver
Cogliati B, Yanguas SC, Silva TC da, Aloia TPA, Nogueira MS, Lima MAR de, Chaible LM, Sanches DS, Willebrords J, Maes M, Pereira IVA, Castro IA de, Vinken M, Dagli MLZ. Connexin32 deficiency exacerbates carbon tetrachloride-induced hepatocellular injury and liver fibrosis in mice [Internet]. Toxicology Mechanisms and Methods. 2016 ; 26( 5): 362-370.[citado 2024 nov. 03 ] Available from: https://doi.org/10.1080/15376516.2016.1190991

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