Source: International Journal of Molecular Sciences. Unidades: FMRP, FM
Subjects: ENDOMETRIOSE, ENDOMÉTRIO, ESTRESSE OXIDATIVO, ESPÉCIES REATIVAS DE OXIGÊNIO, CÉLULAS ESTROMAIS
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MALVEZZI, Helena et al. Higher oxidative stress in endometriotic lesions upregulates senescence-associated p16ink4a and β-galactosidase in stromal cells. International Journal of Molecular Sciences, v. 24, n. 2, p. 1-18, 2023Tradução . . Disponível em: https://doi.org/10.3390/ijms24020914. Acesso em: 10 fev. 2026.APA
Malvezzi, H., Cestari, B. A., Meola, J., & Podgaec, S. (2023). Higher oxidative stress in endometriotic lesions upregulates senescence-associated p16ink4a and β-galactosidase in stromal cells. International Journal of Molecular Sciences, 24( 2), 1-18. doi:10.3390/ijms24020914NLM
Malvezzi H, Cestari BA, Meola J, Podgaec S. Higher oxidative stress in endometriotic lesions upregulates senescence-associated p16ink4a and β-galactosidase in stromal cells [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 2): 1-18.[citado 2026 fev. 10 ] Available from: https://doi.org/10.3390/ijms24020914Vancouver
Malvezzi H, Cestari BA, Meola J, Podgaec S. Higher oxidative stress in endometriotic lesions upregulates senescence-associated p16ink4a and β-galactosidase in stromal cells [Internet]. International Journal of Molecular Sciences. 2023 ; 24( 2): 1-18.[citado 2026 fev. 10 ] Available from: https://doi.org/10.3390/ijms24020914
