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Artigo de periodico
Renovascular remodeling and renal injury after extended angiotensin II infusion (2016)
Casare, Fernando Augusto Malavazzi; Thieme, Karina ; Costa-Pessoa, Juliana Martins; Rossoni, Luciana Venturini ; Couto, Gisele Kruger; Fernandes, Fernanda Barrinha; Casarini, Dulce Elena; Oliveira-Souza, Maria
Source: American Journal of Physiology. Renal Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
CASARE, Fernando Augusto Malavazzi et al. Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, v. 310, n. 11, p. F1295-F1307, 2016Tradução . . Disponível em: https://doi.org/10.1152/ajprenal.00471.2015. Acesso em: 07 out. 2024.
APA
Casare, F. A. M., Thieme, K., Costa-Pessoa, J. M., Rossoni, L. V., Couto, G. K., Fernandes, F. B., et al. (2016). Renovascular remodeling and renal injury after extended angiotensin II infusion. American Journal of Physiology. Renal Physiology, 310( 11), F1295-F1307. doi:10.1152/ajprenal.00471.2015
NLM
Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 out. 07 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
Vancouver
Casare FAM, Thieme K, Costa-Pessoa JM, Rossoni LV, Couto GK, Fernandes FB, Casarini DE, Oliveira-Souza M. Renovascular remodeling and renal injury after extended angiotensin II infusion [Internet]. American Journal of Physiology. Renal Physiology. 2016 ; 310( 11): F1295-F1307.[citado 2024 out. 07 ] Available from: https://doi.org/10.1152/ajprenal.00471.2015
Artigo de periodico
Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues (2016)
Victório, Jamaira A.; Fontes, Milene T.; Rossoni, Luciana Venturini ; Davel, Ana P.
Source: Frontiers in Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
VICTÓRIO, Jamaira A. et al. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, v. 7, p. 1-10, 2016Tradução . . Disponível em: https://doi.org/10.3389/fphys.2016.00295. Acesso em: 07 out. 2024.
APA
Victório, J. A., Fontes, M. T., Rossoni, L. V., & Davel, A. P. (2016). Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues. Frontiers in Physiology, 7, 1-10. doi:10.3389/fphys.2016.00295
NLM
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 out. 07 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Vancouver
Victório JA, Fontes MT, Rossoni LV, Davel AP. Different anti-contractile function and nitric oxide production of thoracic and abdominal perivascular adipose tissues [Internet]. Frontiers in Physiology. 2016 ; 7 1-10.[citado 2024 out. 07 ] Available from: https://doi.org/10.3389/fphys.2016.00295
Artigo de periodico
Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension (2015)
Martínez-Martínez, Ernesto; Calvier, Laurent; Fernández-Celis, Amaya; Rousseau, Elodie; Jurado-López, Raquel; Rossoni, Luciana Venturini ; Jaisser, Frederic; Zannad, Faiez; Rossignol, Patrick; Cachofeiro, Victoria; López-Andrés, Natalia
Source: Hypertension. Unidade: ICB
Subjects: FISIOLOGIA, FISIOLOGIA CARDIOVASCULAR, HIPERTENSÃO, HIPERTENSÃO, INIBIDORES QUÍMICOS
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ABNT
MARTÍNEZ-MARTÍNEZ, Ernesto et al. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, v. 66, n. 4, p. 767-775, 2015Tradução . . Disponível em: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876. Acesso em: 07 out. 2024.
APA
Martínez-Martínez, E., Calvier, L., Fernández-Celis, A., Rousseau, E., Jurado-López, R., Rossoni, L. V., et al. (2015). Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension. Hypertension, 66( 4), 767-775. doi:10.1161/HYPERTENSIONAHA.115.05876
NLM
Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 out. 07 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
Vancouver
Martínez-Martínez E, Calvier L, Fernández-Celis A, Rousseau E, Jurado-López R, Rossoni LV, Jaisser F, Zannad F, Rossignol P, Cachofeiro V, López-Andrés N. Galectin-3 blockade inhibits cardiac inflammation and fibrosis in experimental hyperaldosteronism and hypertension [Internet]. Hypertension. 2015 ; 66( 4): 767-775.[citado 2024 out. 07 ] Available from: https://doi.org/10.1161/HYPERTENSIONAHA.115.05876
Artigo de periodico
Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels (2015)
Peixoto-Neves, Dieniffer; Wang, Qian; Leal-Cardoso, José Henrique; Rossoni, Luciana Venturini ; Jaggar, Jonathan H.
Source: British Journal of Pharmacology. Unidade: ICB
Subjects: FISIOLOGIA, CÉLULAS ENDOTELIAIS, ÓLEOS ESSENCIAIS
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ABNT
PEIXOTO-NEVES, Dieniffer et al. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels. British Journal of Pharmacology, v. 172, n. 14, p. 3484-3494, 2015Tradução . . Disponível em: https://doi.org/10.1111/bph.13156. Acesso em: 07 out. 2024.
APA
Peixoto-Neves, D., Wang, Q., Leal-Cardoso, J. H., Rossoni, L. V., & Jaggar, J. H. (2015). Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels. British Journal of Pharmacology, 172( 14), 3484-3494. doi:10.1111/bph.13156
NLM
Peixoto-Neves D, Wang Q, Leal-Cardoso JH, Rossoni LV, Jaggar JH. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels [Internet]. British Journal of Pharmacology. 2015 ; 172( 14): 3484-3494.[citado 2024 out. 07 ] Available from: https://doi.org/10.1111/bph.13156
Vancouver
Peixoto-Neves D, Wang Q, Leal-Cardoso JH, Rossoni LV, Jaggar JH. Eugenol dilates mesenteric arteries and reduces systemic BP by activating endothelial cell TRPV4 channels [Internet]. British Journal of Pharmacology. 2015 ; 172( 14): 3484-3494.[citado 2024 out. 07 ] Available from: https://doi.org/10.1111/bph.13156
Artigo de periodico
Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause (2015)
Braga, Viviane A. V. N.; Couto, Gisele Kruger; Lazzarin, Mariana C.; Rossoni, Luciana Venturini ; Medeiros, Alessandra
Source: PLOS ONE. Unidade: ICB
Subjects: FISIOLOGIA, EXERCICIO FÍSICO, ÓXIDO NITRICO, MENOPAUSA, ENDOTÉLIO
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ABNT
BRAGA, Viviane A. V. N. et al. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, v. 10, n. 4, p. 1-13, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0125388. Acesso em: 07 out. 2024.
APA
Braga, V. A. V. N., Couto, G. K., Lazzarin, M. C., Rossoni, L. V., & Medeiros, A. (2015). Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause. PLOS ONE, 10( 4), 1-13. doi:10.1371/journal.pone.0125388
NLM
Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 out. 07 ] Available from: https://doi.org/10.1371/journal.pone.0125388
Vancouver
Braga VAVN, Couto GK, Lazzarin MC, Rossoni LV, Medeiros A. Aerobic exercise training prevents the onset of endothelial dysfunction via increased nitric oxide bioavailability and reduced reactive oxygen species in an experimental model of menopause [Internet]. PLOS ONE. 2015 ; 10( 4): 1-13.[citado 2024 out. 07 ] Available from: https://doi.org/10.1371/journal.pone.0125388
Artigo de periodico
Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway (2015)
Davel, Ana P.; Victorio, Jamaira A.; Delbin, Maria Andréia; Fukuda, Livia Emy; Rossoni, Luciana Venturini
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, ENDOTÉLIO, RATOS, ARTÉRIA PULMONAR, ADRENÉRGICOS
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ABNT
DAVEL, Ana P. et al. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, v. 125, p. 49-56, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2015.01.018. Acesso em: 07 out. 2024.
APA
Davel, A. P., Victorio, J. A., Delbin, M. A., Fukuda, L. E., & Rossoni, L. V. (2015). Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway. Life Sciences, 125, 49-56. doi:10.1016/j.lfs.2015.01.018
NLM
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Vancouver
Davel AP, Victorio JA, Delbin MA, Fukuda LE, Rossoni LV. Enhanced endothelium-dependent relaxation of rat pulmonary artery following β-adrenergic overstimulation: involvement of the NO/cGMP/VASP pathway [Internet]. Life Sciences. 2015 ; 125 49-56.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.lfs.2015.01.018
Artigo de periodico
Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway (2014)
Wenceslau, Camilla F.; Rossoni, Luciana Venturini
Source: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
WENCESLAU, Camilla F. e ROSSONI, Luciana Venturini. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, v. 32, n. 3, p. 542-555, 2014Tradução . . Disponível em: https://doi.org/10.1097/HJH.0000000000000059. Acesso em: 07 out. 2024.
APA
Wenceslau, C. F., & Rossoni, L. V. (2014). Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway. Journal of Hypertension, 32( 3), 542-555. doi:10.1097/HJH.0000000000000059
NLM
Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
Vancouver
Wenceslau CF, Rossoni LV. Rostafuroxin ameliorates endothelial dysfunction and oxidative stress in resistance arteries from deoxycorticosterone acetate-salt hypertensive rats: the role of Na+K+-ATPase/cSRC pathway [Internet]. Journal of Hypertension. 2014 ; 32( 3): 542-555.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/HJH.0000000000000059
Artigo de periodico
Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats (2014)
Fontes, M. T.; Silva, T. L. B. T.; Mota, M. M.; Barreto, A. S.; Rossoni, Luciana Venturini ; Santos, M. R. V.
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FONTES, M. T. et al. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, v. 94, n. 1, p. 24-29, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.017. Acesso em: 07 out. 2024.
APA
Fontes, M. T., Silva, T. L. B. T., Mota, M. M., Barreto, A. S., Rossoni, L. V., & Santos, M. R. V. (2014). Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, 94( 1), 24-29. doi:10.1016/j.lfs.2013.11.017
NLM
Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
Vancouver
Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
Artigo de periodico
Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus (2014)
Kinoshita, Paula Fernanda; Yshii, Lidia Mitiko; Vasconcelos, Andrea Rodrigues; Orellana, Ana Maria Marques; Lima, Larissa de Sá; Davel, Ana Paula Couto; Rossoni, Luciana Venturini ; Kawamoto, Elisa Mitiko ; Scavone, Cristoforo
Source: Journal of Neuroinflammation. Unidade: ICB
Subjects: FARMACOLOGIA, HIPOCAMPU DE ANIMAL, RATOS WISTAR, INFLAMAÇÃO
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ABNT
KINOSHITA, Paula Fernanda et al. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, v. 11, n. 1, p. 1-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/s12974-014-0218-z. Acesso em: 07 out. 2024.
APA
Kinoshita, P. F., Yshii, L. M., Vasconcelos, A. R., Orellana, A. M. M., Lima, L. de S., Davel, A. P. C., et al. (2014). Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus. Journal of Neuroinflammation, 11( 1), 1-12. doi:10.1186/s12974-014-0218-z
NLM
Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 out. 07 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
Vancouver
Kinoshita PF, Yshii LM, Vasconcelos AR, Orellana AMM, Lima L de S, Davel APC, Rossoni LV, Kawamoto EM, Scavone C. Signaling function of Na,K-ATPase induced by ouabain against LPS as an inflammation model in hippocampus [Internet]. Journal of Neuroinflammation. 2014 ; 11( 1): 1-12.[citado 2024 out. 07 ] Available from: https://doi.org/10.1186/s12974-014-0218-z
Artigo de periodico
Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats (2014)
Davel, Ana Paula; Couto, Gisele Kruger; Wenceslau, Camila Ferreira; Peres, Emilia Cristina; Xavier, Fabiano Elias; Rossoni, Luciana Venturini
Source: Hormone Molecular Biology and Clinical Investigation. Unidades: ICB, HU
Subjects: FISIOLOGIA, BIOFÍSICA, ENDOTÉLIO VASCULAR, RATOS, AORTA DE ANIMAL
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ABNT
DAVEL, Ana Paula et al. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, v. 18, n. 2, p. 113-122, 2014Tradução . . Disponível em: https://doi.org/10.1515/hmbci-2013-0058. Acesso em: 07 out. 2024.
APA
Davel, A. P., Couto, G. K., Wenceslau, C. F., Peres, E. C., Xavier, F. E., & Rossoni, L. V. (2014). Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats. Hormone Molecular Biology and Clinical Investigation, 18( 2), 113-122. doi:10.1515/hmbci-2013-0058
NLM
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 out. 07 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Vancouver
Davel AP, Couto GK, Wenceslau CF, Peres EC, Xavier FE, Rossoni LV. Enhanced Na⁺, K⁺-ATPase activity and endothelial modulation decrease phenylephrine-induced contraction in aorta from ouabain-treated normotensive and hypertensive rats [Internet]. Hormone Molecular Biology and Clinical Investigation. 2014 ; 18( 2): 113-122.[citado 2024 out. 07 ] Available from: https://doi.org/10.1515/hmbci-2013-0058
Artigo de periodico
Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells (2013)
Lima, Larissa de Sá; Kawamoto, Elisa Mitiko ; Munhoz, Carolina Demarchi ; Kinoshita, Paula Fernanda; Orellana, Ana Maria Marques; Curi, Rui; Rossoni, Luciana Venturini ; Avellar, M. C. W.; Scavone, Cristoforo
Source: Neuropharmacology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 07 out. 2024.
APA
Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
NLM
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Vancouver
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 out. 07 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Artigo de periodico
Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats (2013)
Blanco-Rivero, Javier; Roque, Fernanda R.; Sastre, Esther; Caracuel, Laura; Couto, Gisele Kruger; Avendaño, María Soledad; Paula, Suliana Mesquita; Rossoni, Luciana Venturini ; Salaices, Mercedes; Balfagón, Gloria
Source: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BLANCO-RIVERO, Javier et al. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, v. 31, n. 5, p. 916-926, 2013Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32835f749c. Acesso em: 07 out. 2024.
APA
Blanco-Rivero, J., Roque, F. R., Sastre, E., Caracuel, L., Couto, G. K., Avendaño, M. S., et al. (2013). Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, 31( 5), 916-926. doi:10.1097/HJH.0b013e32835f749c
NLM
Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
Vancouver
Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
Artigo de periodico
Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide (2013)
Breithaupt-Faloppa, A. C.; Fantozzi, Evelyn Thaís; Assis-Ramos, M. M.; Vitoretti, Luana Beatriz; Couto, Gisele Kruger; Rossoni, Luciana Venturini ; Oliveira-Filho, Ricardo Martins; Vargaftig, Bernardo Boris; Lima, Wothan Tavares de
Source: Shock. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
BREITHAUPT-FALOPPA, A. C. et al. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide. Shock, v. 40, n. 3, p. 203-209, 2013Tradução . . Disponível em: https://doi.org/10.1097/SHK.0b013e3182a01e24. Acesso em: 07 out. 2024.
APA
Breithaupt-Faloppa, A. C., Fantozzi, E. T., Assis-Ramos, M. M., Vitoretti, L. B., Couto, G. K., Rossoni, L. V., et al. (2013). Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide. Shock, 40( 3), 203-209. doi:10.1097/SHK.0b013e3182a01e24
NLM
Breithaupt-Faloppa AC, Fantozzi ET, Assis-Ramos MM, Vitoretti LB, Couto GK, Rossoni LV, Oliveira-Filho RM, Vargaftig BB, Lima WT de. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide [Internet]. Shock. 2013 ; 40( 3): 203-209.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/SHK.0b013e3182a01e24
Vancouver
Breithaupt-Faloppa AC, Fantozzi ET, Assis-Ramos MM, Vitoretti LB, Couto GK, Rossoni LV, Oliveira-Filho RM, Vargaftig BB, Lima WT de. Protective effect of estradiol on acute lung inflammation Induced by an intestinal ischemic insult is dependent on nitric oxide [Internet]. Shock. 2013 ; 40( 3): 203-209.[citado 2024 out. 07 ] Available from: https://doi.org/10.1097/SHK.0b013e3182a01e24
Artigo de periodico
Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay (2012)
Bocalini, Danilo Sales; Dos-Santos, Leonardo; Antonio, Ednei Luiz; Santos, Alexandra Alberta dos; Davel, Ana Paula; Rossoni, Luciana Venturini ; Vassallo, Dalton Valentim; Tucci, Paulo José Ferreira
Source: Arquivos Brasileiros de Cardiologia. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BOCALINI, Danilo Sales et al. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay. Arquivos Brasileiros de Cardiologia, v. 98, n. 3, p. 243-251, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0066-782X2012005000016. Acesso em: 07 out. 2024.
APA
Bocalini, D. S., Dos-Santos, L., Antonio, E. L., Santos, A. A. dos, Davel, A. P., Rossoni, L. V., et al. (2012). Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay. Arquivos Brasileiros de Cardiologia, 98( 3), 243-251. doi:10.1590/S0066-782X2012005000016
NLM
Bocalini DS, Dos-Santos L, Antonio EL, Santos AA dos, Davel AP, Rossoni LV, Vassallo DV, Tucci PJF. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay [Internet]. Arquivos Brasileiros de Cardiologia. 2012 ; 98( 3): 243-251.[citado 2024 out. 07 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
Vancouver
Bocalini DS, Dos-Santos L, Antonio EL, Santos AA dos, Davel AP, Rossoni LV, Vassallo DV, Tucci PJF. Myocardial remodeling after large infarcts in rat converts post rest-potentiation in force decay [Internet]. Arquivos Brasileiros de Cardiologia. 2012 ; 98( 3): 243-251.[citado 2024 out. 07 ] Available from: https://doi.org/10.1590/S0066-782X2012005000016
Artigo de periodico
Increased vascular contractility and oxidative stress in β₂-adrenoceptor knockout mice: the role of NADPH oxidase (2012)
Davel, Ana Paula Couto; Ceravolo, Graziela Scalianti; Wenceslau, Camilla Ferreira; Carvalho, Maria Helena Catelli de; Brum, Patricia Chakur; Rossoni, Luciana Venturini
Source: Journal of Vascular Research. Unidades: ICB, EEFE
Subjects: FISIOLOGIA, FARMACOLOGIA, FISIOLOGIA RESPIRATÓRIA E CIRCULATÓRIA, ADRENORECEPTORES
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAVEL, Ana Paula Couto et al. Increased vascular contractility and oxidative stress in β₂-adrenoceptor knockout mice: the role of NADPH oxidase. Journal of Vascular Research, v. 49, n. 4, p. 342-352, 2012Tradução . . Disponível em: https://doi.org/10.1159/000337486. Acesso em: 07 out. 2024.
APA
Davel, A. P. C., Ceravolo, G. S., Wenceslau, C. F., Carvalho, M. H. C. de, Brum, P. C., & Rossoni, L. V. (2012). Increased vascular contractility and oxidative stress in β₂-adrenoceptor knockout mice: the role of NADPH oxidase. Journal of Vascular Research, 49( 4), 342-352. doi:10.1159/000337486
NLM
Davel APC, Ceravolo GS, Wenceslau CF, Carvalho MHC de, Brum PC, Rossoni LV. Increased vascular contractility and oxidative stress in β₂-adrenoceptor knockout mice: the role of NADPH oxidase [Internet]. Journal of Vascular Research. 2012 ; 49( 4): 342-352.[citado 2024 out. 07 ] Available from: https://doi.org/10.1159/000337486
Vancouver
Davel APC, Ceravolo GS, Wenceslau CF, Carvalho MHC de, Brum PC, Rossoni LV. Increased vascular contractility and oxidative stress in β₂-adrenoceptor knockout mice: the role of NADPH oxidase [Internet]. Journal of Vascular Research. 2012 ; 49( 4): 342-352.[citado 2024 out. 07 ] Available from: https://doi.org/10.1159/000337486

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