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Artigo de periodico
Postprandial increase in glucagon-like peptide-1 is blunted in severe heart failure (2020)
Arruda-junior, Daniel F; Martins, Flavia L; Salles, Thiago Almeida; Jensen, Leonardo; Dariolli, Rafael; Antonio, Ednei L; Santos, Leonardo dos; Crajoinas, Renato O; Krieger, Jose Eduardo ; Girardi, Adriana Castello Costa
Source: Clinical science. Unidade: FM
Subjects: HINSUFICIÊNCIA CARDÍACA, INSUFICIÊNCIA CARDÍACA, INFARTO, FISIOLOGIA
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ABNT
ARRUDA-JUNIOR, Daniel F et al. Postprandial increase in glucagon-like peptide-1 is blunted in severe heart failure. Clinical science, v. 134, n. 9, p. 1081-1094, 2020Tradução . . Disponível em: https://doi.org/10.1042/CS20190946. Acesso em: 18 jul. 2024.
APA
Arruda-junior, D. F., Martins, F. L., Salles, T. A., Jensen, L., Dariolli, R., Antonio, E. L., et al. (2020). Postprandial increase in glucagon-like peptide-1 is blunted in severe heart failure. Clinical science, 134( 9), 1081-1094. doi:10.1042/CS20190946
NLM
Arruda-junior DF, Martins FL, Salles TA, Jensen L, Dariolli R, Antonio EL, Santos L dos, Crajoinas RO, Krieger JE, Girardi ACC. Postprandial increase in glucagon-like peptide-1 is blunted in severe heart failure [Internet]. Clinical science. 2020 ; 134( 9): 1081-1094.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1042/CS20190946
Vancouver
Arruda-junior DF, Martins FL, Salles TA, Jensen L, Dariolli R, Antonio EL, Santos L dos, Crajoinas RO, Krieger JE, Girardi ACC. Postprandial increase in glucagon-like peptide-1 is blunted in severe heart failure [Internet]. Clinical science. 2020 ; 134( 9): 1081-1094.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1042/CS20190946
Artigo de periodico
Optimisation of perioperative cardiovascular management to improve surgical outcome II (OPTIMISE II) trial: study protocol for a multicentre international trial of cardiac output-guided fluid therapy with low-dose inotrope infusion compared with usual care in patients undergoing major elective gastrointestinal surgery (2019)
Edwards, Mark R; Forbes, Gordon; Macdonald, Neil; Berdunov, Vladislav; Mihaylova, Borislava; Dias, Priyanthi; Thomson, Ann; Grocott, Michael P. W; Mythen, Monty G; Hajjar, Ludhmila Abrahão
Source: Bmj open. Unidade: FM
Subjects: HEMODINÂMICA, FISIOLOGIA, HOSPITALIZAÇÃO
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ABNT
EDWARDS, Mark R et al. Optimisation of perioperative cardiovascular management to improve surgical outcome II (OPTIMISE II) trial: study protocol for a multicentre international trial of cardiac output-guided fluid therapy with low-dose inotrope infusion compared with usual care in patients undergoing major elective gastrointestinal surgery. Bmj open, v. 9, n. 1, 2019Tradução . . Disponível em: https://doi.org/10.1136/bmjopen-2018-023455. Acesso em: 18 jul. 2024.
APA
Edwards, M. R., Forbes, G., Macdonald, N., Berdunov, V., Mihaylova, B., Dias, P., et al. (2019). Optimisation of perioperative cardiovascular management to improve surgical outcome II (OPTIMISE II) trial: study protocol for a multicentre international trial of cardiac output-guided fluid therapy with low-dose inotrope infusion compared with usual care in patients undergoing major elective gastrointestinal surgery. Bmj open, 9( 1). doi:10.1136/bmjopen-2018-023455
NLM
Edwards MR, Forbes G, Macdonald N, Berdunov V, Mihaylova B, Dias P, Thomson A, Grocott MPW, Mythen MG, Hajjar LA. Optimisation of perioperative cardiovascular management to improve surgical outcome II (OPTIMISE II) trial: study protocol for a multicentre international trial of cardiac output-guided fluid therapy with low-dose inotrope infusion compared with usual care in patients undergoing major elective gastrointestinal surgery [Internet]. Bmj open. 2019 ; 9( 1):[citado 2024 jul. 18 ] Available from: https://doi.org/10.1136/bmjopen-2018-023455
Vancouver
Edwards MR, Forbes G, Macdonald N, Berdunov V, Mihaylova B, Dias P, Thomson A, Grocott MPW, Mythen MG, Hajjar LA. Optimisation of perioperative cardiovascular management to improve surgical outcome II (OPTIMISE II) trial: study protocol for a multicentre international trial of cardiac output-guided fluid therapy with low-dose inotrope infusion compared with usual care in patients undergoing major elective gastrointestinal surgery [Internet]. Bmj open. 2019 ; 9( 1):[citado 2024 jul. 18 ] Available from: https://doi.org/10.1136/bmjopen-2018-023455
Artigo de periodico
The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids (2019)
Felizardo, Raphael José Ferreira; Watanabe, Ingrid Kazue Mizuno; Dardi, Patrizia; Rossoni, Luciana Venturini ; Câmara, Niels Olsen Saraiva
Source: Pharmacological Research. Unidade: ICB
Subjects: FISIOLOGIA, IMUNOLOGIA, NEFROPATIAS, HIPERTENSÃO, INFLAMAÇÃO, ÁCIDOS GRAXOS, METABÓLITOS, TOXINAS, ENDOTOXINAS
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ABNT
FELIZARDO, Raphael José Ferreira et al. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids. Pharmacological Research, v. 141, p. 366-377, 2019Tradução . . Disponível em: https://doi.org/10.1016/j.phrs.2019.01.019. Acesso em: 18 jul. 2024.
APA
Felizardo, R. J. F., Watanabe, I. K. M., Dardi, P., Rossoni, L. V., & Câmara, N. O. S. (2019). The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids. Pharmacological Research, 141, 366-377. doi:10.1016/j.phrs.2019.01.019
NLM
Felizardo RJF, Watanabe IKM, Dardi P, Rossoni LV, Câmara NOS. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids [Internet]. Pharmacological Research. 2019 ; 141 366-377.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.phrs.2019.01.019
Vancouver
Felizardo RJF, Watanabe IKM, Dardi P, Rossoni LV, Câmara NOS. The interplay among gut microbiota, hypertension and kidney diseases: The role of short-chain fatty acids [Internet]. Pharmacological Research. 2019 ; 141 366-377.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.phrs.2019.01.019
Artigo de periodico
Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes (2018)
Cruz, Maysa Mariana ; Lopes, Andressa Bolsoni ; Crisma, Amanda Rabello ; Sá, Roberta Dourado Cavalcante da Cunha de; Kuwabara, Wilson Mitsuo Tatagiba ; Curi, Rui ; Andrade, Paula Bresciani Martins de; Vale, Maria Isabel Cardoso Alonso
Source: Lipids in Health and Disease. Unidade: ICB
Subjects: FISIOLOGIA, GLICOSE, OXIGÊNIO, ÁCIDOS GRAXOS, BIOENERGÉTICA, MITOCÔNDRIAS
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ABNT
CRUZ, Maysa Mariana et al. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids in Health and Disease, v. 17, n. 1, p. 12 , 2018Tradução . . Disponível em: https://doi.org/10.1186/s12944-018-0710-z. Acesso em: 18 jul. 2024.
APA
Cruz, M. M., Lopes, A. B., Crisma, A. R., Sá, R. D. C. da C. de, Kuwabara, W. M. T., Curi, R., et al. (2018). Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes. Lipids in Health and Disease, 17( 1), 12 . doi:10.1186/s12944-018-0710-z
NLM
Cruz MM, Lopes AB, Crisma AR, Sá RDC da C de, Kuwabara WMT, Curi R, Andrade PBM de, Vale MICA. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes [Internet]. Lipids in Health and Disease. 2018 ; 17( 1): 12 .[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/s12944-018-0710-z
Vancouver
Cruz MM, Lopes AB, Crisma AR, Sá RDC da C de, Kuwabara WMT, Curi R, Andrade PBM de, Vale MICA. Palmitoleic acid (16:1n7) increases oxygen consumption, fatty acid oxidation and ATP content in white adipocytes [Internet]. Lipids in Health and Disease. 2018 ; 17( 1): 12 .[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/s12944-018-0710-z
Artigo de periodico
The role of Nox2-derived ROS in the development of cognitive impairment after sepsis (2014)
Hernandes, Marina S.; D'Ávila, Joana C.; Trevelin, Silvia C.; Reis, Patricia A.; Kinjo, Erika R.; Lopes, Lucia Rossetti ; Castro-Faria-Neto, Hugo C.; Cunha, Fernando de Queiroz; Britto, Luiz Roberto Giorgetti de ; Bozza, Fernando A.
Source: Journal of Neuroinflammation. Unidades: ICB, FMRP
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
HERNANDES, Marina S. et al. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, v. 11, n. 01, p. 01-12, 2014Tradução . . Disponível em: https://doi.org/10.1186/1742-2094-11-36. Acesso em: 18 jul. 2024.
APA
Hernandes, M. S., D'Ávila, J. C., Trevelin, S. C., Reis, P. A., Kinjo, E. R., Lopes, L. R., et al. (2014). The role of Nox2-derived ROS in the development of cognitive impairment after sepsis. Journal of Neuroinflammation, 11( 01), 01-12. doi:10.1186/1742-2094-11-36
NLM
Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/1742-2094-11-36
Vancouver
Hernandes MS, D'Ávila JC, Trevelin SC, Reis PA, Kinjo ER, Lopes LR, Castro-Faria-Neto HC, Cunha F de Q, Britto LRG de, Bozza FA. The role of Nox2-derived ROS in the development of cognitive impairment after sepsis [Internet]. Journal of Neuroinflammation. 2014 ; 11( 01): 01-12.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/1742-2094-11-36
Artigo de periodico
The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure (2014)
Ruchaya, P. J.; Antunes, Vagner Roberto ; Paton, J. F. R.; Murphy, D.; Yao, S. T.
Source: Experimental Physiology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
RUCHAYA, P. J. et al. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, v. 99, n. 1, p. 111-122, 2014Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2013.075432. Acesso em: 18 jul. 2024.
APA
Ruchaya, P. J., Antunes, V. R., Paton, J. F. R., Murphy, D., & Yao, S. T. (2014). The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure. Experimental Physiology, 99( 1), 111-122. doi:10.1113/expphysiol.2013.075432
NLM
Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432
Vancouver
Ruchaya PJ, Antunes VR, Paton JFR, Murphy D, Yao ST. The cardiovascular actions of fractalkine/CX3CL1 in the hypothalamic paraventricular nucleus are attenuated in rats with heart failure [Internet]. Experimental Physiology. 2014 ; 99( 1): 111-122.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/expphysiol.2013.075432
Artigo de periodico
Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats (2014)
Fontes, M. T.; Silva, T. L. B. T.; Mota, M. M.; Barreto, A. S.; Rossoni, Luciana Venturini ; Santos, M. R. V.
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FONTES, M. T. et al. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, v. 94, n. 1, p. 24-29, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.017. Acesso em: 18 jul. 2024.
APA
Fontes, M. T., Silva, T. L. B. T., Mota, M. M., Barreto, A. S., Rossoni, L. V., & Santos, M. R. V. (2014). Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats. Life Sciences, 94( 1), 24-29. doi:10.1016/j.lfs.2013.11.017
NLM
Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
Vancouver
Fontes MT, Silva TLBT, Mota MM, Barreto AS, Rossoni LV, Santos MRV. Resistance exercise acutely enhances mesenteric artery insulin-induced relaxation in healthy rats [Internet]. Life Sciences. 2014 ; 94( 1): 24-29.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.017
Artigo de periodico
Possible crosstalk between leptin and proclactin during pregnancy (2014)
Nagaishi, V. S.; Cardinali, L. I.; Zampieri, Thais Tessari; Furigo, Isadora Clivatti; Metzger, Martin Andreas ; Donato Junior, José
Source: Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
NAGAISHI, V. S. et al. Possible crosstalk between leptin and proclactin during pregnancy. Neuroscience, v. 259, p. 71-83, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.11.050. Acesso em: 18 jul. 2024.
APA
Nagaishi, V. S., Cardinali, L. I., Zampieri, T. T., Furigo, I. C., Metzger, M. A., & Donato Junior, J. (2014). Possible crosstalk between leptin and proclactin during pregnancy. Neuroscience, 259, 71-83. doi:10.1016/j.neuroscience.2013.11.050
NLM
Nagaishi VS, Cardinali LI, Zampieri TT, Furigo IC, Metzger MA, Donato Junior J. Possible crosstalk between leptin and proclactin during pregnancy [Internet]. Neuroscience. 2014 ; 259 71-83.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.11.050
Vancouver
Nagaishi VS, Cardinali LI, Zampieri TT, Furigo IC, Metzger MA, Donato Junior J. Possible crosstalk between leptin and proclactin during pregnancy [Internet]. Neuroscience. 2014 ; 259 71-83.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.11.050
Artigo de periodico
Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats (2013)
Sabino-Silva, Robinson; Okamoto, Maristela Mitiko; David-Silva, Aline; Mori, Cristina Rosana; Freitas, Helayne Soares; Machado, Ubiratan Fabres
Source: Diabetology and Metabolic Syndrome. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SABINO-SILVA, Robinson et al. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, v. 5, n. 1, p. 1-5, 2013Tradução . . Disponível em: https://doi.org/10.1186/1758-5996-5-64. Acesso em: 18 jul. 2024.
APA
Sabino-Silva, R., Okamoto, M. M., David-Silva, A., Mori, C. R., Freitas, H. S., & Machado, U. F. (2013). Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats. Diabetology and Metabolic Syndrome, 5( 1), 1-5. doi:10.1186/1758-5996-5-64
NLM
Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/1758-5996-5-64
Vancouver
Sabino-Silva R, Okamoto MM, David-Silva A, Mori CR, Freitas HS, Machado UF. Increased SGLT1 expression in salivary gland ductal cells correlates with hyposalivation in diabetic and hypertensive rats [Internet]. Diabetology and Metabolic Syndrome. 2013 ; 5( 1): 1-5.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1186/1758-5996-5-64
Artigo de periodico
BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease (2013)
Real, Caroline Cristiano; Ferreira, A. F. B.; Chaves-Kirsten, Gabriela Pena; Torrão, Andréa da Silva ; Pires, R. S.; Britto, Luiz Roberto Giorgetti de
Source: Neuroscience. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
REAL, Caroline Cristiano et al. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, v. 237, p. 118-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.01.060. Acesso em: 18 jul. 2024.
APA
Real, C. C., Ferreira, A. F. B., Chaves-Kirsten, G. P., Torrão, A. da S., Pires, R. S., & Britto, L. R. G. de. (2013). BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease. Neuroscience, 237, 118-129. doi:10.1016/j.neuroscience.2013.01.060
NLM
Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060
Vancouver
Real CC, Ferreira AFB, Chaves-Kirsten GP, Torrão A da S, Pires RS, Britto LRG de. BDNF receptor blockade hinders the beneficial effects of exercise in a rat model of parkinson’s disease [Internet]. Neuroscience. 2013 ; 237 118-129.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.01.060
Artigo de periodico
Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells (2013)
Lima, Larissa de Sá; Kawamoto, Elisa Mitiko ; Munhoz, Carolina Demarchi ; Kinoshita, Paula Fernanda; Orellana, Ana Maria Marques; Curi, Rui; Rossoni, Luciana Venturini ; Avellar, M. C. W.; Scavone, Cristoforo
Source: Neuropharmacology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
LIMA, Larissa de Sá et al. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, v. 73, p. 327-336, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuropharm.2013.06.006. Acesso em: 18 jul. 2024.
APA
Lima, L. de S., Kawamoto, E. M., Munhoz, C. D., Kinoshita, P. F., Orellana, A. M. M., Curi, R., et al. (2013). Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells. Neuropharmacology, 73, 327-336. doi:10.1016/j.neuropharm.2013.06.006
NLM
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Vancouver
Lima L de S, Kawamoto EM, Munhoz CD, Kinoshita PF, Orellana AMM, Curi R, Rossoni LV, Avellar MCW, Scavone C. Ouabain activates NFkB through an NMDA signaling pathway in cultured cerebellar cells [Internet]. Neuropharmacology. 2013 ; 73 327-336.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuropharm.2013.06.006
Artigo de periodico
Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats (2013)
Silva, Ariel S. da; Dias, Lucinara D.; Borges, Júlia F.; Markoski, Melissa M.; Souza, Martina S. de; Irigoyen, Maria C.; Machado, Ubiratan Fabres ; Schaan, Beatriz D.
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SILVA, Ariel S. da et al. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, v. 91, n. 24-26, p. 1174-1179, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.05.001. Acesso em: 18 jul. 2024.
APA
Silva, A. S. da, Dias, L. D., Borges, J. F., Markoski, M. M., Souza, M. S. de, Irigoyen, M. C., et al. (2013). Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats. Life Sciences, 91( 24-26), 1174-1179. doi:10.1016/j.lfs.2013.05.001
NLM
Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001
Vancouver
Silva AS da, Dias LD, Borges JF, Markoski MM, Souza MS de, Irigoyen MC, Machado UF, Schaan BD. Renal GLUT1 reduction depends on angiotensin-converting enzyme inhibition in diabetic hypertensive rats [Internet]. Life Sciences. 2013 ; 91( 24-26): 1174-1179.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.05.001
Artigo de periodico
Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats (2013)
Blanco-Rivero, Javier; Roque, Fernanda R.; Sastre, Esther; Caracuel, Laura; Couto, Gisele Kruger; Avendaño, María Soledad; Paula, Suliana Mesquita; Rossoni, Luciana Venturini ; Salaices, Mercedes; Balfagón, Gloria
Source: Journal of Hypertension. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
BLANCO-RIVERO, Javier et al. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, v. 31, n. 5, p. 916-926, 2013Tradução . . Disponível em: https://doi.org/10.1097/HJH.0b013e32835f749c. Acesso em: 18 jul. 2024.
APA
Blanco-Rivero, J., Roque, F. R., Sastre, E., Caracuel, L., Couto, G. K., Avendaño, M. S., et al. (2013). Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats. Journal of Hypertension, 31( 5), 916-926. doi:10.1097/HJH.0b013e32835f749c
NLM
Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
Vancouver
Blanco-Rivero J, Roque FR, Sastre E, Caracuel L, Couto GK, Avendaño MS, Paula SM, Rossoni LV, Salaices M, Balfagón G. Aerobic exercise training increases neuronal nitric oxide release and bioavailability and decreases noradrenaline release inmesenteric artery from spontaneously hypertensive rats [Internet]. Journal of Hypertension. 2013 ; 31( 5): 916-926.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1097/HJH.0b013e32835f749c
Artigo de periodico
Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland (2013)
Villela, Darine; Lima, Larissa de Sá; Peres, Rafael; Peliciari-Garcia, Rodrigo Antonio; Amaral, Fernanda Gaspar do; Cipolla-Neto, José; Scavone, Cristoforo ; Afeche, Solange Castro
Source: Life Sciences. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
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ABNT
VILLELA, Darine et al. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, v. 94, n. 2, p. 122-129, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.11.004. Acesso em: 18 jul. 2024.
APA
Villela, D., Lima, L. de S., Peres, R., Peliciari-Garcia, R. A., Amaral, F. G. do, Cipolla-Neto, J., et al. (2013). Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland. Life Sciences, 94( 2), 122-129. doi:10.1016/j.lfs.2013.11.004
NLM
Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
Vancouver
Villela D, Lima L de S, Peres R, Peliciari-Garcia RA, Amaral FG do, Cipolla-Neto J, Scavone C, Afeche SC. Norepinephrine activates NF-κB transcription factor in cultured rat pineal gland [Internet]. Life Sciences. 2013 ; 94( 2): 122-129.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.11.004
Artigo de periodico
Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine (2013)
Totola, Leonardo Tedesco; Alves, T. B.; Takakura, Ana Carolina ; Ferreira-Neto, Hildebrando Candido; Antunes, Vagner Roberto ; Menani, J. V.; Colombari, E.; Moreira, Thiago dos Santos
Source: Neuroscience. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TOTOLA, Leonardo Tedesco et al. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine. Neuroscience, v. 250, p. 80-91, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2013.06.065. Acesso em: 18 jul. 2024.
APA
Totola, L. T., Alves, T. B., Takakura, A. C., Ferreira-Neto, H. C., Antunes, V. R., Menani, J. V., et al. (2013). Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine. Neuroscience, 250, 80-91. doi:10.1016/j.neuroscience.2013.06.065
NLM
Totola LT, Alves TB, Takakura AC, Ferreira-Neto HC, Antunes VR, Menani JV, Colombari E, Moreira T dos S. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine [Internet]. Neuroscience. 2013 ; 250 80-91.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.06.065
Vancouver
Totola LT, Alves TB, Takakura AC, Ferreira-Neto HC, Antunes VR, Menani JV, Colombari E, Moreira T dos S. Commissural nucleus of the solitary tract regulates the antihypertensive effects elicited by moxonidine [Internet]. Neuroscience. 2013 ; 250 80-91.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.neuroscience.2013.06.065
Artigo de periodico
Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets (2013)
Marçal, Anderson Carlos; Camporez, João Paulo Gabriel; Lima-Salgado, Thais M.; Cintra, D. E.; Akamine, Eliana Hiromi ; Ribeiro, L. M.; Almeida, F. N.; Zanuto, R. P.; Curi, Rui; Boldrini, Silvia de Campos; Liberti, Edson Aparecido; Fiamoncini, Jarlei ; Hirabara, S. M.; Deschamps, F. C.; Carpinelli, Angelo Rafael ; Carvalho, Carla Roberta de Oliveira
Source: British Journal of Nutrition. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MARÇAL, Anderson Carlos et al. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, v. 109, n. 12, p. 2154-2165, 2013Tradução . . Disponível em: https://doi.org/10.1017/S0007114512004576. Acesso em: 18 jul. 2024.
APA
Marçal, A. C., Camporez, J. P. G., Lima-Salgado, T. M., Cintra, D. E., Akamine, E. H., Ribeiro, L. M., et al. (2013). Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets. British Journal of Nutrition, 109( 12), 2154-2165. doi:10.1017/S0007114512004576
NLM
Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1017/S0007114512004576
Vancouver
Marçal AC, Camporez JPG, Lima-Salgado TM, Cintra DE, Akamine EH, Ribeiro LM, Almeida FN, Zanuto RP, Curi R, Boldrini S de C, Liberti EA, Fiamoncini J, Hirabara SM, Deschamps FC, Carpinelli AR, Carvalho CR de O. Changes in food intake, metabolic parameters and insulin resistance are induced by an isoenergetic, medium-chain fatty acid diet and are associated with modifications in insulin signalling in isolated rat pancreatic islets [Internet]. British Journal of Nutrition. 2013 ; 109( 12): 2154-2165.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1017/S0007114512004576
Artigo de periodico
Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats (2013)
David-Silva, Aline; Freitas, Helayne Soares de; Okamoto, Maristela M.; Sabino-Silva, Robinson; Schaan, Beatriz D.; Machado, Ubiratan Fabres
Source: Life Sciences. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DAVID-SILVA, Aline et al. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, v. 93, n. 22, p. 805-813, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2013.10.011. Acesso em: 18 jul. 2024.
APA
David-Silva, A., Freitas, H. S. de, Okamoto, M. M., Sabino-Silva, R., Schaan, B. D., & Machado, U. F. (2013). Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats. Life Sciences, 93( 22), 805-813. doi:10.1016/j.lfs.2013.10.011
NLM
David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011
Vancouver
David-Silva A, Freitas HS de, Okamoto MM, Sabino-Silva R, Schaan BD, Machado UF. Hepatocyte nuclear factors 1α/4α and forkhead box A2 regulate the solute carrier 2A2 (Slc2a2) gene expression in the liver and kidney of diabetic rats [Internet]. Life Sciences. 2013 ; 93( 22): 805-813.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.lfs.2013.10.011
Artigo de periodico
Control of breathing and blood pressure by parafacial neurons in conscious rats (2013)
Takakura, Ana Carolina ; Moreira, Thiago dos Santos ; De Paula, Patrícia M.; Menani, José V.; Colombari, Eduardo
Source: Experimental Physiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
TAKAKURA, Ana Carolina et al. Control of breathing and blood pressure by parafacial neurons in conscious rats. Experimental Physiology, v. 98, n. 1, p. 304-315, 2013Tradução . . Disponível em: https://doi.org/10.1113/expphysiol.2012.065128. Acesso em: 18 jul. 2024.
APA
Takakura, A. C., Moreira, T. dos S., De Paula, P. M., Menani, J. V., & Colombari, E. (2013). Control of breathing and blood pressure by parafacial neurons in conscious rats. Experimental Physiology, 98( 1), 304-315. doi:10.1113/expphysiol.2012.065128
NLM
Takakura AC, Moreira T dos S, De Paula PM, Menani JV, Colombari E. Control of breathing and blood pressure by parafacial neurons in conscious rats [Internet]. Experimental Physiology. 2013 ; 98( 1): 304-315.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/expphysiol.2012.065128
Vancouver
Takakura AC, Moreira T dos S, De Paula PM, Menani JV, Colombari E. Control of breathing and blood pressure by parafacial neurons in conscious rats [Internet]. Experimental Physiology. 2013 ; 98( 1): 304-315.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/expphysiol.2012.065128
Artigo de periodico-carta/editorial
Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? (2013)
Moreira, Thiago dos Santos ; Menani, Jose V.; Colombari, Eduardo; Takakura, Ana Carolina
Source: Journal of Physiology. Unidade: ICB
Subjects: FISIOLOGIA, FARMACOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MOREIRA, Thiago dos Santos et al. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat?. Journal of Physiology. Londres: Instituto de Ciências Biomédicas, Universidade de São Paulo. Disponível em: https://doi.org/10.1113/jphysiol.2012.244640. Acesso em: 18 jul. 2024. , 2013
APA
Moreira, T. dos S., Menani, J. V., Colombari, E., & Takakura, A. C. (2013). Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? Journal of Physiology. Londres: Instituto de Ciências Biomédicas, Universidade de São Paulo. doi:10.1113/jphysiol.2012.244640
NLM
Moreira T dos S, Menani JV, Colombari E, Takakura AC. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? [Internet]. Journal of Physiology. 2013 ; 591( 3): 745-746.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/jphysiol.2012.244640
Vancouver
Moreira T dos S, Menani JV, Colombari E, Takakura AC. Is carotid body input the only critical mechanism involved in hypertension in spontaneously hypertensive rat? [Internet]. Journal of Physiology. 2013 ; 591( 3): 745-746.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1113/jphysiol.2012.244640
Artigo de periodico
Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells (2013)
Costa, E. S.; Faiad, O. J.; Landgraf, R. G.; Ferreira, A. K.; Brigatte, P.; Curi, Rui; Cury, Y.
Source: Toxicon. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
COSTA, E. S. et al. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, v. 74, p. 167-178, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.toxicon.2013.08.057. Acesso em: 18 jul. 2024.
APA
Costa, E. S., Faiad, O. J., Landgraf, R. G., Ferreira, A. K., Brigatte, P., Curi, R., & Cury, Y. (2013). Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells. Toxicon, 74, 167-178. doi:10.1016/j.toxicon.2013.08.057
NLM
Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057
Vancouver
Costa ES, Faiad OJ, Landgraf RG, Ferreira AK, Brigatte P, Curi R, Cury Y. Involvement of formyl peptide receptors in the stimulatory effect of crotoxin on macrophages co-cultivated with tumour cells [Internet]. Toxicon. 2013 ; 74 167-178.[citado 2024 jul. 18 ] Available from: https://doi.org/10.1016/j.toxicon.2013.08.057

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