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Artigo de periodico
Estrogen Receptor 1 (ESR1) enhances Slc2a4/GLUT4 expression by a SP1 cooperative mechanism (2018)
Andrade, João Nilton Barreto; Fátima, Luciana Alves de; Campello, Raquel Saldanha; Guedes, José Augusto Cipriano; Freitas, Helayne Soares de; Okamoto, Maristela Mitiko; Machado, Ubiratan Fabres
Source: International Journal of Medical Sciences. Unidade: ICB
Subjects: ESTRÓGENOS, GLICOSE, GLICEMIA, INSULINA, TECIDO ADIPOSO, WESTERN BLOTTING
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ANDRADE, João Nilton Barreto et al. Estrogen Receptor 1 (ESR1) enhances Slc2a4/GLUT4 expression by a SP1 cooperative mechanism. International Journal of Medical Sciences, v. 15, n. 12, p. 1320-1328, 2018Tradução . . Disponível em: https://doi.org/10.7150/ijms.26774. Acesso em: 30 jun. 2024.
APA
Andrade, J. N. B., Fátima, L. A. de, Campello, R. S., Guedes, J. A. C., Freitas, H. S. de, Okamoto, M. M., & Machado, U. F. (2018). Estrogen Receptor 1 (ESR1) enhances Slc2a4/GLUT4 expression by a SP1 cooperative mechanism. International Journal of Medical Sciences, 15( 12), 1320-1328. doi:10.7150/ijms.26774
NLM
Andrade JNB, Fátima LA de, Campello RS, Guedes JAC, Freitas HS de, Okamoto MM, Machado UF. Estrogen Receptor 1 (ESR1) enhances Slc2a4/GLUT4 expression by a SP1 cooperative mechanism [Internet]. International Journal of Medical Sciences. 2018 ; 15( 12): 1320-1328.[citado 2024 jun. 30 ] Available from: https://doi.org/10.7150/ijms.26774
Vancouver
Andrade JNB, Fátima LA de, Campello RS, Guedes JAC, Freitas HS de, Okamoto MM, Machado UF. Estrogen Receptor 1 (ESR1) enhances Slc2a4/GLUT4 expression by a SP1 cooperative mechanism [Internet]. International Journal of Medical Sciences. 2018 ; 15( 12): 1320-1328.[citado 2024 jun. 30 ] Available from: https://doi.org/10.7150/ijms.26774
Artigo de periodico
High-fat diet affects gut nutrients transporters in hypo and hyperthyroid mice by PPAR-a independent mechanism (2018)
Losacco, Mariana Cerqueira; Almeida, Carolina Fernanda Theodora de; Hijo, Andressa Harumi Torelli ; Souza, Paula Bargi de ; Gama, Patrícia ; Nunes, Maria Tereza ; Silva, Francemilson Goulart da
Source: Life Sciences. Unidade: ICB
Subjects: HORMÔNIOS TIREOIDIANOS, HIPOTIREOIDISMO ANIMAL, INTESTINO DE ANIMAL, NUTRIENTES PARA ANIMAIS, CAMUNDONGOS, DIETA ANIMAL
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LOSACCO, Mariana Cerqueira et al. High-fat diet affects gut nutrients transporters in hypo and hyperthyroid mice by PPAR-a independent mechanism. Life Sciences, v. 202, p. 35-43, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.03.053. Acesso em: 30 jun. 2024.
APA
Losacco, M. C., Almeida, C. F. T. de, Hijo, A. H. T., Souza, P. B. de, Gama, P., Nunes, M. T., & Silva, F. G. da. (2018). High-fat diet affects gut nutrients transporters in hypo and hyperthyroid mice by PPAR-a independent mechanism. Life Sciences, 202, 35-43. doi:10.1016/j.lfs.2018.03.053
NLM
Losacco MC, Almeida CFT de, Hijo AHT, Souza PB de, Gama P, Nunes MT, Silva FG da. High-fat diet affects gut nutrients transporters in hypo and hyperthyroid mice by PPAR-a independent mechanism [Internet]. Life Sciences. 2018 ; 202 35-43.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.03.053
Vancouver
Losacco MC, Almeida CFT de, Hijo AHT, Souza PB de, Gama P, Nunes MT, Silva FG da. High-fat diet affects gut nutrients transporters in hypo and hyperthyroid mice by PPAR-a independent mechanism [Internet]. Life Sciences. 2018 ; 202 35-43.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.03.053
Artigo de periodico
Triiodothyronine differentially modulates the LH and FSH synthesis and secretion in male rats (2018)
Romano, Renata Marino ; Souza, Paula Bargi de ; Ract, Erika Lia Brunetto; Silva, Francemilson Goulart da ; Salgado, Renato de Mayrinck; Zorn, Telma Maria Tenorio; Nunes, Maria Tereza
Source: Endocrine. Unidade: ICB
Subjects: RATOS, HIPOTIREOIDISMO ANIMAL, RATOS, RIBOSSOMOS, HORMÔNIOS TIREOIDIANOS, GONADOTROFINAS
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ROMANO, Renata Marino et al. Triiodothyronine differentially modulates the LH and FSH synthesis and secretion in male rats. Endocrine, v. 59, n. 1, p. 191-202, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12020-017-1487-y. Acesso em: 30 jun. 2024.
APA
Romano, R. M., Souza, P. B. de, Ract, E. L. B., Silva, F. G. da, Salgado, R. de M., Zorn, T. M. T., & Nunes, M. T. (2018). Triiodothyronine differentially modulates the LH and FSH synthesis and secretion in male rats. Endocrine, 59( 1), 191-202. doi:10.1007/s12020-017-1487-y
NLM
Romano RM, Souza PB de, Ract ELB, Silva FG da, Salgado R de M, Zorn TMT, Nunes MT. Triiodothyronine differentially modulates the LH and FSH synthesis and secretion in male rats [Internet]. Endocrine. 2018 ; 59( 1): 191-202.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s12020-017-1487-y
Vancouver
Romano RM, Souza PB de, Ract ELB, Silva FG da, Salgado R de M, Zorn TMT, Nunes MT. Triiodothyronine differentially modulates the LH and FSH synthesis and secretion in male rats [Internet]. Endocrine. 2018 ; 59( 1): 191-202.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s12020-017-1487-y
Artigo de periodico
Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling (2018)
Vrechi, Talita Aparecida de Moraes; Crunfli, Fernanda; Costa, Andressa Pereira; Torrão, Andréa da Silva
Source: Neurotoxicity Research. Unidade: ICB
Subjects: CANNABIS, RETÍCULO ENDOPLASMÁTICO, IMMUNOBLOTTING, DEGENERAÇÃO NEURAL, INFLAMAÇÃO, MORTE CEREBRAL, NEURÔNIOS, SISTEMA NERVOSO CENTRAL
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VRECHI, Talita Aparecida de Moraes et al. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling. Neurotoxicity Research, v. 33, n. 4, p. 846-855, 2018Tradução . . Disponível em: https://doi.org/10.1007/s12640-017-9839-1. Acesso em: 30 jun. 2024.
APA
Vrechi, T. A. de M., Crunfli, F., Costa, A. P., & Torrão, A. da S. (2018). Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling. Neurotoxicity Research, 33( 4), 846-855. doi:10.1007/s12640-017-9839-1
NLM
Vrechi TA de M, Crunfli F, Costa AP, Torrão A da S. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling [Internet]. Neurotoxicity Research. 2018 ; 33( 4): 846-855.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s12640-017-9839-1
Vancouver
Vrechi TA de M, Crunfli F, Costa AP, Torrão A da S. Cannabinoid receptor type 1 agonist ACEA protects neurons from death and attenuates endoplasmic reticulum stress-related apoptotic pathway signaling [Internet]. Neurotoxicity Research. 2018 ; 33( 4): 846-855.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s12640-017-9839-1
Artigo de periodico
Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis (2018)
Gonçalves, Guilherme Lopes; Pessoa, Juliana Martins Costa; Thieme, Karina ; Lins, Bruna Bezerra; Souza, Maria Oliveira de
Source: Scientific reports. Unidade: ICB
Subjects: NEFROPATIAS, ALBUMINAS, RATOS, CÉLULAS EPITELIAIS, APOPTOSE, IMUNOFLUORESCÊNCIA EM ANIMAL, CITOMETRIA DE FLUXO, IMMUNOBLOTTING, PROTEÍNAS QUINASES, ATIVAÇÃO ENZIMÁTICA
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GONÇALVES, Guilherme Lopes et al. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-36933-9. Acesso em: 30 jun. 2024.
APA
Gonçalves, G. L., Pessoa, J. M. C., Thieme, K., Lins, B. B., & Souza, M. O. de. (2018). Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis. Scientific reports, 8. doi:10.1038/s41598-018-36933-9
NLM
Gonçalves GL, Pessoa JMC, Thieme K, Lins BB, Souza MO de. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 30 ] Available from: https://doi.org/10.1038/s41598-018-36933-9
Vancouver
Gonçalves GL, Pessoa JMC, Thieme K, Lins BB, Souza MO de. Intracellular albumin overload elicits endoplasmic reticulum stress and PKC-delta/p38 MAPK pathway activation to induce podocyte apoptosis [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 30 ] Available from: https://doi.org/10.1038/s41598-018-36933-9
Artigo de periodico
Dehydroepiandrosterone supplementation is not beneficial in the late postmenopausal period in diet-induced obese rats (2018)
Teixeira, Caio Jordão ; Ribeiro, Luciene Maria; Veras, Katherine Maria de Araújo ; Araujo, Layanne Cabral de Cunha ; Curi, Rui ; Carvalho, Carla Roberta de Oliveira
Source: Life Sciences. Unidade: ICB
Subjects: GLICOSE, INSULINA, ESTEROIDES, RATOS WISTAR, MENOPAUSA, MÚSCULO ESQUELÉTICO, ENVELHECIMENTO
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ABNT
TEIXEIRA, Caio Jordão et al. Dehydroepiandrosterone supplementation is not beneficial in the late postmenopausal period in diet-induced obese rats. Life Sciences, v. 202, p. 110-116, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.03.052. Acesso em: 30 jun. 2024.
APA
Teixeira, C. J., Ribeiro, L. M., Veras, K. M. de A., Araujo, L. C. de C., Curi, R., & Carvalho, C. R. de O. (2018). Dehydroepiandrosterone supplementation is not beneficial in the late postmenopausal period in diet-induced obese rats. Life Sciences, 202, 110-116. doi:10.1016/j.lfs.2018.03.052
NLM
Teixeira CJ, Ribeiro LM, Veras KM de A, Araujo LC de C, Curi R, Carvalho CR de O. Dehydroepiandrosterone supplementation is not beneficial in the late postmenopausal period in diet-induced obese rats [Internet]. Life Sciences. 2018 ; 202 110-116.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.03.052
Vancouver
Teixeira CJ, Ribeiro LM, Veras KM de A, Araujo LC de C, Curi R, Carvalho CR de O. Dehydroepiandrosterone supplementation is not beneficial in the late postmenopausal period in diet-induced obese rats [Internet]. Life Sciences. 2018 ; 202 110-116.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.03.052
Artigo de periodico
Palmitate-induced Slc2a4/GLUT4 downregulation in L6 muscle cells: evidence of inflammatory and endoplasmic reticulum stress involvement (2018)
Silva, Patrícia Ebersbach; Poletto, Ana Claudia ; Silva, Aline David; Seraphim, Patricia Monteiro ; Anhe, Gabriel Forato ; Passarelli, Marisa ; Furuya, Daniela Tomie ; Machado, Ubiratan Fabres
Source: Lipids in health and disease. Unidades: FM, ICB
Subjects: RATOS, ADIPOSIDADE, OBESIDADE, RESISTÊNCIA À INSULINA
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SILVA, Patrícia Ebersbach et al. Palmitate-induced Slc2a4/GLUT4 downregulation in L6 muscle cells: evidence of inflammatory and endoplasmic reticulum stress involvement. Lipids in health and disease, v. 17, 2018Tradução . . Disponível em: https://doi.org/10.1186/s12944-018-0714-8. Acesso em: 30 jun. 2024.
APA
Silva, P. E., Poletto, A. C., Silva, A. D., Seraphim, P. M., Anhe, G. F., Passarelli, M., et al. (2018). Palmitate-induced Slc2a4/GLUT4 downregulation in L6 muscle cells: evidence of inflammatory and endoplasmic reticulum stress involvement. Lipids in health and disease, 17. doi:10.1186/s12944-018-0714-8
NLM
Silva PE, Poletto AC, Silva AD, Seraphim PM, Anhe GF, Passarelli M, Furuya DT, Machado UF. Palmitate-induced Slc2a4/GLUT4 downregulation in L6 muscle cells: evidence of inflammatory and endoplasmic reticulum stress involvement [Internet]. Lipids in health and disease. 2018 ; 17[citado 2024 jun. 30 ] Available from: https://doi.org/10.1186/s12944-018-0714-8
Vancouver
Silva PE, Poletto AC, Silva AD, Seraphim PM, Anhe GF, Passarelli M, Furuya DT, Machado UF. Palmitate-induced Slc2a4/GLUT4 downregulation in L6 muscle cells: evidence of inflammatory and endoplasmic reticulum stress involvement [Internet]. Lipids in health and disease. 2018 ; 17[citado 2024 jun. 30 ] Available from: https://doi.org/10.1186/s12944-018-0714-8
Artigo de periodico
Metaplasticity in the visual cortex: crosstalk between visual experience and reactive oxygen species (2018)
Oliveira, José Francis de; Higa, Guilherme Shigueto Vilar; Dati, Lívia Mendonça Munhoz; Shieh, Ianê Carvalho; De Pasquale, Roberto
Source: Journal of Neuroscience. Unidade: ICB
Subjects: PLASTICIDADE NEURONAL, CÓRTEX VISUAL, SINAPSE, CAMUNDONGOS, OXIGÊNIO
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OLIVEIRA, José Francis de et al. Metaplasticity in the visual cortex: crosstalk between visual experience and reactive oxygen species. Journal of Neuroscience, v. 38, n. 25, p. 5649-5665, 2018Tradução . . Disponível em: https://doi.org/10.1523/JNEUROSCI.2617-17.2018. Acesso em: 30 jun. 2024.
APA
Oliveira, J. F. de, Higa, G. S. V., Dati, L. M. M., Shieh, I. C., & De Pasquale, R. (2018). Metaplasticity in the visual cortex: crosstalk between visual experience and reactive oxygen species. Journal of Neuroscience, 38( 25), 5649-5665. doi:10.1523/JNEUROSCI.2617-17.2018
NLM
Oliveira JF de, Higa GSV, Dati LMM, Shieh IC, De Pasquale R. Metaplasticity in the visual cortex: crosstalk between visual experience and reactive oxygen species [Internet]. Journal of Neuroscience. 2018 ; 38( 25): 5649-5665.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1523/JNEUROSCI.2617-17.2018
Vancouver
Oliveira JF de, Higa GSV, Dati LMM, Shieh IC, De Pasquale R. Metaplasticity in the visual cortex: crosstalk between visual experience and reactive oxygen species [Internet]. Journal of Neuroscience. 2018 ; 38( 25): 5649-5665.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1523/JNEUROSCI.2617-17.2018
Artigo de periodico
Exercise training abrogates age‐dependent loss of hypothalamic oxytocinergic circuitry and maintains high parasympathetic activity (2018)
Santos, Carla Rocha dos; Ruggeri, Adriana; Ceroni, Alexandre; Michelini, Lisete Compagno
Source: Journal of Neuroendocrinology. Unidade: ICB
Subjects: HIPOTÁLAMO, TRONCO CEREBRAL, OCITOCINA, ANGIOTENSINAS, NEUROANATOMIA, SISTEMA NERVOSO AUTÔNOMO, EXERCÍCIO FÍSICO
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SANTOS, Carla Rocha dos et al. Exercise training abrogates age‐dependent loss of hypothalamic oxytocinergic circuitry and maintains high parasympathetic activity. Journal of Neuroendocrinology, v. 30, n. 8, p. 13 , 2018Tradução . . Disponível em: https://doi.org/10.1111/jne.12601. Acesso em: 30 jun. 2024.
APA
Santos, C. R. dos, Ruggeri, A., Ceroni, A., & Michelini, L. C. (2018). Exercise training abrogates age‐dependent loss of hypothalamic oxytocinergic circuitry and maintains high parasympathetic activity. Journal of Neuroendocrinology, 30( 8), 13 . doi:10.1111/jne.12601
NLM
Santos CR dos, Ruggeri A, Ceroni A, Michelini LC. Exercise training abrogates age‐dependent loss of hypothalamic oxytocinergic circuitry and maintains high parasympathetic activity [Internet]. Journal of Neuroendocrinology. 2018 ;30( 8): 13 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.1111/jne.12601
Vancouver
Santos CR dos, Ruggeri A, Ceroni A, Michelini LC. Exercise training abrogates age‐dependent loss of hypothalamic oxytocinergic circuitry and maintains high parasympathetic activity [Internet]. Journal of Neuroendocrinology. 2018 ;30( 8): 13 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.1111/jne.12601
Artigo de periodico
Repercussions of hypo and hyperthyroidism on the heart circadian clock (2018)
Garcia, Rodrigo Antonio Peliciari ; Souza, Paula Bargi de ; Young, Martin Elliot; Nunes, Maria Tereza
Source: Chronobiology International. Unidade: ICB
Subjects: HIPOTIREOIDISMO ANIMAL, HIPERTIREOIDISMO, RITMOS BIOLÓGICOS, RITMOS CIRCADIANOS ANIMAL, RATOS, GLICOSE, CORAÇÃO, ÁCIDOS GRAXOS, EXPRESSÃO GÊNICA, MIOCÁRDIO
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ABNT
GARCIA, Rodrigo Antonio Peliciari et al. Repercussions of hypo and hyperthyroidism on the heart circadian clock. Chronobiology International, v. 35, n. 2, p. 147-159, 2018Tradução . . Disponível em: https://doi.org/10.1080/07420528.2017.1388253. Acesso em: 30 jun. 2024.
APA
Garcia, R. A. P., Souza, P. B. de, Young, M. E., & Nunes, M. T. (2018). Repercussions of hypo and hyperthyroidism on the heart circadian clock. Chronobiology International, 35( 2), 147-159. doi:10.1080/07420528.2017.1388253
NLM
Garcia RAP, Souza PB de, Young ME, Nunes MT. Repercussions of hypo and hyperthyroidism on the heart circadian clock [Internet]. Chronobiology International. 2018 ; 35( 2): 147-159.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1080/07420528.2017.1388253
Vancouver
Garcia RAP, Souza PB de, Young ME, Nunes MT. Repercussions of hypo and hyperthyroidism on the heart circadian clock [Internet]. Chronobiology International. 2018 ; 35( 2): 147-159.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1080/07420528.2017.1388253
Artigo de periodico
The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior (2018)
Teixeira, Lívia Clemente Motta ; Nils, Aline Vilar Machado; Battagello, Daniella Sabino ; Diniz, Giovanne Baroni ; Silva, Jéssica Andrade da ; Silva Junior, José Sinésio; Matos, Raphael Afonso ; Amaral, Fernanda Gaspar do; Xavier, Gilberto Fernando ; Bittencourt, Jackson Cioni ; Reiter, Russel J. ; Lucassen, Paul John ; Korosi, Aniko ; Cipolla Neto, José
Source: Hormones and Behavior. Unidades: IB, ICB
Subjects: MELATONINA, GLÂNDULA PINEAL, GRAVIDEZ, FOTOPERIODISMO, FETO, PARÂMETROS DE RESPOSTAS, LACTAÇÃO
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ABNT
TEIXEIRA, Lívia Clemente Motta et al. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, v. 105, p. 146-156, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.yhbeh.2018.08.006. Acesso em: 30 jun. 2024.
APA
Teixeira, L. C. M., Nils, A. V. M., Battagello, D. S., Diniz, G. B., Silva, J. A. da, Silva Junior, J. S., et al. (2018). The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior. Hormones and Behavior, 105, 146-156. doi:10.1016/j.yhbeh.2018.08.006
NLM
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Vancouver
Teixeira LCM, Nils AVM, Battagello DS, Diniz GB, Silva JA da, Silva Junior JS, Matos RA, Amaral FG do, Xavier GF, Bittencourt JC, Reiter RJ, Lucassen PJ, Korosi A, Cipolla Neto J. The absence of maternal pineal melatonin rhythm during pregnancy and lactation impairs offspring physical growth, neurodevelopment, and behavior [Internet]. Hormones and Behavior. 2018 ; 105 146-156.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.yhbeh.2018.08.006
Artigo de periodico
Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD (2018)
Araujo, Layanne Cabral da Cunha ; Feitosa, Karla Barroso; Murata, Gilson Masahiro ; Furigo, Isadora Clivatti ; Teixeira, Simone Aparecida ; Monaco, Camila Ferraz Lucena ; Ribeiro, Luciene Maria; Muscará, Marcelo Nicolás (Orientador) ; Costa, Soraia Katia Pereira ; Donato Junior, José ; Bordin, Silvana ; Curi, Rui ; Carvalho, Carla Roberta de Oliveira
Source: Scientific reports. Unidade: ICB
Subjects: PLANTAS MEDICINAIS, HEPATOPATIAS, INSULINA, EXTRATOS (FORMAS FARMACÊUTICAS), GLICOSE, CAMUNDONGOS, HOMEOSTASE
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ARAUJO, Layanne Cabral da Cunha et al. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD. Scientific reports, v. 8, 2018Tradução . . Disponível em: https://doi.org/10.1038/s41598-018-29044-y. Acesso em: 30 jun. 2024.
APA
Araujo, L. C. da C., Feitosa, K. B., Murata, G. M., Furigo, I. C., Teixeira, S. A., Monaco, C. F. L., et al. (2018). Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD. Scientific reports, 8. doi:10.1038/s41598-018-29044-y
NLM
Araujo LC da C, Feitosa KB, Murata GM, Furigo IC, Teixeira SA, Monaco CFL, Ribeiro LM, Costa SKP, Donato Junior J, Bordin S, Curi R, Carvalho CR de O. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 30 ] Available from: https://doi.org/10.1038/s41598-018-29044-y
Vancouver
Araujo LC da C, Feitosa KB, Murata GM, Furigo IC, Teixeira SA, Monaco CFL, Ribeiro LM, Costa SKP, Donato Junior J, Bordin S, Curi R, Carvalho CR de O. Uncaria tomentosa improves insulin sensitivity and inflammation in experimental NAFLD [Internet]. Scientific reports. 2018 ; 8[citado 2024 jun. 30 ] Available from: https://doi.org/10.1038/s41598-018-29044-y
Artigo de periodico
NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance (2018)
Crunfli, Fernanda ; Mazucanti, Caio Henrique Yokoyama ; Moraes, Ruan Carlos Macêdo de ; Costa, Andressa Pereira ; Rodrigues, Alice Cristina ; Scavone, Cristoforo ; Torrão, Andréa da Silva
Source: Journal of Alzheimer's Disease. Unidade: ICB
Subjects: INSULINA, DOENÇA DE ALZHEIMER, NEURÔNIOS, PROTEÍNAS QUINASES, METABOLISMO ENERGÉTICO, RECEPTORES DE INSULINA
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ABNT
CRUNFLI, Fernanda et al. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, v. 65, n. 4, p. 1427-1443, 2018Tradução . . Disponível em: https://doi.org/10.3233/JAD-180284. Acesso em: 30 jun. 2024.
APA
Crunfli, F., Mazucanti, C. H. Y., Moraes, R. C. M. de, Costa, A. P., Rodrigues, A. C., Scavone, C., & Torrão, A. da S. (2018). NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance. Journal of Alzheimer's Disease, 65( 4), 1427-1443. doi:10.3233/JAD-180284
NLM
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 jun. 30 ] Available from: https://doi.org/10.3233/JAD-180284
Vancouver
Crunfli F, Mazucanti CHY, Moraes RCM de, Costa AP, Rodrigues AC, Scavone C, Torrão A da S. NO-dependent akt inactivation by S-nitrosylation as a possible mechanism of STZ-induced neuronal insulin resistance [Internet]. Journal of Alzheimer's Disease. 2018 ; 65( 4): 1427-1443.[citado 2024 jun. 30 ] Available from: https://doi.org/10.3233/JAD-180284
Artigo de periodico
Minocycline alters expression of inflammatory markers in autonomic brain areas and ventilatory responses induced by acute hypoxia (2018)
Silva, Talita de Melo e; Chaar, Laiali Jurdi El ; Silva, Reinaldo Correia da; Takakura, Ana Carolina ; Câmara, Niels Olsen Saraiva ; Antunes, Vagner Roberto ; Moreira, Thiago dos Santos
Source: Experimental Physiology. Unidade: ICB
Subjects: ANTIBIÓTICOS, ANÓXIA, INFLAMAÇÃO, BIOMARCADORES, METALOPROTEINASES, ENZIMAS PROTEOLÍTICAS, ATIVAÇÃO ENZIMÁTICA, SISTEMA NERVOSO AUTÔNOMO
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ABNT
SILVA, Talita de Melo e et al. Minocycline alters expression of inflammatory markers in autonomic brain areas and ventilatory responses induced by acute hypoxia. Experimental Physiology, v. 103, n. 6, p. 884-895, 2018Tradução . . Disponível em: https://doi.org/10.1113/EP086780. Acesso em: 30 jun. 2024.
APA
Silva, T. de M. e, Chaar, L. J. E., Silva, R. C. da, Takakura, A. C., Câmara, N. O. S., Antunes, V. R., & Moreira, T. dos S. (2018). Minocycline alters expression of inflammatory markers in autonomic brain areas and ventilatory responses induced by acute hypoxia. Experimental Physiology, 103( 6), 884-895. doi:10.1113/EP086780
NLM
Silva T de M e, Chaar LJE, Silva RC da, Takakura AC, Câmara NOS, Antunes VR, Moreira T dos S. Minocycline alters expression of inflammatory markers in autonomic brain areas and ventilatory responses induced by acute hypoxia [Internet]. Experimental Physiology. 2018 ; 103( 6): 884-895.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1113/EP086780
Vancouver
Silva T de M e, Chaar LJE, Silva RC da, Takakura AC, Câmara NOS, Antunes VR, Moreira T dos S. Minocycline alters expression of inflammatory markers in autonomic brain areas and ventilatory responses induced by acute hypoxia [Internet]. Experimental Physiology. 2018 ; 103( 6): 884-895.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1113/EP086780
Artigo de periodico
Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats (2018)
Oliveira, Ariclécio Cunha de ; Sertié, Sandra Andreotti ; Sertie, Rogério Antônio Laurato ; Campaña, Amanda Baron; Proença, André Ricardo Gomes de ; Vasconcelos, Renata Prado; Oliveira, Keciany Alves de ; Souza, Andrelina Noronha Coelho de ; Donato Junior, José ; Lima, Fabio Bessa
Source: Life Sciences. Unidade: ICB
Subjects: MELATONINA, INSULINA, GLICEMIA, TECIDO ADIPOSO, GORDURAS, HIPOTÁLAMO, SANGUE
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ABNT
OLIVEIRA, Ariclécio Cunha de et al. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats. Life Sciences, v. 199, p. 158-166, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.lfs.2018.02.040. Acesso em: 30 jun. 2024.
APA
Oliveira, A. C. de, Sertié, S. A., Sertie, R. A. L., Campaña, A. B., Proença, A. R. G. de, Vasconcelos, R. P., et al. (2018). Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats. Life Sciences, 199, 158-166. doi:10.1016/j.lfs.2018.02.040
NLM
Oliveira AC de, Sertié SA, Sertie RAL, Campaña AB, Proença ARG de, Vasconcelos RP, Oliveira KA de, Souza ANC de, Donato Junior J, Lima FB. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats [Internet]. Life Sciences. 2018 ; 199 158-166.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.02.040
Vancouver
Oliveira AC de, Sertié SA, Sertie RAL, Campaña AB, Proença ARG de, Vasconcelos RP, Oliveira KA de, Souza ANC de, Donato Junior J, Lima FB. Combined treatment with melatonin and insulin improves glycemic control, white adipose tissue metabolism and reproductive axis of diabetic male rats [Internet]. Life Sciences. 2018 ; 199 158-166.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.lfs.2018.02.040
Artigo de periodico
Brain STAT5 signaling modulates learning and memory formation (2018)
Furigo, Isadora Clivatti; Melo, Helen Maciqueira; Silva, Natalia de Menezes Lyra e; Lobo, Angela Maria Ramos; Teixeira, Pryscila Dryelle Sousa; Buonfiglio, Daniella do Carmo; Wasinski, Frederick; Lima Filha, Eliana Rosa; Higuti, Eliza; Peroni, Cibele Nunes; Bartolini, Paolo; Soares, Carlos Roberto Jorge; Metzger, Martin Andreas ; Felice, Fernanda Guarino De; Donato Junior, José
Source: Brain Structure and Function. Unidade: ICB
Subjects: CITOCINAS, MEMÓRIA, CÉREBRO, APRENDIZAGEM ANIMAL, METABOLISMO ANIMAL, LIPÍDEOS, HIPOCAMPU DE ANIMAL, HIPOTÁLAMO
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ABNT
FURIGO, Isadora Clivatti et al. Brain STAT5 signaling modulates learning and memory formation. Brain Structure and Function, v. 223, n. 5, p. 2229-2241, 2018Tradução . . Disponível em: https://doi.org/10.1007/s00429-018-1627-z. Acesso em: 30 jun. 2024.
APA
Furigo, I. C., Melo, H. M., Silva, N. de M. L. e, Lobo, A. M. R., Teixeira, P. D. S., Buonfiglio, D. do C., et al. (2018). Brain STAT5 signaling modulates learning and memory formation. Brain Structure and Function, 223( 5), 2229-2241. doi:10.1007/s00429-018-1627-z
NLM
Furigo IC, Melo HM, Silva N de ML e, Lobo AMR, Teixeira PDS, Buonfiglio D do C, Wasinski F, Lima Filha ER, Higuti E, Peroni CN, Bartolini P, Soares CRJ, Metzger MA, Felice FGD, Donato Junior J. Brain STAT5 signaling modulates learning and memory formation [Internet]. Brain Structure and Function. 2018 ; 223( 5): 2229-2241.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s00429-018-1627-z
Vancouver
Furigo IC, Melo HM, Silva N de ML e, Lobo AMR, Teixeira PDS, Buonfiglio D do C, Wasinski F, Lima Filha ER, Higuti E, Peroni CN, Bartolini P, Soares CRJ, Metzger MA, Felice FGD, Donato Junior J. Brain STAT5 signaling modulates learning and memory formation [Internet]. Brain Structure and Function. 2018 ; 223( 5): 2229-2241.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1007/s00429-018-1627-z
Artigo de periodico
Liraglutide modulates gut microbiota and reduces NAFLD in obese mice (2018)
Moreira, Gabriela Virginia; Azevedo, Flávia Figueiredo; Ribeiro, Luciene Maria; Santos, Andrey dos; Guadagnini, Dioze; Gama, Patrícia ; Liberti, Edson Aparecido; Saad, Mário José Abdalla; Carvalho, Carla Roberta de Oliveira
Source: Journal of Nutritional Biochemistry. Unidade: ICB
Subjects: FÁRMACOS SINTÉTICOS, DIABETES MELLITUS, RESISTÊNCIA À INSULINA, CAMUNDONGOS, OBESIDADE, PERDA DE PESO, DOENÇAS METABÓLICAS, MODELOS ANIMAIS DE DOENÇAS, FLORA
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ABNT
MOREIRA, Gabriela Virginia et al. Liraglutide modulates gut microbiota and reduces NAFLD in obese mice. Journal of Nutritional Biochemistry, v. 62, p. 143-154, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.jnutbio.2018.07.009. Acesso em: 30 jun. 2024.
APA
Moreira, G. V., Azevedo, F. F., Ribeiro, L. M., Santos, A. dos, Guadagnini, D., Gama, P., et al. (2018). Liraglutide modulates gut microbiota and reduces NAFLD in obese mice. Journal of Nutritional Biochemistry, 62, 143-154. doi:10.1016/j.jnutbio.2018.07.009
NLM
Moreira GV, Azevedo FF, Ribeiro LM, Santos A dos, Guadagnini D, Gama P, Liberti EA, Saad MJA, Carvalho CR de O. Liraglutide modulates gut microbiota and reduces NAFLD in obese mice [Internet]. Journal of Nutritional Biochemistry. 2018 ; 62 143-154.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.07.009
Vancouver
Moreira GV, Azevedo FF, Ribeiro LM, Santos A dos, Guadagnini D, Gama P, Liberti EA, Saad MJA, Carvalho CR de O. Liraglutide modulates gut microbiota and reduces NAFLD in obese mice [Internet]. Journal of Nutritional Biochemistry. 2018 ; 62 143-154.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.jnutbio.2018.07.009
Artigo de periodico
Melatonin absence leads to long-term leptin resistance and overweight in rats (2018)
Buonfiglio, Daniella do Carmo; Parthimos, Rafaela Panaiotta; Ferreira, Rosana Fátima Dantas; Silva, Raysa Cerqueira; Gomes, Guilherme; Silva, Jéssica Andrade da; Lobo, Angela Maria Ramos; Amaral, Fernada Gaspar do; Matos, Raphael Afonso de; Silva Junior, José Sinésio; Teixeira, Lívia Clemente Motta; Donato Junior, José ; Reiter, Russel J; Cipolla Neto, José
Source: Frontiers in Endocrinology. Unidade: ICB
Subjects: MELATONINA, LEPTINA, RATOS WISTAR, BALANÇO DE ENERGIA, HIPOTÁLAMO, TECIDO ADIPOSO, NEUROPEPTÍDEOS
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ABNT
BUONFIGLIO, Daniella do Carmo et al. Melatonin absence leads to long-term leptin resistance and overweight in rats. Frontiers in Endocrinology, v. 9, p. 12 , 2018Tradução . . Disponível em: https://doi.org/10.3389/fendo.2018.00122. Acesso em: 30 jun. 2024.
APA
Buonfiglio, D. do C., Parthimos, R. P., Ferreira, R. F. D., Silva, R. C., Gomes, G., Silva, J. A. da, et al. (2018). Melatonin absence leads to long-term leptin resistance and overweight in rats. Frontiers in Endocrinology, 9, 12 . doi:10.3389/fendo.2018.00122
NLM
Buonfiglio D do C, Parthimos RP, Ferreira RFD, Silva RC, Gomes G, Silva JA da, Lobo AMR, Amaral FG do, Matos RA de, Silva Junior JS, Teixeira LCM, Donato Junior J, Reiter RJ, Cipolla Neto J. Melatonin absence leads to long-term leptin resistance and overweight in rats [Internet]. Frontiers in Endocrinology. 2018 ; 9 12 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.3389/fendo.2018.00122
Vancouver
Buonfiglio D do C, Parthimos RP, Ferreira RFD, Silva RC, Gomes G, Silva JA da, Lobo AMR, Amaral FG do, Matos RA de, Silva Junior JS, Teixeira LCM, Donato Junior J, Reiter RJ, Cipolla Neto J. Melatonin absence leads to long-term leptin resistance and overweight in rats [Internet]. Frontiers in Endocrinology. 2018 ; 9 12 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.3389/fendo.2018.00122
Artigo de periodico
PPARγ is a major regulator of branched-chain amino acid blood levels and catabolism in white and brown adipose tissues (2018)
Blanchard, Pierre Gilles; Moreira, Rafael Junges; Castro, Érique de; Caron, Alexandre; Côté, Marie France; Andrade, Maynara Lucca; Silva, Tiago Eugênio Oliveira da; Silva, Milene Ortiz; Peixoto, Álbert Souza; Ruas, France Anne Dias; Gélinas, Yves; Cota, Renata Guerra de Sá; Deshaies, Yves; Festuccia, William Tadeu Lara
Source: Metabolism: Clinical and Experimental. Unidade: ICB
Subjects: TECIDO ADIPOSO, INSULINA, OBESIDADE, CAMUNDONGOS, PEROXISSOMOS, METABOLISMO ENERGÉTICO, AMINOÁCIDOS, GRUPOS SANGUÍNEOS
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
BLANCHARD, Pierre Gilles et al. PPARγ is a major regulator of branched-chain amino acid blood levels and catabolism in white and brown adipose tissues. Metabolism: Clinical and Experimental, v. 89, p. 27-38, 2018Tradução . . Disponível em: https://doi.org/10.1016/j.metabol.2018.09.007. Acesso em: 30 jun. 2024.
APA
Blanchard, P. G., Moreira, R. J., Castro, É. de, Caron, A., Côté, M. F., Andrade, M. L., et al. (2018). PPARγ is a major regulator of branched-chain amino acid blood levels and catabolism in white and brown adipose tissues. Metabolism: Clinical and Experimental, 89, 27-38. doi:10.1016/j.metabol.2018.09.007
NLM
Blanchard PG, Moreira RJ, Castro É de, Caron A, Côté MF, Andrade ML, Silva TEO da, Silva MO, Peixoto ÁS, Ruas FAD, Gélinas Y, Cota RG de S, Deshaies Y, Festuccia WTL. PPARγ is a major regulator of branched-chain amino acid blood levels and catabolism in white and brown adipose tissues [Internet]. Metabolism: Clinical and Experimental. 2018 ; 89 27-38.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.metabol.2018.09.007
Vancouver
Blanchard PG, Moreira RJ, Castro É de, Caron A, Côté MF, Andrade ML, Silva TEO da, Silva MO, Peixoto ÁS, Ruas FAD, Gélinas Y, Cota RG de S, Deshaies Y, Festuccia WTL. PPARγ is a major regulator of branched-chain amino acid blood levels and catabolism in white and brown adipose tissues [Internet]. Metabolism: Clinical and Experimental. 2018 ; 89 27-38.[citado 2024 jun. 30 ] Available from: https://doi.org/10.1016/j.metabol.2018.09.007
Artigo de periodico
Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity (2018)
Cardoso, Vanessa Gerolde; Gonçalves, Guilherme Lopes; Pessoa, Juliana Martins da Costa; Thieme, Karina ; Lins, Bruna Bezerra; Casare, Fernando Augusto Malavazzi; Ponte, Mariana Charleaux de; Câmara, Niels Olsen Saraiva ; Souza, Maria Oliveira de
Source: BMC Nephrology. Unidade: ICB
Subjects: ANGIOTENSINA II, APOPTOSE, CITOMETRIA DE FLUXO, IMUNOFLUORESCÊNCIA EM ANIMAL, IMMUNOBLOTTING, RATOS WISTAR, CAMUNDONGOS
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ABNT
CARDOSO, Vanessa Gerolde et al. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity. BMC Nephrology, v. 19, n. 1, p. 12 , 2018Tradução . . Disponível em: https://doi.org/10.1186/s12882-018-0968-4. Acesso em: 30 jun. 2024.
APA
Cardoso, V. G., Gonçalves, G. L., Pessoa, J. M. da C., Thieme, K., Lins, B. B., Casare, F. A. M., et al. (2018). Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity. BMC Nephrology, 19( 1), 12 . doi:10.1186/s12882-018-0968-4
NLM
Cardoso VG, Gonçalves GL, Pessoa JM da C, Thieme K, Lins BB, Casare FAM, Ponte MC de, Câmara NOS, Souza MO de. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity [Internet]. BMC Nephrology. 2018 ; 19( 1): 12 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.1186/s12882-018-0968-4
Vancouver
Cardoso VG, Gonçalves GL, Pessoa JM da C, Thieme K, Lins BB, Casare FAM, Ponte MC de, Câmara NOS, Souza MO de. Angiotensin II-induced podocyte apoptosis is mediated by endoplasmic reticulum stress/PKC-δ/p38 MAPK pathway activation and trough increased Na+/H+ exchanger isoform 1 activity [Internet]. BMC Nephrology. 2018 ; 19( 1): 12 .[citado 2024 jun. 30 ] Available from: https://doi.org/10.1186/s12882-018-0968-4

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