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Artigo de periodico
Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway (2018)
Senger, Nathalia ; Melo, Marcos Barrouin; Diniz, Gabriela Placoná ; Santos, Maria José Campagnole dos ; Santos, Robson Augusto Souza dos; Barreto-Chaves, Maria Luiza Morais
Source: Clinical Science. Unidade: ICB
Subjects: ANGIOTENSINAS, CORAÇÃO, HIPERTIREOIDISMO, SISTEMA RENINA-ANGIOTENSINA, HORMÔNIOS TIREOIDIANOS, ANGIOTENSINA II, ANATOMIA
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ABNT
SENGER, Nathalia et al. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, v. 132, n. 11, p. 1117-1133, 2018Tradução . . Disponível em: https://doi.org/10.1042/CS20171606. Acesso em: 29 jun. 2024.
APA
Senger, N., Melo, M. B., Diniz, G. P., Santos, M. J. C. dos, Santos, R. A. S. dos, & Barreto-Chaves, M. L. M. (2018). Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway. Clinical Science, 132( 11), 1117-1133. doi:10.1042/CS20171606
NLM
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1042/CS20171606
Vancouver
Senger N, Melo MB, Diniz GP, Santos MJC dos, Santos RAS dos, Barreto-Chaves MLM. Angiotensin-(1–7) reduces cardiac effects of thyroid hormone by GSK3Β/NFATc3 signaling pathway [Internet]. Clinical Science. 2018 ; 132( 11): 1117-1133.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1042/CS20171606
Artigo de periodico
MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone (2017)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Moreno, Camila Rodrigues; Senger, Nathalia; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Cellular Physiology. Unidade: ICB
Subjects: ANATOMIA, HIPERTIREOIDISMO, HORMÔNIOS TIREOIDIANOS, RNA DE TRANSFERÊNCIA, SISTEMA RENINA-ANGIOTENSINA, RATOS WISTAR, BLOQUEIO CARDÍACO
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ABNT
DINIZ, Gabriela Placoná et al. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, v. 232, n. 12, p. 3360-3368, 2017Tradução . . Disponível em: https://doi.org/10.1002/jcp.25781. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Lino, C. A., Moreno, C. R., Senger, N., & Barreto-Chaves, M. L. M. (2017). MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone. Journal of Cellular Physiology, 232( 12), 3360-3368. doi:10.1002/jcp.25781
NLM
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1002/jcp.25781
Vancouver
Diniz GP, Lino CA, Moreno CR, Senger N, Barreto-Chaves MLM. MicroRNA-1 overexpression blunts cardiomyocyte hypertrophy elicited by thyroid hormone [Internet]. Journal of Cellular Physiology. 2017 ; 232( 12): 3360-3368.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1002/jcp.25781
Artigo de periodico
S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone (2017)
Takano, Ana Paula Cremasco ; Munhoz, Carolina Demarchi ; Moriscot, Anselmo Sigari ; Gupta, Sudhiranjan; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Molecular Medicine. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAKANO, Ana Paula Cremasco et al. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, v. 95, n. 6, p. 671-682, 2017Tradução . . Disponível em: https://doi.org/10.1007/s00109-017-1511-y. Acesso em: 29 jun. 2024.
APA
Takano, A. P. C., Munhoz, C. D., Moriscot, A. S., Gupta, S., & Barreto-Chaves, M. L. M. (2017). S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone. Journal of Molecular Medicine, 95( 6), 671-682. doi:10.1007/s00109-017-1511-y
NLM
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Vancouver
Takano APC, Munhoz CD, Moriscot AS, Gupta S, Barreto-Chaves MLM. S100A8/MYD88/NF-қB: a novel pathway involved in cardiomyocyte hypertrophy driven by thyroid hormone [Internet]. Journal of Molecular Medicine. 2017 ; 95( 6): 671-682.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s00109-017-1511-y
Artigo de periodico
Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy (2017)
Diniz, Gabriela Placoná ; Huang, Zhan-Peng; Liu, Jianming; Chen, Jinghai; Ding, Jian; Fonseca, Renata Inzinna; Barreto-Chaves, Maria Luiza Morais ; Donato Junior, José ; Hu, Xiaoyun; Wang, Da-Zhi
Source: Clinical Science. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
DINIZ, Gabriela Placoná et al. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, v. 131, n. 24, p. 2885-2900, 2017Tradução . . Disponível em: https://doi.org/10.1042/CS20171368. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Huang, Z. -P., Liu, J., Chen, J., Ding, J., Fonseca, R. I., et al. (2017). Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy. Clinical Science, 131( 24), 2885-2900. doi:10.1042/CS20171368
NLM
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1042/CS20171368
Vancouver
Diniz GP, Huang Z-P, Liu J, Chen J, Ding J, Fonseca RI, Barreto-Chaves MLM, Donato Junior J, Hu X, Wang D-Z. Loss of microRNA-22 prevents high-fat diet induced dyslipidemia and increases energy expenditure without affecting cardiac hypertrophy [Internet]. Clinical Science. 2017 ; 131( 24): 2885-2900.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1042/CS20171368
Artigo de periodico
Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy (2016)
Diniz, Gabriela Placoná ; Senger, Nathalia; Carneiro-Ramos, Marcela S.; Santos, Robson A. S.; Barreto-Chaves, Maria Luiza Morais
Source: Therapeutic Advances in Cardiovascular Disease. Unidade: ICB
Assunto: ANATOMIA
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ABNT
DINIZ, Gabriela Placoná et al. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, v. 10, n. 4, p. 192-202, 2016Tradução . . Disponível em: https://doi.org/10.1177/1753944715623228. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Senger, N., Carneiro-Ramos, M. S., Santos, R. A. S., & Barreto-Chaves, M. L. M. (2016). Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy. Therapeutic Advances in Cardiovascular Disease, 10( 4), 192-202. doi:10.1177/1753944715623228
NLM
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1177/1753944715623228
Vancouver
Diniz GP, Senger N, Carneiro-Ramos MS, Santos RAS, Barreto-Chaves MLM. Cardiac ACE2/angiotensin 1-7/Mas receptor axis is activated in thyroid hormone-induced cardiac hypertrophy [Internet]. Therapeutic Advances in Cardiovascular Disease. 2016 ; 10( 4): 192-202.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1177/1753944715623228
Artigo de periodico
Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor (2015)
Diniz, Gabriela Placoná ; Lino, Caroline Antunes; Guedes, Elaine Castilho; Moreira, Luana do Nascimento; Barreto-Chaves, Maria Luiza Morais
Source: Basic Research in Cardiology. Unidade: ICB
Subjects: ANATOMIA, HORMÔNIOS TIREOIDIANOS, SISTEMA RENINA-ANGIOTENSINA, ANGIOTENSINA II
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ABNT
DINIZ, Gabriela Placoná et al. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, v. 110, n. 5, p. 1-11, 2015Tradução . . Disponível em: https://doi.org/10.1007/s00395-015-0504-7. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Lino, C. A., Guedes, E. C., Moreira, L. do N., & Barreto-Chaves, M. L. M. (2015). Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor. Basic Research in Cardiology, 110( 5), 1-11. doi:10.1007/s00395-015-0504-7
NLM
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Vancouver
Diniz GP, Lino CA, Guedes EC, Moreira L do N, Barreto-Chaves MLM. Cardiac microRNA-133 is down-regulated in thyroid hormone-mediated cardiac hypertrophy partially via Type 1 Angiotensin II receptor [Internet]. Basic Research in Cardiology. 2015 ; 110( 5): 1-11.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s00395-015-0504-7
Artigo de periodico
An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies (2014)
Ceravolo, Graziela S.; Montezano, Augusto C.; Jordão, Maria T.; Akamine, Eliana Hiromi ; Costa, Tiago Januário da; Takano, Ana Paula Cremasco ; Fernandes, Denise C.; Barreto-Chaves, Maria Luiza Morais ; Laurindo, Francisco R.; Tostes, Rita de Cássia; Fortes, Zuleica Bruno; Chopard, Renato Paulo; Touyz, Rhian M.; Carvalho, Maria Helena Catelli de
Source: PLOS ONE. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
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ABNT
CERAVOLO, Graziela S. et al. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, v. 9, n. 11, 2014Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0111117. Acesso em: 29 jun. 2024.
APA
Ceravolo, G. S., Montezano, A. C., Jordão, M. T., Akamine, E. H., Costa, T. J. da, Takano, A. P. C., et al. (2014). An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies. PLOS ONE, 9( 11). doi:10.1371/journal.pone.0111117
NLM
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Vancouver
Ceravolo GS, Montezano AC, Jordão MT, Akamine EH, Costa TJ da, Takano APC, Fernandes DC, Barreto-Chaves MLM, Laurindo FR, Tostes R de C, Fortes ZB, Chopard RP, Touyz RM, Carvalho MHC de. An Interaction of renin-angiotensin and kallikrein-kinin systems contributes to vascular hypertrophy in angiotensin II-induced hypertension: in vivo and in vitro studies [Internet]. PLOS ONE. 2014 ; 9( 11):[citado 2024 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0111117
Artigo de periodico
Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion (2013)
Tavares, Felix Meira; Silva, Ivson Bezerra da; Gomes, Dayane Aparecida; Barreto-Chaves, Maria Luiza Morais
Source: Cardiovascular Drugs and Therapy. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAVARES, Felix Meira et al. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion. Cardiovascular Drugs and Therapy, v. 27, n. 5, p. 393-402, 2013Tradução . . Disponível em: https://doi.org/10.1007/s10557-013-6473-x. Acesso em: 29 jun. 2024.
APA
Tavares, F. M., Silva, I. B. da, Gomes, D. A., & Barreto-Chaves, M. L. M. (2013). Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion. Cardiovascular Drugs and Therapy, 27( 5), 393-402. doi:10.1007/s10557-013-6473-x
NLM
Tavares FM, Silva IB da, Gomes DA, Barreto-Chaves MLM. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 5): 393-402.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s10557-013-6473-x
Vancouver
Tavares FM, Silva IB da, Gomes DA, Barreto-Chaves MLM. Angiotensin II Type 2 Receptor (AT2R) is associated with increased tolerance of the hyperthyroid heart to ischemia-reperfusion [Internet]. Cardiovascular Drugs and Therapy. 2013 ; 27( 5): 393-402.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1007/s10557-013-6473-x
Artigo de periodico
AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth (2013)
Takano, Ana Paula Cremasco ; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 29 jun. 2024.
APA
Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
NLM
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Vancouver
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Artigo de periodico
M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats (2013)
Rozanski, Andrei; Takano, Ana Paula Cremasco ; Kato, Patricia N.; Soares, Antonio G.; Lellis-Santos, Camilo; Campos, Juliane Cruz; Ferreira, Julio Cesar Batista ; Barreto-Chaves, Maria Luiza Morais ; Moriscot, Anselmo Sigari
Source: Molecular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
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ABNT
ROZANSKI, Andrei et al. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats. Molecular Endocrinology, v. 27, n. 12, p. 2055-2065, 2013Tradução . . Disponível em: https://doi.org/10.1210/me.2013-1018. Acesso em: 29 jun. 2024.
APA
Rozanski, A., Takano, A. P. C., Kato, P. N., Soares, A. G., Lellis-Santos, C., Campos, J. C., et al. (2013). M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats. Molecular Endocrinology, 27( 12), 2055-2065. doi:10.1210/me.2013-1018
NLM
Rozanski A, Takano APC, Kato PN, Soares AG, Lellis-Santos C, Campos JC, Ferreira JCB, Barreto-Chaves MLM, Moriscot AS. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats [Internet]. Molecular Endocrinology. 2013 ; 27( 12): 2055-2065.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1210/me.2013-1018
Vancouver
Rozanski A, Takano APC, Kato PN, Soares AG, Lellis-Santos C, Campos JC, Ferreira JCB, Barreto-Chaves MLM, Moriscot AS. M-Protein is down-regulated in cardiac hypertrophy driven by thyroid hormone in rats [Internet]. Molecular Endocrinology. 2013 ; 27( 12): 2055-2065.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1210/me.2013-1018
Artigo de periodico
miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation (2013)
Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
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ABNT
DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
NLM
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Vancouver
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Artigo de periodico
Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism (2013)
Carrillo-Sepúlveda, Maria Alícia; Ceravolo, Graziela S.; Furstenau, Cristina R.; Monteiro, Priscilla de Souza; Bruno-Fortes, Zuleica; Carvalho, Maria Helena Catelli de; Laurindo, Francisco R.; Tostes, Rita de Cássia; Webb, R. Clinton; Barreto-Chaves, Maria Luiza Morais
Source: PLOS ONE. Unidades: ICB, FMRP
Subjects: ANATOMIA, FARMACOLOGIA
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ABNT
CARRILLO-SEPÚLVEDA, Maria Alícia et al. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism. PLOS ONE, v. 8, n. 4, p. 1-9, 2013Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0061982. Acesso em: 29 jun. 2024.
APA
Carrillo-Sepúlveda, M. A., Ceravolo, G. S., Furstenau, C. R., Monteiro, P. de S., Bruno-Fortes, Z., Carvalho, M. H. C. de, et al. (2013). Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism. PLOS ONE, 8( 4), 1-9. doi:10.1371/journal.pone.0061982
NLM
Carrillo-Sepúlveda MA, Ceravolo GS, Furstenau CR, Monteiro P de S, Bruno-Fortes Z, Carvalho MHC de, Laurindo FR, Tostes R de C, Webb RC, Barreto-Chaves MLM. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism [Internet]. PLOS ONE. 2013 ;8( 4): 1-9.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0061982
Vancouver
Carrillo-Sepúlveda MA, Ceravolo GS, Furstenau CR, Monteiro P de S, Bruno-Fortes Z, Carvalho MHC de, Laurindo FR, Tostes R de C, Webb RC, Barreto-Chaves MLM. Emerging role of angiotensin type 2 receptor (AT2R)/Akt/NO pathway in vascular smooth muscle cell in the hyperthyroidism [Internet]. PLOS ONE. 2013 ;8( 4): 1-9.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1371/journal.pone.0061982
Artigo de periodico
New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression (2012)
Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco ; Brunetto-Ract, Erika Lia; Silva, Francemilson Goulart da ; Nunes, Maria Tereza ; Barreto-Chaves, Maria Luiza Morais
Source: Journal of Molecular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná et al. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, v. 49, n. 1, p. 11-20, 2012Tradução . . Disponível em: https://doi.org/10.1530/JME-11-0141. Acesso em: 29 jun. 2024.
APA
Diniz, G. P., Takano, A. P. C., Brunetto-Ract, E. L., Silva, F. G. da, Nunes, M. T., & Barreto-Chaves, M. L. M. (2012). New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression. Journal of Molecular Endocrinology, 49( 1), 11-20. doi:10.1530/JME-11-0141
NLM
Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1530/JME-11-0141
Vancouver
Diniz GP, Takano APC, Brunetto-Ract EL, Silva FG da, Nunes MT, Barreto-Chaves MLM. New insight into the mechanisms associated with the rapid effect of T3 on AT1R expression [Internet]. Journal of Molecular Endocrinology. 2012 ; 49( 1): 11-20.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1530/JME-11-0141
Artigo de periodico
Thyroid hormones are involved in 5′-nucleotidase modulation in soluble fraction of cardiac tissue (2012)
Cotomacci, Guilherme; Sarkis, João J. F.; Fürstenau, Cristina R.; Barreto-Chaves, Maria Luiza Morais
Source: Life Sciences. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COTOMACCI, Guilherme et al. Thyroid hormones are involved in 5′-nucleotidase modulation in soluble fraction of cardiac tissue. Life Sciences, v. 91, n. 3-4, p. 137-142, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2012.06.023. Acesso em: 29 jun. 2024.
APA
Cotomacci, G., Sarkis, J. J. F., Fürstenau, C. R., & Barreto-Chaves, M. L. M. (2012). Thyroid hormones are involved in 5′-nucleotidase modulation in soluble fraction of cardiac tissue. Life Sciences, 91( 3-4), 137-142. doi:10.1016/j.lfs.2012.06.023
NLM
Cotomacci G, Sarkis JJF, Fürstenau CR, Barreto-Chaves MLM. Thyroid hormones are involved in 5′-nucleotidase modulation in soluble fraction of cardiac tissue [Internet]. Life Sciences. 2012 ; 91( 3-4): 137-142.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.lfs.2012.06.023
Vancouver
Cotomacci G, Sarkis JJF, Fürstenau CR, Barreto-Chaves MLM. Thyroid hormones are involved in 5′-nucleotidase modulation in soluble fraction of cardiac tissue [Internet]. Life Sciences. 2012 ; 91( 3-4): 137-142.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.lfs.2012.06.023
Artigo de periodico
Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system (2011)
Araujo, A. S. R.; Diniz, Gabriela Placoná ; Seibel, F. E. R.; Branchini, G.; Ribeiro, M. F. M.; Brum, I. S.; Khaper, N.; Barreto-Chaves, Maria Luiza Morais ; Belló-Klein, A.
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, A. S. R. et al. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, v. 333, n. 1, p. 78-84, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.018. Acesso em: 29 jun. 2024.
APA
Araujo, A. S. R., Diniz, G. P., Seibel, F. E. R., Branchini, G., Ribeiro, M. F. M., Brum, I. S., et al. (2011). Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, 333( 1), 78-84. doi:10.1016/j.mce.2010.12.018
NLM
Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
Vancouver
Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
Artigo de periodico
The crosstalk between thyroid hormones and the Renin–Angiotensin System (2010)
Barreto-Chaves, Maria Luiza Morais ; Carrillo-Sepúlveda, Maria Alícia; Carneiro-Ramos, Marcela Sorelli; Gomes, Dayane Aparecida; Diniz, Gabriela Placoná
Source: Vascular Pharmacology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BARRETO-CHAVES, Maria Luiza Morais et al. The crosstalk between thyroid hormones and the Renin–Angiotensin System. Vascular Pharmacology, v. 52, n. 3-4, p. 166-170, 2010Tradução . . Disponível em: https://doi.org/10.1016/j.vph.2009.10.009. Acesso em: 29 jun. 2024.
APA
Barreto-Chaves, M. L. M., Carrillo-Sepúlveda, M. A., Carneiro-Ramos, M. S., Gomes, D. A., & Diniz, G. P. (2010). The crosstalk between thyroid hormones and the Renin–Angiotensin System. Vascular Pharmacology, 52( 3-4), 166-170. doi:10.1016/j.vph.2009.10.009
NLM
Barreto-Chaves MLM, Carrillo-Sepúlveda MA, Carneiro-Ramos MS, Gomes DA, Diniz GP. The crosstalk between thyroid hormones and the Renin–Angiotensin System [Internet]. Vascular Pharmacology. 2010 ; 52( 3-4): 166-170.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.vph.2009.10.009
Vancouver
Barreto-Chaves MLM, Carrillo-Sepúlveda MA, Carneiro-Ramos MS, Gomes DA, Diniz GP. The crosstalk between thyroid hormones and the Renin–Angiotensin System [Internet]. Vascular Pharmacology. 2010 ; 52( 3-4): 166-170.[citado 2024 jun. 29 ] Available from: https://doi.org/10.1016/j.vph.2009.10.009

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