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FOGAÇA, Manoela V. e CAMPOS, Alline Cristina de e GUIMARÃES, Francisco Silveira. Cannabidiol and 5-HT1A receptors. Neuropathology of drug addictions and substance misuse. Volume 1. Tradução . Massachusetts: Academic Press, 2016. . . Acesso em: 31 maio 2024.
APA
Fogaça, M. V., Campos, A. C. de, & Guimarães, F. S. (2016). Cannabidiol and 5-HT1A receptors. In Neuropathology of drug addictions and substance misuse. Volume 1. Massachusetts: Academic Press.
NLM
Fogaça MV, Campos AC de, Guimarães FS. Cannabidiol and 5-HT1A receptors. In: Neuropathology of drug addictions and substance misuse. Volume 1. Massachusetts: Academic Press; 2016. [citado 2024 maio 31 ]
Vancouver
Fogaça MV, Campos AC de, Guimarães FS. Cannabidiol and 5-HT1A receptors. In: Neuropathology of drug addictions and substance misuse. Volume 1. Massachusetts: Academic Press; 2016. [citado 2024 maio 31 ]
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OLIVEIRA-PAULA, Gustavo H. e LACCHINI, Riccardo e TANUS-SANTOS, José Eduardo. Endothelial nitric oxide synthase: from biochemistry and gene structure to clinical implications of NOS3 polymorphisms. Gene, v. 2, n. 3, p. 584–599, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.gene.2015.09.061. Acesso em: 31 maio 2024.
APA
Oliveira-Paula, G. H., Lacchini, R., & Tanus-Santos, J. E. (2016). Endothelial nitric oxide synthase: from biochemistry and gene structure to clinical implications of NOS3 polymorphisms. Gene, 2( 3), 584–599. doi:10.1016/j.gene.2015.09.061
NLM
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Endothelial nitric oxide synthase: from biochemistry and gene structure to clinical implications of NOS3 polymorphisms [Internet]. Gene. 2016 ; 2( 3): 584–599.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.gene.2015.09.061
Vancouver
Oliveira-Paula GH, Lacchini R, Tanus-Santos JE. Endothelial nitric oxide synthase: from biochemistry and gene structure to clinical implications of NOS3 polymorphisms [Internet]. Gene. 2016 ; 2( 3): 584–599.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.gene.2015.09.061
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RITTER, Alessandra Mileni Versuti et al. Association of mineralocorticoid receptor polymorphism l180V with left ventricular hypertrophy in resistant hypertension. American Journal of Hypertension, v. 29, n. 2, p. 245-250, 2016Tradução . . Disponível em: https://doi.org/10.1093/ajh/hpv070. Acesso em: 31 maio 2024.
APA
Ritter, A. M. V., Fontana, V., Faria, A. P. C. de, Modolo, R., Barbaro, N. R., Sabbatini, A. R., et al. (2016). Association of mineralocorticoid receptor polymorphism l180V with left ventricular hypertrophy in resistant hypertension. American Journal of Hypertension, 29( 2), 245-250. doi:10.1093/ajh/hpv070
NLM
Ritter AMV, Fontana V, Faria APC de, Modolo R, Barbaro NR, Sabbatini AR, Peres H, Biagi C, Silva P, Lopes PC, Tanus-Santos JE, Coelho EB, Moreno H. Association of mineralocorticoid receptor polymorphism l180V with left ventricular hypertrophy in resistant hypertension [Internet]. American Journal of Hypertension. 2016 ; 29( 2): 245-250.[citado 2024 maio 31 ] Available from: https://doi.org/10.1093/ajh/hpv070
Vancouver
Ritter AMV, Fontana V, Faria APC de, Modolo R, Barbaro NR, Sabbatini AR, Peres H, Biagi C, Silva P, Lopes PC, Tanus-Santos JE, Coelho EB, Moreno H. Association of mineralocorticoid receptor polymorphism l180V with left ventricular hypertrophy in resistant hypertension [Internet]. American Journal of Hypertension. 2016 ; 29( 2): 245-250.[citado 2024 maio 31 ] Available from: https://doi.org/10.1093/ajh/hpv070
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PINKE, Karen Henriette et al. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1. Immunobiology, v. 221, n. 2, p. 220-227, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.imbio.2015.09.004. Acesso em: 31 maio 2024.
APA
Pinke, K. H., Lima, H. G. de, Cunha, F. Q., & Lara, V. S. (2016). Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1. Immunobiology, 221( 2), 220-227. doi:10.1016/j.imbio.2015.09.004
NLM
Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1 [Internet]. Immunobiology. 2016 ; 221( 2): 220-227.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.imbio.2015.09.004
Vancouver
Pinke KH, Lima HG de, Cunha FQ, Lara VS. Mast cells phagocyte Candida albicans and produce nitric oxide by mechanisms involving TLR2 and Dectin-1 [Internet]. Immunobiology. 2016 ; 221( 2): 220-227.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.imbio.2015.09.004
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GOBIRA, P. H. et al. Role of the endocannabinoid 2-arachidonoylglycerol in aversive responses mediated by the dorsolateral periaqueductal grey. European Neuropsychopharmacology, v. 26, n. 1, p. 15-22, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.euroneuro.2015.11.014. Acesso em: 31 maio 2024.
APA
Gobira, P. H., Almeida-Santos, A. F., Guimarães, F. S., Moreira, F. A., & Aguiar, D. C. (2016). Role of the endocannabinoid 2-arachidonoylglycerol in aversive responses mediated by the dorsolateral periaqueductal grey. European Neuropsychopharmacology, 26( 1), 15-22. doi:10.1016/j.euroneuro.2015.11.014
NLM
Gobira PH, Almeida-Santos AF, Guimarães FS, Moreira FA, Aguiar DC. Role of the endocannabinoid 2-arachidonoylglycerol in aversive responses mediated by the dorsolateral periaqueductal grey [Internet]. European Neuropsychopharmacology. 2016 ; 26( 1): 15-22.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.euroneuro.2015.11.014
Vancouver
Gobira PH, Almeida-Santos AF, Guimarães FS, Moreira FA, Aguiar DC. Role of the endocannabinoid 2-arachidonoylglycerol in aversive responses mediated by the dorsolateral periaqueductal grey [Internet]. European Neuropsychopharmacology. 2016 ; 26( 1): 15-22.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.euroneuro.2015.11.014
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FOGAÇA, Manoela V et al. Different roles of CB1 and CB2 receptors in the pro-neurogenic effect of cannabidiol in chronically stressed mice. 2016, Anais.. Antibes: ISDN, 2016. . Acesso em: 31 maio 2024.
APA
Fogaça, M. V., Coelho, L. D., Campos, A. C. de, & Guimarães, F. S. (2016). Different roles of CB1 and CB2 receptors in the pro-neurogenic effect of cannabidiol in chronically stressed mice. In Programme Booklet. Antibes: ISDN.
NLM
Fogaça MV, Coelho LD, Campos AC de, Guimarães FS. Different roles of CB1 and CB2 receptors in the pro-neurogenic effect of cannabidiol in chronically stressed mice. Programme Booklet. 2016 ;[citado 2024 maio 31 ]
Vancouver
Fogaça MV, Coelho LD, Campos AC de, Guimarães FS. Different roles of CB1 and CB2 receptors in the pro-neurogenic effect of cannabidiol in chronically stressed mice. Programme Booklet. 2016 ;[citado 2024 maio 31 ]
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FRANCHIN, Marcelo et al. Neovestitol, an isoflavonoid isolated from Brazilian red propolis, reduces acute and chronic inflammation: involvement of nitric oxide and IL-6. Scientific Reports, v. 6 art. 36401, 2016Tradução . . Disponível em: https://doi.org/10.1038/srep36401. Acesso em: 31 maio 2024.
APA
Franchin, M., Colón, D. F., Cunha, M. G. da, Castanheira, F. V. S., Saraiva, A. L. L., Bueno-Silva, B., et al. (2016). Neovestitol, an isoflavonoid isolated from Brazilian red propolis, reduces acute and chronic inflammation: involvement of nitric oxide and IL-6. Scientific Reports, 6 art. 36401. doi:10.1038/srep36401
NLM
Franchin M, Colón DF, Cunha MG da, Castanheira FVS, Saraiva ALL, Bueno-Silva B, Alencar SM de, Cunha TM, Rosalen PL. Neovestitol, an isoflavonoid isolated from Brazilian red propolis, reduces acute and chronic inflammation: involvement of nitric oxide and IL-6 [Internet]. Scientific Reports. 2016 ; 6 art. 36401[citado 2024 maio 31 ] Available from: https://doi.org/10.1038/srep36401
Vancouver
Franchin M, Colón DF, Cunha MG da, Castanheira FVS, Saraiva ALL, Bueno-Silva B, Alencar SM de, Cunha TM, Rosalen PL. Neovestitol, an isoflavonoid isolated from Brazilian red propolis, reduces acute and chronic inflammation: involvement of nitric oxide and IL-6 [Internet]. Scientific Reports. 2016 ; 6 art. 36401[citado 2024 maio 31 ] Available from: https://doi.org/10.1038/srep36401
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FRANCHIN, Marcelo et al. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, v. 79, n. 7, p. 1828-1833, 2016Tradução . . Disponível em: https://doi.org/10.1021/acs.jnatprod.6b00263. Acesso em: 31 maio 2024.
APA
Franchin, M., Rosalen, P. L., Cunha, M. G. da, Silva, R. L., Colón, D. F., Bassi, G. S., et al. (2016). Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB. Journal of Natural Products, 79( 7), 1828-1833. doi:10.1021/acs.jnatprod.6b00263
NLM
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 maio 31 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
Vancouver
Franchin M, Rosalen PL, Cunha MG da, Silva RL, Colón DF, Bassi GS, Alencar SM de, Ikegaki M, Alves Filho JCF, Cunha F de Q, Beutler JA, Cunha TM. Cinnamoyloxy-mammeisin isolated from geopropolis attenuates inflammatory process by inhibiting cytokine production: involvement of MAPK, AP-1, and NF-kB [Internet]. Journal of Natural Products. 2016 ; 79( 7): 1828-1833.[citado 2024 maio 31 ] Available from: https://doi.org/10.1021/acs.jnatprod.6b00263
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KUNTZE, Luciana Bärg et al. Ventral hippocampus modulates bradycardic response to peripheral chemoreflex activation in awake rats. Experimental Physiology, v. 101, n. 4, 2016Tradução . . Disponível em: https://doi.org/10.1113/ep085393. Acesso em: 31 maio 2024.
APA
Kuntze, L. B., Ferreira-Junior, N. C., Lagatta, D. C., & Resstel, L. B. M. (2016). Ventral hippocampus modulates bradycardic response to peripheral chemoreflex activation in awake rats. Experimental Physiology, 101( 4). doi:10.1113/ep085393
NLM
Kuntze LB, Ferreira-Junior NC, Lagatta DC, Resstel LBM. Ventral hippocampus modulates bradycardic response to peripheral chemoreflex activation in awake rats [Internet]. Experimental Physiology. 2016 ; 101( 4):[citado 2024 maio 31 ] Available from: https://doi.org/10.1113/ep085393
Vancouver
Kuntze LB, Ferreira-Junior NC, Lagatta DC, Resstel LBM. Ventral hippocampus modulates bradycardic response to peripheral chemoreflex activation in awake rats [Internet]. Experimental Physiology. 2016 ; 101( 4):[citado 2024 maio 31 ] Available from: https://doi.org/10.1113/ep085393
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CARREGARO, Vanessa et al. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, v. 2016, 2016Tradução . . Disponível em: https://doi.org/10.1155/2016/9626427. Acesso em: 31 maio 2024.
APA
Carregaro, V., Napimoga, M. H., Peres, R. S., Benevides, L., Sacramento, L. A., Pinto, L. G., et al. (2016). Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells. Mediators of Inflammation, 2016. doi:10.1155/2016/9626427
NLM
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 maio 31 ] Available from: https://doi.org/10.1155/2016/9626427
Vancouver
Carregaro V, Napimoga MH, Peres RS, Benevides L, Sacramento LA, Pinto LG, Grespan R, Cunha TM, Silva JS da, Cunha F de Q. Therapeutic treatment of arthritic mice with 15-deoxy Δ12,14 -prostaglandin J2 (15d-PGJ2) ameliorates disease through the suppression of Th17 cells and the induction of CD4+ CD25- FOXP3+ cells [Internet]. Mediators of Inflammation. 2016 ; 2016[citado 2024 maio 31 ] Available from: https://doi.org/10.1155/2016/9626427
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LEITE, Letícia N. et al. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, v. 789, p. 334-343, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.ejphar.2016.07.045. Acesso em: 31 maio 2024.
APA
Leite, L. N., Gonzaga, N. A., Simplicio, J. A., Vale, G. T. do, Carballido, J. M., Alves Filho, J. C. F., & Tirapelli, C. R. (2016). Pharmacological characterization of the mechanisms underlying the vascular effects of succinate. European Journal of Pharmacology, 789, 334-343. doi:10.1016/j.ejphar.2016.07.045
NLM
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
Vancouver
Leite LN, Gonzaga NA, Simplicio JA, Vale GT do, Carballido JM, Alves Filho JCF, Tirapelli CR. Pharmacological characterization of the mechanisms underlying the vascular effects of succinate [Internet]. European Journal of Pharmacology. 2016 ; 789 334-343.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.ejphar.2016.07.045
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BIAGIONI, Audrey Franceschi et al. 5-Hydroxytryptamine 1A receptors in the dorsomedial hypothalamus connected to dorsal raphe nucleus inputs modulate defensive behaviours and mediate innate fear-induced antinociception. European Neuropsychopharmacology, v. 26, n. 3, p. 532-545, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.euroneuro.2015.12.032. Acesso em: 31 maio 2024.
APA
Biagioni, A. F., Oliveira, R. C. de, Oliveira, R. de, Silva, J. A. da, Anjos-Garcia, T. dos, Roncon, C. M., et al. (2016). 5-Hydroxytryptamine 1A receptors in the dorsomedial hypothalamus connected to dorsal raphe nucleus inputs modulate defensive behaviours and mediate innate fear-induced antinociception. European Neuropsychopharmacology, 26( 3), 532-545. doi:10.1016/j.euroneuro.2015.12.032
NLM
Biagioni AF, Oliveira RC de, Oliveira R de, Silva JA da, Anjos-Garcia T dos, Roncon CM, Corrado AP, Zangrossi Júnior H, Coimbra NC. 5-Hydroxytryptamine 1A receptors in the dorsomedial hypothalamus connected to dorsal raphe nucleus inputs modulate defensive behaviours and mediate innate fear-induced antinociception [Internet]. European Neuropsychopharmacology. 2016 ; 26( 3): 532-545.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.euroneuro.2015.12.032
Vancouver
Biagioni AF, Oliveira RC de, Oliveira R de, Silva JA da, Anjos-Garcia T dos, Roncon CM, Corrado AP, Zangrossi Júnior H, Coimbra NC. 5-Hydroxytryptamine 1A receptors in the dorsomedial hypothalamus connected to dorsal raphe nucleus inputs modulate defensive behaviours and mediate innate fear-induced antinociception [Internet]. European Neuropsychopharmacology. 2016 ; 26( 3): 532-545.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.euroneuro.2015.12.032
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MOLON, Rafael Scaf de et al. Rheumatoid arthritis exacerbates the severity of osteonecrosis of the Jaws (ONJ) in mice: a randomized, prospective, controlled animal study. Journal of Bone and Mineral Research, v. 31, n. 8, p. 1596-1607, 2016Tradução . . Disponível em: https://doi.org/10.1002/jbmr.2827. Acesso em: 31 maio 2024.
APA
Molon, R. S. de, Hsu, C., Bezouglaia, O., Dry, S. M., Pirih, F. Q., Soundia, A., et al. (2016). Rheumatoid arthritis exacerbates the severity of osteonecrosis of the Jaws (ONJ) in mice: a randomized, prospective, controlled animal study. Journal of Bone and Mineral Research, 31( 8), 1596-1607. doi:10.1002/jbmr.2827
NLM
Molon RS de, Hsu C, Bezouglaia O, Dry SM, Pirih FQ, Soundia A, Cunha F de Q, Cirelli JA, Aghaloo TL, Tetradis S. Rheumatoid arthritis exacerbates the severity of osteonecrosis of the Jaws (ONJ) in mice: a randomized, prospective, controlled animal study [Internet]. Journal of Bone and Mineral Research. 2016 ; 31( 8): 1596-1607.[citado 2024 maio 31 ] Available from: https://doi.org/10.1002/jbmr.2827
Vancouver
Molon RS de, Hsu C, Bezouglaia O, Dry SM, Pirih FQ, Soundia A, Cunha F de Q, Cirelli JA, Aghaloo TL, Tetradis S. Rheumatoid arthritis exacerbates the severity of osteonecrosis of the Jaws (ONJ) in mice: a randomized, prospective, controlled animal study [Internet]. Journal of Bone and Mineral Research. 2016 ; 31( 8): 1596-1607.[citado 2024 maio 31 ] Available from: https://doi.org/10.1002/jbmr.2827
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VILA-VERDE, C. et al. Nitric oxide in the prelimbic medial prefrontal cortex is involved in the anxiogenic-like effect induced by acute restraint stress in rats. Neuroscience, v. 320, p. 30-42, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.neuroscience.2016.01.040. Acesso em: 31 maio 2024.
APA
Vila-Verde, C., Marinho, A. L. Z., Lisboa, S. F., & Guimarães, F. S. (2016). Nitric oxide in the prelimbic medial prefrontal cortex is involved in the anxiogenic-like effect induced by acute restraint stress in rats. Neuroscience, 320, 30-42. doi:10.1016/j.neuroscience.2016.01.040
NLM
Vila-Verde C, Marinho ALZ, Lisboa SF, Guimarães FS. Nitric oxide in the prelimbic medial prefrontal cortex is involved in the anxiogenic-like effect induced by acute restraint stress in rats [Internet]. Neuroscience. 2016 ; 320 30-42.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.01.040
Vancouver
Vila-Verde C, Marinho ALZ, Lisboa SF, Guimarães FS. Nitric oxide in the prelimbic medial prefrontal cortex is involved in the anxiogenic-like effect induced by acute restraint stress in rats [Internet]. Neuroscience. 2016 ; 320 30-42.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.neuroscience.2016.01.040
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AMARAL, Jefferson H. e TANUS-SANTOS, José Eduardo. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta]. Clinica Chimica Acta. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. Disponível em: https://doi.org/10.1016/j.cca.2016.08.020. Acesso em: 31 maio 2024. , 2016
APA
Amaral, J. H., & Tanus-Santos, J. E. (2016). Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta]. Clinica Chimica Acta. Amsterdam: Faculdade de Medicina de Ribeirão Preto, Universidade de São Paulo. doi:10.1016/j.cca.2016.08.020
NLM
Amaral JH, Tanus-Santos JE. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta] [Internet]. Clinica Chimica Acta. 2016 ; 462 40.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.cca.2016.08.020
Vancouver
Amaral JH, Tanus-Santos JE. Mechanisms involved in cardiovascular protection associated with a vegetarian diet. [Carta] [Internet]. Clinica Chimica Acta. 2016 ; 462 40.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.cca.2016.08.020
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BREUER, Aviva et al. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects. PLOS ONE, v. 11, n. 8, 2016Tradução . . Disponível em: https://doi.org/10.1371/journal.pone.0162087. Acesso em: 31 maio 2024.
APA
Breuer, A., Haj, C. G., Fogaça, M. V., Gomes, F. V., Silva, N. R., Pedrazzi, J. F., et al. (2016). Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects. PLOS ONE, 11( 8). doi:10.1371/journal.pone.0162087
NLM
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects [Internet]. PLOS ONE. 2016 ; 11( 8):[citado 2024 maio 31 ] Available from: https://doi.org/10.1371/journal.pone.0162087
Vancouver
Breuer A, Haj CG, Fogaça MV, Gomes FV, Silva NR, Pedrazzi JF, Del Bel EA, Hallak JEC, Crippa JA de S, Zuardi AW, Mechoulam R, Guimarães FS. Correction: Fluorinated cannabidiol derivatives: enhancement of activity in mice models predictive of anxiolytic, antidepressant and antipsychotic effects [Internet]. PLOS ONE. 2016 ; 11( 8):[citado 2024 maio 31 ] Available from: https://doi.org/10.1371/journal.pone.0162087
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NASCIMENTO, Manuela S. et al. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis. The Journal of Infectious Diseases, v. 214, n. 11, p. 1647-1657, 2016Tradução . . Disponível em: https://doi.org/10.1093/infdis/jiw446. Acesso em: 31 maio 2024.
APA
Nascimento, M. S., Ferreira, M. D., Quirino, G. F. S., Maruyama, S. R., Krishnaswamy, J. K., Liu, D., et al. (2016). NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis. The Journal of Infectious Diseases, 214( 11), 1647-1657. doi:10.1093/infdis/jiw446
NLM
Nascimento MS, Ferreira MD, Quirino GFS, Maruyama SR, Krishnaswamy JK, Liu D, Berlink J, Fonseca DM da, Zamboni DS, Carregaro V, Almeida RP, Cunha TM, Eisenbarth SS, Silva JS da. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis [Internet]. The Journal of Infectious Diseases. 2016 ; 214( 11): 1647-1657.[citado 2024 maio 31 ] Available from: https://doi.org/10.1093/infdis/jiw446
Vancouver
Nascimento MS, Ferreira MD, Quirino GFS, Maruyama SR, Krishnaswamy JK, Liu D, Berlink J, Fonseca DM da, Zamboni DS, Carregaro V, Almeida RP, Cunha TM, Eisenbarth SS, Silva JS da. NOD2-RIP2-mediated signaling helps shape adaptive immunity in visceral Leishmaniasis [Internet]. The Journal of Infectious Diseases. 2016 ; 214( 11): 1647-1657.[citado 2024 maio 31 ] Available from: https://doi.org/10.1093/infdis/jiw446
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ALVES FILHO, José Carlos Farias e PÅLSSON-MCDERMOTT, Eva M. Pyruvate kinase M2: a potential target for regulating inflammation. Frontiers in Immunology, v. 7, 2016Tradução . . Disponível em: https://doi.org/10.3389/fimmu.2016.00145. Acesso em: 31 maio 2024.
APA
Alves Filho, J. C. F., & Pålsson-McDermott, E. M. (2016). Pyruvate kinase M2: a potential target for regulating inflammation. Frontiers in Immunology, 7. doi:10.3389/fimmu.2016.00145
NLM
Alves Filho JCF, Pålsson-McDermott EM. Pyruvate kinase M2: a potential target for regulating inflammation [Internet]. Frontiers in Immunology. 2016 ; 7[citado 2024 maio 31 ] Available from: https://doi.org/10.3389/fimmu.2016.00145
Vancouver
Alves Filho JCF, Pålsson-McDermott EM. Pyruvate kinase M2: a potential target for regulating inflammation [Internet]. Frontiers in Immunology. 2016 ; 7[citado 2024 maio 31 ] Available from: https://doi.org/10.3389/fimmu.2016.00145
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
CAMPOS, Alline Cristina de et al. Absence of gut microbiota influences lipopolysaccharide-induced behavioral changes in mice. Behavioural Brain Research, v. 312, p. 186-194, 2016Tradução . . Disponível em: https://doi.org/10.1016/j.bbr.2016.06.027. Acesso em: 31 maio 2024.
APA
Campos, A. C. de, Rocha, N. P., Nicoli, J. R., Vieira, L. Q., Teixeira, M. M., & Teixeira, A. L. (2016). Absence of gut microbiota influences lipopolysaccharide-induced behavioral changes in mice. Behavioural Brain Research, 312, 186-194. doi:10.1016/j.bbr.2016.06.027
NLM
Campos AC de, Rocha NP, Nicoli JR, Vieira LQ, Teixeira MM, Teixeira AL. Absence of gut microbiota influences lipopolysaccharide-induced behavioral changes in mice [Internet]. Behavioural Brain Research. 2016 ; 312 186-194.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.bbr.2016.06.027
Vancouver
Campos AC de, Rocha NP, Nicoli JR, Vieira LQ, Teixeira MM, Teixeira AL. Absence of gut microbiota influences lipopolysaccharide-induced behavioral changes in mice [Internet]. Behavioural Brain Research. 2016 ; 312 186-194.[citado 2024 maio 31 ] Available from: https://doi.org/10.1016/j.bbr.2016.06.027
A citação é gerada automaticamente e pode não estar totalmente de acordo com as normas
ABNT
FREITAS, Renato Leonardo de et al. µ1-Opioid receptors in the dorsomedial and ventrolateral columns of the periaqueductal grey matter are critical for the enhancement of post-ictal antinociception. Synapse, v. 70, n. 12, p. 519-530, 2016Tradução . . Disponível em: https://doi.org/10.1002/syn.21926. Acesso em: 31 maio 2024.
APA
Freitas, R. L. de, Medeiros, P., Khan, A. U., & Coimbra, N. C. (2016). µ1-Opioid receptors in the dorsomedial and ventrolateral columns of the periaqueductal grey matter are critical for the enhancement of post-ictal antinociception. Synapse, 70( 12), 519-530. doi:10.1002/syn.21926
NLM
Freitas RL de, Medeiros P, Khan AU, Coimbra NC. µ1-Opioid receptors in the dorsomedial and ventrolateral columns of the periaqueductal grey matter are critical for the enhancement of post-ictal antinociception [Internet]. Synapse. 2016 ; 70( 12): 519-530.[citado 2024 maio 31 ] Available from: https://doi.org/10.1002/syn.21926
Vancouver
Freitas RL de, Medeiros P, Khan AU, Coimbra NC. µ1-Opioid receptors in the dorsomedial and ventrolateral columns of the periaqueductal grey matter are critical for the enhancement of post-ictal antinociception [Internet]. Synapse. 2016 ; 70( 12): 519-530.[citado 2024 maio 31 ] Available from: https://doi.org/10.1002/syn.21926
