Source: Programme and Abstracts. Conference titles: Cancer and Metabolism: Pathways to the Future. Unidade: FCFRP
Subjects: ESTRESSE OXIDATIVO, APOPTOSE, CARCINOMA DE CÉLULAS ESCAMOSAS
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PESTANA, Cezar Rangel e CURTI, Carlos e LEOPOLDINO, Andréia Machado. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. Edinburgh: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo. . Acesso em: 30 jul. 2024. , 2010APA
Pestana, C. R., Curti, C., & Leopoldino, A. M. (2010). SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. Edinburgh: Faculdade de Ciências Farmacêuticas de Ribeirão Preto, Universidade de São Paulo.NLM
Pestana CR, Curti C, Leopoldino AM. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. 2010 ;[citado 2024 jul. 30 ]Vancouver
Pestana CR, Curti C, Leopoldino AM. SET protein accumulation prevents oxidative stress-induced apoptosis/mitochondrial dysfunction with potential implication in tumorigenesis. Programme and Abstracts. 2010 ;[citado 2024 jul. 30 ]
