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Artigo de periodico
Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation (2014)
Gomes, Patrícia R.; Graciano, Maria Fernanda; Pantaleão, Lucas Carminatti; Rennó, André L.; Rodrigues, Sandra Campos; Velloso, Lício Augusto; Latorraca, Márcia Q.; Carpinelli, Angelo Rafael ; Anhê, Gabriel F.; Bordin, Silvana
Source: American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. Unidade: ICB
Subjects: FISIOLOGIA, GLICOSE, ESTERÓIDES
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ABNT
GOMES, Patrícia R. et al. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, v. 306, n. 1, p. E109-E120, 2014Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00364.2013. Acesso em: 01 nov. 2024.
APA
Gomes, P. R., Graciano, M. F., Pantaleão, L. C., Rennó, A. L., Rodrigues, S. C., Velloso, L. A., et al. (2014). Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology, 306( 1), E109-E120. doi:10.1152/ajpendo.00364.2013
NLM
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Vancouver
Gomes PR, Graciano MF, Pantaleão LC, Rennó AL, Rodrigues SC, Velloso LA, Latorraca MQ, Carpinelli AR, Anhê GF, Bordin S. Long-term disruption of maternal glucose homeostasis induced by prenatal glucocorticoid treatment correlates with miR-29 upregulation [Internet]. American Journal of Physiology. Endocrinology, Metabolism and Gastrointestinal Physiology. 2014 ; 306( 1): E109-E120.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpendo.00364.2013
Artigo de periodico
Staphylococcus aureus enterotoxins A and B inhibit human and mice eosinophil chemotaxis and adhesion in vitro (2014)
Squebola-Cola, Dalize Maria; De Mello, Glaucia C.; Anhê, Gabriel F.; Condino-Neto, Antonio; De Souza, Ivani A.; Antunes, Edson
Source: International Immunopharmacology. Unidade: ICB
Subjects: IMUNOLOGIA, STAPHYLOCOCCUS, ENTEROTOXINAS, QUIMIOTAXIA, CAMUNDONGOS
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ABNT
SQUEBOLA-COLA, Dalize Maria et al. Staphylococcus aureus enterotoxins A and B inhibit human and mice eosinophil chemotaxis and adhesion in vitro. International Immunopharmacology, v. 23, n. 2, p. 664-667, 2014Tradução . . Disponível em: https://doi.org/10.1016/j.intimp.2014.10.020. Acesso em: 01 nov. 2024.
APA
Squebola-Cola, D. M., De Mello, G. C., Anhê, G. F., Condino-Neto, A., De Souza, I. A., & Antunes, E. (2014). Staphylococcus aureus enterotoxins A and B inhibit human and mice eosinophil chemotaxis and adhesion in vitro. International Immunopharmacology, 23( 2), 664-667. doi:10.1016/j.intimp.2014.10.020
NLM
Squebola-Cola DM, De Mello GC, Anhê GF, Condino-Neto A, De Souza IA, Antunes E. Staphylococcus aureus enterotoxins A and B inhibit human and mice eosinophil chemotaxis and adhesion in vitro [Internet]. International Immunopharmacology. 2014 ; 23( 2): 664-667.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.intimp.2014.10.020
Vancouver
Squebola-Cola DM, De Mello GC, Anhê GF, Condino-Neto A, De Souza IA, Antunes E. Staphylococcus aureus enterotoxins A and B inhibit human and mice eosinophil chemotaxis and adhesion in vitro [Internet]. International Immunopharmacology. 2014 ; 23( 2): 664-667.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.intimp.2014.10.020
Artigo de periodico
Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats (2013)
Faria, Juliana A.; Kinote, Andrezza; Ignacio-Souza, Letícia M.; Araújo, Thiago M. de; Razolli, Daniela S.; Doneda, Diego L.; Paschoal, Lívia B.; Lellis-Santos, Camilo; Bertolini, Gisele L.; Velloso, Lício A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: American Journal of Physiology, Endocrinology and Methabolism. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FARIA, Juliana A. et al. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, v. 305, n. 2, p. E230-E242, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajpendo.00094.2013. Acesso em: 01 nov. 2024.
APA
Faria, J. A., Kinote, A., Ignacio-Souza, L. M., Araújo, T. M. de, Razolli, D. S., Doneda, D. L., et al. (2013). Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats. American Journal of Physiology, Endocrinology and Methabolism, 305( 2), E230-E242. doi:10.1152/ajpendo.00094.2013
NLM
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Vancouver
Faria JA, Kinote A, Ignacio-Souza LM, Araújo TM de, Razolli DS, Doneda DL, Paschoal LB, Lellis-Santos C, Bertolini GL, Velloso LA, Bordin S, Anhê GF. Melatonin acts through MT1/MT2 receptors to activate hypothalamic Akt and suppress hepatic gluconeogenesis in rats [Internet]. American Journal of Physiology, Endocrinology and Methabolism. 2013 ; 305( 2): E230-E242.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajpendo.00094.2013
Artigo de periodico
Airway exposure to staphylococcal enterotoxin A potentiates allergen-induced bone marrow eosinophilia and trafficking to peripheral blood and airways (2013)
Squebola-Cola, Dalize M.; Mello, Glaucia C.; Pissinatti, Lorenzo; Schenka, André A.; Anhê, Gabriel F.; DeSouza, Ivani A.; Condino-Neto, Antonio; Antunes, Edson
Source: American Journal of Physiology: Lung Cellular and Molecular Physiology. Unidade: ICB
Assunto: IMUNOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
SQUEBOLA-COLA, Dalize M. et al. Airway exposure to staphylococcal enterotoxin A potentiates allergen-induced bone marrow eosinophilia and trafficking to peripheral blood and airways. American Journal of Physiology: Lung Cellular and Molecular Physiology, v. 304, n. 10, p. L639-L645, 2013Tradução . . Disponível em: https://doi.org/10.1152/ajplung.00025.2013. Acesso em: 01 nov. 2024.
APA
Squebola-Cola, D. M., Mello, G. C., Pissinatti, L., Schenka, A. A., Anhê, G. F., DeSouza, I. A., et al. (2013). Airway exposure to staphylococcal enterotoxin A potentiates allergen-induced bone marrow eosinophilia and trafficking to peripheral blood and airways. American Journal of Physiology: Lung Cellular and Molecular Physiology, 304( 10), L639-L645. doi:10.1152/ajplung.00025.2013
NLM
Squebola-Cola DM, Mello GC, Pissinatti L, Schenka AA, Anhê GF, DeSouza IA, Condino-Neto A, Antunes E. Airway exposure to staphylococcal enterotoxin A potentiates allergen-induced bone marrow eosinophilia and trafficking to peripheral blood and airways [Internet]. American Journal of Physiology: Lung Cellular and Molecular Physiology. 2013 ; 304( 10): L639-L645.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajplung.00025.2013
Vancouver
Squebola-Cola DM, Mello GC, Pissinatti L, Schenka AA, Anhê GF, DeSouza IA, Condino-Neto A, Antunes E. Airway exposure to staphylococcal enterotoxin A potentiates allergen-induced bone marrow eosinophilia and trafficking to peripheral blood and airways [Internet]. American Journal of Physiology: Lung Cellular and Molecular Physiology. 2013 ; 304( 10): L639-L645.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1152/ajplung.00025.2013
Artigo de periodico
Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats (2013)
Gazzoto Filho, Ademir; Kinote, Andrezza; Pereira, Daniel J,; Rennó, André; Santos, Rodrigo C. dos; Ferreira-Melo, Silvia E.; Velloso, Licio A.; Bordin, Silvana ; Anhê, Gabriel F.; Moreno Junior, Heitor
Source: European Journal Pharmacology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
GAZZOTO FILHO, Ademir et al. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats. European Journal Pharmacology, v. 700, n. 1-3, p. 201-209, 2013Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2012.11.059. Acesso em: 01 nov. 2024.
APA
Gazzoto Filho, A., Kinote, A., Pereira, D. J., Rennó, A., Santos, R. C. dos, Ferreira-Melo, S. E., et al. (2013). Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats. European Journal Pharmacology, 700( 1-3), 201-209. doi:10.1016/j.ejphar.2012.11.059
NLM
Gazzoto Filho A, Kinote A, Pereira DJ, Rennó A, Santos RC dos, Ferreira-Melo SE, Velloso LA, Bordin S, Anhê GF, Moreno Junior H. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats [Internet]. European Journal Pharmacology. 2013 ; 700( 1-3): 201-209.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.ejphar.2012.11.059
Vancouver
Gazzoto Filho A, Kinote A, Pereira DJ, Rennó A, Santos RC dos, Ferreira-Melo SE, Velloso LA, Bordin S, Anhê GF, Moreno Junior H. Infliximab prevents increased systolic blood pressure and upregulates the AKT/eNOS pathway in the aorta of spontaneously hypertensive rats [Internet]. European Journal Pharmacology. 2013 ; 700( 1-3): 201-209.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1016/j.ejphar.2012.11.059
Artigo de periodico
The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion (2012)
Lellis-Santos, Camilo; Sakamoto, Luciano H.; Bromati, Carla Rodrigues; Nogueira, Tatiane Cristina de Araújo; Leite, Adriana R.; Yamanaka, Tatiana S.; Kinote, Andrezza; Anhê, Gabriel F.; Bordin, Silvana
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
LELLIS-SANTOS, Camilo et al. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, v. 153, n. 8, p. 3668-3678, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1135. Acesso em: 01 nov. 2024.
APA
Lellis-Santos, C., Sakamoto, L. H., Bromati, C. R., Nogueira, T. C. de A., Leite, A. R., Yamanaka, T. S., et al. (2012). The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion. Endocrinology, 153( 8), 3668-3678. doi:10.1210/en.2012-1135
NLM
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1210/en.2012-1135
Vancouver
Lellis-Santos C, Sakamoto LH, Bromati CR, Nogueira TC de A, Leite AR, Yamanaka TS, Kinote A, Anhê GF, Bordin S. The regulation of Rasd1 expression by glucocorticoids and prolactin controls peripartum maternal insulin secretion [Internet]. Endocrinology. 2012 ; 153( 8): 3668-3678.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1210/en.2012-1135
Artigo de periodico
Maternal melatonin programs the daily pattern of energy metabolism in adult offspring (2012)
Ferreira, Danilo S.; Amaral, Fernanda Gaspar do; Mesquita, Caroline C.; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Turati, Ariane de Oliveira; Santos, Laila Romagueira Bichara; Sollon. Carolina S.; Gomes, Patricia Rodrigues Lourenço; Faria, Juliana A.; Cipolla-Neto, José; Bordin, Silvana ; Anhê, Gabriel F.
Source: PLOS ONE. Unidade: ICB
Assunto: FISIOLOGIA
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ABNT
FERREIRA, Danilo S. et al. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring. PLOS ONE, v. 7, n. 6, p. 1-10, 2012Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0038795. Acesso em: 01 nov. 2024.
APA
Ferreira, D. S., Amaral, F. G. do, Mesquita, C. C., Barbosa, A. P. L., Lellis-Santos, C., Turati, A. de O., et al. (2012). Maternal melatonin programs the daily pattern of energy metabolism in adult offspring. PLOS ONE, 7( 6), 1-10. doi:10.1371/journal.pone.0038795
NLM
Ferreira DS, Amaral FG do, Mesquita CC, Barbosa APL, Lellis-Santos C, Turati A de O, Santos LRB, Sollon. Carolina S., Gomes PRL, Faria JA, Cipolla-Neto J, Bordin S, Anhê GF. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring [Internet]. PLOS ONE. 2012 ; 7( 6): 1-10.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0038795
Vancouver
Ferreira DS, Amaral FG do, Mesquita CC, Barbosa APL, Lellis-Santos C, Turati A de O, Santos LRB, Sollon. Carolina S., Gomes PRL, Faria JA, Cipolla-Neto J, Bordin S, Anhê GF. Maternal melatonin programs the daily pattern of energy metabolism in adult offspring [Internet]. PLOS ONE. 2012 ; 7( 6): 1-10.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1371/journal.pone.0038795
Artigo de periodico
Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels (2012)
Kinote, Andrezza; Faria, Juliana A.; Roman, Erika A.; Solon, Carina; Razolli, Daniela S.; Ignacio-Souza, Letícia M.; Sollon, Carolina S.; Nascimento, Lucas F.; Araújo, Thiago M. de; Barbosa, Ana Paula L.; Lellis-Santos, Camilo; Velloso, Licio A.; Bordin, Silvana ; Anhê, Gabriel F.
Source: Endocrinology. Unidade: ICB
Assunto: FISIOLOGIA
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
KINOTE, Andrezza et al. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, v. 153, n. 8, p. 3633-3645, 2012Tradução . . Disponível em: https://doi.org/10.1210/en.2012-1341. Acesso em: 01 nov. 2024.
APA
Kinote, A., Faria, J. A., Roman, E. A., Solon, C., Razolli, D. S., Ignacio-Souza, L. M., et al. (2012). Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels. Endocrinology, 153( 8), 3633-3645. doi:10.1210/en.2012-1341
NLM
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1210/en.2012-1341
Vancouver
Kinote A, Faria JA, Roman EA, Solon C, Razolli DS, Ignacio-Souza LM, Sollon CS, Nascimento LF, Araújo TM de, Barbosa APL, Lellis-Santos C, Velloso LA, Bordin S, Anhê GF. Fructose-induced hypothalamic AMPK activation stimulates hepatic PEPCK and gluconeogenesis due to Increased corticosterone levels [Internet]. Endocrinology. 2012 ; 153( 8): 3633-3645.[citado 2024 nov. 01 ] Available from: https://doi.org/10.1210/en.2012-1341

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