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Artigo de periodico
Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback (2018)
Souza, Paula Bargi de ; Silva, Francemilson Goulart da ; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: TRIIODOTIRONINA, HORMÔNIOS TIREOIDIANOS, EXPRESSÃO GÊNICA, LINFÓCITOS T, RNA MENSAGEIRO, RIBOSSOMOS, PROTEÍNAS DA MEMBRANA
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ABNT
SOUZA, Paula Bargi de e SILVA, Francemilson Goulart da e NUNES, Maria Tereza. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback. Molecular and Cellular Endocrinology, v. 478, p. 45-52, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2018.07.006. Acesso em: 30 set. 2024.
APA
Souza, P. B. de, Silva, F. G. da, & Nunes, M. T. (2018). Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback. Molecular and Cellular Endocrinology, 478, 45-52. doi:10.1016/j.mce.2018.07.006
NLM
Souza PB de, Silva FG da, Nunes MT. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback [Internet]. Molecular and Cellular Endocrinology. 2018 ; 478 45-52.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2018.07.006
Vancouver
Souza PB de, Silva FG da, Nunes MT. Posttranscriptional actions of triiodothyronine on Tshb expression in TαT1 cells: new insights into molecular mechanisms of negative feedback [Internet]. Molecular and Cellular Endocrinology. 2018 ; 478 45-52.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2018.07.006
Artigo de periodico
NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice (2017)
Souza, Arnaldo H. de; Santos, Laila R. B.; Roma, Leticia P.; Bensellam, Mohammed; Carpinelli, Angelo Rafael ; Jonas, Jean-Christophe
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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SOUZA, Arnaldo H. de et al. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, v. 439, p. 354-362, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.09.022. Acesso em: 30 set. 2024.
APA
Souza, A. H. de, Santos, L. R. B., Roma, L. P., Bensellam, M., Carpinelli, A. R., & Jonas, J. -C. (2017). NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice. Molecular and Cellular Endocrinology, 439, 354-362. doi:10.1016/j.mce.2016.09.022
NLM
Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
Vancouver
Souza AH de, Santos LRB, Roma LP, Bensellam M, Carpinelli AR, Jonas J-C. NADPH oxidase-2 does not contribute to β-cell glucotoxicity in cultured pancreatic islets from C57BL/6J mice [Internet]. Molecular and Cellular Endocrinology. 2017 ; 439 354-362.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.09.022
Artigo de periodico
MicroRNAs in thyroid development, function and tumorigenesis (2017)
Fuziwara, Cesar Seigi; Kimura, Edna Teruko
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: HISTOLOGIA
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FUZIWARA, Cesar Seigi e KIMURA, Edna Teruko. MicroRNAs in thyroid development, function and tumorigenesis. Molecular and Cellular Endocrinology, v. 456, p. 44-50, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.12.017. Acesso em: 30 set. 2024.
APA
Fuziwara, C. S., & Kimura, E. T. (2017). MicroRNAs in thyroid development, function and tumorigenesis. Molecular and Cellular Endocrinology, 456, 44-50. doi:10.1016/j.mce.2016.12.017
NLM
Fuziwara CS, Kimura ET. MicroRNAs in thyroid development, function and tumorigenesis [Internet]. Molecular and Cellular Endocrinology. 2017 ; 456 44-50.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.12.017
Vancouver
Fuziwara CS, Kimura ET. MicroRNAs in thyroid development, function and tumorigenesis [Internet]. Molecular and Cellular Endocrinology. 2017 ; 456 44-50.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.12.017
Artigo de periodico
Role of microRNAs in endocrine cancer metastasis (2017)
Lima, Cilene Rebouças de ; Gomes, Cibele Crastequini; Santos, Marinilce Fagundes dos
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: HISTOLOGIA
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ABNT
LIMA, Cilene Rebouças de e GOMES, Cibele Crastequini e SANTOS, Marinilce Fagundes dos. Role of microRNAs in endocrine cancer metastasis. Molecular and Cellular Endocrinology, v. 456, p. 62-75, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.03.015. Acesso em: 30 set. 2024.
APA
Lima, C. R. de, Gomes, C. C., & Santos, M. F. dos. (2017). Role of microRNAs in endocrine cancer metastasis. Molecular and Cellular Endocrinology, 456, 62-75. doi:10.1016/j.mce.2017.03.015
NLM
Lima CR de, Gomes CC, Santos MF dos. Role of microRNAs in endocrine cancer metastasis [Internet]. Molecular and Cellular Endocrinology. 2017 ; 456 62-75.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.03.015
Vancouver
Lima CR de, Gomes CC, Santos MF dos. Role of microRNAs in endocrine cancer metastasis [Internet]. Molecular and Cellular Endocrinology. 2017 ; 456 62-75.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.03.015
Artigo de periodico
Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products (2017)
Fabre, Nelly T.; Thieme, Karina ; Silva, Karolline S.; Catanozi, Sergio; Cavaleiro, Ana Mercedes; Pinto jr, Danilo A. C.; Maristela M. Okamoto,; Morais, Mychel Raony P. T.; Falquetto, Bárbara ; Zorn, Telma M.; Machado, Ubiratan Fabres ; Passarelli, Marisa; Correa-Giannella, Maria Lúcia
Source: Molecular and Cellular Endocrinology. Unidades: FM, ICB
Subjects: FÍGADO, DOENÇA CRÔNICA, INSULINA (RESISTÊNCIA), RATOS
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ABNT
FABRE, Nelly T. et al. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products. Molecular and Cellular Endocrinology, v. 447, p. 116-124, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.02.035. Acesso em: 30 set. 2024.
APA
Fabre, N. T., Thieme, K., Silva, K. S., Catanozi, S., Cavaleiro, A. M., Pinto jr, D. A. C., et al. (2017). Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products. Molecular and Cellular Endocrinology, 447, 116-124. doi:10.1016/j.mce.2017.02.035
NLM
Fabre NT, Thieme K, Silva KS, Catanozi S, Cavaleiro AM, Pinto jr DAC, Maristela M. Okamoto, Morais MRPT, Falquetto B, Zorn TM, Machado UF, Passarelli M, Correa-Giannella ML. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products [Internet]. Molecular and Cellular Endocrinology. 2017 ; 447 116-124.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.02.035
Vancouver
Fabre NT, Thieme K, Silva KS, Catanozi S, Cavaleiro AM, Pinto jr DAC, Maristela M. Okamoto, Morais MRPT, Falquetto B, Zorn TM, Machado UF, Passarelli M, Correa-Giannella ML. Hormetic modulation of hepatic insulin sensitivity by advanced glycation end products [Internet]. Molecular and Cellular Endocrinology. 2017 ; 447 116-124.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.02.035
Artigo de periodico
STAT5 signaling in kisspeptin cells regulates the timing of puberty (2017)
Silveira, Marina Augusto; Furigo, Isadora C.; Zampieri, Thais T.; Bohlen, Tabata M.; Paula, Daniella G. de; Franci, Celso Rodrigues; Donato Junior, José ; Frazão, Renata
Source: Molecular and Cellular Endocrinology. Unidades: FMRP, ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
SILVEIRA, Marina Augusto et al. STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, v. 448, p. 55-65, 2017Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2017.03.024. Acesso em: 30 set. 2024.
APA
Silveira, M. A., Furigo, I. C., Zampieri, T. T., Bohlen, T. M., Paula, D. G. de, Franci, C. R., et al. (2017). STAT5 signaling in kisspeptin cells regulates the timing of puberty. Molecular and Cellular Endocrinology, 448, 55-65. doi:10.1016/j.mce.2017.03.024
NLM
Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
Vancouver
Silveira MA, Furigo IC, Zampieri TT, Bohlen TM, Paula DG de, Franci CR, Donato Junior J, Frazão R. STAT5 signaling in kisspeptin cells regulates the timing of puberty [Internet]. Molecular and Cellular Endocrinology. 2017 ; 448 55-65.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2017.03.024
Artigo de periodico
Brain STAT5 signaling and behavioral control (2016)
Furigo, Isadora C.; Ramos-Lobo, Angela M.; Frazão, Renata ; Donato Junior, José
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
FURIGO, Isadora C. et al. Brain STAT5 signaling and behavioral control. Molecular and Cellular Endocrinology, v. 438, p. 70-76, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.04.019. Acesso em: 30 set. 2024.
APA
Furigo, I. C., Ramos-Lobo, A. M., Frazão, R., & Donato Junior, J. (2016). Brain STAT5 signaling and behavioral control. Molecular and Cellular Endocrinology, 438, 70-76. doi:10.1016/j.mce.2016.04.019
NLM
Furigo IC, Ramos-Lobo AM, Frazão R, Donato Junior J. Brain STAT5 signaling and behavioral control [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 70-76.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.04.019
Vancouver
Furigo IC, Ramos-Lobo AM, Frazão R, Donato Junior J. Brain STAT5 signaling and behavioral control [Internet]. Molecular and Cellular Endocrinology. 2016 ; 438 70-76.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.04.019
Artigo de periodico
Fatness rather than leptin sensitivity determines the timing of puberty in female mice (2016)
Bohlen, Tabata M.; Silveira, Marina A.; Zampieri, Thaís T.; Frazão, Renata ; Donato Junior, José
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: ANATOMIA, FISIOLOGIA
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ABNT
BOHLEN, Tabata M. et al. Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, v. 423, p. 11-21, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.12.022. Acesso em: 30 set. 2024.
APA
Bohlen, T. M., Silveira, M. A., Zampieri, T. T., Frazão, R., & Donato Junior, J. (2016). Fatness rather than leptin sensitivity determines the timing of puberty in female mice. Molecular and Cellular Endocrinology, 423, 11-21. doi:10.1016/j.mce.2015.12.022
NLM
Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022
Vancouver
Bohlen TM, Silveira MA, Zampieri TT, Frazão R, Donato Junior J. Fatness rather than leptin sensitivity determines the timing of puberty in female mice [Internet]. Molecular and Cellular Endocrinology. 2016 ; 423 11-21.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2015.12.022
Artigo de periodico
Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway (2016)
Serrano-Nascimento, Caroline; Nicola, Juan Pablo; Teixeira, Silvania da Silva; Poyares, Leonice Lourenço; Lellis-Santos, Camilo; Bordin, Silvana ; Masini-Repiso, Ana Maria; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
SERRANO-NASCIMENTO, Caroline et al. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, v. 426, p. 73-90, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2016.02.006. Acesso em: 30 set. 2024.
APA
Serrano-Nascimento, C., Nicola, J. P., Teixeira, S. da S., Poyares, L. L., Lellis-Santos, C., Bordin, S., et al. (2016). Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway. Molecular and Cellular Endocrinology, 426, 73-90. doi:10.1016/j.mce.2016.02.006
NLM
Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
Vancouver
Serrano-Nascimento C, Nicola JP, Teixeira S da S, Poyares LL, Lellis-Santos C, Bordin S, Masini-Repiso AM, Nunes MT. Excess iodide downregulates Na(+)/I(-) symporter gene transcription through activation of PI3K/Akt pathway [Internet]. Molecular and Cellular Endocrinology. 2016 ; 426 73-90.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2016.02.006
Artigo de periodico
T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion (2015)
Souza, Paula Bargi de; Romano, Renata Marino; Silva, Francemilson Goulart da ; Brunetto, Erika Lia; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Subjects: FISIOLOGIA, GLICOPROTEINAS, GLÂNDULA PITUITÁRIA, RATOS, SECREÇÃO ANIMAL
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ABNT
SOUZA, Paula Bargi de et al. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, v. 409, p. 73-81, 2015Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2015.04.002. Acesso em: 30 set. 2024.
APA
Souza, P. B. de, Romano, R. M., Silva, F. G. da, Brunetto, E. L., & Nunes, M. T. (2015). T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion. Molecular and Cellular Endocrinology, 409, 73-81. doi:10.1016/j.mce.2015.04.002
NLM
Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
Vancouver
Souza PB de, Romano RM, Silva FG da, Brunetto EL, Nunes MT. T(3) rapidly regulates several steps of alpha subunit glycoprotein (CGA) synthesis and secretion in the pituitary of male rats: Potential repercussions on TSH, FSH and LH secretion [Internet]. Molecular and Cellular Endocrinology. 2015 ; 409 73-81.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2015.04.002
Artigo de periodico
POD-1 binding to the E-box sequence inhibits SF-1 and StAR expression in human adrenocortical tumor cells (2013)
França, Monica Malheiros; Ferraz-de-Souza, Bruno; Santos, Mariza Gerdulo; Lerario, Antonio Marcondes; Latronico, Ana Claudia; Kuick, Rork D.; Hammer, Gary D.; Lotfi, Claudimara Ferini Pacicco
Source: Molecular and Cellular Endocrinology. Unidades: FM, ICB
Subjects: NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS, BIOLOGIA CELULAR, EXPRESSÃO GÊNICA, BIOLOGIA MOLECULAR, CARCINOMA (IMUNOLOGIA), REAÇÃO EM CADEIA POR POLIMERASE
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ABNT
FRANÇA, Monica Malheiros et al. POD-1 binding to the E-box sequence inhibits SF-1 and StAR expression in human adrenocortical tumor cells. Molecular and Cellular Endocrinology, v. 371, n. 1-2, p. 140-147, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2012.12.029. Acesso em: 30 set. 2024.
APA
França, M. M., Ferraz-de-Souza, B., Santos, M. G., Lerario, A. M., Latronico, A. C., Kuick, R. D., et al. (2013). POD-1 binding to the E-box sequence inhibits SF-1 and StAR expression in human adrenocortical tumor cells. Molecular and Cellular Endocrinology, 371( 1-2), 140-147. doi:10.1016/j.mce.2012.12.029
NLM
França MM, Ferraz-de-Souza B, Santos MG, Lerario AM, Latronico AC, Kuick RD, Hammer GD, Lotfi CFP. POD-1 binding to the E-box sequence inhibits SF-1 and StAR expression in human adrenocortical tumor cells [Internet]. Molecular and Cellular Endocrinology. 2013 ; 371( 1-2): 140-147.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2012.12.029
Vancouver
França MM, Ferraz-de-Souza B, Santos MG, Lerario AM, Latronico AC, Kuick RD, Hammer GD, Lotfi CFP. POD-1 binding to the E-box sequence inhibits SF-1 and StAR expression in human adrenocortical tumor cells [Internet]. Molecular and Cellular Endocrinology. 2013 ; 371( 1-2): 140-147.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2012.12.029
Artigo de periodico
N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex (2013)
Mendonça, Pedro Omori Ribeiro de; Liria, Cleber Wanderlei; Machini, Maria Teresa ; Lotfi, Claudimara Ferini Pacicco
Source: Molecular and Cellular Endocrinology. Unidades: IQ, ICB
Subjects: CÓRTEX ADRENAL, HORMÔNIO ADRENOCORTICOTRÓFICO
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ABNT
MENDONÇA, Pedro Omori Ribeiro de et al. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, v. 37, n. 1-2, p. 166-173, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2012.11.017. Acesso em: 30 set. 2024.
APA
Mendonça, P. O. R. de, Liria, C. W., Machini, M. T., & Lotfi, C. F. P. (2013). N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex. Molecular and Cellular Endocrinology, 37( 1-2), 166-173. doi:10.1016/j.mce.2012.11.017
NLM
Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
Vancouver
Mendonça POR de, Liria CW, Machini MT, Lotfi CFP. N-`POMC IND. 1-28´ increases cyclin D expression and inhibits `P27 POT. kip1´ in the adrenal cortex [Internet]. Molecular and Cellular Endocrinology. 2013 ; 37( 1-2): 166-173.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2012.11.017
Artigo de periodico
Proteins differentially expressed in human beta-cells-enriched pancreatic islet cultures and human insulinomas (2013)
Terra, Letícia F.; Teixeira, Priscila C.; Wailemann, Rosangela A. M.; Zelanis, André; Palmisano, Giuseppe ; Cunha-Neto, Edecio; Kalil, Jorge; Larsen, Martin R.; Labriola, Leticia ; Sogayar, Mari Cleide
Source: Molecular and Cellular Endocrinology. Unidades: ICB, FM, IQ
Subjects: PANCREATITE, ESPECTROMETRIA DE MASSAS, BIOLOGIA MOLECULAR, FENÓTIPOS, PARASITOLOGIA
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ABNT
TERRA, Letícia F. et al. Proteins differentially expressed in human beta-cells-enriched pancreatic islet cultures and human insulinomas. Molecular and Cellular Endocrinology, v. 381, n. 1-2, p. 16-25, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.07.004. Acesso em: 30 set. 2024.
APA
Terra, L. F., Teixeira, P. C., Wailemann, R. A. M., Zelanis, A., Palmisano, G., Cunha-Neto, E., et al. (2013). Proteins differentially expressed in human beta-cells-enriched pancreatic islet cultures and human insulinomas. Molecular and Cellular Endocrinology, 381( 1-2), 16-25. doi:10.1016/j.mce.2013.07.004
NLM
Terra LF, Teixeira PC, Wailemann RAM, Zelanis A, Palmisano G, Cunha-Neto E, Kalil J, Larsen MR, Labriola L, Sogayar MC. Proteins differentially expressed in human beta-cells-enriched pancreatic islet cultures and human insulinomas [Internet]. Molecular and Cellular Endocrinology. 2013 ; 381( 1-2): 16-25.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.07.004
Vancouver
Terra LF, Teixeira PC, Wailemann RAM, Zelanis A, Palmisano G, Cunha-Neto E, Kalil J, Larsen MR, Labriola L, Sogayar MC. Proteins differentially expressed in human beta-cells-enriched pancreatic islet cultures and human insulinomas [Internet]. Molecular and Cellular Endocrinology. 2013 ; 381( 1-2): 16-25.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.07.004
Artigo de periodico
AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth (2013)
Takano, Ana Paula Cremasco ; Diniz, Gabriela Placoná ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
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ABNT
TAKANO, Ana Paula Cremasco e DINIZ, Gabriela Placoná e BARRETO-CHAVES, Maria Luiza Morais. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, v. 376, n. 1-2, p. 43-50, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.05.024. Acesso em: 30 set. 2024.
APA
Takano, A. P. C., Diniz, G. P., & Barreto-Chaves, M. L. M. (2013). AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth. Molecular and Cellular Endocrinology, 376( 1-2), 43-50. doi:10.1016/j.mce.2013.05.024
NLM
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Vancouver
Takano APC, Diniz GP, Barreto-Chaves MLM. AMPK signaling pathway is rapidly activated by T3 and regulates the cardiomyocyte growth [Internet]. Molecular and Cellular Endocrinology. 2013 ; 376( 1-2): 43-50.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.05.024
Artigo de periodico
miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation (2013)
Diniz, Gabriela Placoná ; Takano, Ana Paula Cremasco ; Barreto-Chaves, Maria Luiza Morais
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
DINIZ, Gabriela Placoná e TAKANO, Ana Paula Cremasco e BARRETO-CHAVES, Maria Luiza Morais. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, v. 374, n. 1-2, p. 117-124, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2013.04.010. Acesso em: 30 set. 2024.
APA
Diniz, G. P., Takano, A. P. C., & Barreto-Chaves, M. L. M. (2013). miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation. Molecular and Cellular Endocrinology, 374( 1-2), 117-124. doi:10.1016/j.mce.2013.04.010
NLM
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Vancouver
Diniz GP, Takano APC, Barreto-Chaves MLM. miRNA-208a and miRNA-208b are triggered in thyroid hormone-induced cardiac hypertrophy – Role of type 1 Angiotensin II receptor (AT1R) on miRNA-208a/α-MHC modulation [Internet]. Molecular and Cellular Endocrinology. 2013 ; 374( 1-2): 117-124.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2013.04.010
Artigo de periodico
New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells (2012)
Serrano-Nascimento, Caroline; Calil-Silveira, Jamile; Silva, Francemilson Goulart da ; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SERRANO-NASCIMENTO, Caroline et al. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells. Molecular and Cellular Endocrinology, v. 349, n. 2, p. 154-161, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2011.09.036. Acesso em: 30 set. 2024.
APA
Serrano-Nascimento, C., Calil-Silveira, J., Silva, F. G. da, & Nunes, M. T. (2012). New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells. Molecular and Cellular Endocrinology, 349( 2), 154-161. doi:10.1016/j.mce.2011.09.036
NLM
Serrano-Nascimento C, Calil-Silveira J, Silva FG da, Nunes MT. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells [Internet]. Molecular and Cellular Endocrinology. 2012 ; 349( 2): 154-161.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2011.09.036
Vancouver
Serrano-Nascimento C, Calil-Silveira J, Silva FG da, Nunes MT. New insights about the posttranscriptional mechanisms triggered by iodide excess on sodium/iodide symporter (NIS) expression in PCCl3 cells [Internet]. Molecular and Cellular Endocrinology. 2012 ; 349( 2): 154-161.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2011.09.036
Artigo de periodico
Insulin-like growth factor system on adrenocortical tumorigenesis (2012)
Ribeiro, Tamaya Castro; Latronico, Ana Claudia
Source: Molecular and Cellular Endocrinology. Unidade: FM
Subjects: FENÓTIPOS, BIOLOGIA MOLECULAR, FATORES DE CRESCIMENTO, PROGNÓSTICO, CARCINOMA, BIOMARCADORES, NEOPLASIAS DAS GLÂNDULAS SUPRARRENAIS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
RIBEIRO, Tamaya Castro e LATRONICO, Ana Claudia. Insulin-like growth factor system on adrenocortical tumorigenesis. Molecular and Cellular Endocrinology, v. 351, n. 1, p. 96-100, 2012Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2011.09.042. Acesso em: 30 set. 2024.
APA
Ribeiro, T. C., & Latronico, A. C. (2012). Insulin-like growth factor system on adrenocortical tumorigenesis. Molecular and Cellular Endocrinology, 351( 1), 96-100. doi:10.1016/j.mce.2011.09.042
NLM
Ribeiro TC, Latronico AC. Insulin-like growth factor system on adrenocortical tumorigenesis [Internet]. Molecular and Cellular Endocrinology. 2012 ; 351( 1): 96-100.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2011.09.042
Vancouver
Ribeiro TC, Latronico AC. Insulin-like growth factor system on adrenocortical tumorigenesis [Internet]. Molecular and Cellular Endocrinology. 2012 ; 351( 1): 96-100.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2011.09.042
Artigo de periodico
the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E (2011)
Mendonça, Pedro Omori Ribeiro de; Lotfi, Claudimara Ferini Pacicco
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
MENDONÇA, Pedro Omori Ribeiro de e LOTFI, Claudimara Ferini Pacicco. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E. Molecular and Cellular Endocrinology, v. 336, n. 1-2, p. 156-161, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.012. Acesso em: 30 set. 2024.
APA
Mendonça, P. O. R. de, & Lotfi, C. F. P. (2011). the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E. Molecular and Cellular Endocrinology, 336( 1-2), 156-161. doi:10.1016/j.mce.2010.12.012
NLM
Mendonça POR de, Lotfi CFP. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E [Internet]. Molecular and Cellular Endocrinology. 2011 ; 336( 1-2): 156-161.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.12.012
Vancouver
Mendonça POR de, Lotfi CFP. the proliferative effect of synthetic N-POMC 1-28 peptides in rat adrenal cortex: a possible role for cyclin E [Internet]. Molecular and Cellular Endocrinology. 2011 ; 336( 1-2): 156-161.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.12.012
Artigo de periodico
T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats (2011)
Silva, Francemilson Goulart da ; Souza, Paula Bargi de; Nunes, Maria Tereza
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SILVA, Francemilson Goulart da e SOUZA, Paula Bargi de e NUNES, Maria Tereza. T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats. Molecular and Cellular Endocrinology, v. 332, n. 1-2, p. 277-282, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.11.005. Acesso em: 30 set. 2024.
APA
Silva, F. G. da, Souza, P. B. de, & Nunes, M. T. (2011). T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats. Molecular and Cellular Endocrinology, 332( 1-2), 277-282. doi:10.1016/j.mce.2010.11.005
NLM
Silva FG da, Souza PB de, Nunes MT. T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats [Internet]. Molecular and Cellular Endocrinology. 2011 ; 332( 1-2): 277-282.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.11.005
Vancouver
Silva FG da, Souza PB de, Nunes MT. T3 rapidly modulates TSHβ mRNA stability and translational rate in the pituitary of hypothyroid rats [Internet]. Molecular and Cellular Endocrinology. 2011 ; 332( 1-2): 277-282.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.11.005
Artigo de periodico
Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system (2011)
Araujo, A. S. R.; Diniz, Gabriela Placoná ; Seibel, F. E. R.; Branchini, G.; Ribeiro, M. F. M.; Brum, I. S.; Khaper, N.; Barreto-Chaves, Maria Luiza Morais ; Belló-Klein, A.
Source: Molecular and Cellular Endocrinology. Unidade: ICB
Assunto: ANATOMIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
ARAUJO, A. S. R. et al. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, v. 333, n. 1, p. 78-84, 2011Tradução . . Disponível em: https://doi.org/10.1016/j.mce.2010.12.018. Acesso em: 30 set. 2024.
APA
Araujo, A. S. R., Diniz, G. P., Seibel, F. E. R., Branchini, G., Ribeiro, M. F. M., Brum, I. S., et al. (2011). Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system. Molecular and Cellular Endocrinology, 333( 1), 78-84. doi:10.1016/j.mce.2010.12.018
NLM
Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018
Vancouver
Araujo ASR, Diniz GP, Seibel FER, Branchini G, Ribeiro MFM, Brum IS, Khaper N, Barreto-Chaves MLM, Belló-Klein A. Reactive oxygen and nitrogen species balance in the determination of thyroid hormones-induced cardiac hypertrophy mediated by renin–angiotensin system [Internet]. Molecular and Cellular Endocrinology. 2011 ; 333( 1): 78-84.[citado 2024 set. 30 ] Available from: https://doi.org/10.1016/j.mce.2010.12.018

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