ReP USP - Resultado da busca

Artigo de periodico
The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer (2017)
Smuczeka, Basilio; Santos, Emerson de S.; Siqueira, Adriane S.; Pinheiro, Joao J. V.; Freitas, Vanessa Morais ; Jaeger, Ruy Gastaldoni
Fonte: Experimental Cell Research. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SMUCZEKA, Basilio et al. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, v. 358, n. 2, p. 323-334, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.yexcr.2017.07.005. Acesso em: 25 jul. 2024.
APA
Smuczeka, B., Santos, E. de S., Siqueira, A. S., Pinheiro, J. J. V., Freitas, V. M., & Jaeger, R. G. (2017). The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer. Experimental Cell Research, 358( 2), 323-334. doi:10.1016/j.yexcr.2017.07.005
NLM
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Vancouver
Smuczeka B, Santos E de S, Siqueira AS, Pinheiro JJV, Freitas VM, Jaeger RG. The laminin-derived peptide C16 regulates GPNMB expression and function in breast cancer [Internet]. Experimental Cell Research. 2017 ; 358( 2): 323-334.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1016/j.yexcr.2017.07.005
Artigo de periodico
A total transcriptome profiling method for plasma-derived extracellular vesicles: applications for liquid biopsies (2017)
Amorim, Maria G.; Valieris, Renan; Drummond, Rodrigo D.; Pizzi, Melissa P.; Freitas, Vanessa Morais ; Sinigaglia-Coimbra, Rita; Calin, George A.; Pasqualini, Renata; Arap, Wadih; Silva, Israel T.; Dias-Neto, Emmanuel; Nunes, Diana N.
Fonte: Scientific Reports. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
AMORIM, Maria G. et al. A total transcriptome profiling method for plasma-derived extracellular vesicles: applications for liquid biopsies. Scientific Reports, v. 7, n. 1, p. 1-11, 2017Tradução . . Disponível em: https://doi.org/10.1038/s41598-017-14264-5. Acesso em: 25 jul. 2024.
APA
Amorim, M. G., Valieris, R., Drummond, R. D., Pizzi, M. P., Freitas, V. M., Sinigaglia-Coimbra, R., et al. (2017). A total transcriptome profiling method for plasma-derived extracellular vesicles: applications for liquid biopsies. Scientific Reports, 7( 1), 1-11. doi:10.1038/s41598-017-14264-5
NLM
Amorim MG, Valieris R, Drummond RD, Pizzi MP, Freitas VM, Sinigaglia-Coimbra R, Calin GA, Pasqualini R, Arap W, Silva IT, Dias-Neto E, Nunes DN. A total transcriptome profiling method for plasma-derived extracellular vesicles: applications for liquid biopsies [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1038/s41598-017-14264-5
Vancouver
Amorim MG, Valieris R, Drummond RD, Pizzi MP, Freitas VM, Sinigaglia-Coimbra R, Calin GA, Pasqualini R, Arap W, Silva IT, Dias-Neto E, Nunes DN. A total transcriptome profiling method for plasma-derived extracellular vesicles: applications for liquid biopsies [Internet]. Scientific Reports. 2017 ; 7( 1): 1-11.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1038/s41598-017-14264-5
Artigo de periodico
AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility (2016)
Silva, Thaiomara Alves; Smuczek, Basílio; Valadão, Iuri Cordeiro; Dzik, Luciana Machado; Iglesia, Rebeca Piatniczka; Cruz, Mário Costa; Pereira, André Zelanis Palitot; Siqueria, Adriane Sousa de; Serrano, Solange Maria de Toledo; Goldberg, Gary S; Jaeger, Ruy Gastaldoni ; Freitas, Vanessa Morais
Fonte: OncoTarget. Unidade: ICB
Assuntos: CARCINOMA, TUMOR CARCINOIDE, FIBROBLASTOS, ONCOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Thaiomara Alves et al. AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility. OncoTarget, v. 7, n. 31, p. 49998-50016, 2016Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.10307. Acesso em: 25 jul. 2024.
APA
Silva, T. A., Smuczek, B., Valadão, I. C., Dzik, L. M., Iglesia, R. P., Cruz, M. C., et al. (2016). AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility. OncoTarget, 7( 31), 49998-50016. doi:10.18632/oncotarget.10307
NLM
Silva TA, Smuczek B, Valadão IC, Dzik LM, Iglesia RP, Cruz MC, Pereira AZP, Siqueria AS de, Serrano SM de T, Goldberg GS, Jaeger RG, Freitas VM. AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility [Internet]. OncoTarget. 2016 ; 7( 31): 49998-50016.[citado 2024 jul. 25 ] Available from: https://doi.org/10.18632/oncotarget.10307
Vancouver
Silva TA, Smuczek B, Valadão IC, Dzik LM, Iglesia RP, Cruz MC, Pereira AZP, Siqueria AS de, Serrano SM de T, Goldberg GS, Jaeger RG, Freitas VM. AHNAK enables mammary carcinoma cells to produce extracellular vesicles that increase neighboring fibroblast cell motility [Internet]. OncoTarget. 2016 ; 7( 31): 49998-50016.[citado 2024 jul. 25 ] Available from: https://doi.org/10.18632/oncotarget.10307
Artigo de periodico
Progesterone acts via the progesterone receptor to induce adamts proteases in ovarian cancer cells (2016)
Lima, Maíra A.; Silva, Suély V.; Freitas, Vanessa Morais
Fonte: Journal of Ovarian Research. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LIMA, Maíra A. e SILVA, Suély V. e FREITAS, Vanessa Morais. Progesterone acts via the progesterone receptor to induce adamts proteases in ovarian cancer cells. Journal of Ovarian Research, v. 9, n. 9, p. 1-11, 2016Tradução . . Disponível em: https://doi.org/10.1186/s13048-016-0219-x. Acesso em: 25 jul. 2024.
APA
Lima, M. A., Silva, S. V., & Freitas, V. M. (2016). Progesterone acts via the progesterone receptor to induce adamts proteases in ovarian cancer cells. Journal of Ovarian Research, 9( 9), 1-11. doi:10.1186/s13048-016-0219-x
NLM
Lima MA, Silva SV, Freitas VM. Progesterone acts via the progesterone receptor to induce adamts proteases in ovarian cancer cells [Internet]. Journal of Ovarian Research. 2016 ; 9( 9): 1-11.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1186/s13048-016-0219-x
Vancouver
Lima MA, Silva SV, Freitas VM. Progesterone acts via the progesterone receptor to induce adamts proteases in ovarian cancer cells [Internet]. Journal of Ovarian Research. 2016 ; 9( 9): 1-11.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1186/s13048-016-0219-x
Artigo de periodico
Laminin-111 peptide C16 regulates invadopodia activity of malignant cells through β1 integrin, Src and ERK 1/2 (2016)
Siqueira, Adriane Sousa de; Pinto, Monique Rodrigues Pereira; Cruz, Mário Costa; Smuczek, Basilio; Cruz, Karen Steponavicius Piedade; Barbuto, José Alexandre Marzagão ; Hoshino, Daisuke; Weaver, Alissa M; Freitas, Vanessa Morais ; Jaeger, Ruy Gastaldoni
Fonte: OncoTarget. Unidade: ICB
Assuntos: PEPTÍDEOS, INTEGRINAS, CARCINOMA DE CÉLULAS ESCAMOSAS, FIBROSSARCOMA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SIQUEIRA, Adriane Sousa de et al. Laminin-111 peptide C16 regulates invadopodia activity of malignant cells through β1 integrin, Src and ERK 1/2. OncoTarget, v. 7, n. 30, p. 47904-4717, 2016Tradução . . Disponível em: https://doi.org/10.18632/oncotarget.10062. Acesso em: 25 jul. 2024.
APA
Siqueira, A. S. de, Pinto, M. R. P., Cruz, M. C., Smuczek, B., Cruz, K. S. P., Barbuto, J. A. M., et al. (2016). Laminin-111 peptide C16 regulates invadopodia activity of malignant cells through β1 integrin, Src and ERK 1/2. OncoTarget, 7( 30), 47904-4717. doi:10.18632/oncotarget.10062
NLM
Siqueira AS de, Pinto MRP, Cruz MC, Smuczek B, Cruz KSP, Barbuto JAM, Hoshino D, Weaver AM, Freitas VM, Jaeger RG. Laminin-111 peptide C16 regulates invadopodia activity of malignant cells through β1 integrin, Src and ERK 1/2 [Internet]. OncoTarget. 2016 ; 7( 30): 47904-4717.[citado 2024 jul. 25 ] Available from: https://doi.org/10.18632/oncotarget.10062
Vancouver
Siqueira AS de, Pinto MRP, Cruz MC, Smuczek B, Cruz KSP, Barbuto JAM, Hoshino D, Weaver AM, Freitas VM, Jaeger RG. Laminin-111 peptide C16 regulates invadopodia activity of malignant cells through β1 integrin, Src and ERK 1/2 [Internet]. OncoTarget. 2016 ; 7( 30): 47904-4717.[citado 2024 jul. 25 ] Available from: https://doi.org/10.18632/oncotarget.10062
Artigo de periodico
ADAMTS-1 is found in the nuclei of normal and tumoral breast cells (2016)
Silva, Suély V.; Lima, Maíra A.; Cella, Nathalie ; Jaeger, Ruy Gastaldoni ; Freitas, Vanessa Morais
Fonte: PLOS ONE. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Suély V. et al. ADAMTS-1 is found in the nuclei of normal and tumoral breast cells. PLOS ONE, v. 11, n. 10, p. 1-17, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0165061. Acesso em: 25 jul. 2024.
APA
Silva, S. V., Lima, M. A., Cella, N., Jaeger, R. G., & Freitas, V. M. (2016). ADAMTS-1 is found in the nuclei of normal and tumoral breast cells. PLOS ONE, 11( 10), 1-17. doi:10.1371/journal.pone.0165061
NLM
Silva SV, Lima MA, Cella N, Jaeger RG, Freitas VM. ADAMTS-1 is found in the nuclei of normal and tumoral breast cells [Internet]. PLOS ONE. 2016 ; 11( 10): 1-17.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0165061
Vancouver
Silva SV, Lima MA, Cella N, Jaeger RG, Freitas VM. ADAMTS-1 is found in the nuclei of normal and tumoral breast cells [Internet]. PLOS ONE. 2016 ; 11( 10): 1-17.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0165061
Artigo de periodico
Obesity impairs lactation performance in mice by inducing prolactin resistance (2016)
Buonfiglio, Daniella C.; Ramos-Lobo, Angela M.; Freitas, Vanessa Morais ; Zampieri, Thais T.; Nagaishi, Vanessa S.; Magalhães, Magna; Cipolla-Neto, José; Cella, Nathalie ; Donato Junior, José
Fonte: Scientific Reports. Unidade: ICB
Assuntos: FISIOLOGIA, HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BUONFIGLIO, Daniella C. et al. Obesity impairs lactation performance in mice by inducing prolactin resistance. Scientific Reports, v. 6, n. 22421, p. 1-13, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep22421. Acesso em: 25 jul. 2024.
APA
Buonfiglio, D. C., Ramos-Lobo, A. M., Freitas, V. M., Zampieri, T. T., Nagaishi, V. S., Magalhães, M., et al. (2016). Obesity impairs lactation performance in mice by inducing prolactin resistance. Scientific Reports, 6( 22421), 1-13. doi:10.1038/srep22421
NLM
Buonfiglio DC, Ramos-Lobo AM, Freitas VM, Zampieri TT, Nagaishi VS, Magalhães M, Cipolla-Neto J, Cella N, Donato Junior J. Obesity impairs lactation performance in mice by inducing prolactin resistance [Internet]. Scientific Reports. 2016 ; 6( 22421): 1-13.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1038/srep22421
Vancouver
Buonfiglio DC, Ramos-Lobo AM, Freitas VM, Zampieri TT, Nagaishi VS, Magalhães M, Cipolla-Neto J, Cella N, Donato Junior J. Obesity impairs lactation performance in mice by inducing prolactin resistance [Internet]. Scientific Reports. 2016 ; 6( 22421): 1-13.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1038/srep22421
Artigo de periodico
EGFR signaling regulates maspin/serpinB5 phosphorylation and nuclear localization in mammary epithelial cells (2016)
Longhi, Mariana Tamazato; Magalhães, Magna; Reina, Jeffrey; Freitas, Vanessa Morais ; Cella, Nathalie
Fonte: PLOS ONE. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
LONGHI, Mariana Tamazato et al. EGFR signaling regulates maspin/serpinB5 phosphorylation and nuclear localization in mammary epithelial cells. PLOS ONE, v. 11, n. 7, p. 1-15, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0159856. Acesso em: 25 jul. 2024.
APA
Longhi, M. T., Magalhães, M., Reina, J., Freitas, V. M., & Cella, N. (2016). EGFR signaling regulates maspin/serpinB5 phosphorylation and nuclear localization in mammary epithelial cells. PLOS ONE, 11( 7), 1-15. doi:10.1371/journal.pone.0159856
NLM
Longhi MT, Magalhães M, Reina J, Freitas VM, Cella N. EGFR signaling regulates maspin/serpinB5 phosphorylation and nuclear localization in mammary epithelial cells [Internet]. PLOS ONE. 2016 ; 11( 7): 1-15.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0159856
Vancouver
Longhi MT, Magalhães M, Reina J, Freitas VM, Cella N. EGFR signaling regulates maspin/serpinB5 phosphorylation and nuclear localization in mammary epithelial cells [Internet]. PLOS ONE. 2016 ; 11( 7): 1-15.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0159856
Artigo de periodico
Chemogenomic study of carboplatin in Saccharomyces cerevisiae: inhibition of the NEDDylation process overcomes cellular resistance mediated by HuR and cullin proteins (2015)
Sousa, Graziele Fonseca de; Lima, Maíra de Assis; Custodio, Debora Fernandes; Freitas, Vanessa Morais ; Monteiro, Gisele
Fonte: PLOS ONE. Unidades: ICB, FCF
Assuntos: SACCHAROMYCES, ANTINEOPLÁSICOS, LEVEDURAS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SOUSA, Graziele Fonseca de et al. Chemogenomic study of carboplatin in Saccharomyces cerevisiae: inhibition of the NEDDylation process overcomes cellular resistance mediated by HuR and cullin proteins. PLOS ONE, v. 10, n. 12, p. 1-18 e0145377, 2015Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0145377. Acesso em: 25 jul. 2024.
APA
Sousa, G. F. de, Lima, M. de A., Custodio, D. F., Freitas, V. M., & Monteiro, G. (2015). Chemogenomic study of carboplatin in Saccharomyces cerevisiae: inhibition of the NEDDylation process overcomes cellular resistance mediated by HuR and cullin proteins. PLOS ONE, 10( 12), 1-18 e0145377. doi:10.1371/journal.pone.0145377
NLM
Sousa GF de, Lima M de A, Custodio DF, Freitas VM, Monteiro G. Chemogenomic study of carboplatin in Saccharomyces cerevisiae: inhibition of the NEDDylation process overcomes cellular resistance mediated by HuR and cullin proteins [Internet]. PLOS ONE. 2015 ; 10( 12): 1-18 e0145377.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0145377
Vancouver
Sousa GF de, Lima M de A, Custodio DF, Freitas VM, Monteiro G. Chemogenomic study of carboplatin in Saccharomyces cerevisiae: inhibition of the NEDDylation process overcomes cellular resistance mediated by HuR and cullin proteins [Internet]. PLOS ONE. 2015 ; 10( 12): 1-18 e0145377.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0145377
Artigo de periodico
A novel cell line derived from pleomorphic adenoma expresses MMP2, MMP9, TIMP1, TIMP2, and shows numeric chromosomal anomalies (2014)
Falcão, Aline Semblano Carreira; Kataoka, Maria Sueli da Silva; Ribeiro, Nélson Antonio Bailão; Diniz Jr, José Antonio Picanço; Alves Jr, Sérgio Melo; Ribeiro, André L.; Siqueira, Adriane Sousa de; Silva, Artur Luiz da; Ramos, Rommel Thiago Jucá; Freitas, Vanessa Morais ; Jaeger, Ruy Gastaldoni ; Pinheiro, João J. V.
Fonte: PLOS ONE. Unidade: ICB
Assuntos: HISTOLOGIA, CROMOSSOMOS (ANORMALIDADES)
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FALCÃO, Aline Semblano Carreira et al. A novel cell line derived from pleomorphic adenoma expresses MMP2, MMP9, TIMP1, TIMP2, and shows numeric chromosomal anomalies. PLOS ONE, v. 9, n. 8, p. 1-15, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0105231. Acesso em: 25 jul. 2024.
APA
Falcão, A. S. C., Kataoka, M. S. da S., Ribeiro, N. A. B., Diniz Jr, J. A. P., Alves Jr, S. M., Ribeiro, A. L., et al. (2014). A novel cell line derived from pleomorphic adenoma expresses MMP2, MMP9, TIMP1, TIMP2, and shows numeric chromosomal anomalies. PLOS ONE, 9( 8), 1-15. doi:10.1371/journal.pone.0105231
NLM
Falcão ASC, Kataoka MS da S, Ribeiro NAB, Diniz Jr JAP, Alves Jr SM, Ribeiro AL, Siqueira AS de, Silva AL da, Ramos RTJ, Freitas VM, Jaeger RG, Pinheiro JJV. A novel cell line derived from pleomorphic adenoma expresses MMP2, MMP9, TIMP1, TIMP2, and shows numeric chromosomal anomalies [Internet]. PLOS ONE. 2014 ; 9( 8): 1-15.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0105231
Vancouver
Falcão ASC, Kataoka MS da S, Ribeiro NAB, Diniz Jr JAP, Alves Jr SM, Ribeiro AL, Siqueira AS de, Silva AL da, Ramos RTJ, Freitas VM, Jaeger RG, Pinheiro JJV. A novel cell line derived from pleomorphic adenoma expresses MMP2, MMP9, TIMP1, TIMP2, and shows numeric chromosomal anomalies [Internet]. PLOS ONE. 2014 ; 9( 8): 1-15.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0105231
Artigo de periodico
Brain intraventricular injection of Amyloid-β in Zebrafish embryo impairs cognition and increases tau phosphorylation, effects reversed by lithium (2014)
Nery, Laura Roesler; Eltz, Natalia Silva; Hackman, Cristiana; Fonseca, Raphaela; Altenhofen, Stefani; Guerra, Heydi Noriega; Freitas, Vanessa Morais ; Bonan, Carla Denise; Vianna, Monica Ryff Moreira Roca
Fonte: PLOS ONE. Unidade: ICB
Assuntos: HISTOLOGIA, EMBRIOLOGIA ANIMAL, LITIO, TELEOSTEI
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
NERY, Laura Roesler et al. Brain intraventricular injection of Amyloid-β in Zebrafish embryo impairs cognition and increases tau phosphorylation, effects reversed by lithium. PLOS ONE, v. 9, n. 9, p. 1-7, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0105862. Acesso em: 25 jul. 2024.
APA
Nery, L. R., Eltz, N. S., Hackman, C., Fonseca, R., Altenhofen, S., Guerra, H. N., et al. (2014). Brain intraventricular injection of Amyloid-β in Zebrafish embryo impairs cognition and increases tau phosphorylation, effects reversed by lithium. PLOS ONE, 9( 9), 1-7. doi:10.1371/journal.pone.0105862
NLM
Nery LR, Eltz NS, Hackman C, Fonseca R, Altenhofen S, Guerra HN, Freitas VM, Bonan CD, Vianna MRMR. Brain intraventricular injection of Amyloid-β in Zebrafish embryo impairs cognition and increases tau phosphorylation, effects reversed by lithium [Internet]. PLOS ONE. 2014 ; 9( 9): 1-7.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0105862
Vancouver
Nery LR, Eltz NS, Hackman C, Fonseca R, Altenhofen S, Guerra HN, Freitas VM, Bonan CD, Vianna MRMR. Brain intraventricular injection of Amyloid-β in Zebrafish embryo impairs cognition and increases tau phosphorylation, effects reversed by lithium [Internet]. PLOS ONE. 2014 ; 9( 9): 1-7.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1371/journal.pone.0105862
Artigo de periodico
Expression of molecules related to AKT pathway as putative regulators of ameloblastoma local invasiveness (2013)
Cecim, Rodolpho L.; Carmo, Hicso A. F.; Kataoka, Maria Sueli da Silva ; Freitas, Vanessa Morais ; Alves Júnior, Sérgio de Melo ; Pedreira, Erick N. ; Jaeger, Ruy Gastaldoni ; Pinheiro, João J. V.
Fonte: Journal of Oral Pathology & MedicinePlosne. Unidade: ICB
Assuntos: BIOLOGIA CELULAR, NEOPLASIAS MAXILOMANDIBULARES, AMELOBLASTOMA, TUMORES ODONTOGÊNICOS, CITOPLASMA, NÚCLEO CELULAR, TRANSDUÇÃO DE SINAL CELULAR, CÉLULAS CULTIVADAS DE TUMOR, PROTEÍNAS PROTO-ONCOGÊNICAS, CÉLULAS ESTROMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CECIM, Rodolpho L. et al. Expression of molecules related to AKT pathway as putative regulators of ameloblastoma local invasiveness. Journal of Oral Pathology & MedicinePlosne, v. Fe 2013, p. 143-147, 2013Tradução . . Disponível em: https://doi.org/10.1111/jop.12103. Acesso em: 25 jul. 2024.
APA
Cecim, R. L., Carmo, H. A. F., Kataoka, M. S. da S., Freitas, V. M., Alves Júnior, S. de M., Pedreira, E. N., et al. (2013). Expression of molecules related to AKT pathway as putative regulators of ameloblastoma local invasiveness. Journal of Oral Pathology & MedicinePlosne, Fe 2013, 143-147. doi:10.1111/jop.12103
NLM
Cecim RL, Carmo HAF, Kataoka MS da S, Freitas VM, Alves Júnior S de M, Pedreira EN, Jaeger RG, Pinheiro JJV. Expression of molecules related to AKT pathway as putative regulators of ameloblastoma local invasiveness [Internet]. Journal of Oral Pathology & MedicinePlosne. 2013 ; Fe 2013 143-147.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1111/jop.12103
Vancouver
Cecim RL, Carmo HAF, Kataoka MS da S, Freitas VM, Alves Júnior S de M, Pedreira EN, Jaeger RG, Pinheiro JJV. Expression of molecules related to AKT pathway as putative regulators of ameloblastoma local invasiveness [Internet]. Journal of Oral Pathology & MedicinePlosne. 2013 ; Fe 2013 143-147.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1111/jop.12103
Artigo de periodico
MYC, FBXW7 and TP53 copy number variation and expression in Gastric Cancer (2013)
Calcagno, Danielle Queiroz; Freitas, Vanessa Morais ; Leal, Mariana Ferreira; Souza, Carolina Rosal Teixeira; Demachki, Samia; Montenegro, Raquel; Assumpção, Paulo Pimentel; Khayat, André Salim; Smith, Marília de Arruda Cardoso; Santos, Andrea Kely Campos Ribeiro dos; Burbano, Rommel Rodriguez
Fonte: BMC Gastroenterology. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CALCAGNO, Danielle Queiroz et al. MYC, FBXW7 and TP53 copy number variation and expression in Gastric Cancer. BMC Gastroenterology, v. 13, p. 1-10, 2013Tradução . . Disponível em: https://doi.org/10.1186/1471-230X-13-141. Acesso em: 25 jul. 2024.
APA
Calcagno, D. Q., Freitas, V. M., Leal, M. F., Souza, C. R. T., Demachki, S., Montenegro, R., et al. (2013). MYC, FBXW7 and TP53 copy number variation and expression in Gastric Cancer. BMC Gastroenterology, 13, 1-10. doi:10.1186/1471-230X-13-141
NLM
Calcagno DQ, Freitas VM, Leal MF, Souza CRT, Demachki S, Montenegro R, Assumpção PP, Khayat AS, Smith M de AC, Santos AKCR dos, Burbano RR. MYC, FBXW7 and TP53 copy number variation and expression in Gastric Cancer [Internet]. BMC Gastroenterology. 2013 ; 13 1-10.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1186/1471-230X-13-141
Vancouver
Calcagno DQ, Freitas VM, Leal MF, Souza CRT, Demachki S, Montenegro R, Assumpção PP, Khayat AS, Smith M de AC, Santos AKCR dos, Burbano RR. MYC, FBXW7 and TP53 copy number variation and expression in Gastric Cancer [Internet]. BMC Gastroenterology. 2013 ; 13 1-10.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1186/1471-230X-13-141
Artigo de periodico
Anti-Angiogenic and anti-Metastatic activity of synthetic phosphoethanolamine (2013)
Ferreira, Adilson Kleber ; Freitas, Vanessa Morais ; Levy, Débora ; Ruiz, Jorge Luiz Mária; Bydlowski, Sérgio Paulo ; Rici, Rose Eli Grassi ; Ribeiro Filho, Otaviano Mendonça; Chierice, Gilberto Orivaldo ; Maria, Durvanei Augusto
Fonte: PLOS ONE. Unidades: ICB, FM, FMVZ, IQSC
Assuntos: METÁSTASE NEOPLÁSICA, CAMUNDONGOS, CARCINOMA, NEOPLASIAS PULMONARES, NEOPLASIAS RENAIS, TÚBULOS RENAIS PROXIMAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FERREIRA, Adilson Kleber et al. Anti-Angiogenic and anti-Metastatic activity of synthetic phosphoethanolamine. PLOS ONE, v. 8, n. 3, p. 14 , 2013Tradução . . Disponível em: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0057937. Acesso em: 25 jul. 2024.
APA
Ferreira, A. K., Freitas, V. M., Levy, D., Ruiz, J. L. M., Bydlowski, S. P., Rici, R. E. G., et al. (2013). Anti-Angiogenic and anti-Metastatic activity of synthetic phosphoethanolamine. PLOS ONE, 8( 3), 14 . doi:10.1371/journal.pone.0057937
NLM
Ferreira AK, Freitas VM, Levy D, Ruiz JLM, Bydlowski SP, Rici REG, Ribeiro Filho OM, Chierice GO, Maria DA. Anti-Angiogenic and anti-Metastatic activity of synthetic phosphoethanolamine [Internet]. PLOS ONE. 2013 ; 8( 3): 14 .[citado 2024 jul. 25 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0057937
Vancouver
Ferreira AK, Freitas VM, Levy D, Ruiz JLM, Bydlowski SP, Rici REG, Ribeiro Filho OM, Chierice GO, Maria DA. Anti-Angiogenic and anti-Metastatic activity of synthetic phosphoethanolamine [Internet]. PLOS ONE. 2013 ; 8( 3): 14 .[citado 2024 jul. 25 ] Available from: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0057937
Artigo de periodico
Retinoic acid and cAMP inhibit rat hepatocellular carcinoma cell proliferation and enhance cell differentiation (2012)
Ionta, Marisa; Rosa, M. C.; Almeida, R. B.; Freitas, Vanessa Morais ; Rezende-Teixeira, P.; Machado-Santelli, Glaucia Maria
Fonte: Brazilian Journal of Medical and Biological Research. Unidade: ICB
Assunto: HISTOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
IONTA, Marisa et al. Retinoic acid and cAMP inhibit rat hepatocellular carcinoma cell proliferation and enhance cell differentiation. Brazilian Journal of Medical and Biological Research, v. 45, n. 8, p. 721-729, 2012Tradução . . Disponível em: https://doi.org/10.1590/S0100-879X2012007500087. Acesso em: 25 jul. 2024.
APA
Ionta, M., Rosa, M. C., Almeida, R. B., Freitas, V. M., Rezende-Teixeira, P., & Machado-Santelli, G. M. (2012). Retinoic acid and cAMP inhibit rat hepatocellular carcinoma cell proliferation and enhance cell differentiation. Brazilian Journal of Medical and Biological Research, 45( 8), 721-729. doi:10.1590/S0100-879X2012007500087
NLM
Ionta M, Rosa MC, Almeida RB, Freitas VM, Rezende-Teixeira P, Machado-Santelli GM. Retinoic acid and cAMP inhibit rat hepatocellular carcinoma cell proliferation and enhance cell differentiation [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 8): 721-729.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1590/S0100-879X2012007500087
Vancouver
Ionta M, Rosa MC, Almeida RB, Freitas VM, Rezende-Teixeira P, Machado-Santelli GM. Retinoic acid and cAMP inhibit rat hepatocellular carcinoma cell proliferation and enhance cell differentiation [Internet]. Brazilian Journal of Medical and Biological Research. 2012 ; 45( 8): 721-729.[citado 2024 jul. 25 ] Available from: https://doi.org/10.1590/S0100-879X2012007500087

Unidades USP

ReP

Filtros

Departamento

Autores

Autores USP

Ano de publicação

Assuntos

Título da fonte

Indexado em

Afiliação dos autores externos não normalizada